Surgical diseases of small animals Flashcards

1
Q

Diseases of eyelid, conjunctiva and third eyelid

A
Entropion 
Congenital 
-inward folding of the eyelid often seen in chow chow 
Acquired 
-Trauma, muscle spasm 
Irritation of cornea
T: eye drops, tacking suture 
Ectropion 
Congenital 
-Outward folding of eyelid, often seen in bulldogs, great danes 
Acquired 
-paralysis of CN VII (facial)
Exposure to environment 
T: Y-U suture, modified Khunt

Trichiasis
Ectopic hair growth on conjunctiva –> irritation of cornea

Blepharitis

  • B: Chlamydia, mycoplasma
  • V: Calici, distemper
  • P: thelaria
  • Non-Inf: Photosensitivity, allergies, vitiligo

Hordeolum
Inflammation of harderian glands, meibomian glands

Cherry eye
Prolapse of third eyelid + hypertrophy
Spaniels
T: Tacking suture

Conjunctivitis
Inf: distemper, herpes, chlamydia, thelaria, dermatophytosis
Allergic
Mechanical - Chemical, entropion, trichiasis,

KCS - lack of tear quality or quantity
Infectious Conjunctivitis

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2
Q

Diseases of the cornea - keratitis, ulceration, perforation

A

Ulcerative keratitis
Loss of stroma
-superficial, deep, perforating
-Mechanical: trichiasis, entropion, FB, Tear film deficiency
-Infectious: produce proteases that melt the cornea (malacia)
T: conjunctival flap NO STEROIDS

Boxer ulcer
-Spontaneous superficial ulcer of cornea

Deep keratitis
CAV-1 (inf. hepatitis)
-Corneal opacity “blue eye”

KCS
-lack of tear film quant and qual 
-hormonal (hypothyroidism, DM, cushings)
-infectious 
-congenital (MUC5A)
-Nervous - loss of parasympathetic innervation 
Dx: STT-2. TFBUT
T: artifical tears (pilocarpine) and parasympathomimetics 

Pannus (chronic superficial keratitis)
-Grey opacity of cornea
-Auto-immune (german shepherds) triggered by viral Ag’s or UV light
T:Keratectomy

Lacerations/Trauma
Penetrating - enters cornea but does not even anterior chamber
Perforating - Through and through
T: sutures

Cs: Epiphora, keratitis, blepharospasm
Dx: STT, slit lamp, TFBUT, rose-bengal

T:
Superficial keratectomy
Keratoplasty
Conjunctival flap

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3
Q

Diseases of the iris and retina

A

Uvea (Iris, chroid, cilliary body)

Congenital defects 
Heterochromia 
Persistent pupillary membrane - should regress after birth 
Uveal cyst 
Collie eye 
-aplasia of retina 

Anterior uveitis
Inflammation of uveal tract - iritis, iridocyclitis
Ant, Post, Int, Pan

Hyphema
Blood in AC
Trauma, clotting issues, glaucoma

Retinal dysplasia
Congenital (collie eye)
Acquired - herpes, toxoplasma, CAV-1, Calici, Distemper

Progressive retinal atrophy
-Breed disposition - setters, poodles
cGMP –> toxic to retina

Cs: Night –> full blindness, slow pupillary reflex, bumping obstacles

Dx: Maze or tracking test
OCT, ophthalmoscopy

T:Mydriatics (uvetits), CCS, ATB

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4
Q

Diseases of the lacrimal glands and ducts and KCS

A
Lacrimal gland (CN VII)
Harderian glands 
Meibomian glands (zeis)

KCS
Deficiency of tear film quality or quantity
Acquired - hypothyroidism, DM, CNVII damage (parasympathetic), infectious
Congenital - brachycephalic ocular syndrome
Acute - conjunctivitis, ulceration
Chronic - hyperkeratisation
Dx: STT <15-25mm, rose-Bengal (stains epithelium)
T:pilocarpine

Mucin deficiency

  • Shi-tzu’s
  • Normal STT
  • TFBUT
Imperforate puncta lacrimalia 
-medial canthus location 
-rains tears as well as the ICA
-Congenital - spaniel 
-Acquired - inflammation (pus)
Dx: cannulate + flush
T: Excise --> Cannulate 

Obstruction of nasolacrimal duct
Lacrimal puncta –> nasolacrimal duct –> nose and throat
Acquired: inflammation
Dx: Jones test

Dacryocystitis 
Inflammation of lacrimal duct 
2nd to obstruction of NLD
Dx: X-Ray
T: conjunctivorhinostomy 

Cs: epiphora, tear staining, fisulation

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5
Q

Glaucoma

A

Increased IOP –> visual defects –> blindness

AH drainage is blocked (ICA + puncta lacrimalia)
Excess production

Results in buphthalmos, exophthalmos –> rupture of descemet’s membrane (corneal striations)

Primary - Breed (Brachy)
Secondary - Disease process
-Acute: corneal oedema, mydriasis
-Chronic: buphthalmos, luxated lens , corneal striations

