Unit 1: Pain Theory, Perception, and Physical Agent Modalities Flashcards

1
Q

Pain

A

Described as an unpleasant and emotional experience association with actual or potential tissue damage

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2
Q

OT and Pain

A

Address pain which:
-Limits patients ability to participate in occupational roles
-Limits function
-Limits ROM/Strength
-Affects patients: Physically, Emotionally, and Mentally

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3
Q

Managing pain enhances:

A

-Healing of soft tissue
-Movement of Body Structures
-Functional Tasks
-Participation in: home, work, hobbies, sports, school, daily activities

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4
Q

Pain Symptoms

A

-Most common reason for seeking medical attention
Symptoms may contribute to:
-Structural deficits
-Protective guarding
-Anxiety/decreased sleep
-Edema
-Joint stiffness
-Tissue shortening
-Poor positioning
-Compensatory movements

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5
Q

Pain Affects

A

May provoke fear, anger, withdrawal, or
anguish

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6
Q

Pain symptoms are generally related to:

A

-Trauma
-Injured tissue
-Inflammation
-Muscular/neurological conditions
-Degenerative disease
-Pathological conditions

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7
Q

Why pain?

A

Pain serves as a protective, warning device designed to protect the body from injury

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8
Q

PAMS control pain in various ways including:

A

◦ Inhibiting inflammatory response
◦ Altering nerve conduction
◦ Increasing endorphin levels
◦ Inhibiting pain transmission at spinal cord

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9
Q

PAMS Advantages

A

-Fewer and less severe side effects than pharmacological agents
-Do not cause sedation and can allow individuals to work and drive.
-PAMs can be used in conjunction with patient home exercise programs

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10
Q

OT Goals (Pain)

A

-Diminish/resolve pain symptoms
-Understand pathology causing pain .
-Modify the patient’s perception of discomfort
-Maximize function within the patient’s limitations

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11
Q

Types of Pain

A

-Acute: Immediate and last 24-48 hrs
-Chronic: Lasts longer than 3-6 months
-Referred: Can be stemming from another part of body where true injury has taken place

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12
Q

Acute Pain

A

-Generally less than 6 months duration
-Underlying pathology can be identified
-Lasts as long as the noxious stimuli persists
-Associated with tissue damage
-Affects daily function
Occurs with sudden onset

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13
Q

Acute Pain Treatment

A

Physical Agent Modalities (heat or cooling) are the most effective when treating patients in the acute stage of pain

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14
Q

Treatment Goals for Acute Pain

A

-Reduce inflammation
-Facilitate resolution of pain
-Modify transmission of pain from peripheral system to central nervous system
-Educate patient regarding pain management
-Restore function

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15
Q

Chronic Pain

A

-Pain that persists beyond the normal time for tissue healing
-Starts as acute pain
-Pain lasting longer than 3-6 months
-Result of activation of dysfunctional,
neurological or psychological responses

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16
Q

Chronic pain may contribute to…

A

-Increased dependence on other
-Changes in meaningful relationships
-Decreased function
-Limitations with daily activities/roles
-Decreasing the risk of chronic pain with early identification of and prevention strategies may reduce the need for prolonged treatment

17
Q

Referred Pain

A

-Experiencing pain in one region when the actual tissue damage is in another region
Pain may be referred from………..
-One joint to another
-Peripheral nerve to distal innervation
-Internal organ to musculoskeletal tissue
-Trigger points

18
Q

Structures involved in Pain

A

-Peripheral Nociceptors
-Peripheral Nerve
-Central Nervous System
-Cortex/Consciousness
-Neuropathic pain

19
Q

Nociceptors

A

Sensory receptors specific to pain are located:
- Skin: sharp, pricking, tingling, Localized
- Viscera (internal organs): Aching
- Musculoskeletal Skeletal: Heavy/achy
Responsive and Sensitive
-Thermal changes
-Mechanical distortion
-Chemical components

20
Q

Peripheral Nerve FIbers (pain pathways)

A

-Specific peripheral nerves (C, A delta fibers) transmit to the spinal cord (T cells) then to
specific tracts to the brain to interpret pain.
-Specific nerve endings (nociceptors), respond to painful stimuli.

21
Q

Peripheral Nerve Pathways

A

Nociceptors: Pain Transmitting Afferent Nerve Fibers
- C fibers: Small unmyelinated fibers (80%) - slow, chronic pain
- A delta fibers: Small myelinated fibers (20 - fast, acute pain
-Transmit the sensation of pain to the spinal cord within specific tracts to the brain

22
Q

Gate Control Theory (Pain Modulation)

A

(Melzack and Wall (1965))
-PAM interventions partially control pain
by activating non-nociceptive sensory nerves.
-Inhibiting the activation of transmitting pain cells closes the gate to pain.

23
Q

Pain Assessments

A

-Visual Analog
-Numeric Scale
-FACES Assessment for Children
-Body Diagrams

24
Q

Response to Pain

A

-Behavior to the response of pain is an adapted behavior that involves learning and memory
-Individuals respond differently to pain
-Perception of pain is subjective
-Subjective pain maybe difficult to evaluate and treat

25
Q

Pain Aspects

A

-Physiologic: Tissue damage is source of pain
-Affective: Individual emotions related to pain, psychological state in response to pain
-Cognitive: How the patient reports the pain
and knowledge about the cause of their
pain
-Behavioral: Expression of pain
-Perceptual: Sensations are conveyed to the brain to alert patient of pain

26
Q

Treatment of Pain

A

-OT’s treating patients with pain have an understanding of the primary characteristics and interventions that can affect all facets of functional performance in life roles and tasks.
-Integrating PAMS and various treatment modalities allows for improved outcomes and quality of life.