Test 2 Flashcards

1
Q

What are the three main types of shock?

A

Cardiogenic, hypovolemic, and distributive

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2
Q

What are three compensatory mechanisms of shock?

A

Neural, hormonal, and chemical

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3
Q

What are the four stages of shock?

A

Initial (usually asymptomatic), compensatory, progressive (when compensatory mechanisms fail), and refractory

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4
Q

What are effects of hypoperfusion in shock in each system (8)? (brain, heart, lungs, GI, GU, skin, hematologic, and metabolic)

A
Brain: ALOC
Heart: ischemia-->infarction
Lungs: VQ abnormalities
GI: impaired mobility
GU: decreased GFR
Skin: vasocontriction
Hematologic: fibronolysis
Metabolic: anaerobic metabolism
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5
Q

A potentially fatal result of septic shock relating to the cascade of inflammation and coagulation includes:

A

Impaired fibrinolysis which results in the formation of clots

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6
Q

What is the difference between “warm shock” and “cold shock”?

A

Warm shock often goes undetected. By the time cold shock ensues, the patient is screwed….
Compensated–>uncompensated

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7
Q

What are the four components of SIRS?

A
>38 C or <36 C
HR > 90 BPM
RR >20 PaCO2 <32
WBC >12,000 or <4,000 or > 10%
*NEED 2 TO QUALIFY AS SIRS
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8
Q

What happens to SvO2 in septic shock?

A

Decreases

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9
Q

DIC occurs as a result of which conditions:

A

Trauma, sepsis, severe disease

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10
Q

Describe the process of DIC:

A

An overabundance of fibrin degradation substance, results in difficulty clotting–at risk for hemorrhage.

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11
Q

How do you treat DIC?

A

Platelets and FFP

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12
Q

What are the four causes of acute resp failure? And describe each

A
  • Failure of ventilation- inability to get air in and out (ie. obstruction)
  • Failure of oxygenation- inability of oxygenation to pass through alveoli (ARDS, pneumonia, COPD, high altitude)
  • Failure of oxygen delivery- inability of oxygen to perfuse the tissues (anemia, hypoxemia)
  • Failure of cellular oxygen utilization- impaired cellular function (carbon monoxide poisoning, alcohol)
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13
Q

What are the two types of ARF and which conditions are they associated with?

A

Hypoxemic (Type I)- ARDS

Hypercarbia (Type II)- COPD

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14
Q

Explain the difference between dead space and shunting

A

Dead space is the inspired air which doesn’t make it to the alveoli to participate in gas exchange.
Shunting is unoxygenated blood which is returned to the heart.

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15
Q

What are the three hallmark signs of an asthma attack?

A

Bronchospasm
Inflammation
Mucosal edema

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16
Q

Describe “blue bloaters” and “pink puffers”

A

Blue bloaters: chronic bronchitis patients (cyanotic, overweight)
Pink puffers: emphysema patients (barrel chest, pursed lip breathing)

17
Q

What are the two ways the brain detects when to breathe?

A
  1. ) Build-up of CO2

2. ) Low Oxygen sats (hypoxia)

18
Q

What do COPD patients rely on to know when to breathe?

A

Low O2 sats

19
Q

What are teaching points for COPD patients? (6)

A

Smoking cessation, exercise, proper nutrition, avoid irritants, flu shots, regular check ups

20
Q

On a vent, how do you increase Oxygen?

A

Increase FiO2, increase PEEP

21
Q

On a vent, how do you decrease CO2?

A

Increase respirations and volume

22
Q

Non-cardiogenic pulmonary edema is synonymous with?

A

ARDS

23
Q

What are the diagnostic criteria for ARDS?

A

Dyspnea, tachypnea, refractory hypoxia w/ resp alkalosis, bilateral infiltrates (fuzzy white), Tracheal/Plasma Protein ratio