Dx: Tonometry (15-25mm/Hg) >30mm/Hg = glaucoma
Gonioscopy, ophthalmoscopy

T:
AH outflow: Pilocarpine (parasympathomimetics), Adrenaline (sympathomimetics)

Decrease production - Acetazolamide (carbonic anhydrase inhibitors - sulfonamide derivatives)

YAG laser
Cryotherapy
Gonio-Implant (widens NLD)
Enucleation

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6
Q

Diseases of the lens. Neoplasia of the eye

A

Cataracts
Opacity of lens

Causes

  • hereditary
  • DM + hormonal
  • Infectious
  • Traumatic/iatrogenic
  • Changes in IOP

Classification

  • Onset: Juvenile, congenital, senior
  • Location: capsular, subcapsular, cortical

Stage

  • Incipient: focal opacity
  • Immature: lens enlargement
  • Mature: Total lens opacity
  • Hypermature: liquefaction

Dx: ophthalmoscopy, USG

T: mydriatics, USG breakdown

Len sub/luxation
Luxation - total lack of attachment
Sub-Lux: partial loss of zonular attachment
Anterior or posterior

Causes:
-Changed by glaucoma and trauma

Cs:
luxation - blocks ICA/PL --> glaucoma 
Posterior - assymptommatic 
Sub-lux: Aphatic cresent 
T: emulsification and removal 

Neoplasia
Eyelid (common) - skin tumours
-Adeno, melano, histio, papillo

Orbital
-Osteosarcoma, fibrosarcoma

Corneal + limbal
Melanoma

Uveal
Melanoma + lymphosarcoma

Cs: exophthalmos, buphthalmos, strabismus, hyphema, glaucoma, iridocyclitis

Dx: Biopsy

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7
Q

Proptosis of the eyeball. Blindness

A

Proptosis
Protrusion of the eye (similar to exophthalmos)

Congenital - Mucocele
Acquired - Myositis, FB, Neoplasia

Proptosis causes inflammation + haemorrhage –> further displacement –> positive feedback

Can result in ulcers, optical nerve damage, KCS

T: Artificial tears, pilocarpine, canthotomy, tarsorrhaphy

Blindness
Partial or complete
Dx: lack of menace response, delayed pupillary response, Maze test, tracking test

Rods - Night vision
Cones - Day vision

Types
Central-cortical: Occipital damage

Acute: Retinal aplasia, optic nerve damage, lens luxation

Progressive vision loss (chronic lesions): corneal oedema, KCS, cataracts, retinal necrosis

Congenital vision loss: Collie eye, pannus, retinal dysplasia, anophthalmus

Cs: anisocoria, mydriasis, loss/delayed pupillary light reflex

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8
Q

Evaluation and therapeutic surgical methods for eye diseases. Diagnosis and Therapy.

A
Exam
Discharge?
Colour
Pupil size 
Symettry 
Cloudiness?

Eliminate skin or dental disease

Ophthalmic exam 
Behaviour + vision 
-Tracking: cotton ball 
-Obstacle/Maze: navigation
-Placing: pre-empt 

Neuropathic exam

  • Menace - 2 + 3
  • Pupillary - 2,3,4,7
  • Dazzle: should avoid
  • Palpebral - CN 5, 6, 7

Tear film test
STT/STT-2 - 15-25mm/min
Phenol red - like STT (18-25mm/15 seconds)
TFBUT
Jones test - patency
Fluorescein - observe corneal defect
Rose-Bengal test: Degenerated cornea (herpes, corneal ulcers)

IOP - tonometry 
15-25mm/Hg
Lower - uveitis 
Higher - Glaucoma
Manual (retropulsion) - assess with hands 
Schoitz - old school
Tonometer - tonovet 

Opthalmoscopy
Direct - fundus + behind eye
Indirect - Upside down
Fundus cam - iphone + lens

Gonioscopy
Goniolens to assess ICA

USG
Retrobulbar lesions, lens luxation or cataracts

Surgical procedures

  • Eyelid laceration - figure 8
  • Tacking eyelid issues
  • Y-U repair: entropion
  • Modified Khunt (similar to Y-U)
  • Excision
  • Enucleation
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9
Q

Surgical diseases of the pinnae and external ear canal. Methods of treatment. Lateral resection of the external ear canal. Partial and total ablation of the ear canal. Osteotomy of the bulla tympani

A

Otitis
Ext - Ear canal
Media - tympanic membrane + eustachian tube (Horner’s)
Int - semi-circular canals and ossicles (Vestibular)
–Cats: Ascending
–Dogs: Descending
Hyperplastica –> ossificans

Othaematoma auris 
Causes: 
-otitis externa
-Trauma + fighting 
-Cushing's 
-Pendulous pinnae 

T: puncture + drain –> Aspiration + CCS

S-Shaped incision –> ligate bleeders –> Ligate ear with horizontal mattress (not over incision) + buttons (to elivated pressure on the sutures)

