Sz Structure

Question 1

DA hypothesis

Answer 1

DA hypothesis
E.g. Falkai
However, Farde, must be other factors

Simplistic - genetic explanation
E.g. Gottesman and shields

However, useful applications
E.g. Antipsychotics target DA

Testable
E.g. Lindstroem PET scan Farde and Falkai
However, not necessarily available to all psych - limiting applicability

Conc -> despite simplistic not saying only exp clearly identifying a possible factor

Question 2

Genetic explanation

Answer 2

Basic idea - family etc
E.g. Gottesman and shields

However too simplistic
E.g. Cog explanation

However, testable
E.g. Illustrative

Question 3

Cognitive explanation

Answer 3

Sz due to faulty cognitive processes, lack metarepresentation
E.g. Knoblich

However, simplistic
Genetic studies
E.g. Gottesman and shields

However, applications good
E.g. CBT as a treatment for Sz

Another criticism is lacks testability as although we can test to see if someone has these, we cannot physically see the processes going on in the brain

Question 4

Biological treatments

Answer 4

Antipsychotics
E.g. Leucht

Typical and Atypical
E.g. Hartling

Practical
E.g. Illustrative

Question 5

Psychological treatments

Answer 5

CBT

Talking therapy, change faulty cognition by:
Identifying what they are and what caused them
Challenging them
Replacing them with healthy ones

Clearly cognitive component
Normalising, belief modification, focussing and reattribution
E.g. Kingdon 35/65 after 5 years

Less practical than drugs
E.g. 6 weeks - 6 months ting