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Neuromotor II > TBI > Flashcards

TBI Flashcards

1
Q

What age demographics are most likely to sustain a TBI?

A
  1. Children ages 0 to 4 years
  2. young adults ages 15-24 years
  3. adults 65 years and older
    - incidence is greater in males in all age groups
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2
Q

What are the parameters for loss of consciousness, alteration of consciousness, post traumatic amnesia, GCS score, and neuroimaging for a mild TBI?

A 
loss of consciousness = 0-30min
alteration of consciousness = brief < 24hrs
post traumatic amnesia = 0-1d
GCS score = 13-15
neuroimaging = normal
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3
Q

What are the parameters for loss of consciousness, alteration of consciousness, post traumatic amnesia, GCS score, and neuroimaging for a moderate TBI?

A 
loss of consciousness = 30min - 24hrs
alteration of consciousness = >24hrs
post traumatic amnesia = 2-7d
GCS score = 9-12
neuroimaging = normal or abnormal
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4
Q

What are the parameters for loss of consciousness, alteration of consciousness, post traumatic amnesia, GCS score, and neuroimaging for a severe TBI?

A 
loss of consciousness = >24hrs
alteration of consciousness = >24hrs
post traumatic amnesia = >7d
GCS score = 3-8
neuroimaging = normal or abnormal
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5
Q

What classifies as an open vs closed TBI?

A

Open = penetrating injury, dura compromised

Closed = non penetrating, dura uncompromised

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6
Q

primary issues of head trauma?

A
  1. Skull fracture
  2. Contusions of gray matter - coup or contrecoup (may lead to 2* issues)
  3. Diffuse white matter (axonal) damage
  4. Hematom
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7
Q

secondary issues of head trauma?

A
  1. Anoxia
  2. Ischemia
  3. Swelling/Increased intracranial pressure (ICP)
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8
Q

What kind of skull fx can occur?

A
  1. Linear
  2. Depressed
  3. Compound - breaks off
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9
Q

What are signs of a basilar fracture?

A
  1. Raccoon eyes – always bilaterally, bleeding at base of skull
  2. Battle’s sign – bruising behind ear; middle of the base fracture
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10
Q

What are features of an orbital fracture?

A
  • eye socket
  • unilateral bruising (usually)
  • sometimes called a ”blowout” fracture
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11
Q

Severing of axons because of shearing forces caused by rapid rotational and acceleration/ deceleration movement; Sudden loss of consciousness - May be prolonged and cause coma; Main mechanism of injury in those with moderate or severe TBI; High mortality rate; Widespread microscopic damage (Often there are minimal findings on initial CT and MRI)

A

Diffuse white matter (axonal) damage

- see more decerebrate posturing - worst prognosis

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12
Q

What type of hematoma?

  • Tear in the meningeal artery between skull and dura
  • Usually from a focused blow to the head
  • Fixed and dilated pupil on same side
  • Emergency surgery
  • Best prognosis if treated early
A

Acute epidural

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13
Q

What type of hematoma?

  • Venous rupture between dura and the arachnoid
  • High frequency of seizures
  • Emergency surgery
  • Poor prognosis
  • High mortality rate
A

Acute subdural

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14
Q

What type of hematoma?

  • Any location in the brain
  • Arteries and veins
  • Neurological deficits
  • Poor prognosis
  • High mortality rate
A

Intracerebral hematoma

  • in the brain itself
  • sx of a stroke, looks like hemorrhagic stroke
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15
Q

What does the following series of events cause?

  • Starts with anoxia
  • Leads to ischemic tissue damage
  • Leads to body responding by compensatory vasodilation
  • Leads to increased swelling and increased ICP resulting in anoxia
A

Secondary issues

  • Cell death results from a chain of cellular events
  • Develop over hours and days
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16
Q

What is normal ICP? When does it cause neurological damage and death?