Bandage with capistrum

Wounds/Trauma
Superficial, deep, perforating
Debridge edges, suture –> ATb
Bandage with capistrum

FB’s
Grass seeds
Otoscope + forceps

Neoplasia
Papiloma, fibroma, sarcoma

Surgical treatment
Lateral ear resection
-Vertical ear canal hyperplasia
-non-responsive otitis ext

Vertical ear ablation
-when vertical canal is diseased but horizontal is fine

Total ear canal ablation

  • Vertical + medial ear canal
  • Drain is affixed to prevent sebum build up
  • Otitis hyperplastica (ossificans)

Lateral bulla osteotomy

  • Done alongside TECA (TECA-BO)
  • Removal of secretory epithelium
  • Use rongeur (avoid retroauricular vein and ossicles) to express bulla

Vertical bulla osteotomy
-Mandible rami –> ventral midline (2cm from centre on affect side)
Avoid hypoglossal nerve and muscle + lingual artery

Cartilage (concha) or vertical ear canal graft

  • Use in hard palate repair from celft palate
  • Use cartilage from pinnae or 2/3 of vertical ear canal
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10
Q

Surgical diseases of the salivary glands and ducts.

A

Mucoceles
Inflammation of gland or duct cause leeching and accumulation of saliva

Location
Cervical - dysphagia and swelling
Sublingual ("Ranula")
Pharyngeal - coughing
Zygomatic - exophthalmos 

T:Drain + marsupialised (cutting a slit and suturing the edges open)

Mandibular + subligual :
Incision at level of ear, removal of both glands must be done
Zygogatic:
Incise at dorsal zygomatic arch
Move globe dorsally
Parotid:
Incise between ramus and jugular bifurcation

Sialadentits
trauma or systemic infection
Swelling –> fistulation
T: drain and marsupialise

Sialoliths 
Calcified stone in salivary duct
Formed from inflammatory cells or ascending FB
Dx: imaging 
T: forceps 

Fistula
Trauma to salivary glands finds it hard to heal due to constantly salivary drip

Ptylism
Primary - hypersialoism
Secondary - Infectious process, swallowing disorder, peridontal disease
DDx important in rabies

T:Anti-drool cheilioplasty

  • cut at 2nd premolar (1/206)
  • lower lip sutured into upper lip with mucosal fold suture
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11
Q

Diseases of the lip. Mouth diseases. Gingivitis. Tartar. Neoplasia of the soft and hard tissue of the mouth. Clef palate. Oronasal fistulation. Surgical diseases of the nose

A

LIPS
Lip fold dermatitis (intertrigo)
Spaniels + St.Bernards

Feline eosinophilic complex 
3 Lesions 
-Plaque 
-Granuloma 
-Indolent ulcer 

Allergic aetiology with localisation on hard palate

MOUTH
Stomatitis
Increased immune activity due to tartar build up on teeth

Contact ulcer on upper lip by carnacial

Gingivitis
Inflammation of gums –> first stage of peridontal disease
Bacteria –> plaque –> Ca2+ –> Tartar
T:Scale and polish

Cs: ptyalism, bruxism, dysphagia, chewing on one side

Peridontal disease
Inflammation of the structure holding the tooth due to excess tartar (plaque –> tartar 2 weeks)

Cs: Peridontal pockets, gingival rescession

Formation of peridontal pockets allows anaerobic bacteria (fuscobact) infiltration
T: Excision, gingivectomy

Cleft lip/palate
Failure of palatine fusion higher occurrence in brachycephalics

Primary - lip
Secondary - hard palate (oronasal fistula)
T: Palate guard. Surgery >12 weeks

Oronasal fistula
Congenital as a result of secondary hard palate clefting
Acquired - FB, chronic rhinitis, iatrogenic during canine tooth removal
Cs: milk leakage, aspiration pneumonia
T:
Debride and cover
-Advancing flap
-Rotational flap
-Double flap (hard palate and lip mucosa)

T:
HARD PALATE
Von Langenbeck technique (sliding pidical flaps)
Overlapping flap (tuck flap into pocket)
SOFT PALATE
Flap from nasal wall, tuck into lip mucosa
Z-cheilioplasty

Neoplasia 
Odontoblastoma 
Ameloblastoma 
odontoblastic fibroma 
Epulis - boxers 

Tonsilitis/adenoma
-Tonsilectomy

NOSE
Stenotic nares 
Part of BS
Mouth breathing 
T: Alar wing resection, punch resection
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12
Q

Diseases of the teeth - oligodontia, polyodontia, brachygnathia, pulpitis

A
Tooth anatomy 
Enamel
Dentin 
Cementin 
pulp 
Periodontal ligament 

Teething: 4 mnths –> 7mnths
Diphyodont

Dental formula:
Cats: 3/3 1/1 3/2 1/1
Dogs: 3/3 1/1 4/4 2/3

Tridan system:
1 - Upper R, 2 - Upper L
3 - Lower L, 4 - Lower R
Start at the incisor (X01)

