Tricuspid Flashcards
Presentation of advanced TR
- elevated “c-V”
- a systolic murmur at the lower sternal border that increases in intensity with inspiration.
- pulsatile liver edge
Severe TR - Echocardiographic findings
- Central jet area >10.0 cm2
- Vena contracta width >0.70 cm
- CW jet density and contour: dense, triangular with early peak
- Hepatic vein flow: systolic reversal
ECHO findings of Tricuspid regurgitation
Central jet area?
Central jet area >10.0 cm2
Severe TR ECHO Vena contracta
Vena contracta width >0.70 cm
Tricuspid valve ECHO consistent with Normal RV function
Normal RV systolic function is defined by several parameters, including:
- TAPSE: >16 mm
- tricuspid valve annular velocity (S’) >10.0cm/s
- RV end-systolic area <20.0 cm2
- fractional area change >35%.
Initial medical therapy with right side heart failure
Diuretics can be used to decrease volume over-
load in these patients.
Loop diuretics are typically provided and may relieve systemic congestion, but their use can be limited by worsening low-flow syndrome.
Aldosterone antagonists may be of additive benefit, especially in the setting of hepatic congestion, which may promote secondary hyperaldosteronism.
Mortality associated with Reoperation for severe, isolated TR after left-sidedvalve surgery?
Reoperation for severe, isolated TR after left-sided
valve surgery is associated with a perioperative
mortality rate of 10% to 25%.
Left uncorrected at the time of left-sided valve sur-
gery, mild or moderate degrees of functional TR may
progress how often?
Left uncorrected at the time of left-sided valve sur-
gery, mild or moderate degrees of functional TR may
progress over time in approximately 25% of patients and result in reduced long-term functional outcome and survival.
Risk factors for worsening TR at the time of left side surgery
Risk factors for persistence and/or progression of
TR include:
- tricuspid annulus dilation:
- > 40 mm diameter or 21 mm/m2 on preoperative TTE
- degree of RV dysfunction/remodeling
- leaflet tethering height
- pulmonary artery
- hypertension
- AF
- nonmyxomatous etiology of MR
- intra-annular RV pacemaker or implantable cardioverterdefi brillator leads
.