Theory Flashcards
ISBAR Handover
Introduction and Identity
Situation
Background
Assessment and Actions
Recommendation
What are risk factors for stroke?
hypertension, diabetes, increased cholesterol, poor diet (increased alcohol, salt, saturated fats), smoking, obesity, lack of regular exercise, genetics, cardiac disease (e.g., AF)
What is the most common stroke presentation and why?
MCA infarct - this is because the MCA is a direct continuation of the internal carotid/common carotid artery. Therefor emboli from the heart can travel up this pathway and lodge in the MCA.
What are the key impairments found in a person with a L MCA infarct?
R hemiplegia (UL>LL)
R hemisensory loss (UL>LL)
R homonymous hemianopia
Dysphasia
Dyspraxia
Criteria for PPC (as per Scholes 2005)
Combination of 4 or more of the following criteria:
- CXR report of collapse and/or consolidation
- Raised maximum oral temperature >38 degrees on more than one consecutive post-op day
- SpO2 <90% on more than one consecutive post-op day
- Productive of yellow or green sputum which is different to pre-op assessment
- Presence of infection on sputum culture report
- An otherwise unexplained white cell count >11x10^9/L or prescription of antibiotic specific for respiratory infection
- New abnormal breath sounds which are different to pre-op assessment
Physicians diagnosis of post-op pulmonary complication
Define Hemineglect
Neglect is a disorder of spatial attention defined as failure to attend to one side of the body and/or environment (either sensory, visual or auditory input), however primary sensation is in tact. It is associated with poorer prognosis for functional outcomes.
Cause: strongly correlated w/R (non-dominant) parietal lobe damage
Signs of a scaphoid fracture
1 - Palpate scaphoid in snuff box
2 - compress push Mc down onto scaphoid
3 - ulna deviation overpressure
(+ve for pain)
dysmetria
dysmetria = inaccurate amplitude of movement and displaced force
dyspraxia (apraxia)
a disorder of skilled voluntary movement not attributed to motor, sensory or perceptual disorders
(ie. difficulty motor planning)
cause = associated w/frontal, parietal and temporal lobe damage
dysdiadochokinesia
Difficultly performing rapid alternating movements (RAM)
Cause = ataxia
agnosia
inability to interpret and thus understand sensory information, however primary sensation is intact
cause = parietal/occipital lobe damage
rigidity
an increase in muscle tone leading to resistance to passive movement throughout ROM
- Lead-pipe = constant
- cog-wheel = episodes/jerky
spasticity
motor disorder characterised by a VELOCITY DEPENDENT increase in tonic stretch reflexes (muscle tone) w/exaggerated tendon jerk
Cause: upper motor neuron (UMN) lesion
Clonus
involuntary, repeated, rhythmic muscle contractions
Homonymous hemianopia (HH)
visual field deficit whereby half (L or R) of the visual field is lost
Rebound Phenomena
dysfunction in agonist/antagonist relationship (ie. ability to brake movement)
cause: ataxia
Dysphagia
difficultly swallowing (gag reflex)
dexterity
fine motor skills (especially hands)
What are the common cardiorespiratory physiotherapy problems?
1 - impaired airway clearance
2 - dyspnoea (increase WOB)
3 - decreased Ex. tol.
