TBI Flashcards

1
Q

TBI

A
  • Rapid injury to brain: stroke, external inflicted trauma, infection, parasite etc.
  • Typical cause: MVA, falls, sports, violence, knife wounds, blunt force trauma
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2
Q

Types of TBI (2)

A
  1. Force inflicted wounds
  • Closed head wound (most common)
  • Open Head wound: penetrating/ crushing (least common)
  1. Encephalopathey
  • Anoxic
  • Metabolic
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3
Q

Concussion

A
  • Closed head wound
  • Mild TBI
  • Causes: blunt force, deceleration, angular rotation
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4
Q

Pathophysiology of Concussion

(what happens)

A
  • Brain bounces inside cranium rubbing dura mater and bone, causing hematoma (blood collection like bruise) and swelling.
  • Brain stretches from base,cause tissue to rub together. Axons become distended (mild DAI)
  • Brain presses down or streches brainstem, temp shutting it down.
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5
Q

Symptoms of Concussion

A
  • Headache
  • Nausea
  • Sleepiness
  • Vomitting
  • Loss of orientation (more disoriented the worse)
  • Glas Coma Scale of 12 and up
  • LoC for less than 30
  • Amnesia for less than 24 hr
  • normal pupillary response less than 5 mm more than 3 mm
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6
Q

Tx for Concussion

A
  • Usually no treat as long as no focal injury non CT
  • ice on contusions
  • monitor vital signs for 24 hr
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7
Q

Prognosis for Concussion

A
  • Excellent prog
  • multiple concussions can lead to lasting impairment
  • one concussion cause permanent impairment in short term mem, attention, encoding, recall, concentration.
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8
Q

Closed head Wounds

Epidural

A
  1. Blunt force: smack to skull, on sides, cause epidural or subdural hematoma
  2. Epidural: blood collects btwn skull & dura mater. Convex blood collection on CT
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9
Q

Closed Head Wounds

Subdural Hematoma

A
  • blood collects btwn skull & arachnoid mater
  • often venous bleed
  • crescent shape on CT
  • more common
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10
Q

Deceleration Injury

A
  • Head moving in 1 direction, quickly stopped, sent in other direction
  • Causes: MVA, falls, blunt force
  • Coup-contre coup=Brain travelling in direction of inertia, bounces back against skull to hit opposite side
  • common in frontal/occipital, temporal/temporal
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11
Q

Rotational Injury

A
  • Head twisted by impact, parts of brain/skull rub against each other
  • Brain rubs against skull/meninges: veins in arachnoid mater rupture, causing subdural/subarachnoid hemorrhage
  • Shearing injury: Brain rub against itself: Axons get stretched/torn (DAI), causing petechial/intraparenchymal hemorrhage
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12
Q

Diffuse Axonal Injury

A
  • Neurons stretched by deceleration/ abraded by shearing
  • Damage from DAI can occur after initial injury when broken axons release apoptic factors (cell suicide)
  • very difficult to see in brain scan bc microscopic
  • POOR prognosis
  • predictive of coma
  • more evident DAI on brain scan=higher mortality
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13
Q

Open Head Wounds

A
  1. Penetrating
    * cranium penetrated-by skull, blunt or piercing object (bullet)
  2. Crushing
  • Skull crushed btwn objects causing widespread fracture/ brain visibility
  • symptoms same as closed w/ higher risk of infectoin & pneumocephalus (air entering brain cavity disrupting pressure)
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14
Q

Extra Axial Bleed

A

Bleed occuring inside skull but outside brain tissue

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15
Q

Herniation

A
  • Occurs during Extra axial bleeds
  • ICP inc, brain swell, blood/meninges push brain in places it souldn’t go.
  • Tentorium=area of dura mater seperating cerebellum/cerebrum
  • Uncus=inferior medial temporal lobe
  • Cingulate gyrus=medialportion of cerebrum (around corpus cal)
  • Foramen magnum=big hole at bottom of cranium where spinal cord/brainstem meet
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16
Q

Supratentorial Herniation

A
  • Herniation above tentorium
  • Subfalcine=most common. Cingulate gyrus pushed under falx cerebri
  • Central=midbrain pushed down tentorium
  • Transcalvarial=brain matter pushed through defect in cranium
  • Uncal=medial temporal lobe pushed through tentorium
17
Q

Infratentorial Herniation

A
  • Herniation below tentorium
  • Upward Cerbellar=cerebellar pushed up tentorium
  • Tonsillar=downward cerebellar, pushed through for mag
18
Q

Symptoms of TBI

A
  • Skull fracturing, skull depression
  • worsening headache, then slip in2 coma/unconsciousness=bad prognosis
  • confusion
  • nausea/vomiting
  • altered mental stat/loss of orientation
  • ICP inc–>symptoms intensify–>herniation, midline shift, mass effect, pupillary irregularity=prognosis worsens
  • potential seizure & AMS
19
Q

Fencing Posture

A
  • Arm flies out as a tonic response to disruption of neurochemistry, with vestibular function involved
  • often in contusion leading to unconsciousness
20
Q

