Trauma Flashcards
What is the leading cause of death for patients ages ____ to _____
Trauma
ages 1 - 45
US
Leading cause of death worldwide between ages 15 and 44?
Still trauma
Name the three components of an initial evaluation
- Rapid Overview
- Primary Survey - ABCDE
- Secondary Survey
Primary Survey
- Airway Patency (obstruction?)
- Breathing
- Circulation (skin temp, color, 2 large bore IVs)
- Disability (GCS)
- Exposure
Airway Patency Assessment
Are they talking?
Agitated? = Hypoxia
Gargling? = Tracheobronchial injury
Flail Chest
3 or more fractured ribs associated w/costrochondral separation
resp insufficiency and hypoxemia over several hours w/deterioration of CXR and ABG
pain management > mechanical ventilation
CPAP or BiPAP may be helpful
Tension Pneumothorax
due to air leaking from the lung, or chest wall into the pleural space
pneumothorax presents in 40% of blunt thoracic injuries
this is why nitrous oxide is c/i in thoracic trauma
Massive hemothorax
> 1500 mL
pneumothorax s/s (6)
hypotension hypoxia tachycardia diminished breath sounds SQ emphysema distended neck veins
GCS Scale - Eye Opening
4 points = spontaneous
3 = to verbal
2 = to pain
1 = not at all
GCS Scale - Verbal
5 = normal conversation 4 = responds, but doesn't make sense 3 = responds, incomprehensible 2 = no words, moans 1 = no response
GCS Scale - Pain
6 = follows commands 5 = localizes pain 4 = withdraws from pain 3 = decorticate 2 = decerebrate 1 = does nothing
GCS < 8
intubate
GCS > 13
mild brain injury
GCS 9 - 12
moderate brain injury
Secondary Survey
Begins after critical life saving actions have begun i/e/ (intubation, chest tubes, fluids)
Focus includes: history, LAMP
DM? Low blood glucose?
Airway Evaluation
Assume patient absolutely requires an airway and cannot be re-awakened electively
Jaw broken
can you mask ventilate? maybe just NRB and intubate from that
Direct airway injury
maybe no edema now.. but there will be later
Airway obstruction considerations (8)
- cervical deformity
- cervical hematoma
- foreign bodies
- dyspnea, hoarseness, stridor, dysphonia
- SQ emphysema + crepitation (tracheal tear?)
- Hemoptysis (no fiberoptic w/active bleeding)
- Tracheal deviation - tension pneumo + death
- JVD - cardiac tamponade
Cardiac tamponade s/s
narrowed pulse pressure
muffled heart sounds
hypoperfusion
Some considerations for airway management (3)
Full stomach
Oral ett > nasal ett b/c airway pressures
C-Spine stability
Major contraindications for a nasal intubation
basilar skull fracture
s/s basilar skull fracture
battle sign
CSF leakage
raccoon eyes
Indication for ETT intubation (7)
- cardiac/resp arrest
- Deteriorating respiratory condition (contusion/pneumo/burns)
- Need for deep sedation
- GCS < 8
- CO poisoning - need that 100% FiO2
- Uncooperative/intoxicated pt
- Transient hyperventilation for a head injury
Tracheotomy
takes longer to perform
need neck extension (c/i c-spine injury)
Cricothyroidotomy contraindications (2)
c/i in those < 12 y.o.
laryngeal damage - c/i
How long can a cricothyroidotomy last?
