Treatment - Soft tissue Flashcards
Aetiology of soft tissue injury
> Overuse - tissue = unable to repair
(symptoms may be due to lack of muscle strength/endurance)
Misuse - abnormal stress (improper form/equipment)
Abuse- overload
Disuse - deconditioned tissue
- Intrinsic factors (inside body) e.g age/genetics/co-ordination
- Extrinsic factors (outside body) e.g training levels/environment/equipment
Cook and Purdum model
Normal tissue + optimal load = strengthened
+
excessive load + individual factors
= reactive tissue and eventually tissue disrepair and degenerative tissue if not adjusted to appropriate modified load
Symptoms for soft tissue injury
> Muscle/Ligament
- Pain = sudden onset
- disability
- swelling
- aggs = stress on tissue via contraction/RoM
> Tendon
- Tear = similar to muscle/ligament
- Disorder = gradual onset
- aggs = loading tendon
- eases = deloading tendon (may be sore after period of rest post loading)
Assessing for soft tissue injury
- AROM vs. PROM (contractile vs inert)
> Muscle/tendon - muscle length
- muscle strength (full RoM + isometric)
- Palpate for tenderness (locate lesion/trigger point)
> Ligament - accessory tests - end feel/pain/apprehension
Treating soft tissue injury
> POLICE = better + faster healing than PRICE
> Tissue needs certain amount of stress to heal better
Specific soft tissue mobilisations (SSTMs)
> Aiming to alter regeneration + remodelling phases (where collagen cross links form)
- providing pain relief via pain gate theory to allow function
- Collagen remodelling
- affect visco-elastic properties
> Types
- physiological
- accessory
- combined
- deep transverse frictions
Physiological SSTM
> Stretching of tissue
- self directed or by therapist
- Hold for ~ 1 min to engage change (possible oscillation to get further RoM)
Accessory SSTM
> Direct pressure
- pressure on tight/painful area
Bowing
- pressure on middle in sideways direction
- good for tendons + scar tissue
Transverse mobilisations
- compressive + transverse forces to superior surface
Combined SSTM
> Both physiological + accessory SSTMs
- ie stretch with manual therapy
Deep transverse frictions
> force perpendicular to fibre orientation > thought to possibly - break down adhesions - increase blood supply - re-orientate fibres - activate cells - may also cause further inflammation *Pressure = nothing vs stress of actual loading
Contra-indications/Precautions
> Open wound > Clotting disorders > DVT > Skin infections > Acute inflammation > Growth plates > skin disorders > Possible cancer > foreign body/implant > rheumatoid arthritis/spondyloarthritis (cause dysregulated response) > Bursitis > Neural tissue
Creep
> Time dependent changes tissue deformation due to external load applied
e.g repeated stretching will lead to long term increase of RoM
increases soft tissue mobility long term
Hysteresis
> Amount of time an increase of RoM is maintained post loading
e.g having stretched to increase RoM - how long that RoM will stay increased before returning to normal
> creating this will create heate, reduce stiffness and restore normal response to loading
Stress - strain curve
> Amount of load to tissue vs amount of deformation