Unit 1- test Flashcards

1
Q

3 inpatient surgical procedures

A
  1. ) emergent- Immediately
  2. ) urgent- schedule ASAP
  3. ) elective
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2
Q

outpatient surgical procedures

A
  • most often elective
  • not acute
  • Ex: cataract removal, hernia repair
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3
Q

pre-operative period Rn role

A
  • begins when pt scheduled for surgery
  • physical/mental assessment
  • blood donations
  • informed consent (signing)
  • teaching
  • discharge planning
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4
Q

pre-operative teaching

A
•pain control
•breathing/spirometer
•ROM
•anti-embolism
•diet
•invasive devices
•anti-anxiety
*demonstrate and then have pt state understanding and demonstrate
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5
Q

intra-operative period Rn role

A
  • monitor for resp. depression
  • maintain body temp
  • equipment count
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6
Q

biggest concern of opioids/sedatives

A

•respiratory depression

*reverse w/ Narcan

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7
Q

hypothermia during surgery

A

•increases chance of surgical/wound infection
•alters metabolism of meds
*why need to give pt warm blanket

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8
Q

malignant hyperthermia

A
•life-threatening complication of exposure anesthetics
•tachycardia/pnea
•elevated body temp
•muscle rigidity
•skin mottling 
•cyanosis
•myobloinuria (muscle protein in urine)
•rise in tidal CO2 and decrease in O2 sat
*tx w/ cold IV and Dantrolene
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9
Q

post-operative Rn role

A
  • evaluate/stabilize pt
  • prevent complications (ABC priority)
  • determine readiness for discharge (ambulate/fluids/VS)
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10
Q

post-op complications

A
  • airway obstruction
  • hypoxia (day 2)
  • hypovolemic schock (massive loss circulating blood)
  • paralytic ileus
  • wound dehiscence or evisceration
  • DVT
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11
Q

general anesthesia

A

•reversible loss of consciousness induced by inhibiting neuronal impulses in CNS
•causes analgesia, amnesia, unconsciousness, and loss of reflexes/tone
•used for major surgery or requiring major muscle rlx
*usually balanced- inhaled, IV, & adjuncts

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12
Q

local anesthesia

A
  • loss of sensation w/o loss of consciousness
  • motor fxn may/may not be affected
  • topical, local infiltration, or regional
  • often supplemented w/ sedative, opioid analgesics, hypnotics
  • risk for aspiration low b/c cough/gag reflex intact
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13
Q

regional anesthesia

A
  • blocks multiple peripheral nerves in specific body region
  • field
  • nerve
  • spinal
  • epidural
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14
Q

moderate (conscious) sedation

A
  • IV delivery of sedative, hypnotic, opioid drugs to reduce LOC for minor procedures w/o pt having discomfort
  • pt responds to verbal stimuli, retains reflexes, and is easily aroused
  • pt maintains airway
  • often used for burn debridement
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15
Q

major components of body fluid

A

1.) intracellular (ICF)- ⅔
2.) extracellular (ECF)- ⅓
•ECF includes intravascular and interstitial
•fluid can move b/t

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16
Q

hypotonic ECF

A
  • osmolarity of less than 270 mOsm/L
  • fluid into cells (lyse)
  • 0.5% NS
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17
Q

hypertonic ECF

A
  • osmolarity of greater than 300 mOsm/L
  • fluid out of cells (crenation)
  • 1.5% NS or 3% NS
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18
Q

isotonic ECF

A
  • osmolarity of 270-300 mOsm/L

* 0.9% NS

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19
Q

major causes of fluid/electrolyte imbalances

A
  • renal/GI/cardiac dysfunction/losses
  • hemorrhage
  • third spacing (ascites/burns)
  • intake
  • fever
  • hormones
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20
Q

aldosterone

A

•excreted by the cortex of the adrenal gland in response to low Na+ levels
•prevents sodium and water loss
*RETAIN fluid (FVD)

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21
Q

anti-diuretic hormone (ADH)

A

•Produced in the posterior pituitary
•acts on kidneys to make kidneys reabsorb more water so that the body retains more fluid
*RETAIN fluid (FVD)

