Test 2 (Mod 3 and 4) Flashcards
Four Heart Valves are
Tricuspid, Pulmonic, Mitral, Aortic
Tricuspid Valve links
Right atrium to right ventricle
Pulmonic Valve links
Right ventricle to pulmonary artery
Mitral Valve links
Left atrium to left ventricle
Aortic Valve links
Left ventricle to aorta
Umbilical Vein carries oxygenated blood from
Placenta to infant
In fetal circulation, blood bypasses the liver through the
Ductus Venosus
In fetal circulation, oxygenated blood flows from right to left atria through the
Foramen Ovale
Closes after birth with change in pressure in cardiac chambers
Foramen Ovale
Causes fetal circulation blood to bypass the lungs, which are nonfunctional at this time
Foramen Ovale
A fistula between aorta and pulmonary artery that allows for mixing of blood
Ductus Arteriosus
Closes after birth, sometimes with first few breaths but may take up to three days
Ductus Arteriosus
Bound to hemoglobin on red blood cells
Oxygen
Facilitate Oxygen
Hematocrit and hemoglobin
Heart rate is sensitive to
Oxygen level
Cardiac output is dependent on ________ until child is 5 years old
Heart rate
A child has an increased risk of heart failure because of
Immature heart that is sensitive to volume or pressure overload, and muscle fibers that are less developed
Reveals size and contour of heart
X-ray
Visualizes characteristics of pulmonary vascular markings
X-ray
Identifies heart structure
Ultrasound (Echocardiography)
Identifies pattern of movement, hemodynamics
Ultrasound (Echocardiography)
Records quality of major electrical activity of heart
Electrocardiogram (ECG)
Identifies dysrhythmias
Electrocardiogram (ECG)
Encourage increased fluid intake to maintain hydration to offset diuretic effect of contrast dye after
Cardiac Catheterization
Heart conditions that do not cause deoxygenation or low oxygen levels; skin and mucous membrane color is usually normal pink
Acyanotic heart defects
Atrial Septal Defect is a defect between
Atria
Septal wall defect allowing blood to flow from left atrium to right atrium, called left to right shunt
Atrial Septal Defect
Often asymptomatic if small defect, dyspnea, fatigue, poor growth, soft systolic murmur, and CHF are all assessment findings of
Atrial Septal Defect
Defect between Ventricles
Ventricular Septal Defect (VSD)
Septal wall incomplete allowing blood to flow from left ventricle to right ventricle (left to right shunt)
Ventricular Septal Defect
Tachypnea, dyspnea, poor growth, reduced fluid intake, palpable thrill, systolic murmur at lower sternal border, large septal defects detected on ECG and radiology, and signs of CHF are all assessment findings of
Ventricular Septal Defect
Teach the need for antibiotic prophylaxis to prevent subacute bacterial endocarditis with
Ventricular Septal Defect
Narrowing of descending aorta and restriction of blood flow leaving the heart are signs of
Coarctation of Aorta
Narrowing or constriction of descending aorta, often near ductus arteriosus. Progressive disorder that leads to CHF.
Coarctation of Aorta
Assessment of ________ may be asymptomatic, have blood pressure difference of 20mm between upper and lower extremities, with upper extremity pressure higher, have brachial and radial pulses full, and femoral pulses weak, have headache, vertigo, and epistaxis, have exercise intolerance, left ventricular hypertrophy, and dyspnea, and can have CVA secondary to hypertension in upper circulation
Coarctation of Aorta
Therapeutic Management for Coarctation of Aorta
- Balloon cardiac catheterization with insertion of endovascular stents. 2. Surgical resection and patch of coarctation. 3. Possible prophylaxis for endocarditis when undergoing surgical or dental procedures. 4. Prior to correction, monitor BP in upper and lower extremities. 5. Rebound hypertension occurs in immediate postoperative period.
Heart conditions that cause blood to contain less oxygen than required; skin and mucous membrane color is usually pale to blue
Cyanotic heart defects
Four defects that combine to allow blood flow to bypass lungs and enter left side of heart, called a right to left shunt
Tetralogy of Fallot
This heart defect leads to unoxygenated blood to enter the body circulation accounting for cyanosis
Tetralogy of Fallot
Four defects: pulmonic stenosis, right ventricular hypertrophy, ventricular septal defect, and overriding aorta, all make up _________
Tetralogy of Fallot
Deficient oxygen in the tissues leads to
acidosis
Hypercyanosis (TET) spells are
transient periods when there is an increase in right to left shunting of blood
TET spells are characterized by:
Hypoxia, pallor, and tachypnea, which may be precipitated by crying, defecation, and feeding; older children will assume squatting position to decrease blood return from the lower extremities.
