Test 1 Flashcards
Cleft Lip and Palate (cause, prevalence):
Failure of processes to merge. Prevelance: Native American, Asian, White, Black.
Lateral: Maxillary and Mandibular.
Oblique: Lateral nasal process and Maxillary.
Cleft Lip: Maxillary and medial nasal.
Median Cleft: Two medial nasal processes.
CP= failure of palatal shelves to fuse (bifid uvula is minimum manifestation) if syndromic its CP only, if nonsyndomic (more common) its CP and CL.
Submucosal cleft= palate shelves get close enough that the mucosa cover it but bones aren’t fused.
Peirre Robin Sequence
CP, Mandibular Micrognathia, glossoptosis
Commissural lip pits
Corners of the mouth, on vermillion border
Paramedian lip pits
Symmetrical just off center pits on the lower lip. Van der woude
Ascher syndrome
Double lip, blepharochalasis, nontoxic thyroid enlargement
Fordyce granules
ectopic sebaceous glands. Raised yellow papules
Leukoedema
White appearance on buccal mucosa, disappears when stretched and comes back when released
Macroglossia
big tongue, beckwidth-weiddermans syndrome. Caused by 1) muscular hypertrophy 2) vascular malformations 3) other (lymphangioma, downs syndrome ect)
Lingual Thyroid
big thyroid in back of throat. Between foramen cecum and epiglottis. Has difficulty swallowing (dysphagia) talking (dysphonia) breathing (dyspnea). Dx by iodine/technetium-99/ct/mri
Geographic tongue:
PMN swelling on tongue. White/yellow serpentine lines that move around. Erythema migrans when not on tongue
Fissured tongue
AKA scrotal tongue. Deep fissured tongue. Pts complain of bad breath, burning, and bad taste
Coronoid hyperplasia
Overgrowth of coronoid. Deviates to ipsilateral side. Typically only during open
Condylar hyperplasia
Overgrowth of the condylar process. Deviates to contralateral side. Even at rest
Stafne defect
Saliva gland gets in mandible formation, radiopacity below the mandibular canal “lingual mandibular salivary gland depression” submandibular gland (mostly serous with some mucins)
Cyst
pathologic cavity lined by epithelium. Typically fluid filled and continue to grow bc of hydrostatic pressure
Globulomaxillary radiolucencies
radiolucency in the anterior maxilla. Commonly is radicular cyst or periapical granuloma. Usually in the lateral incisor area.
Nasopalatine duct cyst
incisive canal cyst” cyst below the incisive papilla. Duct should be less than 6 mm wide.
Dermoid cyst
has dermis structures in the lining (hair, and sebaceous glands)
Lymphoepithelial cyst
FOM (most common), ventral tongue, soft palate. waldeyers ring (palatine tonsils, lingual tonsils, pharyngeal adenoids) White/yellow asymptomatic mass less than 1 cm. has lymph tissue in it.
Progressive hemifacial atrophy
atrophy on one side of the face. Possible hx of trauma. Lyme disease can be a big cause. Look at tongue
Apert syndrome
downward slant of lateral palpebral fissures. Mouth is trapezoid shape. 75% have cleft soft palates or bifid uvula. Syndactyly (malformation of limbs)
Mandibulofacial dysostosis/treacher-collins syndrome
defects of 1 and 2 branchial arches. Coloboma (notch on outer portion of eyelid) hypoplastyic zygomatic arch. micrognathia
Turner’s hyperplasia/turner’s tooth
periapical inflammation of the primary tooth
Congenital syphilis
hutchisons incisors, mulberry molar, interstitial keratitis (blindness), 8 nerve deafness
Post developmental loss of tooth structure (attrition, abrasion, erosion, abfraction):
Attrition: tooth on tooth,
Abrasion: tooth on not tooth.
Erosion: chemical (perimolysis is erosion caused by gastric secretions)
Abfraction: loading causes cervical notches
Hypodontia
one or more missing teeth. anodontia: no teeth oligodontia: six or more missing teeth. If missing a primary tooth you will most likely miss permanent tooth
Hyperdontia
more than normal teeth. Mesiodens= extra tooth between centrals.
Gemination
Count teeth and normal number but one is big and enarged
Fusion
Two teeth have fused together
Concrescence
tooth fusion on the cementum. Extractions are hard, need to do both
Dilaceration
curved root