Opisthotonus Posture

A
  • severe muscle spasms caused by compression of corticoreticular tracts in midbrain
  • Back and head arch back
  • not common in TBI
21
Q

Decorticate Posture

A
  • arms fold into “prayer” position, legs extended and feet turned inwards
  • muscles rigid
  • Indicative of pressure or damage to brainstem, especially motor tracts going through medulla
  • Bad
22
Q

Decerebrate Posture

A
  • arms straighten, hands curl downward and outward. Legs extend, feet turn inwards
  • very rigid, teeth clenched
  • If unconscious and continous, indicates brainstem herniation
  • VERY bad
  • seen frequently for short periods of time in comatose patients with brainstem damage
23
Q

Acute Dx and Prognostic Ind of TBI

A
  • CT: differential Dx for extr vs. intra axial hemorrhage, skull frac, vertebral frac
  • Acute Prog Indicator: age, pupillary response, consciousness, Glas Coma Scale, temp (lower=better), hypoxia (amount of oxygen in blood/brain)
  • Goals: evacuation of blood for decompression, remove dead/dying tissue to prevent infection
24
Q

Primary Tx options for TBI (5)

A
  • Burr-hole: drill hole in cranium, suck out blood for extra axial bleed.
  • Craniotomy: remove part of skull, suck out blood. Extra and some intra axial bleeds. Followed by cranioplasty (reinserting skull/plastic)
  • Craniectomy: remove part of skull. Hemorrhage and decompress of brain tissue & ICP due to swell & herniation.
  • marsupialization=keeping piece of skull in abdomen for cranioplasty
  • Lobectomy: remove part of brain after Wada test to make sure it isnt vital. Cases of severe focal injury
  • Shunt/EVD for drainage of CSF or blood
25
Q

Encephalopathy

A

Anoxic=oxygen not reaching brain

  • Asphyxiation
  • cardiac arrest
  • drowning
  • CO poisoning
  • Acute drug OD

Metabolic=caused by external factor

  • cytotoxic drug use (huffing)
  • lead poisoning

Symptoms/Prog vary widely bc damage to entire brain, hard to spot.

26
Q

Long Term Care/Prog

A
  • Prognosis=extremely variable
  • Long term prog indicators: age, sverity of injury, length/depth of coma, amount of recovery=6mos, level of amnesia.
  • Temporally graded amnesia: amnesia for amount of time before injury. Should give GOAT Test.
  • 10% seizure disorder
  • 50% personality change/mood disorder
27
Q

Long Term Sequelae

A
  • Encephalomalacia: softening of brain tissue by necrosis
  • Heterotrophic Ossification: very rare
  • Periods of dizziness and nausea
  • Photosensitivity/headaches
  • Cranial nerve/visual damage
  • Memory deficits
  • Aphasia
  • anosognosia: Insight and judgment deficits
  • Seizures
  • Personality change
  • Executive functioning deficits in planning, organization, problem-solving
28
Q

TBI Rehab

4 therapies

A
  • Typically four therapies:
    Speech
    Physical
    Occupational
    Cognitive
  • Counseling, Job Placement, Community Re-entry
  • Physical and occupational therapies maintain gains after rehab
  • Cognitive therapy has shown to remit somewhat after termination of therapy, indicating ongoing therapy is required
29
Q

Hemineglect

A
  • disorder that tends to appear after damage to the R Parietal Lobe
  • causes: CVA, TBI, or disease
  • often occurs w/ Left Homonymous Hemianopsia (only seeing out of the Right side of both eyes)
  • common types of neglect are Spatial, Motor, Sensory, maybe Auditory involvement
30
Q

Symptoms of Hemineglect

A
  • Hemianopsia Inattention – can’t see out of the L side, like left side of the world does not exist
  • Anosognosia – doesn’t believe half body belongs to them, not bothered by it
  • Extinction to Double Simultaneous Stimulation – can raise their arms separately, but when asked to raise both, will only raise the R.
31
Q

Hemineglect Personality Change

& Tx

A
  • tempero-parietal junction controls mood/personality function
  • will become nonchalant about neglect

OR

  • become argumentative, angry, non-compliant w/ Tx
  • Tx=Behavioral plans based on contingency management (reinforcement for compliance with therapy)
32
Q

Tx of Hemineglect

A
  • Lighthouse Therapy – teach pt to use head and eyes like a lighthouse, “scanning” left to right. Maintain continuous tactile stimulation on the neglected side, and force to use neglected limbs.
  • Cancellation Tests – used to teach pts to overcome inattention
33
Q

Prognosis of Hemineglect

A
  • not good for recovery

(BUT age=good predictor for recovery – I.e., younger people are-more likely to recover than older)

  • Tx=compensatory (focus on insight and awareness of inattention).
  • remind ind. to groom neglected half
  • Highlight neglected side of important documents
  • Place arrows pointing to neglected side
  • worse the motor and personality components (vs. visual/spatial components)=worse prognosis for recovery and compensation for deficits.