Oligodontia - Brachycephalics have a higher incidence of this. (>6 teeth missing) 0 usually premolars

Polyodontia - “supernumery teeth” usually incisors –> crowding + malocclusion
Cs: Dysphagia, chewing on one side, bruxism, ptyalism

Brachygnathia 
Abnormally short jaw 
Maxilliary - results in crowding 
Mandibular 
T:Bite plates + correction

Pulipitis
Inflammation of pulp
-Trauma
-Infection from decay allowing bacterial infiltration
-periodontal disease
Pulp is enclosed so pressure can cause ischemia and further destroy the tooth

Cs: Dysphagia, swelling, halitosis, weight loss, ptyalism

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13
Q

Fracture of teeth. Extraction of teeth. Endodontia, exodontia. Local analgesia of the head

A

Fractures
Canine and carnacials (4th premolar)
Usually trauma from chewing FB’s

Classification 
Enamel 
Uncomplicated crown 
Complicated crown
Root (bi/tri-furcation)
Uncomplicated crown-root
Complicated crown-root 

Enamel hypoplasia (results in pulp exposure)
-Inf: distemper
-Non-Inf: fluoride excess
Pulp exposure –> infection (pulpitis)

Cs: dysphagia, unilateral chewing, ptyalism, facial oedema
T: ATb, replanting, extration, root canal

Exodontia 
Dental extraction 
Root abscess, fracture, FORL
Elevate + levate
Drill 
Perigingival flap (envelope tech) 
Canine = oronasal fistula risk
Endodontics
Dentistry of the pulp 
-Pulp exposure + pulpitis 
-Abscess, cyst 
-Remove necrotic tissue 
-Prevent apical peridontitis 
-Fill canal with sealant 
Partial maxilectomy/mandibulectomy 
-Oral neoplasia resection 
Localisations 
-Pre-maxilectomy (bilateral rostral)
-Rostral - incisors + canines
-Central - premolars 
-Caudal - molars 
-Hemi: entire side of skull
Anaesthesia in the head 
Rostral maxillary
Caudal maxillary 
Rostral mandibular 
Caudal mandibular
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14
Q

Surgical diseases of the oesophagus. Hiatal hernia. oesophageal feeding tubes

A

Oesophagotomy - opening
Oesophagectomy - removal
Oesophagostomy - placing a tube

Adventisia, musc, sub, mucosa
-Lack of serosa means adhesions are common

Obstruction
Intramural
Foreign bodies 
3 Sites
-Ap. Thor. Cran. 
-Basis cordis 
-Hiatus oesophagi 

Extramural
Neoplasia

Stricture - recurrent obstrcution

Diverticulum

  • Traction
  • Pulsion

Peristalsis –> necrosis –> perforation

Megaoesophagus

  • Acquired: Addion’s, mechanical obstruction
  • congenital: myasthena gravis

Oesophagitis
-FB, acid reflux, pancreatic enzymes

Hiatal hernia 
Cardia - slide hernia 
Fundus - rolling hernia 
Congenital 
Acquired: dyspnea (thoracic pressure), reflux 

Gastroesophageal intussusception
-Similar to hernia but instead of fundus/cardia going into the hiatus the fundus folds into the oesophagus

Oesophageal aclasia
-Failure of sphincter to open at birth

Cs: Regurgitation (not vomiting), ptyalism, halitosis, dysphagia, dyspnea

Dx:
Fluroscopy (to observe size and motility)
Endoscopy
X-Ray

T:
Omeprazole (PPI)
Cimetidine (H2 blocker)
Metoclopramide (peristalsis upregulator) 
Anastomosis  
Oesophagostomy tubes 
-GA + feeding
-7th ICS
-forceps --> press to skin by ramus 
-Pull tube out mouth --> redirect into oesopagus 
Chinese finger trap suture
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15
Q

Acute abdomen. Types. Traumatic, hypovolemic and septic shock. Emergency and critical care

A

Acute abdomen
Sudden abdominal pain seen as distention, V+/D+, shock

Dilation - enlargement with gas without rotation (aerophagia, eating snow + delayed emptying = fermentation)

GDV
Acute life threatening condition 
90-360 degrees 
Duodenum between oesophagus and stomach
Dx:
Radiography (Lat + DV)
-C-shape (stage 1 + 2)
-Double bubble (stage 3)

Blood flow obstruction (VCCau)

  • Portal hypotension
  • GIT ischemia
  • hypovolemic shock

Cs: ptyalism, bloating, tachycardia (hypovolemia), cyanosis

T: 
Fluids + critical care (dexamethasone) 
Omeprazole, cimetidine, metoclopramide
Decompress - ETT, Large IV catheter gastrocentesis
gastrectomy + gastropexy
Benign gastric outflow 
-Pyloric stenosis
-Pyloric hypertrophy (Muscular, mucosal)
Dx: 
Endoscopy > x-ray 
-Can DDx hyperplasia, stenosis, inflammation, FB

