trauma-ppt Flashcards

1
Q

ppts what is trauma

A

Trauma is an injury to human tissues and organ s resulting from the transfer of energy from the environment

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2
Q

what causes injury

A

Injuries are caused by some form of energy that is beyond the body’s resilience to tolerate *

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3
Q

what does the trimodal distribution of trauma pt deaths look like
% of who dies when

A

50% in first hr (often on scene)
25% in first 4hrs (often in emerg)
25% in the weeks 1-5 after initial injury (often in ICU)

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4
Q

common causes of death in first hr after trauma

A

hemmorhage
shock

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5
Q

common causes of death in first 4hrs after trauma

A
hypovolemia
airway obstruction (generally d/t unconscious pt)
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6
Q

common cause of death in 1-5wks after trauma

A

Die from infection, multiple organ dysfx syndrome (from being ischemic, anaerobic metb)

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7
Q

what is the definitive treatment of multiple trauma pts

A

surgery

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8
Q

when are diagnostics a priority of pt care

A

when they give useful info for Tx

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9
Q

how do you calculate the impact force

A

Impact Force = weight x speed

eg weight of car x speed in km you were going

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10
Q

falls > than __ft can be fatal

why?

A

12 ft

risk of traumatic dissection of thoracic aorta

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11
Q

acceleration/deceleration injuries are a form of what type of force

A

blunt

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12
Q

what is a risk if going >50km/h in a vehicle and crashing or stopping abruptly

A
  • when the heart goes forward it creates shearing forces, primarily above the heart, shears on aorta. The heart also bounces back. The aorta has multi layers and they can come apart.
  • MVA >50km/h have high risk of shearing aortic dissection
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13
Q

what kind of injuries or organs might be caused by sudden accel/decel

A
  • damage to liver, heart, aorta, head injury

- the liver and heart are heavy and htey move in the body cavity more

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14
Q

why can blunt force trauma often go without being properly treated

A

blunt forces often have no visible, outer evidence of injury

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15
Q

what are the forms of energy that can cause injury

what is another biomechanism of energy that causes cellular damage

A
Energy
Mechanical
Thermal
Chemical
Electrical
Radiant

-hypoxia

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16
Q

if a pt was in a car accident what kind of injuries from compression might they sustain?
what reaction might the person have in this situation and what organs could it affect?

A

The lap belt comes up around abdm and can pop bowel up through diaphragm
people tend to hold their breath and clamp their glottis shut, this creates a seal on the lungs and can contribute to a pneumothorax when suddenly compressed

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17
Q

what structures do rotational injuries often occur to

A

limbs generally also head/neck/spine, trunk

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18
Q

when a structure is rotated although it might not break or show external signs of damage what might happen and why is this dangerous

A

the internal strs are damged–>inflm or the damage doesnt allow perfusion–>hypoxia

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19
Q

what causes more damage to the structures around it, a sharp or dull penetrating object

A

dull as it pulls the tissues surrounding it

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20
Q

when are penetrating accidents often missed

A

when the object that entered the person was thin and fine

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21
Q

are bullet wounds generally in a straight line

A

no, theyre designed to spin within the person

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22
Q

what basic questions should you ask yourself about your trauma pt and their accident

A

how much energy, what kind of injury, what organs affected

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23
Q

why might some trauma pts not show their injury until many hours after

A

Sympathetic system will activate. Alpha adrenergic receptors—triage body organs and send blood to most important organs (heart, brain). A lot of your organs dont have blood going to them, youre not hypovolemic although you might present with cold hands etc

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24
Q

if your pt has a skull laceration that is barely bleeding should you be worried

A

yes, this means they had a lot of force to their head and their brain might have been affected or inflm might follow

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25
Q

your pt has a compound bone fx and a laceration thats exposed. What do you do?

A

cover it quickly to prevent infection but dont dress and clean it carefully until pt stable

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26
Q

what is a comminuted fx

A

a bone that has been broken in many places

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27
Q

what is a basal skull fx and what kind of pts might present with this

A

-a fracture to the base of the skull (includes hard palate of the mouth, the middle fossa is around cheeks, posterior fossa is at back of head)
Often assoc w facial trauma or people falling and hitting the back of their head on the sidewalk

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28
Q

what is danger with injury to dura mater

A

WBCs or RBCS or worse entering the arachnoid space

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29
Q

if pt had anterior fossa basal skull fx what part of the craniums 3 components might leak and give you a clue as to the location of their injury

A

the CSF might leak into subcut tissue of the eyes and give them racoon eyes awhile after
CSF might leak out their nose

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30
Q

what does CSF do that water wont on a pillow

what might CSF test positive for that water wont

A

halo
put ina dipstick and it might be positive for glucose
pt might report sweet taste in their mouth if it got in there

