TENDON INJURIES Horses Flashcards

1
Q

Explain the structure and function of the SDFT

A

FUNCTION:

SDFT and SL;

  • reduces hyptoension of the fetlock joint
  • springs to store and release energy during stretching
  • main energy storing structures
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2
Q

explain the structure and function of tendons

A

Structure

Specialised molecular composition –> high strength structure Dense fibrous ECM - High water content - Collagen I predominant - Non-collagenous proteins (GAG, COMP)

Function

  • Postion limb durring locomotion
  • Viscoelastic properties
  • Max stress = tendon rupture
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3
Q

describe collagen

A

Hierachical Struture, fibrils orientated in direction of force application

  • collagen molecules stabilised by chemical crosslinks, hgih tensile strength
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4
Q

Draw a tendon.

(learn this diagram..)

A

should look like this

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5
Q

describe two non-collagenous proteins

A
  1. Proteoglycans
    - regulate fibrillogensis
    - organise matrix
  2. Glycoproteins
    - COMP most abundant; older horses tend to have a lower amound of COMP hench wby there is a higher rate of tendon injuries in older horses.
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6
Q

explain tendon/ ligament epidemiology

A

most common cause of MSI (muscle skeltal injury)

  • 46% of all MSI involve ligamets/tendons
  • SDFT most commonly injured

risk of injury increses with

  • increased age
  • increased speed
  • male horses
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7
Q

what are the two ways tendon injury occurs?

A

occurs in one ot two ways:

  1. overstrain injury (most common)
  2. Truma
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8
Q

explain the mechanisms of overstrain injury

A
  1. accumulation of microdamage overtime
    - increased MMP gene expression
    - limited ability to repair
  2. increased age
    - reduced crimp; reduces strain needed to cause damage
    - reduced tendon fascicle sliding
  3. Hysteresis
    - temperature within core of tendon, recent evidence suggests increased pro-inflammatory mediators greater then 45 celsius
  4. muscle fatigue
    - most tendon injuries occur later in the race
    - muscle fatigue late in race leads to abnormal loading
    - DDFT fatigue; destabilisation of fetlock joint
    - never been followed up in studies if this is true.
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9
Q

List and describe methods for diagnosis

A
  1. Clinical examination
    - palpation
    - lameness examination; but horse may only appear lame for a couple of days following injury and then be sound
  2. Ultrasound examination
    - appearance varies depending of severity of lesion
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10
Q

list and describe rehabilitation programs

A
  1. Initial treatment
    - reduce inflammation; ice/cold water hosing, bandaging, NSAIDs, confinement
  2. Excercise programs
    - uncontrolled excercise; not a good option, tendons will not be conditioned and re-injury rate is high
    - controlled excercise; accompanied by regular ultrasounds, still about 50% re-injury when returned to racing
  3. surgical management
    - not done too often these days
  4. intralesional therapy
    - regenerative medicine
    - stem cells, bone marrow or adipose derived
    - Platelet Rich Plasma; limited evidence that it is of any benefit
  5. shockwave therapy
    - some evidence that it may be harmful not used often.
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