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22
Q

natriuretic peptides (NPs)

A

•released in response to barorecptors in the heart or vascular system detecting increased blood volume
*LOSE fluid (FVE)

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23
Q

renin-angiotension system

A
  • in response to low BP, blood volume, blood O2, and blood osmolarity kidneys excrete renin
  • renin activates angiotension I, which is converted to angiotension II (active form) by ACE
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24
Q

3 fxns of angiotension II

A
  • vasoconstrictor
  • causes nephrons to contract, decreasing UOP
  • causes kidneys to release aldosterone
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25
Q

an ACE inhibitor would be classified as…

A
  • diuretic
  • lowers BP b/c blocks production of angiotension II
  • commonly used for mild HTN
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26
Q

fluid-volume deficit (FVD)

A
  • hypovolemia and dehydration
  • tachy/thready
  • increased hemoconcentration
  • hyperthermia, hyPOtension, confusion, oliguria
  • increased serum osm, Hct, Hgb, urine specific gravity
  • HYPERNATREMIA
  • tx w/ fluid (PO pref.)
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27
Q

hypovolemia vs/ dehydration

A

•hypovolemia is fluid and electrolyte loss, while dehydration is just fluid loss

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28
Q

hypovolemic shock

A
  • decreased oxygen to organ/pressure to organ
  • complication of FVD
  • tx w/ O2, fluids, vasoconstrictors (increase central flow first)
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29
Q

hemoconcentration

A

•decrease in the volume of plasma in relation to # of RBCs
•increase in concentration of circulating RBCs
•increases in Hct, Hgb, electrolytes, and urine specific gravity
*expected in endurance athletes

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30
Q

fluid volume excess (FVE)

A
  • hypervolemia or over-hydration
  • tachy/bounding
  • weight gain/crackles/edema
  • hyPERtension
  • decreased hemoconcentration, Hgb, Hct, serum osm
  • HYPONATREMIA
  • tx w/ Na+, diuretics, O2
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31
Q

expected serum Na+ levels

A
  • 135-145 mEq/L

* imbalances cause NEURO problems (influences conduction rates)

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32
Q

hypernatremia

A
  • Na+ > 145 mEq/L
  • due to increased Na+ or excessive water loss
  • hypertonic serum (crenation)
  • dehydration
  • tx w/ hypo/iso IV
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33
Q

hyponatremia

A
  • Na+ < 135 mEq/L
  • due to decreased Na+ or excessive water concentration in relation to sodium
  • hypotonic serum (lyse)
  • over-hydration
  • tx w/ hyper IV, ACE inhibitors, diuretics
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34
Q

expected K+ levels

A
  • 3.5-5.0 mEq/L

* imbalances cause CARDIAC problems

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35
Q

hyperkalemia

A
  • K+ > 5.0 mEq/L
  • due to increased K+ intake, K+ out of cells (diabetes/tumor lysis), salt sub.
  • EKG shows peaked T waves
  • tx w/ decreased K+ intake, Kayexalate (K+ in stool), diuretics
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36
Q

hypokalemia

A
•K+ < 3.5 mEq
•results from decreased intake, movement of K+ into cells (insulin high), Lasix (furosimide) cause K+ loss
*key s/s is fluttering chest
•EKG shows inverted T waves
•tx w/ PO K+ (never bolus)
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37
Q

expected Ca2+ levels

A
  • 9.0-10.5 mg/dL

* imbalances cause SKELETAL MUSCLE problems

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38
Q

hypocalcemia

A
  • Ca2+ < 9.0 mg/dL
  • bradycardia, hypotension, hyper bowel
  • muscle cramps (SPASMS)
  • risk of seizure/fall
  • Trousseau’s sign- wrist spasm with BP cuff
  • Chvostek’s sign- cheek muscle twitch when tap
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39
Q

hypercalcemia

A
•Ca2+ > 10.5 mg/dL
•lethargy, paresthesia b/c less sensitive to normal stimuli
•mscl WEAKNESS
•causes faster clotting time
*high risk for DVT
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40
Q

