The Digestive System - GI Surgical Emergencies Flashcards
Peritonitis
Infl of serial membrane lining abdo cavity and the organs
Types of peritonitis
Primary - spontaneous
Secondary - to pathology in a visceral organ
Tertiary - persist/ recurs adequate initial treatment
Primary peritonitis
Spontaneous bacterial peritonitis (SBP)
Infection of ascitic fluid which arises in the absence of any other source of sepsis within the peritoneum or the adjacent tissues
Risk factors for primary peritonitis
Co-existence of GIT bleeding
Previous SBP
Low ascitic protein
What is SBP commonly seen in
Alcoholism and cirrhosis
Malignant mets
Hepatitis
CHF
SLE
Usually develops on top of pre-existing ascites
Px of SBP
Pyrexia - seen in 80%
Abdo pain
Peritoneal irritation - pain, rebound tenderness
What is diagnostic for SBP
> 250 polymorphonuclear WBCs in peritoneal fluid
Common organisms in SBP
E. coli
Strep
Enterococci
Treatment of SBP
Broad spectrum abx before culture results
3rd gen cephalosporins
Best-lactam combi e.g. piperacillin
Secondary peritonitis
Spillage of GI or gU micro-organism –. loss of integrity of mucosal barrier
May be disease, perforation, trauma, gangrene, obstruction, malignancy
Diseases that may cause secondary peritonitis
Appendicitis
Diverticulitis
Pancreatitis
IBD
In females, from an infected Fallopian tube or ruptured ovarian cysts
Px of 2’ peritonitis
Local pain from ruptured organ
Pts lie motionless/ curled
Rebound tenderness
Febrile
Death in 2’ peritonitis
Typically Gram-ve rod sepsis and potent endotoxins
Pathogens causing 2’ peritonitis - contamination form upper GI
Gram+ve organisms usually predominate
Incl yeasts, lactobacilli
Gram-ve rods if gastr9c acid suppressed
Pathogens causing 2’ peritonitis - contamination from distal bowel
Polymicrobial incl yeasts
GNR and anaerobes
Pathogens causing 2’ peritonitis - bloodstream/ lymphatic spread
Strep pneumonia
When does 3’ peritonitis occur
Within 48hrs of surgery
What is 3’ peritonitis typically caused by
Multidrug resistance organisms (ESBL, VRE) - difficult to treat
Abscesses
Severe complications of sepsis following surgery
Common sources of pyrexia in a surgical pt
Chest (infection)
Cut (wound infection)
Catheter (UTI)
Collections (abdo, pelvis)
Calves (DVT)
Cannula (infection)
Central line (infection)
CAPD
Continous ambulatory peritoneal dialysis
What does CAPD peritonitis involve
Skin organisms’ vs endogenous flora
Similar to endovascular device infection
Px of CAPD peritonitis
Same as 2’ peritonitis
What would be seen in dialysate in CAPD peritonitis
Cloudy
>100 WBCs, 50%
What is a peritoneal abscess
Infected fluid collection
Encapsulates by fibrinoid exudate, momentum and/or adjacent visceral organs
Loculation
Compartmentalisation of fluid-filled cavities by septa
Where do peritoneal abscesses usually occur
Sub-hepatic
Pelvic region
Paracolic gutter
Half with loculation
What may cause intra-ab abscesses
Faecal spillage from colonic source
Diverticular abscess
Necrotising abscess
Necrotising pancreatitis
Which antimicrobials to use for Gram-ve organisms
Amoxi*
Gentamycin
Co-amoxiclav
Cefuroxime
Cipro
Piperacillin
Which antimicrobials to use Gram+ve organisms
Amoxi
Gentamycin*
Co-amoxiclav
Piperacillin/ tazobactam
Vancomycin
Which antimicrobials to use for anaerobes
Metronidazole
Co-amoxiclav
Piperacillin
Hernia
Abnormal protrusion of a cavities through its wall
Defect in wall creates neck of hernia
Types of abdominal hernias
Epigastric
Umbilical
Spighelian
Excisional
Inguinal
Femoral
Is divarification of recti a hernia
No
There is no defect of wall
Recti shifted away from midline and sheath becomes lax
Who does divarification of recti affect
Seen in women after childbirth
Elderly men
Epidemiology of hernias
9x more common in men (except femoral)
Men have lifetime risk of 27%
Most commonly presents in ages 40-59
Px of hernias
Bulge or mass
Discomfort
Cough impulse
Px of obstructive hernias
N & V
Abdo pain
Distention
Absent bowel sounds
Px of strangulated hernias
Tenderness
Severe pain
Gangrenous mass
Classification of non-reducible hernias
Incarcerated
Obstructed
Strangulated
Spigelian hernias
Occurs between lateral border of rectus abdominis and linea semilunaris
Area of central weakness pushed dup - doesn’t go through anterior sheath
Incisional hernias
Occurs at sites where there gas been a previous surgical incision has been made