Treatment for TET spell
place child in knee-chest position, administer morphine or propranolol and O2
Signs of Tetralogy of Fallot
Clubbing of digits, polycythemia, poor growth, exercise intolerance, systolic murmur in pulmonic area, right ventricular hypertrophy, and TET spells
Prostaglandin E1 is given in order to maintain open ductus arteriosus in treatment of
Tetralogy of Fallot
Aorta arises from right ventricle, and pulmonary artery arises from left ventricle
Transposition of the great vessels
Anomolies exist that increase mixing of blood between two separate circulations; these anomalies promote oxygenation. Right to left shunting occurs.
Transposition of the great vessels
Progressive cyanosis to hypoxia to acidosis. Signs and symptoms of CHF. Tachypnea. Poor feeding. Failure to grow. Echocardiogram identifies misplacement of arteries.
Assessment findings for Transposition of the great vessels
Abnormally small left ventricle noted at birth
Hypoplastic left heart syndrome
Inability of heart to supply oxygen needs of body is a description of
Hypoplastic Left Heart Syndrome
Absent or stenotic mitral and aortic valves, abnormally small left ventricle and aortic arch, major resistance to aortic flow, hypertrophy of right ventricle, with a poor prognosis, all describe
Hypoplastic Left Heart Syndrome
Tachypnea, chest retractions, dyspnea, cyanosis, decreases pulses, poor peripheral perfusion, increases right ventricular implies, echocardiogram indicating small and weak left ventricle, and CHF, are all signs of
Hypoplastic Left Heart Syndrome
Prostaglandin E1 given to prevent closure of patent ductus arteriosus, palliative surgery, transplant, are all interventions for
Hypoplastic left heart syndrome
Survival rate currently at or above 50% for
Hypoplastic Left Heart Syndrome
Systemic inflammatory disease that involves heart and joints; CNS and connective tissue involvement may also occur
Rheumatic Fever
Occurs secondary to infection by group A beta-hemolytic streptococcus; most common in children age 5 years to adolescence
Rheumatic Fever
Follows one to three weeks after a group A beta-hemolytic streptococcal infection
Rheumatic Fever
Must have presence of two major or one major and two minor criteria to diagnose __________using Jones Criteria
Rheumatic Fever
A new murmur, pericardial fruition rub, changes on the ECG; tachycardia may be noted by nurse as a pulse greater than 100 while sleeping, are all symptoms of
Carditis
Involves involuntary movement of limbs; emotional lability and slurred speech may occur; this symptom tends to have a latent period of two months or more from strep infection
Chorea (CNS symptom of Rheumatic Fever)
An erythematous, macular rash that occurs primarily on trunk and proximal limbs; frequently associated with carditis
Erythema Marginatum
non-tender nodules that develop on skin over flexor surfaces of joints and vertebrae
Subcutaneous Nodules
Planning and Interventions for Rheumatic Fever
- Bedrest until ESR returns to normal 2. Use ASA and prednisone as ordered anti-inflammatory agents to reduce inflammation; aspirin will also promote comfort from painful joints. 3. Give PCN as ordered in either an oral daily dose or monthly long-acting injection after recovery from rheumatic heart disease to reduce risk of recurrence of strep infection; erythromycin given if allergic to PCN. 4. Design nursing activities to promote rest and to encourage diversional activities which do not stress heart; maintain bed rest with bathroom privileges.