expected phosphorus level

A
  • 3.0-4.5 mg/dL

* imbalances cause SKELETAL MUSCLE problems

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41
Q

hyphosphatemia

A
  • phos < 3.0 mg/dL
  • same as HYPERCALCEMIA
  • muscle weakness
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42
Q

hyperphasphatemia

A
  • phos > 4.5 mg/dL
  • same as HYPOCALCEMIA
  • muscle spasms
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43
Q

phosphorus food sources

A
  • fish
  • chicken
  • beef
  • pork
  • organ meats
  • nuts
  • whole grains
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44
Q

expected Mg2+ levels

A
  • 1.3-2.1 mEq/L

* abnormalities involve DTRs

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45
Q

hypomagnesemia

A
  • Mg2+ < 1.3 mEq
  • HYPERACTIVE DTRs
  • occurs in conjunction w/ hypocalcemia (spasm)
  • tetany, seizure, psychoses
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46
Q

hypermagnesemia

A
•Mg2+ > 2.1 mEq/L
•HYPOACTIVE DTRs
•occurs in conjunction w/ hypercalcemia (weakness)
•coma, bradycardia, hypotension
*use of laxatives poses major risk
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47
Q

use of laxatives risk

A
  • hypermagnesemia

* hypokalemia

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48
Q

normal Hgb values

A

•m: 14-18 g/dL
•f: 12-16 g/dL
*hemochromatosis- too much Hgb

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49
Q

normal Hct values

A

•m: 50-57 ml/dL (50-54%)
•f: 37-48 ml/dL (37-48%)
*% of packed RBC/dL of blood

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50
Q

normal RBC values

A

•m: 4.7-6.0 million/uL
•f: 4.2-5.4 million/uL
*4.5-6
*polycythemia vera- too many RBCs

51
Q

anemia

A

•abnormally low RBCs, Hgb, and/or hematocrit
•results in diminished O2 carrying capacity and deliver to tissues/organs
*goal of tx is to restore/maintain adequate tissue oxygenation

52
Q

anemia causes

A
  • blood loss
  • inadequate RBC production (hypoproliferative)
  • increased RBC destruction (hemolytic)
  • Fe, folic acit, erythropoietin, and/or B12 deficiency
53
Q

s/s anemia

A
  • tachy, hypotension, HF
  • pallor, cold, brittle nails
  • weakness, fatigue
  • dyspnea, decreased O2 on exertion
  • somnolence, forgetfullness, dizziness
54
Q

Hgb levels in mild anemia

A

•10-14 g/dL

55
Q

Hgb levels in moderate anemia

A

•8-10 g/dL

56
Q

Hgb levels in severe anemia

A

• < 8 g/dL

* < 10 g/dL when clinical manifestations show

57
Q

hemolytic anemia

A
  • due to excessive destruction of RBCs (or SCD)
  • splenomegaly and jaundice
  • followed by acceleration of erythropoises
  • response to trauma, infection, chemical exposure, autoimmune rxns
58
Q

tx hemolytic anemia

A
  • steriods
  • spenectomy
  • chemo
  • plasma exchange
  • immuno-suppressant agents
59
Q

iron-deficiency anemia (IDA)

A
  • due to inadequate intake, most common in children, adolescents, and preggo
  • due to blood loss is common in older adults
  • menstruating women can develop secondary to menorrhagia
  • Hgb < 8 g/dL
  • Fe level 10 micrograms/dL
  • TIBC increase (trying to signal more iron to come)
60
Q

IDA tx

A
  • encourage intake
  • PO iron supplement (w/ OJ or vit C to absorb best)
  • Iron dextran- IM
  • transfusion packed RBCs
  • discontinue anti-acids, coffee, tea b/c impair Fe absorption
61
Q

pernicius anemia

A
  • due to vit B12 (cobalamin) deficiency
  • lack of intrinsic factor found in GI mucosa, which is necessary for B12 (extrinsic factor) absorption
  • low Hgb, Hct, cobalamin, and RBC
  • weakness/fatigue
  • jaundice
  • memory/personality changes
  • paresthesia
62
Q