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31
Q

pt has middle basal fx what sign might you see

A

mastoid sign (bruising behind ear on mstoid process) i think also called battle sign

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32
Q

your pt has a facial injury can you rule out spinal injury

A

no

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33
Q

your pt has a head injury can you rule out spinal injury

A

no

facial head or c spine injury you must rule out first before taking off spinal precautions

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34
Q

which two parts of the spine are most vulnerable to injury or commonly injured

A

lumbosacral junction and c spine

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35
Q

what is an unstable spinal cord injury

A

torn posterior ligament

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36
Q

why do drs inset their fingers into pts rectum in admitting emergent assessment

A

the final dermatome in the body terminates there. If the pt still has the motor and sensation for rectal tone then they have a chance of recovery from spinal injury

if they have no rectal tone and this persists for >24h the chance of recovery is

37
Q

C_ innervates the diaphragm with the ____ nerve

A

C5 phrenic nerv

38
Q

what is the only definitive way to rule out spine injury

only real tx for spinal injury

A

CT scan–could also run hands down spine and next to it and see if pt has pain but this is informal
Sx

39
Q

why are lungs so vulnerable to trauma and how is a pneumothorax corrected

A

-friable, soft, very elastic and always wanting to reoil. they rely on negative pressure to keep them exanded.
a chest tube re-establishes the vacuum in the pleural space

40
Q

what is a tension pneumothorax and what specific mnfts might you see

A

Sometimes an alveoli will rupture and it creates a valve. It will tear the visceral pleura which wil alllow air to escape into pleural space with each inhalation. The pressure will inc until it shifts the heart over, shifts trachea, and the other lung will collapse. This is tension pneumothorax.
mnfts:The pt will look worse every breath, inequal chest expansion, may have tracheal shift

41
Q

what other injury to chest can cuase tension pneumo

A

penetrating

42
Q

what complications can result from broken ribs

A

pt tends to hypoventilate and is at risk of atelectasis pneumonia

43
Q

flail chest is

A

ribs broken in multiple places
In this case the ribs arent moving in a concentric form anymore. Paradoxical movement of the chest. Youll feel a segment moving in the opposite direction

44
Q

Normal p02 is ___% on ROOM AIR ONLY

A

80-100%

45
Q

pts chest tube detaches what do you do

A

dont clamp it as this can give them a pneumo

put end in a bottle of sterile NS

46
Q

what injuries or risks are assoc w solid organs vs hollow organs

A

If you perforate solid organs you bleed. If you perforate hollow organs you get sepsis

47
Q

why might abdm bleeding be hard to detect

A

Under lower ribs are liver and spleen. Organs that are dense and blood filled. Theyre located in a capsule, they can bleed in there. These are very painful injuries as the capsule fills

48
Q

how can you detect a retroperitoneal injury

A

percuss the costovertebral angle

49
Q

how to treat an orthopedic injury

A

rest ice compression elevation

50
Q

qualities of first degree burn

A

1o - Superficial
Dry, red, blanches
Painful

51
Q

qualities of 2nd degree burn

A

2o – partial thickness
Red, moist, blisters
Painful

52
Q

qualities of 3rd degree burn

A

3o – full thickness
Dry, no blanching
painless

53
Q

what makes up the volume of the cranium

A

Volume of intracranium=V of blood (10%)+V CSF (10%) + V brain (80%)

54
Q

what is normal intracranial pressure

A

10mmHg pressure

55
Q

wht is the first thing in cranium to be reduced in inc ICP

A

CSF is displaced. the arachnoid villi are always making CSF and it slows down production and inc reabsorption and shunts it down the spinal canal.

56
Q

what component of the cranium is altered second in compensation of IICP

A

Pt also vasoconstricts…to dec amount of blood in/around cranium

57
Q

inc ICP how are resps affected in early stages

A

carbon dioxide is very potent cerebral vasodilator. The less C02 you have the more vasoconstriction. IF pt is hyperventilating they will dec the amount of C02

58
Q

pt has inc ICP what symptoms might they report and what else might you see

A

horrible headache-from mech of injury as well

vomit center is stimulated early–this inc ICP too so dont want this

59
Q

what are terms to describe compensation and not in ICP.

what happens between these places (s/s)

A

spatial compensation and spatial exhaustion
-inappropriate–>alt consciousness–>sensory changes–>pupil changes

pressure on more advanced outer parts of brain?? PFC? pt becomes inappropriate/behavioural changes

-brain/brainstem gets caught on the foramen magnum the RAS will have circ caught off–>The GCS will change.
Next within the brain stem is the sensory and motor fibres. Youll see unilateral sensory changes (which you cant assess) but you can assess motor functions eg ask them to lift arms, painful stimulus.

