Stroke Intro Flashcards

1
Q

When observing a patient post stroke, what is important to note?

A
  1. Bony prominences
  2. Muscle atrophy
  3. Head position
  4. Shoulder height
  5. Pelvic position – Usually affected side is more posterior in standing
  6. UE & LE position
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2
Q

what is cerebral shock?

A

flaccidity following stroke; moves to spasticity (similar to spinal shock

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3
Q

what is the flexion synergy of the UE?

A
  1. Scapular retraction/elevation
  2. Shoulder abduction/ER
  3. Elbow flexion**/supination
  4. Wrist and finger flexion
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4
Q

what is the extension synergy of the UE?

A
  1. Scapular protraction
  2. Shoulder adduction**/IR
  3. Elbow extension/pronation**
  4. Wrist and finger flexion
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5
Q

what is the flexion synergy of the LE?

A
  1. Hip flexion**, abduction, ER
  2. Knee flexion
  3. Ankle DF/Inv
  4. Toe DF
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6
Q

what is the extension synergy of the LE?

A
  1. Hip extension, adduction**, IR
  2. Knee extension**
  3. Ankle PF**/Inv
  4. Toe PF
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7
Q

what are the stages of motor recovery (synergy patterns) according to Brunnstrom?

A
I. Flaccidity
II. Synergies; some spasticity
III. Marked spasticity
IV. Out of synergy; less spasticity
V. Selective control of movement
VI. Isolated/ coordinated movement
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8
Q

primitive reflex most often seen post stroke; pt may be unable to straighten arm wo/turning head toward that arm; to secure extension of the involved leg, pt can turn head to involved side

A

ATNR

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9
Q

primitive reflex; when pt is supine w/ head up on pillows, arms won’t fully extend & legs are in an extensor pattern; Coming to sitting while flexing head, LE’s go into EXT and are difficult to bend; Transfers – if head extends, legs may flex

A

STNR

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10
Q

primitive reflex most often seen post head injury; Extensor tone increases in supine if head is extended; Head pushes into supporting surface; Resistance to shoulder protraction; Rolling blocked by extensor tone; Sitting - when pt extends head, hips slide forward in chair (May slide out of chair)

A

TLR

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11
Q

primitive reflex most often seen post TBI; When ball of foot is in contact w/the floor, immediate extensor tone; Not a normal standing position - not conducive to regaining balance and equilibrium; Tx: get either entire foot on floor, or at least heel – do NOT want just balls of foot on floor or will cause this to occur

A

Positive Supporting Reaction

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12
Q

Unintentional movements of hemiparetic limb caused by voluntary movements of another limb or other stimuli; Can be cause by yawning, sneezing, or coughing

A

Associated Reactions
- Raimiste’s Phenomenon = Involved LE will ABD and/or ADD if resistance is applied to the uninvolved extremity; Ex: Supine/Sitting – put pillow between pt’s knees – have them ADD univolved side, & involved will kick in

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13
Q

what is the pattern of mm weakness after stroke?

A

usually distal, then proximal

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14
Q

can you use MMT for testing mm strength after stroke?

A
  • Difficult to do due to compensations, spasticity, inability to isolate the muscle; If pt has spasticity – goal is to get functional idea of strength; document “Unable to MMT due to spasticity, but assessed….”
  • MCA - 20% don’t regain use of their UE’s
  • Stroke in General: must keep in mind that only 75-90% of corticospinal fibers cross, so MOST OFTEN you see Contralateral involvement, BUT may have some ipsilateral involvement as well; often we use ‘unaffected side’ as baseline – in these pt’s, ‘unaffected side’ might be weak too
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15
Q

What outcome measure do you use for with pts that are highly involved/ lower level?

A
  1. Five Time Sit to Stand Test
  2. Function in Sitting Test (FIST)
  3. Trunk Impairment Scale
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16
Q

What is pusher syndrome?

A
  • Altered perception of body’s orientation
  • Perceive body as vertical when it is actually tilted 20*
  • Ipsilateral Pushing - active pushing w/stronger extremities toward weak side - Tendency to fall toward weak side
  • visual and vestibular input intact
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17
Q

Where is the lesion when pusher syndrome is present?

A

posterolateral thalamus

18
Q

How should a PT intervene for pusher syndrome?

A
  • working in a corner, push towards wall
  • Having something in front of them to make them more aware
  • Mirror – for visual input so they know when they are doing this

(-) poor outcome – difficult to work on interventions if always push to weak side
(+) brain can compensate – generally will; disappear/ decrease around ~6 mo’s

19
Q

What is the difference between an physiological, household, and community walker?

A

1) Physiological Walker- Walks for exercise only at home or during PT sessions ; Likely requires a good amt of help/assistance; in home, may stand to complete some ADL’s (toileting, etc)
2) Household Walker - Uses walking for home activities; Predictable environment; can getting around their house decently, though may use an AD; won’t walk into clinic for a PT session
3) Community Walker - Enter/leave home, ascend/descend curb, and manage stairs independently, Independent w/some community activities

20
Q

Where is a lesion if there is speech and language impairments?