pernicius anemia tx

A

•parenteral/intranasal admin of B12

63
Q

folic acid deficiency anemia

A
  • decreased RBC, Hgb caused by impaired production r/t decreased folate level
  • higher risk if poor nutrition, malabsorption syndromes (Crohn’s), anticonvulsant/oral contraceptive use, alcoholism, anorexic
64
Q

iron food sources

A
  • red/organ meat
  • leafy greens
  • egg yolks
  • almonds
  • legumes
  • dried fruit
65
Q

vit B12 food sources

A
  • meat/poultry
  • eggs
  • milk products
  • fortified cereals
66
Q

folic acid food sources

A
  • beans/legumes
  • citrus fruits/juice
  • fortified bread, cereals, pasta, etc
67
Q

homologous blood transfusion

A

•from another donor

68
Q

autologous blood transfusion

A
  • pt blood collected in anticipation of future transfusion
  • donated 5 wks- 72 hrs prior to elective surgery
  • eliminates risk of alloimmunization (immune response to antigens)
69
Q

whole blood transfusion

A
  • used for massive blood loss

* pt needs O2-carrying capacity and vol. increase

70
Q

packed RBC transfusion

A
  • whole blood with ⅔ of plasma removed
  • used for severe anemia or moderate blood loss
  • less danger of fluid overload
  • transfusion of choice
  • should not exceed 4 hrs transfusing
71
Q

fresh-frozen plasma transfusion

A
  • anti coagulated clear liquid portion of blood separated from whole blood by centrifugation
  • used to reverse excessive anticoagulation
  • used for clotting factor deficiencies associated w/ hemorrhagic tendency
72
Q

administering transfusion priorities

A
  • ensure type and Rx order
  • make sure no allergy, hx rxn, etc
  • cross match before getting from blood bank
  • verify blood and pt #
  • infuse w/ NS (NOT meds)
  • remain w/ pt for first 15 min
73
Q

hemolytic transfusion rxn

A
  • chills, fever, urticaria (rash), tach, pain/tight chest, SOB, cloudy urine
  • stop blood (keep NS)
74
Q

febrile transfusion rxn

A
  • sudden fever/chills, headache, flushing, anxiety, muscle pain
  • give antipyretics (avoid aspirin)
75
Q

bacterial (sepsis) rxn

A

•rapid onset of hypotension, fever, chills, vomit, diarrhea, and shock
stop transfusion and treat septicemia (abx, IV fluid, vasopressors, steroids)

76
Q

allergic transfusion rxn

A
  • antihistamine admin 15-30 min prior to transfusion to prevent
  • s/s: urticaria, edema of face, asthma attack, flushing/itching
  • tx: stop transfusion and KVO w/ NS
77
Q

circulatory overload transfusion rxn

A
  • higher risk if have renal/cardiac insufficiencies
  • s/s: cough, dyspnea, headache, hypertension, tach, JVD
  • tx: pt upright, O2 therapy, diuretics, morphine
78
Q

pancytopenia

A

•low RBC, WBC, and platelets

79
Q

cerebral vascular accident (CVA)

A
  • disruption in the cerebral blood flow secondary to ischemia from thrombosis, hemorrhage, or embolism
  • aka: stroke, cerebral infarction, or brain attack
80
Q

hemorrhagic CVA

A
  • occur secondary to ruptured artery or aneurysm
  • prognosis poor b/c of amt ischemia and increased ICP caused by collection of blood
  • prognosis better if stroke caught early, bleeding ceased, and clot evacuated
81
Q

thrombotic CVA

A

•occurs secondary to development of blood clot on an atherosclerotic plaque in a cerebral artery
•clot gradually shuts off artery and causes ischemia distal to occlusion
•symptoms evolve over period of hrs-days (often preceded by TIA)
*occlusive

82
Q

embolic CVA

A

•occur secondary to embolus traveling from another part of body to cerebral artery
•brain blood distal to occlusion immediately shut off, causing loc to occur
*occlusive

83
Q

transient ischemic attack (TIA)

A
  • mini stroke (warning stroke)
  • caused by thrombotic clot but blockage is temporary
  • sx occur rapidly but last 1-5 minutes
  • warning signs almost same as stroke
  • usually no permanent damage
84
Q

reversible ischemic neurological deficit (RIND)

