The choking horse Flashcards

1
Q

Approach to ptyalism/drooling & hypersalivation

A
  • General history
  • Focused history
  • Physical exam
  • Morphological abnormalities?
    If no
  • Functional abnormalities
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2
Q

Focused history

A

Diet
- Legume contaminated with fungus (Rhizoctonia leguminicola?) -> hypersalivation due to transient increase in saliva production
- Oral PBTZ given with Clembuterol -> transient period of hypersalivation

Dental
- Speculum, good light source, gag

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3
Q

Obstructive morphological abnormalities

A
  • Retropharyngeal lymphadenopathy
  • Malformation, injury, oedema (pharynx, larynx, oesophagus)
  • Pharyngeal disorders (abscess, cicatrix, inflammation)
  • Laryngeal disorders (epyglottic cysts, RDP Arch)
  • Palate disorders (DDSP, cleft palate)
  • Guttural pouch disorders (tympany & empyema)
  • Oesophageal problems (obstruction & diverticula)
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4
Q

Painful morphological abnormalities

A
  • Teeth (root abscess, broken teeth, abnormal wear)
  • Mandible/maxilla (fractures, trauma)
  • Stomatitis/glossitis
  • Temporohyoid OA (pain, may also cause functional)
  • Temporo-mandibular osteopathy
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5
Q

Stomatitis/glossitis causing choke

A

FB most common (glossitis)
- desiccated plant material/awns
- metal/wire

Ulcerative stomatitis
- bute toxicity
- blester beetle poisoning

Vesicular stomatitis
- viral (Poxvirus & Rhabdovirus serotype: New Jersey, Indiana)

Bacterial
- Actinobacillus lignieresi

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6
Q

Functional abnormalities - neuro exam

A

Infectious
- rabies
- viral encephalitis
- verminous encephalitis & EPM
- botulism & tetanus
- meningitis

CNS trauma
- cerebral damage/oedema
- brainstem haemorrhage

CNS masses
- e.g. cholesteroloma

Toxic
- lead poisoning
- yellow star thistle
- hepatoencephalopathy

Other
- polyneuritis equi
- Grass sickness (dysautonomia)
- THOosteoarthropathy
- guttural pouch (mycosis, neoplasia, empyema)
- Petrous temporal bone fracture/osteomyelitis

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7
Q

Functional abnormalities - look for muscular aetiology

A
  • HYPP
  • NMD
  • PSSM
  • Masseter myositis
  • Hypocalcaemic tetany/eclampsia
  • Myotonia
  • Rectus wapitis ventralis rupture
  • White snake root toxicity
  • Megaoesophagus
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8
Q

Choke - clinical presentation

A
  • regurgitation of food/water/saliva
  • ptyalism
  • dysphagia
  • coughing
  • repeated head extensions or retching
  • anxiety
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9
Q

Choke - other less common clinical signs

A
  • distention
    – left jugular furrow (right side in some horses)
  • crepitus
    – oesophageal rupture
  • CS of complications
    – dehydration
    – rr/abnormal pattern
    – fever
    – other signs of systemic inflammation
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10
Q

Diagnosis

A
  • General physical exam
  • Palpate neck and thoracic inlet
    – both sides but focus on left
    – thoracic inlet a common site of obstructions
  • Oral exam
  • Thoracic auscultation
    – Some may have aspiration pneumonia, esp if have had the obstruction for some time
  • NG intubation
    – Confirms diagnosis and locates the obstruction
  • Clinpath
    – Hydration status (PCV/TP, urea/creatinine)
    – CBC
    -> further information re systemic inflammation
    – Electrolytes
    – Acid-base status
  • Radiography
  • US
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11
Q

Radiography

A
  • Survey plain films
  • Determines extent and location of the obstruction
  • Contrast radiography
    – Barium (iodinated contrast for rupture)
    –> barium paste PO
    –> liquid barium (cuffed NG tube)
    – Double contrast
    –> liquid barium by cuffed NG tube under pressure
    –> Gives best definition
    – Additional information
    –> Rupture
    –> Aspiration pneumonia
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12
Q

Medical management (spontaneous resolution)

A
  • remove all feed & water
    – NPO (nothing per os)
  • IV fluids
  • NSAIDs/analgesics
  • sedation (for relaxation of the oesophagus)
  • oxytocin (for proximal obstruction)
  • ideally place in stall without bedding (at least for 24-48h)
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13
Q

Medical management (assisted resolution)

A
  • oesophageal lavage & drainage
    – sedation
    – maintain head below the thoracic inlet
    – pumping lots of water in via NG tube into oesophagus to try macerate and dissolve obstruction
    – use a double lumen NG tube so can put water through 1 side, empty water and contents through the other lumen
  • aggressive oesophageal lavage
    – cuffed nasotracheal and naso-oesophageal tube to prevent aspiration
    – standing or under GA
  • GA
    – minimises aspiration
    – aids relaxation
    – if impaction hard/difficult/very long
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14
Q

General considerations for medical management

A

Once relieved prevent re-obstruction
- repeat endoscopy to assess mucosal damage
- fusiform dilation predisposes to re-obstruction
- small quantities soft feed 48h post relief (almost liquid for 1st few days)
- provide electrolytes and fresh water
- anti-inflammatories
– care with NSAIDs as may exacerbate the
oesophageal ulcerations
– avoid oral, go IV
- broad spec antibiotics?
– if the obstruction has been there for quite a while, aspiration pneumonia is quite likely
– particularly if find there are signs that indicate systemic inflammation or abnormal thoracic auscultation or abnormal thoracic US -> consider broad spec AB

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15
Q

Surgical - Oesophagotomy (primary closure)

A
  • Incision above/on obstruction
  • Longitudinal oesophagotomy with primary closure
    – standing with tube in place
    – approach dictated by location of lesion
    – laryngeal hemiplegia common complication
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16
Q

Surgical - Oesophagotomy - indications

A
  • if medical management doesn’t resolve it
17
Q

Oesophagostomy

A
  • incision 5cm distal to lesion
    – mucosal sutures
  • indwelling tube
    – into stomach
    – purse string
  • pelleted slurry
  • 2nd intention healing
  • for oesophageal rupture
    – incision close (but distal) to lesion and NG tube into stomach
18
Q

Complications

A
  • aspiration pneumonia
  • oesophageal ulcer (if circumferential stricture)
  • oesophageal stricture
    – if the ulcer is circumferential and deep it has a high change of a stricture being formed down the line)
  • megaoesophagus
  • diverticula
  • oesophageal rupture
19
Q

Ptyalism vs hypersalivation (according to Ignacio)

A

hypersalivation = increase in production

ptyalism = saliva coming out of the mouth (without over/increase in production)

20
Q

Why can acid-base and electrolyte balance be altered in a horse with choke?

A
  • 500kg horse produces ~1L saliva/hr
  • In a day will have 24L deficit
  • Saliva is not just fluid: contains electrolytes
21
Q

What helps diagnose a megaoesophagus?

A
  • contrast radiographs confirm the presence and extent
  • along with endoscopy