Shock
O2 requirement > delivery
Hypovolemic
-Lower blood flow, thready pulse, cold extremities
Due to:
-Haemorrhage
-Kidney failure: RAAS + ADH (vasoconstriction decreased GFR)
-Lungs: Vasoconstriction (oedema in lungs + impaired O2 exchange)

Traumatic shock
-Trauma + burns –> bleeding, vasodilation

Distributive shock (septic)
Excessive vasodilation 
-septic: Endotoxemia or pancreatitis 
-anaphylactic: huge type I inflammatory response 
-No blood loss, but increased intravascular space and lowered filling
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16
Q

Surgery of the stomach. FB’s, Dilation and volvulus. Pericardioperitoneal hernia. Neoplasia of the stomach.

A
Surgery of the stomach 
Gastrectomy - resection
-prefered between vessels 
Gastrotomy - opening 
Gastropexy - attach to abdominal wall 
-Circumcostal loop 
-Muscular flap

Arteria linealis + coeliaca

  • Gastric
  • splenic
  • hepatic

Foreign bodies
Bones/stones
Linear FB’s are more serious (peritonitis if GIT ruptures)

Gastric ulceration
Zollinger-ellinson syndrome: overproduction of acid (gastroma of pancreas)
Erosion (muc + sub) –> ulcer (muc + sub + muscularis)

commonly iatrogenic (steroids + NSAID’s)

Dx: Relies on endoscopy (x-ray cant see erosions)
T: Omeprazole, metoclopramide, cimetidine, Sucralfate (mucous), bicarb

Dilation (pyloric stenosis)
-aerophagia/snow eating
Volvulous 
GDV
C-Shape / double bubble 
90-360 degrees 
Hypovolemic shock 
Oesophagus and duodenum entrap stomach and VCCau

Gastric neoplasia
Adenocarcinoma
Leiomyosarcoma
Pythium insidiosum (phycomycosis)

Laparotomy + celiotomy

  • 3 layer suture
  • -Skin
  • -SubQ
  • -Linea alba + m.rectus ab
Rectus abdominis
Internal obliques
Transversus obliques 
external obliques 
Linea alba
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17
Q

Diaphragmatic hernia. Pericardioperitoneal hernia. Abdominal organ trauma. Umbilical hernia, traumatic hernia.

A

Diaphragmatic hernia
Congenital or acquird
-Increased abdominal pressure –> diaphragmatic rupture
Abdomenal organs –> thorax

Pericardioperitoneal diaphragmatic hernia
-Birth defect of pericardium and diaphragm, often the liver herniates into the pericardial space
T: Repositioning and closure is usually easy as the motion of the heart stops adhesions

Abdominal trauma

  • External Hernia through abdominal wall defect
  • Umbilical hernia
  • -associated with intersex in females and testicle descent in males
  • Internal Hernia through abdominal structure (diaphragm or inguinal canal)
  • May result in peritonitis
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18
Q

Surgical diseases of the pylorus and spleen. pyloroplasty + Splenectomy

A

Pylorus
-Stomach –> SI

Stenosis
Congenital or acquired
-True stenosis or muscular hypertrophy

Obstruction
FB, chronic hypertrophy (stenosis –> obstruction)
Causes ileus + gas dilation

T: Pyloroplasty
-UY-Pyloroplasty

  • Fredet-Ramstead pyloromyotomy (cut through serosa and muscularis to allow muscoa to bulge)
  • Heineke-mikulicz - horizontal
  • Jaboulay pyloroplasty - attach duodenum to stomach (bypass pylorus)
Spleen 
Torsion
-Torsion of splenic artery --> obstructs blood flow 
-Usually occurs with GDV
T:Splenectomy, gastropexy 

Rupture
Neoplasia - haemangiosarcoma
-Blood loss due to abdominal haemorrhage
-Anaemia + hypovolemia

T: Splenectomy

  • Partial
  • Total: ligate splenic arteries
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19
Q

Surgical diseases of the small intestine and cecum. Enterotomy + enterectomy

A

Obstruction
Extraluminal, intraluminal, interluminal
-Mechanical: FB, intussusception, tumors
-Functional: Water loss, nervous, parasympatholytics

Simple obstruction
Causes:
-LI displacement, food impaction, nematode impaction, Neoplasia, Hypertrophy (mural)
-fluid cant pass to LI –> hypovolemia + cardiac failure

-Gas distention –> loss of peristalsis (ileus) + fluid (with protein and ions) leakage –> peritoneum –> hypovolemia + acid-base disbalance

  • -Proximal: Alkalosis
  • -Distal: Acidosis

Incarceration
-Through mesenteric defect

Strangulation
Causes:
-Intussusception, volvulus, Fibrous adhesions, herniation
-Acute abdomen. Vascular compromise –> hypoxia –> necrosis –> perforation

Ileus

  • Arrest of peristalsis
  • Mechanical: Gas distention
  • Functional: electrolytes, myasthenia gravis

Infarct - mesenteric artery

Linear FB
-Partial obstruction

Intussusception
-Usually ileo-cecal junction

Enterotomy

  • Opening of the bowel to remove FB’s
  • Close horizontal to prevent stricture

Enterectomy

  • Anastomosis of intestine for the removal of stenotic, necrotic or fibrous intestine
  • Serosal patching can be used to promote healing
  • Functional end-to-end anastomosis
20
Q

Surgical diseases of the LI. prolapse recti. Colonpexy.