A
  • caused by thrombotic clot, but blockage temporary

* similar to TIA, but lasts longer (up to 24 hrs)

85
Q

recombinant tissue plasminogen activator (rtPA)

A

•thrombolytic NZ (Activase)
•can be used to reverse ischemic stroke (thrombolitic/embolitic) if given w/ 3-4.5 hrs of initial sx
•contraindicated if hemorrhagic stroke or pt on anticoagulants
•have to rule out hemorrhagic stroke w/ MRI before initiating
*only used for clot in brain (not used for DVT)

86
Q

AEDs for CVA

A
  • phenytoin (Dilantin), gabapentin (Neurontin)
  • usually only if patient develops seizures
  • gabapentin may be given for paresthetic pain in affected extremity
87
Q

antiplatelets

A
  • low dose ASA-acetylsalicylic acid (aspirin) given within 24-48 hrs. following a stroke to prevent further clot formation
  • Give within 4.5 hrs. of initial symptoms
88
Q

anticoagulates

A
  • controversial tx for CVA
  • high risk of intracerebral hemorrhage
  • Heparin, enoxaparin (Lovenox), warfarin (Coumadin)
89
Q

CVA complications

A
  • dysphagia
  • aspiration
  • unilateral neglect
  • anosognosia- don’t recognize impairments
90
Q

left hem CVA possible abnormalities

A
  • language, math, and analytic thinking
  • expressive, receptive, global aphasia
  • agnosia- inability to recognize objects
  • alexia- reading diff.
  • agraphia- writing diff.
  • hemianopsia, hemiplegia, hemiparesis
91
Q

right hem CVA possible abnormalities

A

•spatial/depth perception
•proprioception
judgment/impulse control
•hemianopsia, hemiplegia, hemiparesis

92
Q

homonymous hemianopsia

A
  • visual field loss on same side of both eyes
  • left: due to abrasion on right side of brain (has visual pathways for left hemifield of both eyes)
  • right: due to abrasion on left side of brain (has visual pathways for right hemifield of both eyes)
93
Q

cerebrum damage

A

contralateral impact

94
Q

cerebellar damage

A

ipsilateral impact

95
Q

FAST

A
  • facial drooping
  • arm weakness
  • slurred speech (dysarthria)
  • time- call 911
96
Q

ICP increase

A

•due to blood from hemorrhage, cerebral edema
•causes hyperthermia b/c pressure on thalamus, widening of pulse pressure, decreased HR
*normal: 10-15 mmHg

97
Q

seizure

A
  • abrupt, abnormal, excessive, and uncontrolled electrical discharge of neurons within the brain
  • may cause alterations in LOC, motor/sensory ability, and/or behavior
  • can be generalized, partial, or unclassified
98
Q

primary (idiopathic) epilepsy

A
  • chronic recurring abnormal brain electrical activity
  • dx based on hx and labs to rule out other causes
  • considered a syndrome
  • due to imbalance of nts (GABA)
99
Q

generalized seizure

A
  • involves both hemispheres
  • tonic-clonic
  • tonic
  • clonic
  • absence
  • myoclonic
  • atonic
100
Q

tonic-clonic (grand mal) seizure

A

•may (rarely) begin with aura (altered sense)
•begins w/ tonic episode (stiff muscles) and loc
•clonic episode (muscle jerk) follows tonic
•clonic phase followed by postictal phase
*lasts 2-5 min

101
Q

postical phase

A
  • period of confusion, fatigue, agitation, and lethargy following a tonic-clonic seizure
  • lasts up to an hour
102
Q

absence seizure

A
  • most common in children
  • loc lasting few sec
  • blank staring
  • automatisms
  • return to baseline after seizure
103
Q

automatisms

A
  • involuntary behavior
  • lip smacking
  • eye fluttering
  • picking at clothes
104
Q

myoclonic seizure

A
  • generalized
  • brief jerking/stiffening of extremities (sym/asym)
  • lasts for few seconds
105
Q

atonic (akinetic) seizure

A
  • few second period of muscle tone loss
  • followed by period of confusion (postictal)
  • frequently results in falling
  • most resistant to drug therapy
106
Q

complex partial seizure

A
  • automatisms
  • loc for 1-3 min
  • amnesia possible prior to and after seizure
  • temporal lobe usually involved
107
Q

simple partial seizure

A
  • consciousness maintained
  • unusual sensations- aura, deja vu
  • autonomic abnormalities (HR, flushing, epigastric discomfort)
  • unilateral extremity movement
108
Q

unclassified (idiopathic seizure)

