Tiff Test 3 COPY Flashcards

1
Q

HIV attacks immune system by destroying __ and __

A

T cell, and CD4 cells

-Attacks the cell, takes over cell function, replicates

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2
Q

Priority Patient Problems for HIV

A

-Potential for infection: avoid exposure, drug therapy, education, private room, prevention
-Inadequate oxygenation: drug therapy, comfort, rest, support
-Pain: comfort measures, drug therapy, alternative therapy
Inadequate nutrition: nutrition, mouth care, drug therapy
-Diarrhea: minimize diarrhea, restore skin integrity
-Reduced skin integrity
-Confusion: reorientation, safety, support
-Reduced self-esteem: trust, acceptance, encourage self care
-Potential loss of social contact

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3
Q

Systemic Manifestations of Anemia

A

▪ Integumentary
-pallor, cool to touch, brittle nails, intolerance to cold

▪ Cardiovascular
-tachycardia at rest and increases with activity, orthostatic hypotension, murmurs or gallops (when severe)

▪ Respiratory
-dyspnea on exertion, decreased O2 sats

▪ Neurological
-increased fatigue, decreased LOC, headache

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4
Q

Sickle Cell Crisis Interventions

A

1 priority is managing pain

Managing pain:

- IV analgesics: Opiods
- IV hydration
- Hydroxyurea
- Complimentary therapies

Preventing sepsis:

- role of the spleen: immunizations
- antibiotics
- assessments
- lab data

Perfusion:

- Remove restrictive clothing
- Keep room > 72 degrees F
- Keep extremities extended
- Oxygen
- Transfusion

reduce condition that is causing the sickling
IV opioids is the best way to manage pain, PCA
IV hydration- normal saline
IV fluids will hopefully reduce the clumping of the sickle cells
most patients who come in are dehydrated in sickle cell crisis so blood is hypertonic so give NS or hypotonic solution. BP will tell how hydrated they are
Hydroxyurea - medication that can help with sickle cell crisis
oral, reduce number of sickling events
comfortable in bed, extremities extended: get blood flow back, restricted clothing: can encourage clot formation, do not move knees above bed, keep temp above 72: cold causes vasoconstriction, elevated HOB no more than 30 degrees
decreases immune function can lead to sepsis
spleen can not function with our immunity response- get flu vaccine and pneumococcal
o2 given during crisis
transfusion with RBC will dilute hemoglobin s levels and will provide RBC when pt is anemic
*teaching prevention of sickle cell crisis, and when to come in for treatment: early is better!

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5
Q

DI

A
Water metabolism problem caused by an ADH deficiency (either decrease in ADH synthesis or inability of kidneys to respond to ADH)
Classifications
Kidneys:
Do not conserve fluid
Do not reabsorb water
Put out large amounts of dilute urine
Risk for dehydration!
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6
Q

Assessment findings in DI

A

think about dehydration due to massive water loss
skin- dry, cracked, poor skin turgor

lab values- hypernatremia due to loss of water

GU-Increased UOP, dilute, low specific gravity
Cardio-Hypotension, tachy, weak pulses, hemoconcentation (risk for hypernatremia)
Neuro-Great thirst, Decreased LOC (with dehydration)

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7
Q

Management of DI:

A

I/Os, IVF, weights, urine specific gravity
Oral chlorpropamide
Desmopressin acetate
Early detection of dehydration and maintenance of adequate hydration
Lifelong vasopressin therapy with permanent condition
Teach patients to weigh themselves daily to identify weight gain

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8
Q

SIADH Assessment:

A

Decrease in serum sodium levels
CNS/LOC changes
Risk for seizure
Fluid volume overload/water retention:, UOP decrease, Bounding pulses, Risk for heart failure and pulmonary edema

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9
Q

SIADH Interventions

A
Fluid restriction
Drug therapy: diuretics, hypertonic saline (3% saline IV), Vasopressin antagonists
Monitor for fluid overload
Monitor labs
Safe environment
Neurologic assessment
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10
Q

Posted care for hypophysectomy:

A

managing ICP and anything that will delay healing

Monitor neurologic response- change in LOC, vision, mental status, strength in extremities

Assess for post nasal drip- csf leak due to “digging around in brain” (clear drainage, positive for glucose, severe headache)

Elevate HOB- promote drainage to release any ICP

Assess nasal drainage- color, clarity, odor

Avoid coughing soon after surgery- can increase ICP

Assess for meningitis- sx in brain increases risk for meningitis. monitor temp, vitals, change in LOC, headache, neucoregidity

Hormone replacement- lifelong HRT

Avoid bending- decrease ICP

Avoid straining at stool- to decrease ICP

Avoid tooth brushing- can introduce foreign particles, and infection and can cause delay in healing in sx site

