Tutorials Flashcards
name 3 potential triggers of asthma
allergies
exercise
smoke
infections
aetiological factors of MIH
perinatal hypoxia
genetic predisposition
childhood illness e.g measles, rubella
how does the pattern of MIH differ from fluorosis
MIH has an asymmetrical pattern
Fluorosis is diffuse linear patches with clear boundaries in a symmetrical pattern
what teeth are most commonly involved in MIH
one to four FPMs, at least one must be affected for MIH diagnosis
permanent incisors also commonly affected
Other teeth may also present defects
describe the demarcated opacities seen in MIH
only defects more than 1mm should be considered
clearly demarcated opacities presenting with an alteration in the translucency of enamel. Can vary in size, shape and colour - white, creamy or yellow/brown
why is post eruptive enamel breakdown seen on MIH teeth
due to masticatory forces on the weakened enamel, breaks down over time
Loss is often associated with a pre existing demarcated opacity
what shape does dens in dente appear as on a radiograph
white ‘m’ shape
what teeth is most commonly affected by dens in dente
maxillary lateral incisors
difference between MIH and amelogenesis imperfecta
amelogenesis - generally all teeth affected
MIH - not all teeth affected, most commonly just FPM(s) +/- incisors
what is contained within some fluoride varnishes that may trigger asthma
colophony (sticky agent)
treatment option for MIH posterior teeth with no post eruptive breakdown
(child)
fissure sealants
treatment for MIH posterior teeth with post eruptive breakdown
(child)
composite - ideally with no prep
no crowns or veneers in children
FS is insufficient for teeth that have experienced PEB
non invasive treatment option for MIH anterior teeth
microabrasion
resin infiltration
in what circumstances may microabrasion be considered for treating MIH teeth
anterior teeth where aesthetics important
most useful on yellow/ brownish demarcations
superficial lesions
what is used for carrying out microabrasion
18% hydrochloric acid
