Tachycardia Flashcards

1
Q

Which general classification of antiarrhythmics should be avoided when treating Atrial Fibrillation in patients experiencing severe CHF and acute decompensated heart failure (ADHF)?

A
  1. Calcium-channel blockers;

https://foamcast.org/2014/09/21/episode-15-atrial-fibrillationflutter/

Note: “In congestive heart failure, patients with reduced ventricular function may not be able to counteract the inotropic and chronotropic effects of diltiazem, the result being an even higher compromise of function.”

https://en.wikipedia.org/wiki/Diltiazem

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2
Q

Name nine risk factors for Atrial Fibrillation.

A

ATRIAL FIB

A - Alcohol

T - Thyroid (primarily hyperthyroidism)

R - Rheumatic Heart (as well as other valvular heart disease such as mitral valve stenosis)

I - Ischemia

A - Atrial Myxoma

L - Lung (PE, Emphysema)

F - Pheochromocytoma

I - Infection

B - Blood Pressure (HTN)

https://foamcast.org/2014/09/21/episode-15-atrial-fibrillationflutter/

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3
Q

Per FOAMCast, a target heart rate of < ___ BPM is acceptable when attempting rate control of stable atrial fibrillation in the ED.

A
  1. 120;

https://foamcast.org/2014/09/21/episode-15-atrial-fibrillationflutter/

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4
Q

What is the general dose of Diltiazem for rate control of patients experiencing stable atrial fibrillation in an acute setting (first, second and third doses)?

A
  1. 0.25 mg/kg (average 20 mg) IV over 2 min, may repeat after 15 minutes (if HR > 100 bpm) with 0.35 mg/kg (average 25 mg), then 5-15 mg/hr continuous IV drip infusion (for no longer than 24 hours);

https://www.acc.org/tools-and-practice-support/clinical-toolkits/atrial-fibrillation-afib/rate-rhythm-dosing-table/diltiazem

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5
Q

ED cardioversion of stable, new-onset atrial fibrillation is appropriate in a select population, notably, when the onset is < ___ hours. The pooled literature suggests a thromboembolism rate of < ___ under these conditions.

A
  1. 48;

Note: Rosen’s Emergency Medicine allows for cardioversion when onset is < 72 hours.

  • https://foamcast.org/2014/09/21/episode-15-atrial-fibrillationflutter/*
    2. 0.8;

Major Memory System: A patient is cardioverted the safe way, in a Safeway (08 → 0.8).

Cohn BG, Keim SM, Yealy DM. Is Emergency Department Cardioversion of Recent-onset Atrial Fibrillation Safe and Effective? J Emerg Med. 2013;45(1):117–27.

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6
Q

Atrial fibrillation featuring evidence of ___, (e.g., wide, polymorphic atrial fibrillation) should not be treated with AV nodal blocking agents (e.g. adenosine, beta-blockers, calcium-channel blockers, digoxin). Consider synchronized cardioversion or procainamide instead.

A
  1. Pre-excitation syndrome (e.g. WPW);

Note: Blocking the AV node causes unopposed electrical conduction down the accessory pathway. Additionally, the accessory pathway in WPW responds paradoxically to the AV nodal blocking agents by further decreasing its refractory time.

https://foamcast.org/2014/09/21/episode-15-atrial-fibrillationflutter/

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7
Q

_(cardiac rhythm)_ is characterized by an irregular narrow complex tachycardia with p waves of at least three morphologies. As this rhythm can be difficult to differentiate, it is advisable to look for p waves in multiple leads, particularly ___.

A
  1. Multifocal Atrial Tachycardia;
  2. V2;
    * https://foamcast.org/2014/09/21/episode-15-atrial-fibrillationflutter/*
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8
Q

Generally speaking, how should multifocal atrial tachycardia be treated? What treatment should generally be avoided?

A
  1. Treat the underlying cause;
  2. Cardioversion;
    * https://foamcast.org/2014/09/21/episode-15-atrial-fibrillationflutter/*
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9
Q

___ is caused by chaotic, disorderly firing from a second focus within the atria, resulting in uncoordinated atrial contractions. On ECG, there are irregularly irregular narrow QRS complexes. In addition, no discernible ___ waves are noted, rather ___ waves are seen.

A
  1. Atrial fibrillation;
  2. P;
  3. Fibrillatory (aka f waves);
    * https://foamcast.org/2014/09/21/episode-15-atrial-fibrillationflutter/*
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10
Q

The underlying mechanism of WPW involves an ___ between the atria and the ventricles. It is a type of ___ syndrome.