4 - low lung volumes
5 - impaired gas exchange
6 - decrease mobility
7 - respiratory muscle dysfunction
8 - pain
Respiratory Muscle Dysfunction (Cardio PT Problem)
signs: increase WOB, nocturnal symptoms (e.g. orthopnoea)
treatment: respiratory muscle training, relaxed controlled breathing
Orthopnea
Difficultly breathing in supine position
Impaired Gas exchange (Cardio PT Problem)
Signs: decrease SpO2 (<95%), decrease PaO2 (<80mmHG), increase PaCO2 (>45)
treatment: O2 therapy
Re-Assess: SpO2, ABGs, O2 requirements
decreased mobility (Cardio PT Problem)
signs: decreased ROM, inability to complete ADLs/transfers, bed-bound, post-op, acutely unwell (sedated)
treatment: positioning, assist w/transfers, walking
re-assess: assistance required, functional questionnaires
Impaired airway clearance/mucociliary clearance/sputum retention (Cardio PT Problem)
signs: increase sputum production, change in sputum colour, coarse crac ales, febrile, difficult/weak/ineffective cough, CXR consolidation
Treatment: ACBT, PEP, exercise/mobility, autogenic drainage, postural drainage, manual techniques (vibs and percussions), inhalation therapy (nebs)
Re-assess: sputum expectorated (colour and amount), auscultation, CXR, cough, exacerbations (chronic), palpation (fremitus)
Dyspnoea and SOB (increase WOB) (Cardio PT Problem)
signs: patient reported SOB (BORG scale), increase RR, increase accessory muscle use, distress/anxiety, shallow breaths (decrease TV)
Treatment: relaxed controlled breathing, pursed lips breathing, positioning (forward lean), gait aid for mobility, reliever/inhaler education, O2 therapy, exercise (long-term)
Re-Assess: modified BORG scale, MRC dyspnoea scale, QoL measures, SpO2
decrease exercise tolerance (Cardio PT Problem)
signs: decrease exercise (e.g. walking distance) compared to normal, limited by fatigue/SOB
treatment: mobilisation and exercise (endurance and strength), long term cardiac/pulm rehab, education
Re-assess: 6MWT, distane walked, duration, SOB during/after exercise, limiting factors
low lung volumes (Cardio PT Problem)
signs: CXR (shows collapse, fluid etc.), decrease BS on Auscultation, decrease SpO2, decrease bibasal expansion, weak cough
Treatment: upright positioning, NIV (CPAP), deep breathing exercises (+/- SMI)
Re-Assess: CXR, auscultation, cough, bibasal expansion, SpO2
Pain (Cardio PT Problem)
signs: patient reported, VAS/NRS ratings
Treatment: pain-medication, rest
Re-assess: VAS, NRS
What is the difference between pneumothorax and tension pneumothorax? (and on CXR)
Pneumothorax = air in pleural space but it is able to move between lungs and pleural space
Tension pneumothorax = air in pleural space, but the air can not move out of plueral space
On CXR: both have loss of peripheral lung markings w/visible lung edge, however, tension pneumothorax will result in a mediastinal shift AWAY from the affected lung
What are the common features of COPD on a CXR?
1 - Hyperinflation (low/flat diaphragm, increased rib count, flattened ribs)
2 - elongated mediastinum and hilum
3 - increased radio-translucency
4 - barrel-shaped chest
5 - bullae (areas of increased darkness due to trapped air)
6 - decreased vascular markings
What is a meniscus sign? (CXR)
A meniscus sign is a concave line obscuring costophrenic angle and part or all of hemidiaphragm
This is due to fluid collecting at lowest part of chest (in erect position due to gravity) in the case of a PLEURAL EFFUSION (fluid in pleural space)
pleural effusion
the accumulation of fluid in between the parietal and visceral pleura, called the pleural cavity
Crepitus
crepitus describes a popping, clicking or crackling sound in a joint
Pneumothorax
A pneumothorax is a collection of air outside the lung but within the pleural cavity. It occurs when air accumulates between the parietal and visceral pleura inside the chest. The air accumulation can apply pressure on the lung and make it collapse
What is a silhouette sign? (CXR)
a silhouette sign is a loss of normal borders between contents of the thoracic cavity on CXR (including loss of R heart and aortic knuckles). This is usually the result of a mass of the same tissue density as another structure being present and thus obscuring clear borders in the XR
Systematic approach to reading CXR
- check name, date, time and type
- check quality RIP
Rotation: spinner process and clavicles
Inspiration: 6th rib on R and 7th rib on L
Penetration/exposure: vertebrae just visible behind heart - Anatomical structures (ABCDE)
Airways, Breathing, Circulation, Disability/bones, Everything else (attachments)
What are the limitations of an AP CXR? When may an AP image be taken?
An AP CXR may be performed when a pt. is too unwell to stand (e.g. in ICU, emergency)
Disadvatages: heart and mediastinum appear enlarged.
