Syncope Flashcards

1
Q

most common cause of syncope in young and middle aged populations

A

neurocardiogenic (vasovagal or vasodepressor) syncope

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2
Q

most common causes of syncope in elderly

A

LV outflow obstruction or arrhythmias

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3
Q

1st steps to clarify etiology of syncope

A

history, physical exam, medication list.

May need orthostatics, EKG, TTE, and stress test to rule out structural heart dx

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4
Q

PTs with unclear etiology of syncope should get:

A

ambulatory loop recording with holter monitor, mobile telemetry, or implantable loop recorder.

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5
Q

what does upright tilt testing do?

A

limited sensitivity and specificity for vasovagal syncope and doesn’t help guide treatment. ALso hemodynamic response during test doesn’t always reproduce the actual syncopal mechanism.

Only use for recurrent unexplained syncope when cardiac causes are enegative.

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6
Q

initial assessment of syncope and high risk features that merit further evaluation if there is 1 or more present:

A

abnormal ECG (new changes)
history of structural heart dx or heart failure
systolic blood pressure <90 at triage
associated SOB during syncope
hematocrit<30%
advanced age with cardiovascular comorbities
family history of sudden cardiac death

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7
Q

neurocardiogenic syncope is also known as

A

vasovagal syncope

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8
Q

neurocardiogenic syncope clinical features

A

: diarphoresis, nausea, generalized warmth, (signs of an autonomic prodrome)

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9
Q

signs suggestive of sinister etiology for syncope on physical exam:

A

chest pain, abnormal vital signs, abnormal physical exam or signs of injury

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10
Q

management of neurocardiogenic syncope

A

if they have clear vasovagal symptoms, they ned to have reassured if they returned to baseline and discharged as outpatient follow up.

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11
Q

management for sinister syncope

A

needs admission for telemetry if high suspicion of syncope due to cardiac arrhythmia.

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12
Q

Sinister syncope features on clinical history:

A

syncope while sitting or lying supine, exertional syncope, absence of prodrome

ECG abnormalities, prior history of CAD dx

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13
Q

when do we get outpatient ambulatory cardiac monitoring (holter)?

A

syncope is not entirely consistent with neurocardiogenic syncope but there’s a low risk for VT.

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14
Q

When do we get brain imaging and EEG?

A

not for syncope but for diagnosing seizure disorder cause.

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15
Q

twitching while someone passes out

A

vasovagal syncope can have brief tonic clonic movements due to transient global cerebral hypoperfusion but don’t represent a seizure disorder.

No post ictal period after syncope.

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16
Q

who gets admitted to the hospital for telemetry? (indication)

A

seriously ill pts who have may life threatening cardiac arrhtyhmias

17
Q

ambulatory (Holter) electrocardiography (Continuous or intermittent recorders) indications

A

pts with frequent episodes with syncope >1 /week and can store up to 24-48 hrs of data

18
Q

external loop recorder indications

A

syncope free interval >1 /month

19
Q

implantable loop recorder indications

A

recurrent infrequent <1/month syncope to identifiable cause

can provide monitoring for months to years

20
Q

syncope triggered by prolonged standing, emotional distress, painful stimuli and see prodromal symptoms of nausea, warmth, and diaphoresis

A

vasovagal or neurally mediated syncope

21
Q

syncope triggered cough micturation and defecation

A

situational syncope

22
Q

syncope with postural changes in heart rate/blood pressure after standing suddenly

A

orthostatic syncope

23
Q

syncope after exertion or during exercise

A

aortic stenosis,
HCM
anomalous coronary arteries

24
Q

syncope with prior history of CAD, MI, cardiomyopathy, or low EF

A

ventricular arrhythmias

25
Q

syncope with sinus pauses, Increased PR or QRS duration

A

sick sinus syndrome, bradyarrhythmias, AV block

26
Q

syncope with hypokalemia, hypomagnesemia, medications causing increased QT prolongation

A

Torsades de pointes (acquired long QT syndrome)

27
Q

syncope and family history of sudden cardiac death, Increased QT interval, syncope with triggers (exercise, startle, and sleeping)

A

congenital long QT syndrome.

28
Q

diagnosis of postural hypotension

A

also called orthostatic hypotension

within 2-5 minutes of standing from supine position
drop in SBP>20 or drop in DBP>10

29
Q

Cardiac myxoma is a

A

benign neoplasm and 80% located in left atrium

30
Q

clinical features of a cardiac myxoma

A

position dependent mitral valve obstruction
see mid-diastolic murmur
dyspnea and lightheadedness and syncope

embolization of tumor fragments (stroke)

see constitutional symptoms of weight loss

31
Q

diagnosis and management of cardiac myxoma

A

TTE

prompt surgical resection- to avoid embolization and risk sudden death

32
Q

transient vision loss (amaurosis fugax) and see unintentional weight loss and diastolic murmur

A

cardiac myxoma

see fatigue and mimic mitral valve dx with early diastolic sound.

33
Q

carotid sinus hypersensitivity syncope

A

pressure on the carotid sinus by a tight collar nad neck movements while getting dressed in the AM

shaving the neck

34
Q

diagnosis of carotid sinus hypersensitivity

A

carotid sinus massage and passing out or see asystoe for 3 seconds or there’s a fall in SBP>50 mmHg

NO additional work up is needed for diagnosing.

35
Q

DO NOT do a carotid sinus massage and should be AVOIDED in pts who have

A

should avoid carotid sinus massage with ipsilateral carotid artery stenosis or carotid bruits or hx of TIA or stroke in the last three months

36
Q

carotid sinus hypersensitivity treatment

A

permanent pacing