A
  1. Accessory pathway (aka accessory electrical conduction pathway);
  2. Pre-excitation;
    * https://en.wikipedia.org/wiki/Wolff–Parkinson–White_syndrome*
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11
Q

Individuals with WPW have an accessory pathway that communicates between the atria and the ventricles, in addition to the AV node. This accessory pathway is known as the ___.

A
  1. Bundle of Kent;

https://en.wikipedia.org/wiki/Wolff–Parkinson–White_syndrome

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12
Q

If a person with WPW experiences episodes of ___, the ECG shows a rapid polymorphic wide-complex tachycardia (without torsades de pointes). This combination of atrial fibrillation and WPW is considered dangerous, and most antiarrhythmic drugs are contraindicated.

A
  1. Atrial Fibrillation;

https://en.wikipedia.org/wiki/Wolff–Parkinson–White_syndrome

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13
Q

What is the definitive treatment for WPW?

A
  1. Destruction of the abnormal electrical pathway by radiofrequency catheter ablation;

https://en.wikipedia.org/wiki/Wolff–Parkinson–White_syndrome

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14
Q

Name the three key ECG features of pre-excitation syndrome (e.g. WPW).

A
  1. PR Interval < 0.12 sec;
  2. QRS Interval >= 0.12 sec;
  3. Delta Wave;
    * https://ecgwaves.com/topic/pre-excitation-avrt-wolff-parkinson-white-wpw-syndrome/*
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15
Q

Pre-excitation causes secondary ___ changes. The _(same as 1.)_ segment is directed ___ to the ___ wave.

A
  1. ST-T;
  2. Oppositely;
  3. Delta;
    * https://ecgwaves.com/topic/pre-excitation-avrt-wolff-parkinson-white-wpw-syndrome/*
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16
Q

During pre-excitation, a positive delta wave will result in a ___ deflected ST-T segment.

A
  1. Negatively;

Note: ST segment depression and T wave inversion are also typical during pre-excitation.

https://ecgwaves.com/topic/pre-excitation-avrt-wolff-parkinson-white-wpw-syndrome/

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17
Q

Patients with WPW can be asymptomatic or may present with severe tachydysrhythmias. The most common presenting dysrhythmia is ___ (70-80%) and second is ___ (10-30%).

A
  1. AVRT;
  2. Atrial Fibrillation;
    * https://foamcast.org/2014/09/21/episode-15-atrial-fibrillationflutter/*
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18
Q

In WPW-related tachydysrhythmias, the patient can conduct ___ (down the AV node and back up the accessory pathway), ___ (down the accessory pathway and up the AV node) or in both directions.

A
  1. Orthodromically;
  2. Antidromically;
    * https://foamcast.org/2014/09/21/episode-15-atrial-fibrillationflutter/*
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19
Q

Describe the “Modified Valsalva Maneuver” utilized for conversion of stable SVT to Sinus Rhythm in the REVERT trial.

A
  1. In a semi-recumbent position, patients produce 40 mm Hg pressure (which may be approximated in the field by blowing hard enough into a 10 mL syringe to cause the plunger to move) for 15 seconds, and are then repositioned supine with a passive leg raise to 45 degrees immediately after the valsalva strain;

https://rebelem.com/the-revert-trial-a-modified-valsalva-maneuver-to-convert-svt/

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20
Q

With regards to the conversion of stable SVT to Sinus Rhythm one minute after intervention, The REVERT trial demonstrated the efficacy of the Standard Valsalva Maneuver at ___% and the Modified Valsalva Maneuver at ___%.

A
  1. 17;
  2. 43;

Major Memory System: The passive leg raise utilized during a Modified Valsalva Maneuver causes a worm (43) to be squeezed from the patient’s dick (17).

https://rebelem.com/the-revert-trial-a-modified-valsalva-maneuver-to-convert-svt/

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21
Q

___ is a broad term which refers to tachycardias originating above the ventricles, including the regular rhythms of sinus tachycardia, atrial tachycardia, AVNRT, AVRT, and the irregular rhythms of multifocal atrial tachycardia, atrial fibrillation, and some forms of atrial flutter. ___ is a term that is widely considered a synonym, but which generally excludes _(rhythm)_ and focuses primarily on _(rhythm)_ and _(rhythm)_.