A
  • occur fo no known reason

* account for ½ of all seizure activities

109
Q

seizure risks

A
  • increased physical activity
  • stress
  • hyperventilation
  • fatigue
  • excessive caffeine/etoh intake
  • flashing lights
  • chemical exposure
110
Q

hyperventilation

A
  • CO2 decrease (alkalosis- basic)
  • Vasoconstriction b/c low CO2 (and pH) causes CSF to signal vasoconstriction (seizure)
  • decrease in ICP
  • too much O2 b/c no CO2 to compete w/ for Hgb site
111
Q

nursing interventions during seizure

A
  • PRIORITY: keep pt from injury
  • maintain airway (be ready to suction)
  • ease pt to floor
  • turn pt on side
  • remove loose items
  • don’t retrain/put object in mouth
112
Q

status epilepticus

A

•prolonged seizure activity occurring over 30-min time frame
•decreased O2
•inability brain to return to normal fxn
*require immediate tx to proven loss of brain fxn, organ failure, dysrhythmias, etc

113
Q

anticonvulsants (AEDs)

A

•tx to control seizures to some degree
•pt often requires combo for control
•doses adjusted to achieve therapeutic blood levels with least amount of side effects
•oral hygiene important when taking
*CANNOT be stopped abruptly
•educate pt on drug/food interactions (GF juice)

114
Q

diazepam (Valium)

A
  • acute seizure tx to prevent progression into status epileptics
  • anti-convulsant, anti-anxiety, BZD, and skeletal muscle relaxer
  • give 4 mg over 2 min (slow IV push)
115
Q

lorazepam (Ativan)

A
  • acute seizure tx to prevent progression into status epileptics
  • anti-anxiety, BZD, and anti-convulsant
  • give 4 mg over 2 min (slow IV push)
116
Q

phenytoin (Dilantin)

A
  • acute seizure tx to prevent progression into status epileptics (loading dose)
  • anti-convulsant and anti-dysrhythmic
  • AED for maintenance therapy
  • decreases effectiveness of warfarin and oral contraceptives
117
Q

EEG testing for seizure

A
  • hyperventilation- rapid deep breathing
  • photic stimulation- strobe light flashes
  • sleep- temporal lobe epilepsy
118
Q

MACHINE mnemonic

A
•causes of hyperkalemia
Meds (ACE, steroids, beta blockers)
Acidosis
Cellular destruction (burns, trauma)
Hypoaldosteronism, hemolysis
Intake- excessive
Nephrons- renal failure
Excretion- impaired
119
Q

6 L’s mnemonic

A
•signs of hypokalemia
Lethargy
Lethal cardia arrhythmia
Limp muscles
Leg cramps
Low, shallow respirations
Less stool (constipation)
120
Q

FRIED mnemonic

A
•signs of hypernatremia
Fever
Restless
Increased BP
Edema
Decreased UOP
121
Q

MURDER mnemonic

A
•signs of hyperkalemia
Muscle weakness
Urine- oliguria/anuria
Respiratory distress
Decreased cardiac contractility
EKG changes (peaked T waves)
Reflexes hyper or hypo
122
Q

GRAPHIC IDEA mnemonic

A
•causes of hypokalemia
GI losses
Renal losses
Aldosterone excess
Periodic Paralysis
Hypothermia
Insulin excess
Cushing syndrom
Insufficient intake
Diuretics
Elevated beta adrenergic activity
Alkalosis
123
Q

K-BANK mnemonic

A
•K+ increasing agents
K-sparing diuretic
Beta Blocker
ACE Inhibitor
NSAID
K supplement