Treatment - Soft tissue Flashcards

1
Q

Aetiology of soft tissue injury

A

> Overuse - tissue = unable to repair
(symptoms may be due to lack of muscle strength/endurance)
Misuse - abnormal stress (improper form/equipment)
Abuse- overload
Disuse - deconditioned tissue

  • Intrinsic factors (inside body) e.g age/genetics/co-ordination
  • Extrinsic factors (outside body) e.g training levels/environment/equipment
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2
Q

Cook and Purdum model

A

Normal tissue + optimal load = strengthened
+
excessive load + individual factors
= reactive tissue and eventually tissue disrepair and degenerative tissue if not adjusted to appropriate modified load

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3
Q

Symptoms for soft tissue injury

A

> Muscle/Ligament

  • Pain = sudden onset
  • disability
  • swelling
  • aggs = stress on tissue via contraction/RoM

> Tendon

  • Tear = similar to muscle/ligament
  • Disorder = gradual onset
  • aggs = loading tendon
  • eases = deloading tendon (may be sore after period of rest post loading)
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4
Q

Assessing for soft tissue injury

A
  • AROM vs. PROM (contractile vs inert)
    > Muscle/tendon
  • muscle length
  • muscle strength (full RoM + isometric)
  • Palpate for tenderness (locate lesion/trigger point)
    > Ligament
  • accessory tests - end feel/pain/apprehension
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5
Q

Treating soft tissue injury

A

> POLICE = better + faster healing than PRICE

> Tissue needs certain amount of stress to heal better

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6
Q

Specific soft tissue mobilisations (SSTMs)

A

> Aiming to alter regeneration + remodelling phases (where collagen cross links form)

  • providing pain relief via pain gate theory to allow function
  • Collagen remodelling
  • affect visco-elastic properties

> Types

  • physiological
  • accessory
  • combined
  • deep transverse frictions
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7
Q

Physiological SSTM

A

> Stretching of tissue

  • self directed or by therapist
  • Hold for ~ 1 min to engage change (possible oscillation to get further RoM)
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8
Q

Accessory SSTM

A

> Direct pressure
- pressure on tight/painful area
Bowing
- pressure on middle in sideways direction
- good for tendons + scar tissue
Transverse mobilisations
- compressive + transverse forces to superior surface

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9
Q

Combined SSTM

A

> Both physiological + accessory SSTMs

- ie stretch with manual therapy

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10
Q

Deep transverse frictions

A
>  force perpendicular to fibre orientation
> thought to possibly 
- break down adhesions
- increase blood supply
- re-orientate fibres 
- activate cells 
- may also cause further inflammation 
*Pressure = nothing vs stress of actual loading
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11
Q

Contra-indications/Precautions

A
> Open wound
> Clotting disorders
> DVT 
> Skin infections
> Acute inflammation
> Growth plates
> skin disorders
> Possible cancer
> foreign body/implant
> rheumatoid arthritis/spondyloarthritis (cause dysregulated response)
> Bursitis 
> Neural tissue
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12
Q

Creep

A

> Time dependent changes tissue deformation due to external load applied
e.g repeated stretching will lead to long term increase of RoM
increases soft tissue mobility long term

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13
Q

Hysteresis

A

> Amount of time an increase of RoM is maintained post loading
e.g having stretched to increase RoM - how long that RoM will stay increased before returning to normal

> creating this will create heate, reduce stiffness and restore normal response to loading

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14
Q

Stress - strain curve

A

> Amount of load to tissue vs amount of deformation

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