A
  1. Supraventricular Tachycardia (SVT);

  • https://foamcast.org/2015/09/09/episode-34-tachyarrhythmias/*
  • https://www.hopkinsmedicine.org/heart_vascular_institute/conditions_treatments/conditions/supraventricular_tachycardia.html*
    2. Paroxysmal Supraventricular Tachycardia (PSVT);
    3. Sinus Tachycardia;
    4. AVRT;
    5. AVNRT;
  • http://hqmeded-ecg.blogspot.com/2017/08/what-happens-when-you-give-adenosine-to.html*
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22
Q

The Single Syringe Administration of Diluted Adenosine trial proved that when diluted in ___ mL of 0.9% Sodium Chloride solution, the use of prescribed doses of Adenosine for the conversion of stable SVT in adults is at least as effective as traditional techniques (Adenosine flushed afterwards with normal saline).

A
  1. 20;

  • https://foamcast.org/2015/09/09/episode-34-tachyarrhythmias/*
  • McDowell M, Mokszycki R, Greenberg A, et al. Single Syringe Administration of Diluted Adenosine. Acad Emerg Med. 2019 Oct 30. [ePub ahead of print]. [PMID 31665806]*
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23
Q

It is typical for Atrial Flutter to produce ___ flutter waves in V1 that preceed the QRS complexes. It is important to remember that sinus P waves are ___ in V1. This distinction can be used to help differentiate what may appear at first glance to be Sinus Tachycardia from what is actually Atrial Flutter.

A
  1. Upright (aka positive);
  2. Biphasic;

Note: Atrial Flutter with 2:1 conduction is a PSVT mimic.

http://hqmeded-ecg.blogspot.com/2017/08/what-happens-when-you-give-adenosine-to.html

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24
Q

Adenosine’s mechanism of action can be thought of as a “temporary paralyzing” of ___ tissue.

A
  1. Supraventricular;

https://www.jems.com/2012/09/07/patient-cardiac-rhythm-important-ems-ade/

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25
Q

Pharmacologically, adenosine ___-polarizes the cell by stimulating an inward _(electrolyte)_ current and temporarily inhibiting _(electrolyte)_ migration. In doing so, the pacemaker activity of the SA node, spontaneous atrial activity and conduction through the AV node are dramatically slowed or temporarily stopped.

A
  1. Hyper;
  2. Potassium;
  3. Calcium;
    * https://www.jems.com/2012/09/07/patient-cardiac-rhythm-important-ems-ade/*
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26
Q

According to JEMS, adenosine has ___ effects on accessory pathways, such as those seen in WPW syndrome. The Pierce County PCPs, however, state that adenosine ___ inhibit reentry pathways.

A
  1. No;

  • https://www.jems.com/2012/09/07/patient-cardiac-rhythm-important-ems-ade/*
    2. May;
  • Pierce Co. EMS Procedures and PCPs, June 2019, Appendix O, Adenosine*
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27
Q

Under what respiratory condition should adenosine be withheld from a patient with a history of asthma or COPD?

A
  1. Wheezing present prior to adenosine administration;

Note: Adenosine may cause bronchospasm.

https://www.jems.com/2012/09/07/patient-cardiac-rhythm-important-ems-ade/

28
Q

Generally speaking, ___ is the drug of choice for PSVT in pregnant patients.

A
  1. Adenosine;

https://www.jems.com/2012/09/07/patient-cardiac-rhythm-important-ems-ade/

29
Q

What three tachycardic rhythms does JEMS indicate are absolute contraindications to the use of adenosine?

A
  1. Atrial flutter;
  2. Atrial fibrillation;
  3. Sinus tachycardia;
    * https://www.jems.com/2012/09/07/patient-cardiac-rhythm-important-ems-ade/*
30
Q

What rhythms can adenosine safely be used to differentiate (i.e., as a diagnostic aid)? What other two rhythms may be inadvertently differentiated with Adenosine, but should never be treated with the drug?

A
  1. 1SVT with abberancy from VT;

Note: 2Adenosine can convert regular, monomorphic VT to a sinus rhythm, especially in young people, so it does not definitively rule-out VT if conversion of a wide-complex tachycardia is achieved. 1 However, there is a 36-fold increase in the likelihood of a supraventricular origin if the wide complex tachycardia converts to sinus with adenosine and a nine-fold increased likelihood of VT if it doesn’t respond to escalating doses of adenosine.