A
Megacolon
Increase in diameter of colon 
Changes to function --> constipation + hypomotility 
Congenital or acquired 
-Mechanical: Obstruction 
-Neurological: Cauda equina 

Constipation + obstipation

  • C: Passage of dry hardened faeces
  • O: Complete absence of faecal passage

Incontinence
Loss of voluntary control of defecation
Cauda equina, prolapse

Anal/Rectal prolapse
Complete - all layers
Incomplete - mucosa only
Often due to increased abdominal pressure (constipation, dystocia, respiratory disease)
Hypertonic (mannitol) solution can reduce size, lubricate, massage, replace, purse-string suture + colopexy

Diverticulosis
-Development of diverticula in LI –> diverticulitis

Diverticulitis
-Filling of diverticulum with faeces, blood or pus –> diverticulitis –> peritonitis, abdominal haemorrhage

Neoplasia
Polyps
Leiomyoma
Adenoma

Dx: colonoscopy, X-Ray, USG
T: Colectomy (resection)

21
Q

Perineal hernia. Diseases of the perianal glands and perineal neoplasia.

A

Perineal hernias
Uni or bilateral
Often occurs in prostate disease, constipation, straining (tenesmus)
Dx: Palpation/imaging
T: Evacuate faeces, surgical herniorrhaphy + castration

Perianal fistula 
From diverticulitis 
Septic progressive infection of the perianal area (ulceration + abscessation) 
Cs: Moist foul smelling area near anus 
T: graft + anal sacculectomy 

Anal sac neoplasia
Adenocarcinoma
T: excision

Anal sac disease 
Obstruction or infection of the gland 
Overproduction of gland 
Poor muscle tone in obese animals causes improper emptying of gland during defecation 
Cs: pain during defecation 

Anal sacculectomy
Indicated in chronic anal sac impaction
2 types
-Open: incise gland and expose secretory lining
-Closed: Gland kept in tact and bluntly dissected

22
Q

Hernias - general information. Inguino-scrotal hernias

A

Hernia
-Protrusion through a defect

Classification

  • Ext. abdominal hernias - through the abdominal wall
  • Int. abdominal hernias - peritoneal/inguinal
  • True hernia - within peritoneal sac
  • False hernia - non-enclosed
  • Reducible - no adhesions
  • Irreducible - adhesions

Can cause;
incarceration - fluid retention (obstipation)
Strangulation - obstructed blood flow (necrosis)

Inguinal (scrotal in male)

  • Through inguinal canal
  • Usually males after testicles descend
  • rare in females associated with intersex
  • Bladder, SI, uterus

Umbilical

  • due to failure of closure of umbilicus at birth
  • Pulling on umbilical cord during separation

Femoral / ectopic
-Defect in canalis femoralis

Surgical approach

  • Linea alba approach
  • Debride any fat or muscle
  • hernial sac ligation
  • tissue flap appositional suture
  • Hernioraphy
23
Q

Surgical diseases of the trachea. Collapse.

A
Tracheal collapse 
Progressive collapse 
Acquired 
-Megaesophagus + laryngeal paralysis 
Congenital 
Inspiration - Cervical 
Expiration - Thoracic 
"Goose-honking" 
Grades 
1 - 25%
2 - 50%
3 - 75% 
4 - 100% 

Dx:Endoscopy, palpation, x-ray
T: glycosaminoglycan, chondroitin, prosthesis (2+3),
Antitussive, bronchodilator
Tacking sutures (1)

Rupture
Bites, choke chains, overfilling of ET tube cuff

FB 
Cervical + thoracic
Aspiration 
ET tube --> push past --> inflate --> remove with FB
Tracheotomy 

Tracheostomy
Critical care and UR obstruction
-Transverse flap (5-6th ring)

  • Horizontal (3+4, 5+6th ring)
  • Inverted ventral flap (as transverse but lifted upwards)
  • Vertical (as horizontal)
24
Q

Trauma of the chest wall and lungs. Neoplasia of the lung. Pneumothorax, Lobectomy. Thoracocentesis

A

Thoracic trauma

  • Rib trauma –> “Flail chest”
  • Paradoxical breathing (one piece of chest doesn’t move so looks depresed in inspiration and expanded in expiration)

Tumors of lung + thorax
-often 2nd tumors (Mx)
-Bronchoalveolar adenoma
T: Thoracotomy + lobectomy