A multisystem disorder involving vasculitis, also called mucocutaneous lymph node syndrome. Leading cause of acquired heart disease in children
Kawasaki’s Disease
Inflammation of tunica intima or inner lining of arteries and veins
Vasculitis
Acute, systems inflammatory illness involving arteries, unknown cause but generally affects young children; most frequently affected are boys under 5 years of age
Kawasaki’s Disease
Three phases of Kawasaki’s Disease
Acute, subacute, and convalescent
Characterized by fever, conjunctival hyperemia, swollen hands and feet, rash, and enlarged cervical lymph nodes
Acute phase of Kawasaki’s disease
characterized by cracking lips, desquamation of skin on tips of fingers and toes, cardiac disease, and thrombocytosis
Subacute phase of Kawasaki’s disease
Has lingering signs of inflammation
Convalescent phase of Kawasaki’s disease
Fever lasting longer than five days that is unresponsive to antipyretics, conjunctivitis, crusted and fissured lips, swelling of hands and feet, erythema, lymphadenopathy, are all signs of
Stage one Kawasaki’s. (one to two weeks)
Fever diminishes, irritability, anorexia, desquamation of hands and feet, arthritis and arthralgia, cardiovascular manifestations
Subacute stage of Kawasaki’s (two to four weeks)
Drop in ESR and diminishing signs of illness
Convalescent stage of Kawasaki’s (six to eight weeks)
A condition that causes swollen glands and can be caused by infection or cancer
Lymphadenopathy
Treatment for Kawasaki’s Disease
Administer 80 to 100 mg/kg/day ASA as anti platelet agent. Wean child over time to decrease risk of bleeding. Administer gamma globulin (IVIG) once as ordered to reduce risk of coronary artery lesions and aneurysms; may be repeated if fever does not decrease
Nursing management of Kawasaki’s Disease
Promote comfort, small frequent feedings, passive range of motion to extremities, cool baths, gentle oral care, encourage fluids, monitor for complications such as aneurysms, side effects of aspirin therapy: bleeding, GI upset, side effects of IVIG therapy: elevated blood pressure, facial flushing, tightness in chest, monitor temperature, and monitor eyes for conjunctivitis.
noted when clients head is flexed while in the supine position, resulting in involuntary flexion of knees or hips; a positive result is a common sign in meningitis.
Brudzinski’s Sign
level of consciousness in which client cannot be aroused even with painful stimuli
Coma
a late sign of increased intracranial pressure characterized by widening pulse pressure (rising systolic blood pressure with a stable diastolic pressure), bradycardia, and irregular respirations
Cushing’s Triad
bleeding between the dura and the cranium
Epidural Hematoma
force exerted by brain tissue, cerebrospinal fluid, and blood within cranial vault; normal is 4 to 12 mm Hg and is dependent on age in childhood
Intracranial Pressure (ICP)
demonstrated when client’s leg is raised with knee flexed and any resistance or pain is felt; it is a common finding that indicates meningeal irritation in meningitis
Kernig’s Sign
a measure of degree of responsiveness of mind to sensory stimuli; lower levels indicate decreased neurologic functioning; levels can be categorized (in order of decreasing level of function) as confusion, delirium, obtunded, stupor, and coma
Level of Consciousness (LOC)
fibrous membrane that covers brain and lines vertebral canal; consists of three layers: dura, arachnoid, and pia matter
Meninges
a sheath made of a fatty substance that covers axon process of neuron or nerve fibers and increases speed and accuracy of nerve impulses; accounts for gradual development of fine and gross motor skills during early childhood
Myelin
refers to a lack of innervation to an organ
Neurogenic
stiffness of neck or resistance to neck flexion, often seen in infections of central nervous system
Nuchal Rigidity
Client positions self with hyperextension of head and neck; this is seen in meningitis and felt to relieve some discomfort from meningeal irritation
Opisthotonus
Sensitivity to light, seen in some clients with migraine headaches or viral infections such as measles, meningitis, and encephalitis
Photophobia
difference in systolic and diastolic blood pressure
Pulse Pressure
bleeding between dura and cerebrum
Subdural Hematoma
tenseness of muscles, uncoordinated, stiff movements; can be seen as scissoring or crossing of legs; exaggerated reflex reactions
Spasticity
term frequently used to describe characteristics of certain seizures, involving continuous muscle contractions, and alternating contraction and relaxation of muscles
Tonic Clonic
Brain and spinal cord develop during
First trimester
Not mature at birth, but as numbers of glial cells and dendrites increase, refinement continues until about 4 years of age
Nervous system
Posturing seen with severe dysfunction of cerebral cortex; it includes adduction of arms at shoulders, arms flexed at chest, wrists flexed, hands fisted, and lower extremities flexed
Decorticate Posturing
Posturing seen with dysfunction at level of midbrain; it includes rigid extension and pronation of arms and legs