  • 1- https://www.jems.com/articles/print/volume-37/issue-9/patient-care/patient-cardiac-rhythm-important-ems-ade.html*
  • 2- https://lifeinthefastlane.com/cardiovascular-curveball-013/*
    2. Atrial Fibrillation;
    3. Atrial Flutter;

Note: Adenosine reveals flutter waves by eliminating QRS complexes through AV nodal blockade.

Note 2: Per https://elearning.heart.org/scorm/launchsco/28828621/1296, the AHA states “Although it [adenosine] doesn’t terminate Atrial Flutter or Atrial Fibrillation, it will slow AV conduction, allowing for identification of flutter or fibrillation waves, thereby allowing confirmation of the underlying tachyarrhythmia.”

  • https://www.jems.com/2012/09/07/patient-cardiac-rhythm-important-ems-ade/*
  • http://hqmeded-ecg.blogspot.com/2017/08/what-happens-when-you-give-adenosine-to.html*
31
Q

Adenosine is categorized as a US FDA pregnancy category ___ drug.

A
  1. C;

- https://www.drugs.com/pregnancy/adenosine.html

Note: Animal reproduction studies have shown an adverse effect on the fetus and there are no adequate and well-controlled studies in humans, but potential benefits may warrant use of the drug in pregnant women despite potential risks. Use is recommended only if clearly needed and the benefit outweighs the risk.

32
Q

Name the three supraventricular rhythms which represent absolute contraindications to the use of adenosine.

A
  1. Atrial fibrillation;
  2. Atrail flutter;
  3. Sinus tachycardia;

Note: According to the AHA, “

https://www.jems.com/articles/print/volume-37/issue-9/patient-care/patient-cardiac-rhythm-important-ems-ade.html

33
Q

In cases for which there’s any chance that sinus tachycardia is the etiology of a patient’s elevated heart rate, ___ should be administered prior to considering adenosine.

A

1.A fluid bolus (Jems recommends a rapid fluid bolus of 250 cc);

Note: Any slowing by just a few beats per minute (rather than a dramatic conversion to a normal sinus rhythm) confirms the diagnosis of sinus tachycardia due to volume depletion.

https://www.jems.com/articles/print/volume-37/issue-9/patient-care/patient-cardiac-rhythm-important-ems-ade.html

34
Q

Adenosine slows or blocks ___grade (atrial to ventricular) conduction through the AV node but doesn’t affect ___ or ___ tracts like those seen in ___ syndrome.

A
  1. Ante;
  2. Accessory;
  3. Bypass;
  4. WPW;

Note: Because of this, adenosine can be dangerous when given to patients with atrial fibrillation, especially if they have a bypass track. Numerous reports show patients degenerating into rapid atrial fibrillation with rates at 250 or greater and becoming hemodynamically unstable.

https://www.jems.com/articles/print/volume-37/issue-9/patient-care/patient-cardiac-rhythm-important-ems-ade.html

35
Q

Adenosine can convert relatively stable Atrial Flutter with 2:1 conduction and a heart rate of 150 to ___ conduction with a heart rate of ___ and cause rapid clinical decompensation.

A
  1. 1:1;
  2. 300;

Note: Atrial Flutter with 2:1 conduction is a PSVT mimic.

https://www.jems.com/articles/print/volume-37/issue-9/patient-care/patient-cardiac-rhythm-important-ems-ade.html

36
Q

Investigators found that 6 mg of adenosine converted ~___% of patients and that 12 mg converted ~___% of patients without an increase in toxicity.

A
  1. 60 (62.3%);
  2. 90 (91.4%);

Major Memory System: Don Hodges (60) drinks booze (90) until his heart stops.

https://www.jems.com/articles/print/volume-37/issue-9/patient-care/patient-cardiac-rhythm-important-ems-ade.html

37
Q

Atrial Flutter does not use the ___ for part of its re-entrant loop, as do AVNRT and AVRT.

A
  1. AV node;

http://hqmeded-ecg.blogspot.com/2017/08/what-happens-when-you-give-adenosine-to.html

38
Q

Patients with atrial flutter often alternate between fib and flutter, and produce thrombi during episodes of ___. Hence, cardioversion of atrial flutter may involve a risk of thromboembolism if the onset of atrial flutter is not within ___ hours of ED presentation.

