Test 2: vestibulopathies Flashcards

1
Q

describe vestibulopathies

A

peripheral vestibular dysfunction

can be unilateral or bilateral

may require habituation or adaptation or both

CPG applies if VRT is appropriate based on etiology

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2
Q

types of vestibulopathies

A

vestibular neuritis

labyrinthitis

meniere’s disease

acoustic neuroma

superior canal dehiscence syndrome

perilymphatic fistula

labryinthine concussion

ototoxicity

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3
Q

what is a videonystamography

A

video goggles used to track for nystagmus with various conditions

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4
Q

what is a electronystagmography

A

electrodes used around the eye to record electric activity

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5
Q

what is a rotary chair

A

sit in a chair in a dark room that spins around

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6
Q

what is video head impulse test

A

studies VOR at high frequency

more sensitive than clinical head impulse test

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7
Q

what is vestibular evoked myogenic potential

A

applying a repetitive sound stimulus to one ear and then averaging the reaction of the muscle activity in response to each sound click or pulse

CVEMP (cervical mm)

OVEMP (ocular mm)

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8
Q

what is computerized dynamic posturography

A

detects postural sway by measuring shifts in the center of gravity (COG) as a person moves within their limits of stability

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9
Q

what is caloric testing

A

water or air in ear to see electrical activity

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10
Q

describe vestibular neuritis

A

inflammation of balance portion of CN VIII (can be inferior or superior portion)

precipitated by viral illness (usually a few weeks prior)

acute onset vertigo

lasts min to hours

likely have N&V

usually no hearing impact

often unilateral but can be bilateral

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11
Q

diagnosis of vestibular neuritis

A

head impulse test, caloric testing, VEMP

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12
Q

management of vestibular neuritis

A

glucocorticoids in first 3 days since symptom onset

want to reduce the inflammation

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13
Q

is vestibular neuritis responsive to vestibular rehabilitation treatment (VRT)

A

yes

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14
Q

how long until vestibular neuritits improves

A

6 weeks to 3 months

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15
Q

what is labyrinthitis

A

bacterial or viral infection of labryrinth

if bacterial it is generally meningitis

will affect hearing and balance

prolonged vertigo

N&V

will have tinnitis

often unilateral but can be bilateral

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16
Q

diagnosis of labyrinthitis

A

head impulse test

caloric testing

vestibular evoked myogenic potential

will also test CSF, auditory markers, and MRI in case of bacterial

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17
Q

treatment of labyrinthitis

A

bacteria = antibiotics

autoimmune/viral = steroids

18
Q

is labyrinthitis responsive to VRT

19
Q

describe meniere’s disease

A

aka endolymphatic hydrops

ischemia or fibrosis of endolymphatic sac causing abnormalities in endolymph drainage

often the catch all term for vestibular disorders

may have N&V

may have fluctuating hearing loss

may have tinnitus that sounds like roaring

starts unilateral and progresses to bilateral

episodic

20
Q

diagnosis of menieres

A

audiogram is important; will show low frequency hearing loss

may test positive on vestibular hypofunction tests

21
Q

treatment for menieres

A

not curable

2g/day of sodium to control fluids

diuretics to lower extracellular fluid

22
Q

does menieres respond to VRT

A

may respond to VRT at first but need to move to habituation as it gets worse

23
Q

what is an acoustic neuroma

A

aka vestibular schwannoma

benign tumor CN VIII

often presents in the internal auditory canal but can present other places as well

symptoms dependent on tumor location

if in IAC - will have hearing and balance impairment

tumors are slow growing so symptom onset can be slow

typically unilateral

24
Q

diagnosis of acoustic neuroma

A

may be positive on other CN VIII screens like Renne and Webber

MRI and CT needed

25
treatment for acoustic neuroma
sx excision or gamma knife radiation VRT may be helpful post op
26
what is superior canal dehiscence syndrome
thinning or opening on the top of the bone overlying the superior canal symptoms are oscillopsia or vertigo induced by sound often congenital
27
diagnosis of superior canal dehiscence
observing eye movements caused by increased pressure or sound in inner ear or during valsalva
28
treatment for superior canal dehiscence
repair of bony deficit in sx not responsive to VRT
29
what is a perilymphatic fistula
perforation (usually trauma related) of the oval or round windows that disrupts the biochemistry of the ear perilymph leaks into the middle ear resulting in vertigo and hearing loss that are episodic symptoms increase with activity (increase pressure) and decrease with rest hard to diagnose because similar test to other disorders but can increase pressure in inner ear and observe for vertigo
30
medical management of perilymphatic fistula
rest, sx, and VRT
31
describe labyrinthine concussion
concussion of inner ear often cooccurs with brain concussion most common incidence is trauma symptoms = balance problems, dizziness, concussive symptoms, cognitive changes, irritability, and sleep disturbances may have central and peripheral findings can be unilateral or bilateral
32
does labyrinthine concussion respond to VRT
yes with a cognitive component
33
what is ototoxicity
can be chemical or environmental typically bilateral gentamycin (powerful antibiotic), chemo, solvents all can cause symptoms = balance dysfunction and visual dependence won't necessarily have vertigo may co-occur with hearing loss adaptation WILL NOT WORK
34
screen for ototoxicity
VEMPs, calorics, etc
35
adaptation vs habituation vs substitution
adaptation = change in vestibular response to certain stimuli (neuroplastic change where there is a physiological balance of signaling) habituation = decreased response to a stimulus with increased exposure (get used to it) substitution = uptrain other systems
36
CPG for vestibular function high evidence
VRT with acute/subacute/chronic unilateral vestibular hypofunction VRT with bilateral hypofunction supervised VRT VRT to improve quality of life age and gender DO NOT influence outcomes
37
CPG for VRT is against what
saccades and smooth pursuit to improve gaze stability this DOES NOT work
38
CPG for VRT has moderate evidence for what
modalities based training - virtual reality - optokinetic stimulation - platform perturbations - vibrotactile feedback when to stop CR (i.e. plateau)
39
CPG for VRT has weak evidence for
balance dosage gaze stability HEP
40
other notable things that may impact outcomes according to the CPG
early intervention improves outcomes anxiety, vision disturbances, migraines, long term use of vestibular suppressants can also impacy outcomes (i.e. meclazine damages inner ear over time)
41