A

Normal ICP is 5-20 mm Hg
20-40 mm Hg = may contribute to brain damage
> 40 mm Hg = neurological damage and death

  • as it climbs over 20, something has to be done; may remove portion of skull if medication does not work
  • may be monitored by ventriculostomy
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17
Q

What are clinical manifestations of ICP?

A
  1. Headache
  2. nausea
  3. drowsiness
  4. weakness
  5. papilledema
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18
Q

What causes brainstem damage?

A

Downward pressure on foramen magnum

  • Caused from swelling and increased ICP
  • cranial nerve damage occurs with injuries to base of the skull where the nerves emerge directly from brainstem
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19
Q

What. can cause internal carotid artery damage?

A
  1. Gunshot wounds
  2. Blows to the neck
  3. Injuries to face
  4. Cervical hyperextension
20
Q

What. can cause vertebral artery damage?

A
  1. severe cervical hyperextension

2. severe cervical rotation

21
Q

What is the purpose of a neuro exam in a TBI?

A
  1. Determine severity
  2. Determine baseline of injury in order to detect deterioration
  3. Categorize injury
  4. Help with prognosis
22
Q

What are the components of a neuro exam in a TBI?

A
  1. Eye assessment
  2. Posturing
  3. Vocalization
  4. PROM and tone
  5. Active movement - Purposeful or nonpurposeful?
  6. Reaction to tactile/painful stimulation
  7. Clinical Rating Scales
23
Q

What are you looking at during an eye assessment?

A
  1. Size and symmetry of pupils
  2. Response to light
  3. Tracking in response to auditory or visual stimulation
  4. Dysconjugate - asymmetry, inability to turn eyes together in the same direction
24
Q

What’s the purpose of medications for TBIs?

A
  1. Decrease ICP
  2. Hypertension
  3. Anti-Seizure
  4. Muscle spasticity
  5. Behavioral and cognitive functions
25
Q

What categories does the Glasgow coma rating scale assess for scores?

A

Eye Opening = spontaneous> to speech> to pain> no response

Motor Response = Follows motor commands> localizes> withdraws> abnormal flexion> abnormal extension> no response

Verbal Response = oriented> confused conversation> inappropriate words> incomprehensible sounds> no response

26
Q

Assists with prognostic assessment and treatment planning; 23 items, 6 subscales addressing:
Auditory
, Visual
, Motor
, Oromotor/Verbal
, Communication
, Arousal
- takes 30 mins to administer, cut off scores for vegetative state, minimally conscious, and emerging from minimally conscious

A

Coma recovery scale

27
Q

What are the Ranchos Los Amigos Levels of Cognitive Recovery? assesses typical cognitive behavioral recovery

A

I No response: Needs total assistance
II Generalized response: Needs total assistance
III Localized response: Needs total assistance
IV Confused – agitated: Needs maximal assistance
V Confused – inappropriate: Needs maximal assistance
VI Confused – appropriate: Needs mod assistance
VII Automatic – appropriate: Needs min assistance
VIII Purposeful – appropriate: needs SBA

28
Q

Damage to this area of the brain results in:

  • loss of movement
  • repetition of single thought
  • unable to focus on a task
  • mood swings, irritability, impulsiveness
  • changes in social behavior and personality
  • difficulty with problem solving
  • difficulty with language; can’t get words out (aphasia)
A

frontal lobe

29
Q

Damage to this area of the brain results in:

  • difficulty distinguishing L from R
  • lack of awareness or neglect of certain body parts
  • difficulty with eye-hand coordination
  • problems with reading, writing, naming
  • difficulty with math
A

parietal lobe

30
Q

Damage to this area of the brain results in:

  • defects in vision or blind spots
  • blurred vision
  • visual illusions/ hallucinations
  • difficulty reading and writing
A

occipital lobe

31
Q

Damage to this area of the brain results in:

  • difficulty understanding language and speaking
  • difficulty recognizing faces
  • difficulty IDing/ naming objects
  • problems with ST and LT memory
  • changes in sexual behavior
  • increased aggressive behavior
A

temporal lobe

32
Q

Damage to this area of the brain results in:

  • difficulty coordinating fine movements
  • difficulty walking
  • tremors
  • dizziness
  • slurred speech
A

cerebellup

33
Q

Damage to this area of the brain results in:

  • changes in breathing
  • difficulty swallowing food and water
  • problems with balance and movement
  • dizziness and nausea
A

brainstem

34
Q

what is worse? decorticate or decerebrate?