A
  1. Fibrillation;
  2. 48;
    * http://hqmeded-ecg.blogspot.com/2017/08/what-happens-when-you-give-adenosine-to.html*
39
Q

A ___ is a rare benign tumor of the heart. These growths are the most common primary cardiac tumor in adults, and are most commonly found within the ___ atrium.

A
  1. Myxoma;
  2. Left;
    * https://en.wikipedia.org/wiki/Cardiac_myxoma*
40
Q

Rheumatic heart disease is a condition in which permanent damage to _(structures)_ is caused by _(disease)_. This process generally begins with the development of _(disease)_ due to ___ bacteria.

A
  1. Heart valves;
  2. Rheumatic fever;
  3. Strep throat;
  4. Streptococcal (there are many different types);
    * https://chw.org/medical-care/herma-heart/conditions/rheumatic-heart-disease*
41
Q

Name the two most common alterable risk factors for Atrial Fibrillation.

A
  1. Hypertension;
  2. Valvular heart disease (e.g., rheumatic heart disease);
    * https://en.wikipedia.org/wiki/Atrial_fibrillation*
42
Q

Atrial Fibrillation is often treated with medications to slow the heart rate to a near normal range (known as _(therapy)_) or to convert the rhythm to normal sinus rhythm (known as _(therapy)_).

A
  1. Rate control;
  2. Rhythm control;
    * https://en.wikipedia.org/wiki/Atrial_fibrillation*
43
Q

Most persons diagnosed with Atrial Fibrillation are at high risk of _(adverse event)_, and are prescribed ___ medications.

A
  1. Stroke (aka CVA);
  2. Anti-clotting (aka anticoagulants);
    * https://en.wikipedia.org/wiki/Atrial_fibrillation*
44
Q

___ is the most common serious abnormal heart rhythm. Studies performed in 2014 demonstrated that it affects about ___% of the population of the developed world.

A
  1. Atrial Fibrillation;
  2. 2.5;

Major Memory System: Bumba performs vigorous anal (25 → 2.5) on GamGam, their hearts palpitating.

https://en.wikipedia.org/wiki/Atrial_fibrillation

45
Q

The risk of a blood clot forming in the left atrial chamber of the heart, breaking off, and then traveling in the bloodstream can be assessed using the ___ or ___ scores.

A
  1. CHADS2;
  2. CHA2DS2-VASc;
    * https://en.wikipedia.org/wiki/Atrial_fibrillation*
46
Q

Many of the symptoms associated with uncontrolled Atrial Fibrillation are a menifestation of ___ due to the reduced cardiac output.

A
  1. Congestive Heart Failure (CHF);

https://en.wikipedia.org/wiki/Atrial_fibrillation

47
Q

Consumption of ___ beverages does not appear to be associated with Atrial Fibrillation, but excessive consumption of ___ beverages is linked to the disease.

A
  1. Caffeinated;
  2. Alcoholic;
    * https://en.wikipedia.org/wiki/Atrial_fibrillation*
48
Q

In Atrial Fibrillation, the normal regular electrical impulses generated by the sinoatrial node in the right atrium of the heart are overwhelmed by disorganized electrical impulses usually originating in the roots of the ___.

A
  1. Pulmonary veins;

https://en.wikipedia.org/wiki/Atrial_fibrillation

49
Q

The primary pathologic change seen in Atrial Fibrillation is progressive ___ of the atria due primarily to atrial ___.

A
  1. Fibrosis;
  2. Dilation;
    * https://en.wikipedia.org/wiki/Atrial_fibrillation*
50
Q

What are the common abbreviations for Atrial Fibrillation and Atrial Flutter, respectively?

A
  1. AF;
  2. AFL;
    * https://en.wikipedia.org/wiki/Atrial_fibrillation*
51
Q

A ___ is a wearable ambulatory heart monitor that continuously monitors the heart rate and heart rhythm for a short duration, typically 24 hours.

A
  1. Holter monitor;

https://en.wikipedia.org/wiki/Atrial_fibrillation

52
Q

The ___, located in the left atrium, is the place where a blood clot forms in more than 90% of cases in non-valvular (or non-rheumatic) atrial fibrillation.