A PA is usually taken because: cardiac size more accurate, scapula can be out of the way
Pleural Rub (auscultation)
creaking, squeaking, grating or rubbing around during inspiration and expiration due to friction between pleural surfaces
causes: inflammation or infection of pleura
Wheezes (auscultation)
continuous, high-pitched musical tones produced by air vibrating in narrow airway during late inspiration or expiration
Causes: bronchospasm, narrowed airway diameter, mucosal oedema, sputum, foreign body
bronchial breath sounds (auscultation)
normal tracheal sounds heard at lung periphery (louder in periphery)
cause: consolidation
coarse crackles (auscultation)
popping or clicking sounds mainly heard in inspiration (coarse crackles are louder and longer laster and lower pitch
cause: sputum retention, pulmonary oedema, aspiration
Fine crackles (auscultation)
popping or clicking sounds heard mainly on inspiration (fine crackles are shorter, quicker and more prominent on late inspiration)
cause: atelectasis, pulmonary fibrosis
Reduced breath sounds (auscultation)
decreased intensity of breath sounds (heard during inspiration and short periods of expiration)
cause: atelectasis, pneumothorax, pleural effusion
absent breath sounds (auscultation)
no sounds during inspiration or expiration
cause: localised fluid/air in pleural space, obstruction due to large sputum plug or carcinoma
Atelectasis
Atelectasis, the collapse of part or all of a lung, is caused by a blockage of the air passages (bronchus or bronchioles) or by pressure on the lung. Risk factors for atelectasis include anesthesia, prolonged bed rest with few changes in position, shallow breathing and underlying lung disease
Ataxia (+ 8 symptoms)
A general term used to describe abnormal coordination of movements (clumsiness/loss of coordination demonstrated by decrease speed, amplitude, accuracy and force)
8 = dysmetria, rebound phenomen, dysdidochokinesia, intention tremor, dyssynergia, hypotonia, dysarthria, nystagmus
Cause: cerebella, vestibular, or sensory (stroke, MS, CP, TBI, Peripheral neuropathy)
nystagmus
repetitive, uncontrolled movements of the eyes
freezing
difficultly in starting or continuing rhythmic, repetitive movements (e.g., speech, handwriting, gait/FOG)
cause: Parkinsons, (can be triggered by cognitive or emotional challenges)
Akinesia
difficultly initiating movement and freezing during movement
cause: Parkinsons
hypokinesia
decreased amplitude of movement
cause: Parkinsons
Bradykinesia
Slowness of movements (decrease speed)
cause: Parkinsons
dysarthria
motor speech disorder (ie. muscles are damaged, paralysed or weakened
cause: damage to cerebellum
Cardinal signs of Parkinsons
1 - Bradykinesia
2 - Rigidity
3 - Resting Tremor
4 - postural instability (balance deficits)
Dysexecutive syndrome
dysregulation of executive functions such as emotion, motivation, behaviour and cognition
cause: frontal lobe damage
dysphasia
language disorder marked by deficiency in generation (expressive) or comprehension (receptive) of speech
cause: damage to Wernicke’s or Broca’s
Wernicke’s Area
(Left/dominant temporal lobe)
Comprehension of sounds (especially speech)
Damage results in receptive dysphasia
Broca’s area
a region in the frontal lobe of the dominant hemisphere, usually the left, of the brain with functions linked to speech production.
damage results in expressive dysphasia
Signs of PCA infarct
Visual deficits
agnosia
memory deficits
Effects inf- post- parietal lobe and occipital lobe
What structures are present in both the superior and inferior mediastinum?
oesophagus
aorta (arch & descending)
vagus nerve
thoracic duct
lymphatic trunks
OTTAWA Knee rules
pts with acute knee injury an x-ray is only necessary if they have any of:
- over 55yrs of age
- isolated patella tenderness
- tenderness over head of fibula
- have less than 90 knee Flex
- unable to WB 4 steps BOTH initially and in ED (regardless of limp)
OTTAWA Ankle Rules
acute ankle pain
ANKLE PAIN: pts only need x-ray if pain in malleolar zone and any of:
- Tenderness of post- aspect of distal 6 cm of tib or fib
- unable to WB 4 steps initially and in ED
MID-FOOT PAIN: pts only need x-ray if pain in mid foot and any of:
- tenderness on base of 5th metatarsal
- tenderness of navicular
- unable to WB 4 steps initially and in ED
Landmarks of end of Spinal Cord
Spinal cord ends at T12
Dural sac ends at S2
Filum terminale attaches to coccyx (elongation of Pia mater)