Decerebrate Posturing
Corneal, pupillary, muscle-stretch, superficial, and plantar reflexes are all absent in
Deep Coma
A nonprogressive motor disorder of central nervous system (CNS) resulting in alteration in movement and posture
Cerebral Palsy (CP)
Classified as spastic, athetoid, or mixed
Cerebral Palsy
Causes of ________ may include trauma, hemorrhage, anoxia, or infection before, during, or after birth
Cerebral Palsy
One-third of children with this disease will also have some degree of mental retardation, while the other two thirds do not
Cerebral Palsy
Many clients with this disease may require increased caloric intake because of spasticity or increased motor functioning
Cerebral Palsy
Use of adaptive feeding devices and positioning during feedings to decrease risk of aspiration, and the use of a gastrostomy tube may be needed for
Cerebral Palsy
Develop during the first trimester of fetal development; defects can occur at any place along spinal canal
Neural tube defects
Unknown etiology, but may be associated with maternal dietary folic acid deficiency; incidence has decreased with emphasis on folic acid supplementation during pregnancy; the degree of disability is determined by location of defect and amount of spinal nerves encased in sac; the higher the defect, the greater the neurologic dysfunction
Neural Tube Defects
Neural Tube Defects are also known as
Spina Bifida, or Myelodysplasia
Posterior vertebral arches fail to fuse, but there is not herniation of spinal cord or meninges; no loss of function
Spina bifida occulta
Posterior vertebral arches fail to fuse, and there is a saclike protrusion at some point along posterior vertebrae; sac contains meninges and CSF but not spinal cord
Meningocele
Posterior vertebral arches fail to fuse; saclike herniation contains meninges, CSF, as well as a portion of spinal cord or nerve roots; sometimes leakage of CSF occurs
Myelomeningocele
brain and meninges herniate through defect in skull into a sac
Encephalocele
Elevated levels of alpha-fetoprotein (AFP) in maternal serum and fluid obtained by amniocentesis, as well as ultrasound, all lead to
prenatal diagnosis of open spinal defects
Frequent, clean straight catheterization is the preferred method of management for
Neurogenic bladder
High fiber diet, adequate fluid intake, and pattern of evacuation of bowels, some cases of laxative and enema use, are all means of managing
Neurogenic bowel
A condition characterized by imbalance between CSF production and absorption resulting in enlarged ventricles and an increase in ICP; if untreated, this condition can cause permanent brain damage
Hydrocephalus
Congenital causes of this disease include Arnold Chiari malformation associated with myelomeningocele, and can be acquired from meningitis, trauma, or intraventricular hemorrhage in premature infants. Etiology is idiopathic (cause unknown) in up to 50% of cases
Hydrocephalus
Increased head circumference, split cranial sutures, high-pitched cry, bulging fontanel, irritability when awake and seizures, are all assessment findings of
Hydrocephalus
Toddlers and older children suffering from _______, may also present with setting-sun eyes, seizures, irritability, papilledema, decreased LOC, and changes in vital signs (increased BP and widening pulse pressure).
Hydrocephalus
All children with this disease can present with vomiting, lethargy, and Cheyne-Stokes respiratory pattern
Hydrocephalus
Surgical insertion of a tube or “shunt” into ventricles with other end in either the peritoneum or atrium; the most common version is ventriculoperitoneal; preoperatively position client flat and on unoperative side; if held it is important not to allow head to be elevated.
Treatment for Hydrocephalus
Alterations in firing of neurons in brain (cortical neuronal discharge); the result of this discharge depends on where in brain discharge begins and how it spreads
Seizures
Seizure that begins locally in one hemisphere of the brain
Partial Seizure
Seizure that begins in both hemispheres of the brain
Generalized Seizures
Tegretol is commonly used in children for
Seizures
Premature closure of cranial sutures in young children; there is some relationship between this and several inherited syndromes
Craniosynostosis
Premature closure of skull may lead to
increased ICP with subsequent brain damage
Fluid restriction may be prescribed in postop period; head of bed may be ordered to elevation at 30-degree angle to assist in decreasing facial swelling with this disease
Craniosynostosis
Inflammation of meninges; is most common infection of CNS; two primary classifications include viral or aseptic and bacterial
Meningitis
Clinical manifestations of this process in infants and toddlers are irritability, vomiting, and change in appetite
Viral Meningitis
Clinical manifestations for an older child with this process are usually preceded by a nonspecific febrile illness; client presents with headache, malaise, muscle aches, n/v, photophobia, and nuchal/spinal rigidity
Viral Meningitis
