Syncope Flashcards
Extreme lightheadedness, visual sensations, variable degrees of altered consciousness without complete LOC
Near Syncope
transient, complete LOC, inability to maintain postural tone with rapid and complete recovery
syncope
what is considered good syncope?
- several episodes of syncope since their 20-30s
- prodromal sx present before event (lightheaded, hearing loss, room closing in, etc)
- autonomic sx after event (fatigue, pale, sweaty, loss of urine or stool)
- no major injuries
- recent illness (Dehydration)
what is considered the bad syncope?
- Single episode of syncope
- “i just went out”
- “i found myself on the floor”
- syncope with exercise
- syncope lying down
- sustained injury (fractures, contusion, etc.)
- known heart disease
Cardiac-Arrhythmic conditions that are reasons for admissions?
- inheritable CV conditions predisposing to arrythmias (long-QT pattern, brugada pattern)
- sinus bradycardia < 40 beats/min (symptomatic) or pauses > 3 sec
- mobitz II or 2:1 second-degree or third degree atrioventricular block
- sustained supraventricular tachycardia
- sustained ventricular tachycardia
- pacemaker (ICD) malfunction with cardiac pauses
what are predictors of cardiac syncope?
- Palpitations preceding syncope
- quick event, minimal or no symptoms, amestic
- known heart disease; abnormal ECG
- syncope while lying down
- syncope during effort
- due to a mutation in the cardiac sodium channel gene (sodium channelopathy)
- abnormal ECG along with… one or more of the following
- documented ventricular fibrillation (VFO or polymorphic ventricular tachycardia (VT)
- family history of sudden cardiac death < 45 years old
- coveted-type ECGs in family members
- syncope
Brugada
what ECG abnormality is diagnostic or seen brugada syndrome?
- Type 1: Coved type ST- segment elevation
- Type 2 &3 : saddle-back type ST-segment elevation
what are preventable treatment options of brugada
implantable cardioverter defibrillator (ICD)
pacemaker, or specific medicines are available
what are some etiologies of long QT syndrome?
- medications (amiodarone, TCAs, SSRIs, antibiotics, metoclopramide, haloperidol, ondansetron)
- genetic: cardiac ion channel mutation (Na+, K+)
- Myocardial disease: myocardial infarction, 3rd degree HB, cardiomyopathy
- electrolyte abnormalities: low calcium, potassium and/or magnesium
What type of cardiac monitors are available?
- Holter monitor: 24-48hrs
-continuous recording; patient with frequent symptoms
-symptom rhythm correlation with patient event diary - Event Monitor: up to 30 days
-Patient activated or automatically activated
-symptoms “a few times/ month” - Zio Patch: 1 to 2 weeks
-continuous recording
-patient-trigger capability (2 weeks)
- continuous monitoring
- trans-telephonic transmission
- automatic detection of significant arrhythmias
- patient activation
- 2-3 years of longevity
implantable loop recorder
what are intrinsic mechanisms of sick sinus syndrome?
Intrinsic
- Idiopathic degenerative fibrosis (most common)
- ischemia
- cardiomyopathies
- infiltrative diseases (e.g, sarcoidosis, hemochromatosis)
- congenital abnormalities
Extrinsic mechanisms of sick sinus syndrome?
drugs (e.g, digoxin, B-blockers, Ca channel blockers)
autonomic dysfuntion, hypothyroidism, hyperkalemia
when should you suspect aortic stenosis in your patient?
- fatigue, dyspnea, exertional angina, lower extremity edema, near syncope and syncope
- murmur= crescendo-decrescendo SEM (heard best at the right sternal border)