A

cortex of the dominant side

- majority of L sided stroke

21
Q

trouble speaking fluently but their comprehension can be relatively preserved; difficulty producing grammatical sentences and their speech is limited mainly to short utterances; understand speech relatively well

A

Broca’s aphasia

- also known as non-fluent or expressive aphasia

22
Q

form of aphasia the ability to grasp the meaning of spoken words and sentences is impaired, while the ease of producing connected speech is not very affected

A

Wernicke’s aphasia

  • receptive aphasia
  • increase tactile cues; simple sentences; don’t give options; give extra time to respond
23
Q

Mixture of receptive and expressive aphasia

A

global aphasia

- poorer outcomes for rehab

24
Q

difficulty swallowing

A

dysphagia

- mm problem (usually), nerve problem, or both

25
Q

motor speech disorder, indicates movement of jaw and tough impaired

A

dysarthria

- slurred speech

26
Q

What area of the brain is affected when there are visual-perceptual deficits?

A

right parietal cortex
- S and S: unilateral neglect, agnosia (inability to interpret sensation, therefore difficulty recognizing certain people/ objects/ sounds/ shapes, etc

27
Q

What cognitive deficits are seen following stroke?

A
  1. Attention
  2. Memory
  3. Perseveration
  4. Executive function
28
Q

What effect can a stroke have on a pts emotional status?

A
  1. Pseudobulbar affect - Emotional Lability = uncontrollable emotions; uncontrollable crying or euphoria
  2. Apathy - Decreased emotion (may come off as depression, or lack of motivation)
  3. Depression - result of both dealing w/ disability + physical effects
29
Q

Where is the lesion that most likely results in depression?

A

left frontal or right parietal

30
Q

When does depression most often occur following a stroke?

A

6months- 2 years (lasts/ peaks)

- dealing with life changes due to stroke

31
Q

Which side of the brain is affected: Difficulties w/communication; Negative, anxious, depressed, slower, cautious, uncertain, insecure; More realistic about their problems, very aware of impairments

A

Left Brain Stroke

32
Q

Which side of the brain is affected: Unilateral neglect; Indifferent, quick, impulsive, euphoric, poor judgment; Overestimate their abilities, often unaware of impairments

A

Right Brain stroke

  • safety is major concern
  • use bed/ chair alarms
33
Q

Which side of the brain is affected: Short attention span; Emotional lability; Irritability, confusion, restlessness; Psychosis, delusions, or hallucinations

A

Both sides affected

34
Q

What sensation is commonly lost following stroke?

A

Proprioception

  • esp. ankle: affects mobility, balance, ambulation, etc
  • Needs to be in sensory exam!
  • other sensations are impaired, but rarely absent
35
Q

What is the most common distribution of sensation loss/ impairment?

A

Face, UE, and LE together

36
Q

What types of pain are often felt following stroke?

A
  1. Thalamic pain (uncommon) - constant severe, burning type of pain; involving PCA usually and parts of thalamus; spinothalamic systems affected; no great way to treat.. some meds
  2. shoulder pain - more common; involved side = due to mm weakness (weight of arm on joint) and lack of movement; uninvolved side = overcompensation
  3. other joint pain - more common
37
Q

What does bladder function look like in patients with stroke?

A
  1. Incontinence common in acute phase - Catheter should be removed early (UTI)
  2. Can by hyper or hyporeflexic bladder later on
38
Q

What are secondary complications due to stroke?

A
  1. Contractures*
  2. DVTs & PE*
  3. Skin breakdown*
  4. Seizures
  5. Aspiration
  6. Causes issues with dysphagia
  7. Problems with speech – swallow eval
  • KEY is PREVENTION! – PT ROLE, esp. *’d ones; Get pts moving, and early
39
Q

What outcome measures does the neurology section recommend students learn for patients with stroke?

A
  1. 6 MWT
  2. 10 mWT
  3. Action research arm test
  4. Ashworth
  5. BBS
  6. DGI
  7. Fugl-Meyer (motor performance)
  8. FRT
  9. Orpington Prognostic scale
  10. Postural assessment scale for stroke
  11. Stroke Impact Scale
  12. Tardieu spasticity scale
40
Q

What are outcome measures that asses body structure and function following stroke?

A

1, Ashworth Scale

  1. Orpington Prognostic Scale
  2. Fugl-Meyer (motor performance)
  3. Tardieu Spasticity Scale
  4. FTSST
  5. NIH Stroke Scale
41
Q

What are outcome measures that asses activity following stroke?

A
  1. Berg Balance Scale
  2. DGI
  3. Functional Gait Assessment
  4. 10 mWT
  5. 6 MWT
  6. Action Research Arm Test
  7. Arm function
  8. Functional Reach Test
  9. FIM
  10. STREAM
  11. ABC - sometimes more difficult for pts to understand
42
Q

What are outcome measures that asses participation following stroke?

A
  1. Stroke impact scale - the pt’s perception of where they’re at; physical, memory, thinking, etc.
  2. SF36