Clinical manifestations of this process in infants and toddlers include poor feeding/suck, vomiting, high-pitched cry, bulging fontanel, fever or hypothermia depending on maturity of infant’s neurological system, and poor muscle tone and nuchal rigidity
Bacterial Meningitis
Stiffness of the neck
Nuchal Rigidity
Hyperextension of head and neck
Opisthotonus
Positive Kernig or Brudzinski’s sign indicates
Meningeal irritation
ND for Meningitis
Risk for ineffective breathing pattern, pain, risk for injury, risk for ineffective thermoregulation
Treatment for Meningitis
Elevate head of bed 15 to 30 degrees, avoiding neck extention or flexion, and maintaining head in neutral position. Monitor for cerebral edema. restrict oral fluids and monitor IV fluids to prevent fluid overload
Syndrome of inappropriate antidiuretic hormone
SIADH
Normal CSF Values
ICP- 50-100 Appearance- Clear Leucocytes- 0-8 Protein- 14-45 Glucose- 35-75
CSF with Viral Meningitis
ICP- Normal or slightly elevated Appearance- Clear Leukocytes- Slightly elevated Protein- Slightly elevated Glucose- Normal or decreased
CSF with Bacterial Meningitis
ICP- Elevated Appearance- Cloudy Leukocytes- Elevated Protein- Elevated Glucose- Decreased
Clients with this type of meningitis will need to have transmission-based droplet precautions instituted until at least 24 hours of antibiotic therapy have been completed
Bacterial Meningitis
Complications of Meningitis
Seizures, hearing loss, visual alterations
This is treated with IV antibiotics sensitive to causative organism; treatment usually continues for 7 to 14 days; preventative care includes Hib vaccine
Bacterial Meningitis
Those in close contact with a child who has bacterial meningitis may receive
Rifampin (Rifadin) or ciprofloxacin (Cipro) prophylactically
Inflammation of brain
Encephalitis
Classic symptoms of this include an acute febrile illness with neurologic signs
Encephalitis
Usually caused by a viral organism; herpes simplex type 1 is most common cause during neonatal period; enteroviruses are frequently identified as causative agents; nonviral agents include bacteria, parasites, fungi, and rickettsiae
Encephalitis
Fever, h/a, n/v, and signs of an upper respiratory infection are all assessment findings of
Encephalitis
Neurologic symptoms of this process include those of nuchal rigidity, photophobia, and positive Kernig’s and Brudzinski’s signs
Encephalitis
Treatment for Encephalitis
seizure precautions, if bacterial use antibiotics, acyclovir or other antiviral agents used for herpes virus infection
An acutre metabolic encephalopathy of childhood; fatty degeneration of liver leads to liver dysfunction
Reye Syndrome
This process is characterized by five stages: 1. Vomiting and lethargy 2. Combativeness and confusion 3. Coma, decorticate posturing 4. Decerebrate posturing 5. Seizures, loss of deep tendon reflexes, respiratory arrest
Reye Syndrome
Ususally develops after a mild viral illness such as chickenpox
Reye Syndrome
Linked to the use of ASA in children
Reye Syndrome
Hypoglycemia is one of the earliest signs of this syndrome
Reye Syndrome
Child presents with an abrupt change in LOC; hx reveals client recovering from viral disease with sudden onset of vomiting and mental confusion
Reye Syndrome
Liver enzymes and ammonia levels are elevated; blood glucose levels are below normal and prothrombin time is prolonged; bilirubin levels remain normal; liver biopsy shows small fat deposits
Reye Syndrome
Tx for Reye Syndrome
ICU, monitor cerebral edema, fluid restrictions usually instituted; frequent vials and neurological assessments. Monitor lab values for elevated ammonia, acidosis, or hypoglycemia; I&O
Toddler ages
12-36 months
Toddler motor development
Slower physical growth. Reduced demand for calories. Learning to walk. Increased motor skills and lack of experience place the toddler at risk for injury. Child-proofing the home is essential.
Preschooler ages
3-5 years
Preschool motor development
Coordination and muscle strength increase rapidly. Increases in brain size and nerve myelinization. Perfects fine and gross motor skills. Engages in activities such as running cutting with scissors and drawing. By ages 4 or 5, the child is independent and can dress, eat, and go to the bathroom.
Cognitive and Sensory Development for Toddler
Consumed with curiosity. Boundless energy. Tremendous cognitive growth. Piaget’s sensorimotor period. Thinking is immature and limited in its logic. Behavior is characterized by negativsm, ritualism, and egocentrism.
Cognitive and Sensory Development for Preschooler
A critical period for the development of socialization. Children need opportunities to play with others. Learn communication skills. Play is often dramatic, imitative, and creative. Thinking is magical and egocentric. Overwhelmed by guilt if a loved one is injured or ill.
Psychosocial Development for Toddler
Characterized by the struggle for autonomy. Develops a sense of self separate from the parent Autonomy vs shame and doubt Erikson Stage Spend most of their time at play- it is the childs work.