A
  1. Left atrial appendage;

https://en.wikipedia.org/wiki/Atrial_fibrillation

53
Q

Atrial Fibrillation Classification System:

___ - Only one diagnosed episode;

___ - Recurrent episodes that stop on their own in less than seven days;

___ - Recurrent episodes that last more than seven days;

___ - An ongoing long-term episode;

A
  1. First detected;
  2. Paroxysmal;
  3. Persistent;
  4. Permanent;

Note: Episodes that last less than 30 seconds are not considered in this classification system.

https://en.wikipedia.org/wiki/Atrial_fibrillation

54
Q

About ___% of people with Atrial Fibrillation have permanent AF, while ___% have paroxysmal AF and ___% have persistent AF.

A
  1. 50;
  2. 25;
  3. 25;

Major Memory System: Lassie (50) swims down the Nile (25) in an O’Neill (25) wetsuit, her heart palpitating from fear of piranhas.

https://en.wikipedia.org/wiki/Atrial_fibrillation

55
Q

The main goals of treatment of Atrial Fibrillation are to prevent ___ instability and ___. ___ or ___ control are used to achieve the former, whereas ___ is used to decrease the risk of the latter.

A
  1. Circulatory;
  2. Stroke;
  3. Rate;
  4. Rhythm;
  5. Anticoagulation;
    * https://en.wikipedia.org/wiki/Atrial_fibrillation*
56
Q

What does DOAC stand for, with regard to anticoagulation?

A

DOAC

D - Direct

O - Oral

A - Anti

C - Coagulant

https://en.wikipedia.org/wiki/Atrial_fibrillation

57
Q

Name four (of six listed) anticoagulants commonly used for stroke prevention during Atrial Fibrillation.

A
  1. Warfarin (Coumadin);
  2. Heparin (and other drugs ending with the suffix -parin);
  3. Apixaban;
  4. Rivaroxaban;
  5. Edoxaban;
  6. Dabigatran;

Memory Aid for #s 4-6: A pixy flies by a river on the planet Endor, when suddenly da big train whooshes by.

https://en.wikipedia.org/wiki/Atrial_fibrillation

58
Q

What does NOAC stand for, with regard to anticoagulation?

A

NOAC

N - Novel

O - Oral

A - Anti

C - Coagulant

https://www.pharmacytimes.com/contributor/sean-kane-pharmd/2016/09/noac-doac-or-tsoac-what-should-we-call-novel-oral-anticoagulants

59
Q

A(n) ___ is a rare tumor of adrenal gland tissue. It results in the release of too much epinephrine and norepinephrine, hormones that control heart rate, metabolism, and blood pressure.

A
  1. Pheochromocytoma;

https://medlineplus.gov/ency/article/000340.htm

60
Q

With regards to AF, studies suggest that ___ control is more important in the acute setting, whereas ___ control is more important in the chronic phase.

A
  1. Rhythm;
  2. Rate;
    * https://en.wikipedia.org/wiki/Atrial_fibrillation*
61
Q

In the setting of Atrial Fibrillation with Rapid Ventricular Response, intravenous ___ significantly increases the chances of successful rate and rhythm control in the urgent setting without major side-effects.

A
  1. Magnesium;

https://en.wikipedia.org/wiki/Atrial_fibrillation

62
Q

Rate control of Atrial Fibrillation is achieved with medications that work by increasing the degree of block at the level of the ___, decreasing the number of impulses that conduct into the ___.

A
  1. AV node;
  2. Ventricles;
    * https://en.wikipedia.org/wiki/Atrial_fibrillation*
63
Q

Rate control of Atrial Fibrillation can generally be accomplished with ___ blockers, ___ blockers and cardiac ___. In addition to these agents, _(medication)_ has some AV node blocking effects and can be used in individuals when other agents are contraindicated or ineffective (particularly fue to hypotension).

A
  1. Beta (preferably the “cardioselective” beta blockers such as metoprolol);
  2. Calcium channel (non-dihydropyridine, e.g., diltiazem or verapamil);
  3. Glycosides (e.g., digoxin);
  4. Amiodarone;
    * https://en.wikipedia.org/wiki/Atrial_fibrillation*
64
Q

Name two (of four listed) drugs which may be used to chemically cardiovert Atrial Fibrillation.

A
  1. Amiodarone;
  2. Procainamide (especially in pre-excited atrial fibrillation);
  3. Propafenone;
  4. Flecainide;
    * https://en.wikipedia.org/wiki/Atrial_fibrillation*
65
Q

Name two types of ablation which may be considered for rate control of Atrial Fibrillation.

A
  1. Radiofrequency;
  2. Cryoablation;
    * https://en.wikipedia.org/wiki/Atrial_fibrillation*