-The cranial nerves, 1 &2 cant be assessed. 3 is pupil changes gen one will dilate

60
Q

what part of the brain is affected that leads to VS changes in ICP what ICpressure is this around

A

circulatory centre, hypothalamus

around 40mmHg

61
Q

what is ___ triad r/t ICP

A

Irregular respirations (caused by impaired brainstem function) Bradycardia. Systolic hypertension (Widening Pulse Pressure)

62
Q

what do you try to address when treating IICP

what is the ultimate goal

A

Almost all Tx should focus on dec oxygen demand and inc oxygen supply.
want sx to remove clot (or maybe remove P if another cause??)

63
Q

how to manage ICP with meds. what are they addressing?

what are the names or classes that address the limited amount of space

A

lasix
mannitol or 20% osmitrol
hypertonic saline sometimes
all of these dec brain water

carbonic anhydrase inhibitor-CSF
and vasoconstricotrs-blood

64
Q

what other meds beyond treating the ICP do you give that prevents symptoms from worseinging the problem

A
sedatives
anxiolytics
might put in barbituate coma
pain control --i imagine you have to be careful not to cause vasodilation here?
antiemetics
65
Q

beyond meds what do you do for inc ICP

A

dec stimulation–dark environment

might intubate and hypervntilate them to cause vasoonstriction. dont ant Pc02 below 33 as normal is 35-45

66
Q

pt is victim of multi trauma and has bleeds and inc ICP is lasix a good idea

A

lasix–>diuresis which helps dec ICP but if theyre having circulation problem the pros and cons must be weighed

67
Q

what is mannitol and when should mannitol not be used

A

20% osmitrol is a high molecular weight sugar. When pt is hyperglycemic they pee d/t the high osmotic pull. After 5min lg volume. In order for the mannitol to work the cap membranes must be intact. With inflm the pt must not have inc cap permb. Only give mannitol within first 12hr as if after it moves into interstitial space and takes water with it

68
Q

giving which drug interferes with synthesis of CSF

A

a carbonic anhydrase inhibitor CAI-eg diamox

69
Q

what is the ICpressure during spatial exhaustion

A

20-40mmHG

70
Q

how do you address chemical burn

A

flush with copious amounts of water immediately

71
Q

what does a first degree superficial burn look like

A

1o - Superficial
Dry, red, blanches
Painful

72
Q

what does second degree burn look like

aka

A

2o – partial thickness
Red, moist, blisters
Painful

73
Q

3rd degree burn characteristics

aka

A

3o – full thickness
Dry, no blanching
painless

74
Q

how is nervous system affected by burn

A

pain

75
Q

resp effects of burn

A

-airway obstruction

CO poisoning

pulmonary edema

hypoventilation

76
Q

cardiovascular complications from burn

A

hypovolemia

77
Q

Gastrointestinal

complications from burn

A

paralytic ileus

GI bleed

electrolyte imbalance

78
Q

why is it essential to know amount of body burnt

A

to det fluid resuscitation parameters

79
Q

what is the best indicator of cardiac output in a burn pt

A

urine ouput

80
Q

what is the timeframe of % of fluids lost after a burn

A

50 % in first 8 hours post burn

50% over next 16 hours

81
Q

how do you calculate how much fluids someone will lose from burn

A

2-4 ml/kg/% of total body surface area burned (>2nd degree)

2 x 60 kg pt x 40% burn = 4800 ml in 24 hours = 2400 in first 8 hours = 400 ml/ hour x 8 hours

82
Q

how might the kidnys be affected by burn

A

Renal
rhabdomyolysis
shock

83
Q

possible problems from burn to MS system

A

Musculoskeletal

limb loss

wounds infection

84
Q

what is the problem with circumferential burn

A

Circumferential deep full thickness burns of an extremity or around the chest or abdomen should be carefully monitored. Oedema and swelling in the tissue deep to the burn cause the unyielding overlying burnt skin (‘eschar’) to act like a tourniquet. In a limb this will result in interference with distal blood flow.

85
Q

what is silver sulfadiazine

A

anti-infetive used for burn pts

86
Q

how is coagulation affected for burn pt

A

hypercoagulable

from loss of fluids?? from SNS and inc coagulabilty there?

87
Q

what are special issues with a burn

A

Continues to burn
Rapid & excessive inflammation
Very painful
Start volume replacement from time of burn

88
Q

what is curlings ulcer

A

gastric or duodenal erosion

(Stress Ulcer) or a Curling ulcer is an acute gastritis erosion. complication from severe burns when reduced plasma volume leads to ischemia and cell necrosis (sloughing) of the gastric mucosa.