toothwear Flashcards
causes of TSL
caries
trauma
developmental problems
toothwear
causes of NCTSL
trauma
developmental problems
toothwear
what is physiological toothwear?
normal wear associated with normal function
what does physiological toothwear increase with?
age
elderly often have TW - not necessary to tx
what is a normal amount of physiological toothwear per year?
20-38 um
pathological toothwear
remaining tooth/pulpal health compromised
OR
rate in excess of what is expected for that age
OR
pt experiences masticatory/aesthetic deficit
attrition
physiological wearing away of tooth structure as a result of tooth to tooth contact
where are attritive lesions located and why?
occlusal and incisal
contacting surfaces
what does attrition do to the length of incisor and canine crowns?
shortens them
cause of attrition
bruxism - parfct habit
restoration wear in attrition
equal to tooth
progression of attrition
polished facet on cusp/slight flattening of incisal edge
decrease in cusp height and flattening of occlusal inclined planes
flat facets
abrasion
physical wear of tooth substance through an abnormal mechanical process, independent of occlusion
- foreign object/substance repeatedly contacting tooth
site and pattern of abrasion
related to abrasive element
often labial/buccal/cervical on 3,4,5
abrasive lesions
V or O lesions
sharp margin at E edge - D worn preferentially
- notching of incisal edges
main causative factor in abrasion
toothbrush
other causative factors in abrasion
habits/lifestyle/occupation
pins, nails, electrical wire, stripping, fishing line, thread, pipe smoking, E-cigs
erosion
loss of tooth substance by a chemical process that does not involve bacterial action
what is the main cause of pathological toothwear?
erosion
erosion is caused by a chronic exposure of hard tissues to what?
acids (intrinsic or extrinsic)
what is the position and severity of erosion determined by?
source, type and freq of acid exposure
progression of erosion
E loss of surface detail, flat, smooth, shiny. bilateral concave lesions
(not chalky like bacterial acid decalcification)
D exposure
- cupping of occ surfaces (preferential wear)
erosion and restorations
sit proud of tooth
erosion and staining
no tooth staining as gets washed away by acid
in erosion is the base of the lesion in contact with the opposing tooth?
no
what happens to the incisal edges in erosion?
increased translucency - dark
abfraction
the loss of hard tissues from eccentric occlusal forces leading to compressive and tensile stresses at the cervical fulcrum areas of the tooth
2 theories for abfraction
basic cause of all non-carious cervical lesions
multifactorial aetiology - occlusal stress, abrasion, erosion
what is the consequence of abfraction?
pathological loss at the cervical margin where stress concentration
V shaped loss where tooth under tension - sharp rim at ACJ
what is abfraction caused by?
biomechanical loading forces
- flexure and failure of E and D at a location away from loading
- disruption of ordered crystalline structure of E and D by cyclic fatigue
- cracks - chips out
cervical wear causes
multifactorial? abrasion?
likely erosion + abrasion +/- abfraction
no definitive conclusive studies
location of cervical wear
buccal of 4,5,6,7,8
almost never lingually
usually more U
features of cervical wear
goes with good OH
restorations and tooth wear at same rate
what is the cause of almost all toothwear?
multifactorial
what is the most common type of wear in older patients?
physiological
gender pattern of wear
M>F
assessment of toothwear overall
recognise problem grade severity diagnose likely cause(s) monitor progression - active/historic? - preventive measures working/need active Rx tx?
what is a contraindication for complex tx in PDH?
non-regular
poorly motivated
phobic
SH aspects of toothwear
lifestyle stresses - bruxism occupation (drivers grind) alcohol consumption (reflux) diet habits sports (weightlifting, sports gels v acidic)
history - main complaint?
aesthetics
fct difficulties - masticatory efficiency, biting tongue/lips
pain
why is it uncommon to feel pain in toothwear?
unless rapidly progressing/pulpal involvement
as wear usually slow - get secondary dentine and pulp recedes
MH aspects
esp in erosion meds with low pH meds which dry mouth eating disorders alcoholism heartburn - pts not always aware of reflux - may be benign nocturnal GORD hiatus hernia rumination pregnancy - transient - morn sickness, reflux and heartburn GP referral? - get consent
EO exam
TMJ muscles mouth opening ? parotid hypertrophy - if bulimic ? overclosure - if nose and chin approaching each other lip line smile line
EO exam - TMJ
restriction of movement
clicking
crepitus
EO exam - muscles
hypertrophy of masseter
EO exam - mouth opening
restriction (<4cm) - tense muscles?
deviation during movement
assessing occlusion
assess FWS, OVD and RFH - often normal if slow wear
dento-alveolar compensation?
record OB and OJ
stable contacts in centric relation?
what are tooth contacts like in excursive movements?
STs
dry?
buccal keratosis or lingual scalloping? - sign they are likely bruxist
IO exam
occlusion STs OJ perio charting
location
anterior
posterior
generalised
where does toothwear caused by bulimia typically affect?
anterior and palatal
basic severity grading
E only
into D
severe
Smith and Knight tooth wear index grades
0-4
Smith and Knight tooth wear index grade 0
no loss of E surface characteristics
Smith and Knight tooth wear index grade 1
loss of surface E characteristics
Smith and Knight tooth wear index grade 2
B, L and O loss of E, exposing D for <1/3 of the surface
incisal loss of E
minimal D exposure
Smith and Knight tooth wear index grade 3
B, L and O loss of E, exposing D for >1/3 of the surface
incisal loss of E
substantial D exposure
Smith and Knight tooth wear index grade 4
B, L and O complete loss of E, pulpal exposure or exposure of secondary dentine
incisal pulp exposure or exposure of secondary dentine