Trauma Flashcards
Top reasons for shock in trauma patients:
- ) Hypovolemia
- ) Hypovolemia
- ) Hypovolemia
- ) Obstructive (PTX, tamponade, ACS)
- ) Distributive (neurogenic/SCI)
- ) Cardiogenic (pts predisposed to HF can’t compensate)
Shock ABCs. What three things does Dr. Hameed consider when assessing ‘C.’
- Is the patient in shock?
- What type of shock is it?
- How do I reverse this kind of shock?
What is the most reliable marker for end-organ perfusion?
Urine output
ATLS classifications for blood loss:
Class One: 0-15%, 750mL
Class Two: 15-30%, 750-1500mL (tachypnea, tachycardia)
Class Three: 30-40%, 1500-2L (decreased UO, DLOC, Low BP)
Class Four: 40% +, 2L +
What percent of patients with decreased EOP shock will have normal vital signs?
50%
Be sure to check the lactate/base deficit off either an arterial draw or CVC. Don’t use a VBG since it can be confounded by regional ischemia.
Populations that hide shock particularly well:
- Obstetrics
- Pediatrics
- Beta Blockers
Increasing lactate levels in a trauma patient need to be cleared somehow. If the liver is shutting down, where does it go?
Tachypnea isn’t just a compensatory mechanism for decreased cardiac output. It’s an early sign in shock that lactate levels are increasing and need to be blown off.
A base deficit of lower than ______ is often the first sign of hypovolmeic shock.
-3
Lactate will start to increase. Hgb won’t begin to get ‘watered down’ for 1-2 hours post insult.
What do hematocrit/hemoglobin levels do in a rapidly exsangunating shock patient?
Stay the same. It takes time for fluid to be pulled from the extravascular space and cause hemodilution.
“If you throw out half a pitcher of Kool-aid, it still tastes just as sweet.”
What is the target Hgb for actively bleeding trauma patients?
100g/l
Not treating anemia has certain benefits. What are some advantages of anemic blood?
It has increased levels of 2-3DPG which causes a right shift of the oxyhemoglobin dissociation curve. It’s also less vicious so it travels through the capillaries more easily and is also less strain and MVO2 for the heart to pump.
Another detriment of blood transfusion is that it suppresses the immune system.
Would you expect a high/low/normal lactate in regional true ischemia such as ischemic gut?
Low/Normal.
Shock does not affect the body in a homogenous manner. The gut can become ischemic in which case there is no blood going in or out. No blood coming out means no lactate.
The liver can be affected early in the shock process since blood is first shunted away from the skin and gut. What lab values can help support a diagnosis of early shock?
AST and ALT could be elevated due to shock liver.
Three pieces of imaging that should be ordered on every trauma patient:
- CXR
- PXR
- EFAST
What is a Morel-Lavallee lesion?
A closed degloving injury that shears open subcutaneous bridging vessels and presents as a boggy, hemolymphatic mass. (Think motorcyclist sliding on his back across pavement. This serious hemorrhage will not be seen on an EFAST)
What’s the harm with a tourniquet applied too loosely?
It will impair venous return but still allow arterial hemorrhage, thus making blood loss worse.
When it comes to the trauma triad of death, what interventions can be done to prevent worsening coagulopathy?
- Avoid dilutional fluid resuscitation with crystalloids
- Replace volume with a 1:1:1 ratio
- Allow permissive hypotension in penetrating trauma
- Keep pt warm
- Oxygen
- TXA
What 6 factors of shock physiology contribute to worsening coagulopathy?
- Consumption of clotting factors
- Hypothermia
- Acidosis
- Third spacing dilution
- Shock liver
- Fibrinolysis
What three patient populations are permissive hypotension contraindicated in?
TBI
SCI
Obstetrics
*Keep MAP > 80 but SBP < 160
Land marking for chest tube insertion:
- Infamammary fold to between anterior and midaxillary line
- Cut skin/adipose through to rib
- Choke up on scalpel to prevent going to deep, then punch through to lung just superior to 5th rib.
- Barbarically pull tissue away
What electrolytes must be monitored when transfusing large volumes of RBCs?
Ca++ (due to citrate in RBCs)
K+ (in case of lysis during storage)
Fibrinogen is one of the first clotting factors consumed during ongoing massive hemorrhage. How can you replace it?
Cryoprecipitate
- fibrinogen
- factor VII (extrinsic pathway)
- factor VIII (intrinsic pathway)
- von Willebrand factor
What is the future of guiding massive blood transfusions?
ROTEM
You’re treating a trauma patient and you set some parameters. One of them is to treat if the heart rate climbs over 110. Now you’re looking at a sinus tach of 111. The patient is in obvious pain and anxious about flying. Do you let it slide?
No! It is easy to write something off as non-serious, but if you don’t stay on top of a trauma patient, they will deteriorate quickly and you will be left scrambling.