Numbness in the area of the incision

Decreased sense of smell

Vasopressin- at risk for DI

Monitor fluid balance- i/o’s, cardiovascular status

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11
Q

Mineralocorticoids:

A

Fluid and electrolyte balance
Regulated by RAS, K levels, ACTH

Aldosterone:
Regulates Na++ and K+
Promotes sodium and water reabsorption
Promotes potassium excretion

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12
Q

s/s of adrenal crisis:

5 S’s and 3 H’s

A

Super low BP (nothing will bring it up, no fluids or vasopressors)
Sudden pain in stomach, back, or pain
Syncope
Shock
Severe vomiting, diarrhea, headache
Hyponatremia (less than 135) vomiting and diarrhea
Hypoglycemia- cortisol increases BG and if body doesn’t have cortisol will have low BS
Hyperkalemia- greater than 5.1

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13
Q

Nursing management of adrenal crisis:

A
Priority is getting pt stable
Administer cortisol STAT!
Iv fluids
Oral glucocorticoids or mineral (replace aldosterone)
Monitor glucose, K+ and Na+
Prevent infection
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14
Q

ADH: antidiuretic hormone (vasopressin)

A

Regulates water in the body through water retention, constricts arterioles
Renal tubules retain water
Increase adh, kidneys to retain water
Decrease adh, kidneys will get rid of water
Hypothalamus produces adh -> signals to posterior pituitary to secrete and that is where it is stored.

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15
Q

SIADH: syndrome of inappropriate diuretic hormone - INCREASED adh

A
Adh is being produced somewhere other than hypothalamus ot its damaged
Lung cancer is a major cause
Damage to posterior pituitary
Infections
Medications- chlorpropamide
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16
Q

s/s of SIADH

A
Increase ADH: water retention
Fluid overload (edema, weight gain)
HTN
Hypovolemia
Seizures
Tachycardia
Hyponatremia (increase of water in body but sodium stays the same so sodium is diluted)
Confusion - edema of brain
Anorexia- so full of water they do not want to eat
Low UO: high specific gravity
17
Q

management of SIADH

A
Daily weights, strict IO’s, safety
Fluid restriction
Meds:
Loop diuretics- watch out for hypokalemia because they like to deplete K+ levels so check potassium levels. 
Hypertonic iv 3% NS
declomycin
18
Q

adrenal cortex:

A

Adrenal cortex: sex hormones
Releases corticosteroids and sex hormones
Steroid hormones: aldosterone and cortisol
Aldosterone: mineralocorticoid, regulates BP via RAAS, retention of Na+ and secretes K+
Cortisol: glucocorticoid, stress hormone, regulates BG, breakdown fats, carbs, and proteins, helps body deal with stress ex: illness, external stress, increases BP

19
Q

Addisons

A

Decrease secretion of cortisol and aldosterone-

Autoimmune disorder- attacking the cortex due to cancer, TB, or trauma

20
Q

S/S of addisons:

A
Low “steroid” hormones
Sodium and sugar levels will be very low, salt cravings
Tired and weak
Electrolyte imbalance (K+ and Ca+)
Reproductive changes
lOw BO
Increased pigmentation (hyperpigmentation of the skin)
Diarrhea and nausea, depression
21
Q

Management of addisons

A

Monitor for hypoglycemia and hyperkalemia
HRT
Report stress/illness
Can not abruptly stop medication
Medic alert bracelet
Diet high in protein, carbs, and normal Na+
Avoid stress, illness, and strenuous exercise

Watch out for addisonian/adrenal crisis
5 s’s
Sudden pain in stomach, back, and legs
Syncope
Shock
Super low bp
Severe vomiting, diarrhea, and h/a 
Tx for addisonian crisis
Iv cortisol
Iv fluid
22
Q

Thyroid Secretions:

A
T3 and T4  = Thyroid Hormone
Control of metabolism
Exert effects on HR and contractility
Affect RR and drive
Act as insulin antagonists
Increase RBC production
Calcitonin = helps regulate serum calcium and phosphorus levels
23
Q

Calcitonin

A

Thyroid control Ca and phosphorus levels in blood

Calcitonin decreases serum Ca and phosphorus levels by reducing bone resorption (breakdown)

Serum calcium levels control calcitonin section
Low serum CA, suppress calcitonin
High serum CA, increase secretion
Actions opposite of parathyroid hormone

24
Q

Thyroid Storm:

A

s/s:
increases metabolic rate
key manifestations: fever, tachycardia, HTN
anxious, chest pain, n/v, diarrhea
so hyperstimulated can cause dysrhythmias