Pneumothorax
-Spontaneous
-Traumatic
-Closed - Lung broken and air leaks into thorax
-Open - thoracic wall broken allowing air to enter that cannot escape
-Increased thoracic pressure can cause partial or complete lung collapse –> lung tidal volume reducation
Dx: Heart elevated from sternum
T: Thoracocentesis

Thoracocentesis 
6-8ICS
-Fluid: ventral portion position 
-Air: dorsal needle position
-Needle at 45 degrees 
Thoracotomy 
Access the cavity 
Method 
-Intercostal 3-4th ICS 
-Median - Sternum 
-Transcostal - Last rib 

Lobectomy
Partial - Removal of part
Total - An entire lobe

25
Q

Surgical therapy of the kidneys and surgical disease of ureters. Ectopic ureters

A

Kidneys
Trauma
causes rupture in vessels or rupture of parenchyma

Nephrolithiasis
Ca2+ and alkaline urine or UTI (haematogenous or ascending)

Hydronephrosis
Dilation of renal pelvis due to stones/inflammation/stenosis in kidneys or further down urinary tract

Cystic kidney disease 
Polycystic kidneys replace large portions of the parenchyma affecting function 
Genetic predisposition 
Hormonal - PTH + Vasopressin 
Dx: FNA + USG

Surgery
Nephrotomy - biopsy, cystic excision, stone removal

Nephrectomy

  • Complete
  • Partial

Nephrostomy
Cannulisation to create a permanent fistual - bypassing stones, damaged ureter

Pyelolithomy
Opening renal pelvis to remove stones

Ureter 
Ectopic ureters (opening of ureter anywhere but trigoneum of bladder)
Most common cause of urinary incontinence in young dogs. 
Females at 8x higher risk 
Intra/extra-mural 
Cs: urine scalding
Dx: contrast urography (iodine, air) 
T: 
Cystocentesis + ligate 
Nephrostomy 
Ureter obturation/obstruction 
Hypomotility of muscle 
Urolithiasis, neoplasia, urethrospasm 
Post-renal azotemia + uremia 
Dx: contrast x-ray, neurology, myelography 
T: 
Muscle atony - metaclopromide + cisapride (prokinetic) 
Urethrostomy
26
Q

Surgical diseases of the urinary bladder and ureters. Cystotomy, cystectomy, urethrotomy, urethrostomy

A

Bladder
Urolithiasis
Urinary stones (struvite, calcium, urate)
Higher concentration of salts + alkaline urine

Cystitis
Bacteiral, stones, neoplasia
Dx: Bladder becomes thickned

Feline lower UTI / FIC
Congenital 
uroliths 
idiopathic/stress induced 
Primarily males 
Stages
1 - Inital disease 
2 - No Cs but furtherment of 1
3 - PU/PD
4 - Uremic syndrome 

Neoplasia
TCC

Cs: stranguria, haematuria, imaging, palpation

Surgery
Cystotomy - removal of stones, biopsy, ectopic ureters (urethrostomy)
Two layer closure - schneiden + lambert

Cystectomy 
Removal of portion 
-Cut ventral or dorsal 
-suture submucosa NOT MUCOSA 
-Omentoplasty to help healing 

Urethrotomy - stone
Urethrostomy - Premanent urinary diversion due to chronic processes

27
Q

Head and spinal trauma. Critical and emergency care. Diseases of the peripheral nerves of the fore and hind limbs

A
Head traua 
RTA, fighting, falls 
Concussion - traumatised on impact 
Contussion - bleeding from blunt forse 
Coup-contrecoup - bilater contussion 
Diffuse axonal - shaking --> nerve tearing 

Cs: eistaxis, coma, mydriasis
Dx: imagine, increased ICP
T: ABC, benzos

Spinal injury 
IVDD, trauma, ischemia 
Seen vental, dorsal or at ligaments 
Cs: ataxia, spastic, paralysis (depends on location) 
Dx: myelography 
T: ABC, Mannitol, benzos

Critical care
Indications; shock, poisons, burns, seizures, bleeding
ABC care
Airway - remove obstruction + place ET tube
Breathing - breathing patterns, Perform CPR, Thoracocentesis if fluid present
Circulation - HR, CRT, MM colour, pulse intensity

Head trauma 
Supply O2
IV catheter 
-60-90ml/kg/hr (dog)
-45ml/kg/hr (cat)
BP - Colloids, crystaloids 
Glucose - hypoglycaemia 
Seizures - Benzo's 
IC pressure - mannitol 
NO STEROIDS
DO NOT MOVE 
SEDATE IF NEEDED

Peripheral nerve damage
Congenital/hereditary
Acquired
-Toxic, drug, chemical, heavy metals

  • Neuropraxia: disruption in transmission but no muscle atrophy
  • Anoxotmesis: damage and loss of function

