TBI Flashcards
leading cause of TBI
Falls (elderly) vs MVA (children)
TBI demographic
single, white, undereducated, teen to 25 yo, male
cerebral profusion pressure equation
What value keep CPP?
CPP = MAP - ICP
CPP>60mmhg
Keep ICP <20mmhg
Managing ICP
nl range 2-5, keep under 20
- force hyperventilation (decrease PaCO2-> vasoconstriction-> less flow)
- hypothermia
- Meds: IV mannitol or acetazolamide
- emergency- burr hole, craniotomy
Primary injury
Contusion
Diaschisis
DAI
immediate concussive force
brain bruises at “horns” of brain. usually at inf frontal lobe and ant temp lobe
“split across”- damage area damages remote region connected
“Diffuse Axon Injury”
DAI- causes, most common locations
diffuse axonal injury
axon injury 2.2 concussive blat to head and shears axon leading to central white matter damage. white on diffusion weighted MRI. Usually seen at central white matter, brain stem, corpus callosum
DAI grading
Using MRI criteria
1- no focal change
2- focal change
3- brain stem involvement
Secondary injury
biochemical cascade after primary injry
- excitatory toxicity (glutamate): massive neurotransmitte r surge and burn out
- Brain swelling- CT shows decrease ventricle size
- Brain edema - longstanding, blood vessel damage, fluid leak out of brain tissue
Posturing
decorticate
decerebrate
deCORticate: arms flexed, legs extended. lesion at brainstem
Decerebrate: arms extended, leg extended. lesion at midbrain. (worse due to brainstem involvement)
Epidural hematoma
Lens
middle meningeal artery
no midline shift
lucid then downhill
Subdural hematoma
Crescent
bridging vein
bleed between dura and arachnoid
subarachnoid hemorrhage
bleeding in subarachnoid space
rupture AVM or berry aneurysm (usually Acomm or Pcomm)
give nimodipine x 21days for cerebral vasospasm
C1 injury
smell, most common injury
C2
visual field
C7
deficit at ant 2/3 tongue, facial expression, salivation, tears