Trauma II Flashcards
TBI goal of care
-prevention of secondary brain damage resulting from intracranial bleeding, edema, increased ICP, hypoxia and shock
mild TBI
- GCS of 13-15
- short period of observation, usually 24 hours
moderate TBI
- GCS 9-12
- manifested as intracranial lesions that require surgical evacuation
- early CT
- high potential for deterioration requires early intubation and mechanical ventilation
severe TBI
- GCS less than 8
- carries a significant rate of mortality
- care is directed at perfusion of injured brain
severe TBI care guidelines
- after a primary survey approach, maintain CPP 60-70 mmHg at all times
- fluid resuscitation keep euvolemic
- correction of anemia, maintain Hct 30%
- PaCO2 around 35 mmHg
- insertion of ventriculostomy and control ICP (<20 mmHg)
- positional therapy (HOB 15-30 degrees)
- judicious use of analgesics/sedation
- mannitol
- hypertonic saline
TBI airway and ventilatory management
- hyperventilation only if herniation is imminent
- hyperventilate to PaCO2 of 30 if elevated ICP is not responsive to sedatives, CSF drainage, NMBD, osmotic agents, and barbiturate coma
early control of airway in TBI
- orotracheal intubation to maintain SpO2 >90%
- maintain normoventilation to help in the reduction of hypercarbia and hypoxemia
- judicious use of induction agents (Prop and etomidate)
- NMBD to avoid coughing and bucking
establish CV stability in TBI
- avoid intracranial hypertension (ICP > 20)
- avoid systolic hypotension
- placement of A line in addition to standard monitors
- low concentrations of sevo, iso, or des (all cerebral vasodilators so use with caution)
- avoid N2O
management of ICP in OR for TBI
- mannitol 0.25-1 g/kg for control of ICP
- consider hyperosmolar therapy as ordered by surgeon
- corticosteroids have been shown to increase mortality in TBI, so avoid use
mannitol dose
0.25-1 g/kg
SCI causes
MVA
falls
penetrating trauma
SCI incidence
10,000 americans each year
most common location for SCI
low cervical spine (C4-C7)
SCI injuries include
- sensory deficits
- motor deficits
- OR BOTH YIKES
3 factors that determine outcome of SCI patient
- severity of acute injury
- prevention of exacerbation of injury during rescue, transport and hospitalization
- avoidance of hypoxia and hypotension
early SCI treatment focus
-adequate perfusion to prevent secondary injury from forming
when does autonomic hyperreflexia occur?
- complete injury above T5
- occurs in 85% if SCI
SCI management
- treatment = aimed at preservation of perfusion (avoid hypotension or correct immediately)
- avoid hypoxemia (hypoxia and hypercarbia can further accentuate the damage sustained with SCI)
- MAP maintained normal to high
- neurogenic shock can occur (hypotension and bradycardia)
- adequate circulation
- glucocorticoid bolus OK (controversial)
- C spine evaluation
C spine clearance includes
- X ray of all 7 cervical vertebrae
- non-obtunded and non-sedated patient can move their neck without pain
what C spine injuries are commonly missed
- C1-2
- C7-T1
- difficult to image