Systematic ECG Analysis Flashcards
what are the four methods of measuring HR on an ECG?
1) Six Second Method
2) Sequence Method
3) Small Square Method
4) Ruler Method
what is the 6 second method?
of QRS complexes or P waves in a 6 second period x 10 = bpm
what is the sequence method?
choose R wave that falls on a dark line, number the next 6 dark vertical lines on the paper 300, 150, 100, 75, 60, 50.
what is the small square method?
count # of small boxes between P or R waves, divide by 1500 = bpm
what is the ruler method?
use ECG ruler. place 0 at top of R wave, count 3 waves over from first = bpm
what does it mean if a rhythm strip has no P waves?
no atrial depolarization = no contraction = no atrial kick = loss of 15-30% of preload = decreased CO
what does ST depression or elevation mean?
ST depression = myocardial ischemia
ST elevation = myocardial injury or infarction
how do you determine ST elevation or depression?
1) identify isoelectric line
2) identify J point
3) measure 0.06 seconds (1.5 boxes) to the right of the J point
4) if ST segment is above isoelectric line, it is considered elevated and vice versa
what is considered significant for ST elevation/depression?
> 1mm (1 small box) from isoelectric line
why does the QT interval vary with HR?
high HR shortens QT interval, low HR lengthens QT interval
QTc
QT interval corrected for HR
what could occur if the QT interval lengthens?
life threatening dysrhythmias, as the relative refractory period is lengthening therefore increasing the time where a cell is vulnerable to a strong stimulus
what must you do when stating the rhythm?
state the underlying rhythm with all elements
ie) regular sinus rhythm with PVCs
what do you state when you identify the site of impulse formation?
1) what pacemaker site is responsible for the ECG rhythm? SA node, atrial tissue, junctional tissue, ventricles. or a combo.
2) what is the rate of the ECG rhythm, too fast or too slow?
what are implications for O2 supply and demand and CO if the ventricular rate is TOO SLOW
potential for decrease in CO d/t slow rate.
what are implications for O2 supply and demand and CO if the ventricular rate is TOO FAST
- decrease in VFT = decrease in preload and CO
- decrease in coronary artery perfusion w/ potential for myocardial ischemia (coronary arteries fill during diastole, so if heart is always in systole, arteries will have inadequate time to perfuse)
- increase in myocardial O2 demand with potential for myocardial ischemia if O2 supply is not adequate
what are implications for O2 supply and demand and CO if there is NO P WAVE
loss of atrial kick = decreased preload and CO
what are implications for O2 supply and demand and CO if there are ST CHANGES
myocardial ischemia/injury and potential for decrease in contractility, leading to decrease in CO
interventions
- HR too slow: speed it up using drugs or pacemaker
- HR too fast: slow it down using drugs or electrical conversion
- no P wave: chemically or electrically convert rhythm
- ST changes: minimize/prevent ischemia and injury to myocardium (MONA, cath lab)
what are the 13 steps of analyzing an ECG strip?
1) HR
2) rhythm
3) P wave
4) PR interval
5) QRS complex
6) ST segment
7) T wave
8) QT interval
9) identify rhythm
10) site of impulse formation/conduction problem
11) causes
12) implications for O2 supply and demand
13) interventions