SV-Tachycardias: (A flutter & A fib) Flashcards
1
Q
SV-Tachycardias: (A flutter & A fib): Definition
A
- flutter: (of a bird or winged creature) to fly unsteadily or hover by flapping the wings quickly or lightly
- Therefore, atrial flutter is a condition where the atria beat regularly, but faster than usual and more often than the ventricles, so you may have four atrial beats to every one ventricular beat
- fibrillation: (of a muslce, especially in the heart) make a quivering movement due to uncordinated contraction of the individual fibrils
- Therefore, atrial fibrillation is a condition that causes an irregular, and often fast, heartbeat
- Afib is the most common serious abnormal heart rhythm
- So, the difference is that in Aflutter the atria beat regularly whereas in Afib the atria beat irregularly - and they both involve tachyarrhythmia
- Also, Afib may start as other forms of arrhythmia such as Aflutter that then transform into AF
2
Q
SV-Tachycardias: (A flutter & A fib): Aetiology
A
- I think both can be caused by similar things; various cardiac conditions:
- HTN
- Valvular heart disease
- heart failure
- coronary artery disease
- cardiomyopathy
- congenital heart disease
- structural abnormalities of the heart
- may be idiopathic atrial fibrosis
- may be due to scars from atrial surgery or prior atrial ablation sites
- Afib is linked to several forms of cardiovascular disease but may occur in otherwise normal hearts
3
Q
SV-Tachycardias: (A flutter & A fib): Risk factors
A
- Same at aetiology really
- HTN
- therefore obesity etc
- valvular heart disease
- therefore genetics
- heart failure
- coronary artery disease
- cardiomyopathy
- congenital heart disease
- structural abnormalities of the heart
- may be idiopathic atrial fibrosis
- may be due to scars from atrial surgery or prior atrial ablation sites
- HTN
4
Q
SV-Tachycardias: (A flutter & A fib): Pathophysiology
A
- Specifics depends on aetiology
- Aflutter
- Typically initiated by a premature electrical impulse arising in the atria
- atrial flutter is propagated due to differences in refractory periods of atrial tissue.
- This creates electrical activity that moves in a localized self-perpetuating loop, which usually lasts about 200 milliseconds for the complete circuit.
- For each cycle around the loop, an electric impulse results and propagates through the atria
- Afib
- The primary pathologic change seen in Afib is the progressive fibrosis of the atria - fibrosis may also occur in SA or AV node
- this fibrosis can be caused by many of the range of Afib aetiologies
- In AFib, the normal regular electrical impulses generated by the sinoatrial node are overwhelmed by disorganized, rapid electrical discharges produced in the atria and adjacent parts of the pulmonary veins.
- These disorganized waves conduct intermittently through the atrioventricular node, leading to irregular activation of the ventricles that generate the heartbeat.
- The primary pathologic change seen in Afib is the progressive fibrosis of the atria - fibrosis may also occur in SA or AV node
5
Q
SV-Tachycardias: (A flutter & A fib): Cinical manifestations: key presentations, other symptoms and signs
A
- Aflutter
- can sometimes go unnoticed
- but people may notice their heart beating too fast/ hard until the episode is over or the HR is controlled
- if Aflutter is due to aetiology like CAD, patients may experience the related symptoms and Aflutter will be noticed on ECG
- Afib
- can be symptomless
- similar to Aflutter in the sense that patient may simply realise their heart is beating too fast or hard
- occasionally may produce anginal chest pain (if the high heart rate causes the heart’s demand for oxygen to increase beyond the supply of available oxygen (ischemia))
- beyond the above, as Afib is often secondary to other conditions, symptoms of angina/ lung disease/ hyperthyroidism can also be present
6
Q
SV-Tachycardias: (A flutter & A fib): Investigations (diagnosis): 1st line, gold standard & other
A
- Both conditions will often be noticed on ECG during investigation of conditions that are causing more sig symptoms than the Afib or Aflutter alone, ie. CAD/ ischaemia-angina
- Afib
- Irregularly irregular QRS complexes
- Absence of P waves
- QRS usually < 120ms unless patient has other conducting problems
- ECHO if suspected valvular disease
- Aflutter
- Organised ‘sawtooth pattern’
- Important factor is a regular beat
- Murmurs or rubs on auscultation suggest valvular disease, pericarditis, or congenital heart disease
7
Q
SV-Tachycardias: (A flutter & A fib): DDx
A
- Aflutter
- other supraventricular tachycardias
- Afib
- Afib
- atrial flutter
- atrial tachycardia
- paroxysmal supraventricular tachycardia
- Wolff-Parkinson-White syndrome
8
Q
SV-Tachycardias: (A flutter & A fib): Management
A
- In general, Aflutter should be managed in the same way as Afib
- bc both rhythms can lead to atrium clot formation, so,
- anticoagulation agents
- antiplatelet agent
- bc both rhythms can lead to atrium clot formation, so,
- Both rhythms can be associated with dangerously fast heart rates and thus require medication to control the heart rate
- beta blockers
- calcium channel blockers
- and/or rhythm contro
- with class III antiarrhythmics (such as ibutilide or dofetilide)
- Hemodynamically Unstable due to uncontrolled tachycardia:
- immediate heparin
- attempted cardioversion with synchronised DC shock
- Hemodynamically Stable:
- Anti-coagulation (red stroke risk)
-
Rate control - patient not in severe tachycardia but will still have Afib rhythm
- BBs are first line
- CCBs or Digoxin are second line
-
Rhythm control - returns to sinus rhythm, given to patients with reversible causes, or new onset of Afib, achieved by cardioversion
- Pharmacological Cardioversion - amiodarone
- Electrical cardioversion - controlled shock delivered
- Long term BBs for rate and rhythm control
- Definitive treatment is catheter ablation