A

decerebrate rigidity

  • Corticate – corticospinal damage
  • Cerebrate – brainstem damage
35
Q

What are cognitive impairments associated with mild TBI (concussion)?

A
  1. Irritable, distractible
  2. Difficulty with reading and memory
  3. Symptoms of headache, fatigue, dizziness
  4. Changes in personality
  5. Emotional changes
36
Q

Level of consciousness:

  • Drowsy, fall asleep easily
  • Decreased alertness, delayed reactions
  • Once aroused, responds appropriately
A

obtunded/ lethargy

37
Q

Level of consciousness:

  • Near-unconsciousness
  • Aroused briefly by verbal, visual, or painful stimuli
  • Slowed motor or moaning responses to stimuli
A

Stupor

38
Q

Level of consciousness:

  • Not awake and not aware
  • “Unarousable unresponsiveness”
  • No sleep-awake cycle “deep sleep”
A

coma

39
Q

Level of consciousness:

  • Intact eye opening and sleep-awake cycles but not able to speak or obey commands
  • Called a persistent vegetative state if >1 year
A

vegetative state

40
Q

Whats the difference between retrograde and post-traumatic (anterograde) amnesia?

A

Anterograde – time between injury and memories returning, can’t form new memories, used in diagnosing severity

Retrograde – period before injury

41
Q

What are the communication impairments often present in TBI?

A

Normally no aphasia but issues are related to cognitive impairments

  1. Disorganized
  2. Word retrieval difficulties
  3. Socially inappropriate language
  4. Difficulty reading social cues
42
Q

What are the behavioral impairments often present in TBI?

A
  1. Agitation
  2. Anger/Aggression
  3. Denial
  4. Depression
  5. Egocentrism
  6. Excessive Talking
  7. Impulsivity
  8. Inability to Initiate
  9. Irritability
  10. Lability
  11. Memory
  12. Obsessional Behavior
  13. Paranoia
  14. Social Inappropriateness
43
Q

What are strategies for agitated patients?

A
  1. Model calm and controlled behavior
  2. Redirect when necessary
  3. Be consistent with daily routine
  4. Be flexible and vary activity to avoid boredom
  5. Schedule short frequent breaks
  6. Give options to patient so they feel more in control
  7. Do not teach a new complex skill
  8. Do not overstimulate
  9. Always be aware of safety
44
Q

What are some general strategies for working with pts with TBI?

A
  1. BE PATIENT
  2. Establish an open, trusting relationship
  3. Remember anger is often a spontaneous act
  4. Assist pt. in verbalizing what they may be feeling
  5. Seek professional help for suicidal statements
  6. Give consistent feedback and praise
  7. Set clear boundaries
  8. Do not feel obligated to respond to all statements
  9. End conversation at a set amount of time
  10. Teach “stop, think, and then act”
  11. Break tasks into small steps
  12. Don’t argue with paranoid thoughts
  13. Directly confront inappropriate behavior
45
Q

What are techniques to address memory loss?

A
  1. Use compensation skills - Lists, calendars, reminders, alarms, etc
  2. Give information in writing
  3. Use repetition
  4. Periodically “check in” with pt. during treatment - “Explain what I just said” or “Review what we have been working on”
46
Q

What are pre injury prognostic indicators?

A

Education level

Age - neuroplasticity

47
Q

What are post-injury prognostic indicators?

A
  1. Type of injury
  2. Initial GCS score
  3. Pupil reactivity
  4. Other injuries along with TBI
  5. Duration of coma
    6 Duration of amnesia

Neuromotor II (13 decks)

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