Training Prescription for Anaerobic Adaptation Flashcards
What effect does a 6-second sprint have on muscle metabolites?
14% decrease in glycogen concentrations, 13% increase in ATP concentrations, no change on ADP and AMP concentrations, 57% decrease in PCr concentrations
What effect do 10 6-second sprints have on glycogen breakdown and glycolytic rate in skeletal muscles?
91% decrease in glycogenolytic rate, 87% decrease in glycolytic rate, 53% decrease in rate of glycogen degredation
What are the main contributions to ATP production after 1 6-second sprint compared to 10 6-second sprints?
After 1 sprint 49.6% PCr and 44.1% glycolysis, after 10 sprints 80.1% PCr and 16.1% glycolysis, contribution of glycolysis down by the end due to a decrease in glycogen phosphorylase and PFK
Why is glycogen breakdown low after 3 30-second sprints?
Increase in AMP activates PFK, PFK regulates glycolysis, however glucose-6-phosphate accumulates in the muscle reducing glucose concentration gradient, therefore less glucose goes into the muscle increasing blood glucose levels
What are the chronic adaptations of sprint training?
Increased peak power, increased total work done (ability to maintain high power output), PFK max enzyme activity increased by 49%, hexokinase max enzyme activity increased by 56%, no change in glycogen phosphorylase enzyme activity or LDH enzyme activity, oxidative metabolism enzyme increased, VO2max increased roughly 7% after 7 weeks, more mitochondria = more ATP production = more PCr resynthesis
What effect does recovery time have on adaptation in sprint training?
Short recovery improves VO2max and increases mitochondria, long recovery increases sprint performance and PCr resynthesis
Explain the potential mechanism of why sprint training increases oxidative enzyme activity
Sprint training stresses the mitochondria and oxidative enzymes, studies show increase in malate dehydrogenase, succinate dehydrogenase and cytrate synthase after 7 weeks (3 major oxidative enzymes)
