TENDON INJURIES Horses Flashcards
Explain the structure and function of the SDFT
FUNCTION:
SDFT and SL;
- reduces hyptoension of the fetlock joint
- springs to store and release energy during stretching
- main energy storing structures
explain the structure and function of tendons
Structure
Specialised molecular composition –> high strength structure Dense fibrous ECM - High water content - Collagen I predominant - Non-collagenous proteins (GAG, COMP)
Function
- Postion limb durring locomotion
- Viscoelastic properties
- Max stress = tendon rupture
describe collagen
Hierachical Struture, fibrils orientated in direction of force application
- collagen molecules stabilised by chemical crosslinks, hgih tensile strength
Draw a tendon.
(learn this diagram..)
should look like this
describe two non-collagenous proteins
- Proteoglycans
- regulate fibrillogensis
- organise matrix - Glycoproteins
- COMP most abundant; older horses tend to have a lower amound of COMP hench wby there is a higher rate of tendon injuries in older horses.
explain tendon/ ligament epidemiology
most common cause of MSI (muscle skeltal injury)
- 46% of all MSI involve ligamets/tendons
- SDFT most commonly injured
risk of injury increses with
- increased age
- increased speed
- male horses
what are the two ways tendon injury occurs?
occurs in one ot two ways:
- overstrain injury (most common)
- Truma
explain the mechanisms of overstrain injury
- accumulation of microdamage overtime
- increased MMP gene expression
- limited ability to repair - increased age
- reduced crimp; reduces strain needed to cause damage
- reduced tendon fascicle sliding - Hysteresis
- temperature within core of tendon, recent evidence suggests increased pro-inflammatory mediators greater then 45 celsius - muscle fatigue
- most tendon injuries occur later in the race
- muscle fatigue late in race leads to abnormal loading
- DDFT fatigue; destabilisation of fetlock joint
- never been followed up in studies if this is true.
List and describe methods for diagnosis
- Clinical examination
- palpation
- lameness examination; but horse may only appear lame for a couple of days following injury and then be sound - Ultrasound examination
- appearance varies depending of severity of lesion
list and describe rehabilitation programs
- Initial treatment
- reduce inflammation; ice/cold water hosing, bandaging, NSAIDs, confinement - Excercise programs
- uncontrolled excercise; not a good option, tendons will not be conditioned and re-injury rate is high
- controlled excercise; accompanied by regular ultrasounds, still about 50% re-injury when returned to racing - surgical management
- not done too often these days - intralesional therapy
- regenerative medicine
- stem cells, bone marrow or adipose derived
- Platelet Rich Plasma; limited evidence that it is of any benefit - shockwave therapy
- some evidence that it may be harmful not used often.