Systematic ECG Analysis Flashcards

1
Q

what are the four methods of measuring HR on an ECG?

A

1) Six Second Method
2) Sequence Method
3) Small Square Method
4) Ruler Method

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2
Q

what is the 6 second method?

A

of QRS complexes or P waves in a 6 second period x 10 = bpm

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3
Q

what is the sequence method?

A

choose R wave that falls on a dark line, number the next 6 dark vertical lines on the paper 300, 150, 100, 75, 60, 50.

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4
Q

what is the small square method?

A

count # of small boxes between P or R waves, divide by 1500 = bpm

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5
Q

what is the ruler method?

A

use ECG ruler. place 0 at top of R wave, count 3 waves over from first = bpm

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6
Q

what does it mean if a rhythm strip has no P waves?

A

no atrial depolarization = no contraction = no atrial kick = loss of 15-30% of preload = decreased CO

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7
Q

what does ST depression or elevation mean?

A

ST depression = myocardial ischemia
ST elevation = myocardial injury or infarction

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8
Q

how do you determine ST elevation or depression?

A

1) identify isoelectric line
2) identify J point
3) measure 0.06 seconds (1.5 boxes) to the right of the J point
4) if ST segment is above isoelectric line, it is considered elevated and vice versa

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9
Q

what is considered significant for ST elevation/depression?

A

> 1mm (1 small box) from isoelectric line

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10
Q

why does the QT interval vary with HR?

A

high HR shortens QT interval, low HR lengthens QT interval

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11
Q

QTc

A

QT interval corrected for HR

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12
Q

what could occur if the QT interval lengthens?

A

life threatening dysrhythmias, as the relative refractory period is lengthening therefore increasing the time where a cell is vulnerable to a strong stimulus

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13
Q

what must you do when stating the rhythm?

A

state the underlying rhythm with all elements
ie) regular sinus rhythm with PVCs

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14
Q

what do you state when you identify the site of impulse formation?

A

1) what pacemaker site is responsible for the ECG rhythm? SA node, atrial tissue, junctional tissue, ventricles. or a combo.

2) what is the rate of the ECG rhythm, too fast or too slow?

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15
Q

what are implications for O2 supply and demand and CO if the ventricular rate is TOO SLOW

A

potential for decrease in CO d/t slow rate.

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16
Q

what are implications for O2 supply and demand and CO if the ventricular rate is TOO FAST

A
  • decrease in VFT = decrease in preload and CO
  • decrease in coronary artery perfusion w/ potential for myocardial ischemia (coronary arteries fill during diastole, so if heart is always in systole, arteries will have inadequate time to perfuse)
  • increase in myocardial O2 demand with potential for myocardial ischemia if O2 supply is not adequate
17
Q

what are implications for O2 supply and demand and CO if there is NO P WAVE

A

loss of atrial kick = decreased preload and CO

18
Q

what are implications for O2 supply and demand and CO if there are ST CHANGES

A

myocardial ischemia/injury and potential for decrease in contractility, leading to decrease in CO

19
Q

interventions

A
  • HR too slow: speed it up using drugs or pacemaker
  • HR too fast: slow it down using drugs or electrical conversion
  • no P wave: chemically or electrically convert rhythm
  • ST changes: minimize/prevent ischemia and injury to myocardium (MONA, cath lab)
20
Q

what are the 13 steps of analyzing an ECG strip?

A

1) HR
2) rhythm
3) P wave
4) PR interval
5) QRS complex
6) ST segment
7) T wave
8) QT interval
9) identify rhythm
10) site of impulse formation/conduction problem
11) causes
12) implications for O2 supply and demand
13) interventions