The Hip and Thigh Flashcards
name all the disorders of the hip
Osteoarthritis Acetabular Labral Tears Slipped Capital Femoral Epiphysis Leg-Calve-Perthes Disease Hip Dysplasia Avascular Necrosis (AVN at the hip) “Snapping hip” syndrome Trochanteric Bursitis Hip “pointer” Avulsion Fractures
Hip Osteoarthritis
Population?
Causes?
Symptoms?
> 50 yrs
- trauma
- repetative stress
- bony abnormality
- inflammation/ infection
Symptoms:
- progressive stiffness
- AM stiffness which eases with movement
- exacerbated by weight bearing and cold weather
- presents with a capsular pattern: flexion (osseous) the Abduction (iliofemoral log) then internal rotation (ischiofemoral ligament)
- loss of flexion and IR are easiest to assess
Acetabular labral tears Common in what population? Causes? Symptoms/ presentation? Treatment?
- late childhood to adolescence
- Boys> girls
Causes
traum- ie planted foot and twisted on it
-can be gradual and progressive with hip OA
Presentation:
- sharp, deep catching pain in the groin after twisting/ slipping injury
- can be gradual and progressive
- groin pain
- audible click
- mild limitation of ROM
Treat:
-surgery if significant tear
Slipped Capital Femoral Epiphysis
Population?
Cause?
presentation?
pop:
late childhood adolescence
-Boys> girls
Cause
- obesity is factor
- can occur bilaterally
presentation:
limp and decreased ROM
Leg-Calve Perthe’s
Population?
Cause?
Pop:
- younger children
- Boys> girls
Cause:
- avascular necrosis of femoral head leading to collapse and fragmentation
- due to disruption of blood flow to the bony area (idiopathic-unknown)
Hip Dysplasia
Population?
Cause?
-usually noted in first few months of life but can be diagnoses when they start walking
Cause: ligament laxity -from possible connective tissue disorder
-or secondary cause is that femur doesn’t have contact with acetabulum so there is no stress on bone for it to develop properly.
Avascular Necrosis (AVN) What is AVN? Causes? Presentation? Treatment?
what is it?
-interruption of blood supply to bone. Femoral head schema results in death of marrow and osteocytes and usually results in collapse of the necrotic segment.
Causes:
-excessive corticosteroid use and alcohol abuse may account for 90% of new cases
-coagulation secondary to vessel wall injury (e.g. chemotherapy) or thromboembolic event
Traumatic causes:
-femoral neck fracture
-hip dislocation
-slipped capital epiphysis
-high incidence in pregnant women-cmpresses vascular structures
Presentation:
- AVN may present with nonspecific signs and symptoms
- early in disease process, the condition is painless; however, patient ultimately present with pain and limitation of motion.
- pain in localised groin area but manifest to butt, knee, greater trochanter
- exaccerbates with weight bearing and relieved by rest
How is it treated? Limit progression-cant do anything once its set in
- restrict weight bearing
- surgery to alter the collapse of the femoral head
Snapping hip Syndrome
3 types
external internal intra-articular m.c in cycling sports Tense TFL puts increased friction over greater trochanter -snap may be palpable/audible/ both -trochanteric bursitis may occur treatment- get rid of trigger points ago avoid bursitis ad check for underlying tendonitis
Internal:
-when iliopsoas tendon is flexed, abducted and externally rotated and hip moves to a position medial to the hip capsule during extension. - iliopsoas bursitis may ensue
treat- lengthen iliopsoas, clicking is inside