25
Q

Management of thyroid storm:

A
Airway
Antithyroid drugs
Blood pressure drugs
Reduce fever
Monitor cardiac, vital signs
Prevent vascular collapse
Glucocorticoids (Hydrocortisone) to treat/prevent shock
26
Q

Parathyroids:

A

4 small glands
Embedded posterior thyroid
Easily removed by accident in thyroid surgery
Secrete Parathyroid hormone (PTH)
Regulate calcium and phosphorus by acting on bone, kidneys and GI tract

27
Q

Parathyroid Hormone (PTH)

A

-Kidneys
PTH activates vitamin D, increases absorption of CA and phosphorus
Kidney tubules reabsorb CA and put into serum
Bone
Main storage site of CA
PTH increases bone resorption (from bone to blood), thus increasing serum CA

Serum CA levels determine PTH secretion
PTH secretion decreases when CA levels are high
PTH increases when CA levels are low
Work with calcitonin to maintain balance
Inverse relationship with phosphorus

28
Q

Hyperparathyroidism

A
Common cause benign tumor of parathyroid
Manifestations:
High serum Ca+ and hypophosphatemia
Cardiac dysrhythmias
GI problems
Muscle weakness
Pathological fractures
Skeletal pain
Emotional changes
Kidney stones r/t output of Ca and Phos
29
Q

Nonsurgical and Surgical interventions for hyperparathyroidism

A

furosemide- increase ca+ excretion
Prevent injury/safety: because of loss of ca+ and bones are fragile
calcitonin has opposite effect of parathyroid, also can give other drugs that help or block hypercalcemia and block PTH

30
Q

Hypoparathyroidism

A
S/Sx:
High phosphorus
Low Calcium
S/S of tetany
Tremor and Spasms
Changes in mental status 
Cardiac dysrhythmias

Low Ca with hypoparathyroidism

Trousseau’s sign: carpal spasms with BP cuff 3 minutes

Chvostek’s sign: Tap facial nerve (in front of ear) to produce facial spasms

31
Q

AKI INTERVENTIONS

A

Prevention, perfusion, nephrotoxic medications
Monitor intake and output. first indicator there is problem
Monitor vital signs and MAP (CVP, art line)
Daily weights
Labs: trends
Promote fluid balance.
Assess for manifestations of volume excess, pulm edema, electrolyte imbalance
Nutrition therapy
RRT
Drug therapy:
Includes diuretics to increase UOP
Rid body of excess fluid and electrolytes
Fluid challenges to promote kidney perfusion

32
Q

pre renal kidney failure

A

Most common type: hypovolemic shock and heart failure
Reduced perfusion:Results from decreased blood flow to kidney and consequent ischemia of nephrons
Keep MAP > 65mmHg
Restoration of renal blood flow and GFR can rapidly reverse prerenal AKI

33
Q

LABORATORY ASSESSMENT of AKI

A

Serum creatinine
BUN
BUN to serum creatinine ratio: BUN divided by creatine - increased ration increased fluid volume deficit, decreased fluid volume excess
Creatinine clearance: looks at how well creatinine is cleared from kidneys- look at this with meds
Blood osmolarity: concentration of blood, good indicator of hydration (normal range: 285-295)
GFR:
ABGs: change in terms of metabolic acidosis and the kidneys inability to produce and secrete bicarb
Electrolytes
Urinalysis: observing urine looking for quantity, specific gravity, pH, color, ketones

34
Q

post av fistula assessment

A
don't bend arm
don’t use arm
do not take iv, sticks on side
do not take bp
make sure they have distal pulses
don't carry heavy objects
avoid tight clothing
don’t sleep on it
35
Q

Patient Considerations on HD

A
Restricted d/t keeping appt 3-4x/week
Restricted fluids  
Restricted diet
Anemia
Major sleep disturbances
Hypotension
Muscle cramping
H/A, nausea, vomiting r/t cerebral fluid shifts
36
Q

Continuous Renal Replacement Therapy

A

Used for patients too unstable with hypotension for traditional hemodialysis: in ICU settings
Does not cause rapid fluid shifts
Requires access to circulation
Specialized training for nurses to perform
Blood passes through a hemofilter that contains semipermeable membrane

37
Q

lab for hyperparathyroidism

A

High serum Ca+

hypophosphatemia

38
Q

Complications: of PD

A
Complications:
Peritonitis is most common
Serious
Cloudy dialysate
Abdominal pain
Rebound tenderness
Antibiotics
Flushes to abdomen
Unresolved = cath removal = hemodialysis
After approx 1 month, can replace cath
Strict sterile technique is necessary