72 hours
Cricothyrotomy supplies
- Scalpel - 2.5cm vertical incision, keep < 1.3 cm deep
- Dilator
- Tracheal Hook - Keep dilator in until tracheal hook placed
- 10 cc syringe + trach cuff
- Trach tube (< 7 ) or ETT 6.0
Instances you do a cric (5)
macey
- massive trauma to the face
- angioneurotic edema
- supraglottic obstruction
- croup/epiglottitis
- inhalational injury
Gold standard for C-Spine injury
fiberoptic intubation - awake (pt gotta be cooperative tho)
LMA
contraindicated for a definitive airway
etomidate
0.2 - 0.3 mg/kg (if severly compromised, you will still see a drop in BP)
ketamine
2 - 4 mg/kg (c/i in head injury)
4 - 10 mg/kg IM
- direct cardiac depressant; usually masked by catechols*
- great choice for tamponade pt*
Sux
1 - 1.5 mg/kg IV
30 second onset; fasiculate
5 - 12 m duration
c/i w/globe injury or head injury
pre-treat w/rocuronium 5 mg
Roc
1.2 mg/kg IV
30 - 60 s onset; 60 - 90 m duration
may need gentle mask ventilation (MRSI)
Scopalamine
0.4 mg/mL
Dose = 5 - 10 mcg/kg
c/i in pregnancy
How to clear a c-spine pt
Complete xray C1 - C7
pt not obtunded or under influence
manual in-line stabilization
(MILS)
hold mastoid process
Name the 3 factors that influence a penetrating injury
- type of instrument
- velocity
- characteristics of the tissue through which it passes through
laryngotracheal damage s/s (7)
hoarseness muffled voice dyspnea stridor dysphagia cervical pain/tenderness flattened thyroid cartilage
why do you intubate w/c4 or c5 lesion
diaphragm
why do you intubate w/c6 or c7 lesions
pt cannot cough or clear secretions with those
pulmonary contusion
bruising of alveoli allowing protein rich fluid to escape and increase alveolar capillary membrane distance
leading to ARDS
keep pplat < 32
hemothorax
make sure you havee adequate fluids on board prior to ct placement
s/s: hypotension, hypoxemia, tachycardia, increased CVP
anesthesia considerations for a hemothorax
one lung ventilation
use regular ett first
name the 3 types of a pneumothorax
simple
communicating
tension
when do you treat a pneumo
> 20% of the lung collapsed
needle decompression at 2nd/3rd rib - mid clavicle
14 or 16 gauge
initial response to shock
mediated by neuroendocrine system
hypotension - catecholamine + vc
-heart kidney brain preserved
name all the hormones released during shock
renin/AT vasopressin ADH GH glucagon cortisol epi/NE
Lungs + Shock
the destination of inflammatory byproducts that accumulate in capillary beds leading to ARDS
Trigger for MOSF
likely the gut b/c hypoperfusion
coagulopathies explained (4)
hypotension, tissue injury
inflammatory response
endothelial activation of protein C
hyperfibrinolysis d/t APC
what does APC do
blocks factor 5/8 to limit clot propagation in normals.
why do trauma pt have coagulopathies (4)
- dilution
- hypothermia/acidosis
- TBI
- hemorrhagic shock
PRBC Hct?
55%
Plt (50 ml) =
5.5 x 10^10
Plasma =
80% coags
What is base deficit?
reflects the severity of shock, oxygen debt, changes in oxygen delivery, fluid resuscitation adequacy ?
Base deficit of 2 - 5 =
mild shock
base deficit of 6 - 14
modereate shock
what is severe shock
base deficit > 14
blood lactate level
leess specific than base deficit, but still important
elevated levels correlate w/hypoperfusion
normal 0.5 - 1.5 and half life is 3 hours
plasma lactate > 5 =
significant lactic acidosis
how do you assess systemic perfusion?
- vs - not indicate occult hypoperfusion
- UO - can be inaccurate d/t diuretics, renal injury, intoxication
- acid-base balance - confounded by resp. status
- lactate clearance - timing
- CO - need PA cath
- SVO2 - accurate marker, hard to get
- SVV
how long can an IO stay?
2 - 3 days
Goals for EARLY resuscitation (24 - 48h)
- SBP 80 - 100
- HCT 25 - 30
- PTT (25 - 35) and PT (11 - 13) WNL
- PLT > 50
- Normal Ionized Calcium 4.6 - 5.2
- Core temp > 35
- prevent worsening lactate, acidosis
- keep them without pain and under anesthesia
Risks of aggressive volume replacement during early resuscitation
increased BP decreased viscosity decreased hct decreased clotting factor conceentration need more tx lytes out of wack direct immune suppression premature reperfusion
Goals for LATE resuscitation
SBP > 100 and HR < 100
- maintain hct at individualized level
- normalize coags/lytes/body temp
- good urine output
- reverse systemic acidosis
- good CO
- seerial lactate. if it does not decrease, something else. is going on
What is the end point for resuscitation
Lactate < 2mmol
Base Deficit < 3
Gastric intramucosal pH > 7.33
No type and cross - what do you give ?
RH negative, esp if woman is of childbearing eyars
FFP:PRBC
2 u FFP to every 4 u PRBC
what can you mix w.blood
NS
plasmalyte
how fast can you give fluids
1500 mL/m
crystalloid, colloid, PRBCs, plasma
all mix together in reservoir
38 - 40 degrees Celsius
what is the lethal triad
coagulopathy
acidosis
hypothermia
what does hypothermia do ? (6)
- acid base d/o
- coagulopathy (inhibits thrombin and fibrinogen, impairs platelet, abnormal calcium and K hemostasis)
- myocardial dysfunction
- oxy-hgb to left
- decreases metabolism of lactate, citrate, and some anesthetic drugs
- causes vasoconstriction - BP appears higher
29 degrees c PT and PTT
increase by 50%
29 degrees C platelets
decrease 40%