Range from small (few cm) to v large
Incarcerated hernias
Cannot be returned as they have formed adhesions outside but are still viable
Obstructed vs strangulated hernias
Bowel lumen becomes obstructed –> intestinal obstruction
Strangulated - blood flow to bowel stooped (ischaemic)
How are hernias repaired
Identify neck and sac of hernia, open up sac and reduce hernia content intra-ab
Neck is brought back together and covered w/ mesh
Different placement of mesh in hernia repairs
Onlay - over the top
Inlay - in between gap of muscle
Retro-muscular - beneath muscles
Preperitoneal and intraperitoneal - in peritoneal space
Best and worst place to put mesh in hernia repair
Inlay - worst, can be moved if intra-ab pressure increases
Pre/intraperitoneal - strongest, if intra-ab pressure increases, mesh is pushed up against muscle
Ddx for groin hernias
Malignant
Aneurysm caused by IVDUs
Venous swellings
How can we tell the difference between inguinal and femoral hernias
Relation to pubic symphysis
Femoral is below and lateral
Inguinal is above and medial
Features of direct inguinal hernias
Protrudes through Hesselbach trinagle - herniates through external and internal ring
Low rich of strangulation
Common in M
Seen in adults
Features of indirect inguinal hernia
Protrudes through inguinal ring (external only) - failure of processus vaginalis to close
Low risk of strangulation
Common in M
May occur in infants - congenital
Differentiating between direct and indirect inguinal hernia
Locate deep inguinal ring (midway of ASIS and pubic tubercle)
Manually reduce hernia towards deep ring and apply pressure
Ask pt to cough - if reappears, direct
Referral guidelines for hernia
Sx of strangulation/ obstruction
All females
Symptomatic males
Ix if diagnostic uncertainty for hernias
USS
if still unclear, MRI
Operative mx of hernias
Open vs laparoscopic (less painful, reduced recovery)
Incl herniotomy or hernioplasty
When is conservative mx used for hernias
Asymptomatic hernias (typically have wide neck)
Features of femoral hernias
Protrudes below inguinal ligament
High risk of strangulation
More common in F
Ddx for femoral hernias
Lymphadenopathy
Abscess
Femoral artery aneurysm
Lipoma
Inguinal hernia
Are trusses useful for femoral hernias
No - risk of strangulation
Complication of hernia repair
Bleeding
Infection
Recurrence - 10%
C/c pain
Cardinal sx of bowel obstruction
Absolute constipation - obstipation
Vomiting
Pian
Distended abdomen
Richter’s hernia
Only part of bowel walls and lumen herniate, other part remains in peritoneal cavity
Madyl’s hernia
Two diff loops of bowel contained within hernia
Hesselbach’s triangle boundaries
Rectus abdominis - medial
Inferior epigastric vessels - superior/ lateral
Inguinal ligament - inferior
Boundaries of femoral canal
Femoral vein - lateral
Lacunar ligament - medially
Inguinal ligament - anteriorly
Pectineal ligament - posteriorly
A/c abdo pain
Non-traumatic abdo pain lasting 5/7 or less
C/c abdo pain
Recurring or constant abdo pain of 2/12 or more
Sx of a/c or c/c abdo pain
Constant or episodic
Spp pain characteristics
Red flags
Dysphagia/ odynophagia
Palpitation
Chest sx
Signs of a/c or c/c abdo pain
Abdo distention
Tenderness
Grey Turner’s sign
+ve Carnett’s sigan
Rovsing sign
Guarding
Ddx of a/c abdo pain - diffuse
Pancreatitis
Bowel obstruction
Sickle cell crisis
Gastroenteritis
Mesenteric thrombosis
Extras - metabolic disorder , psychogenic illness
Ddx of a/c abdo pain - focal
A/c appendicitis
Biliary colic
A/c pancreatitis
A/c diverticulitis
Extras - ruputured adnexa cysts, ovarian torsion
Ddx of c/c abdo pain
Functional dyspepsia
IBS
IBD
PUD
Diverticular disease
Narcotic bowel syndrome (opined induced GI hyperplasia)
Ix for c/c abdo pain
Tends to be more focussed
Guided by red flag sx and signs
If c/c pancreatitis and 20-49yrs, excl CF, but if >40, excl cancer
Fecal fat/ elastase
Intestinal obstruction
Interruption in normal flow of intestinal content either due to mechanical occlusion of the intestinal lumen or black of peristalsis
Pathophysiology of intestinal obstruction
Mechanical obstruction
Fluid and electrolyte loss
Bacterial translocation and toxaemia
Ischaemia
Perforation and peritonitis
6 Fs of abdo distension
Fat
Foetus
Flatus
Fluid
Faeces
Fulminnat masses
Ddx of SBO - neonates
Duodenal/ jejunal atresia
Meconium ileus