Neurotmesis: complete severance of axon

Dancing doberman

  • Flex and extend the hip
  • develops to pariesis

Distal denerving disease
-Idiopathic in dogs –> muscle axons stripped of myelin

Distal polyneruropathy of rotweillers

28
Q

Diseases of the cervical vertebrae and spinal cord. AAI, wobbler’s, Myelography

A

Atlantoaxial instability
Dens becomes displaced or ligaments fail
Developmental or acquired through trauma
Subluxation results in compression of the spinal cord –> UMN signs in FL and HL

X-Ray shows spinal process of axis over the atlas

T: Neck brace, fusion

Wobbler's syndrome 
Caudal cervical vertebrae (C4-7)
Can result from;
-Instability + subluxation
-Articular process enlargement --> Ligament hypertrophy
-IVDD (protrusion)
-Malformed 

Types
-Dynamic: when moving
-Static: due to spondylosis
Cs: HL ataxia (john wayne)

IVDD
Chondrodystrophic breeds (brachycephalics)
-Extrusion - through annulus fibrosis
-Protrusion - pushes annulus fibrosus (no rupture)

Schiff-sherrington syndrome
T3-L3
FL - Hyperreflexivity
HL - Paralysis

Surgical procedures
Ventral slot - drill to look
Facetectomy - remove cranial and caudal aspects
Foraminotomy - remove roof
Fenestration - Remove nucleus pulposus via annulus fibrosus
Laminectomy

Myelography
Injection of iodine into sub-arachnoid space
0.3-0.45ml
Cervical (cisterna magna)
-Middle ear line, Wings of atlas and occipital protuberance (triangulate)

Lumbar (L4-L6) - lumbosacral space

  • extradural (IVD protrusion)
  • intra-dural-extramedullary (neoplasia)
  • intramedullary (oedema, ischemia myelopathy)

Intramedullary opacification = meningitis/myelomalacia

29
Q

Diseases of IVD. Diseases of thoraco-lumbar vertebrae.

Discospondylitis, DISH, Spondylosis deformans, fractures + luxation

A

C:7 T:13 L:7 S:3 Cau:6-23

Diseases of IVD

  • Protrusion: mass impinging on SC
  • Bulging: Nuc. Pulp pushes on AF
  • Extrusion: NP breaks through AF

Hansen I - Dehydration –> mineralisation of NP and degeneration of AF –> Extrusion

Hansen II - Fibrosis of NP –> normal AF –> bulging (protrusion)

X-Ray: Narrowing and calcification

Diseases of T-L vert
Discospondylosis
-Bacterial infection of vertebrae (Prostate or bladder)
Dx: sclerosis

Spondylosis deformans
-Spinal Instability or luxation can result in osteophyte formation –> spurs can form

Diffuse idiopathic skeletal hyperostosis
-Boxers, calcification along SC assymptomatic until fusion

Fractures

  • Vertebral body
  • Compression fracture

Luxation
-Displacement of bone from jont

Neoplasia
-Meningioma
-Lymphoma 
-Osteosarcoma
-Fibrosarcoma
Dx: Scintigraphy
30
Q

Syndrome cauda equina. Transitional lumbosacral vertebrae.

A

Cauda equina syndrome
-Instability, luxation, articular process enlargement and ligament hypertrophy, protrusion/extrusion
Compress the cauda equina, (nerves of the SC behind the lumbar vert)
T: laminectomy (removal of the doral lamina in the VC to make space for SC)

LS transitional vertebrae
Congenital malformation in which one vertebrae has the characteristics of another
German shepards
-Transitional LS segment: Vert w/ lumbar and sacral features (L7/S1)
-Sacralisation: Lumbar vert with sacral wings (no proc. trans.)

Causes;
-Hip dysplasia, CES, scoliosis

Fractures
Luxation 
Neoplasia
-Osteosarcoma
-Chondrosarcoma
31
Q

Dx of spinal cord diseases. spinal reflexes. imaging Dx, radiography, CT, MRI

A

Dx
Clinical signs
Cerical syndrome (1-5)
UMN signs on FL + HL

Cervico-thoracic syndrome (C6-T2)
UMN - HL, LMN - FL
“Root signature”

Thoraco-lumbar (T3-L3)
UMN - HL
Schiff-sherington (FL extension + HL paralysis)

Lumbosacral (L4-Ca5)
LMN - HL
Incontinance

Imaging

  • X-Ray
  • Myelography (iodine, 3ml - 4.5ml)

CT
-Creates a cross sectional imagine by taking multiple x-rays at different angles and building an image

MRI
-Uses protons and energy release from them to construct an image using magnetic coils that surround the patient to visualise the brain/SC in slices. Gold standard.

Spinal reflexes
Also diagnostic
Reflex testing
-Muscle tone > myotactic reflex > flexor reflexes

Mycotactic (patellar)
Flexor (withdrawl pedal reflex - pinch between digits)

Sensation (panniculus reflex)

C-Biceps reflex
T-Panniculus reflex
L-Patellar reflex
S-Perianal reflex

32
Q

pediatric long bone diseases

A

Panostitis juvenilis
Inflammation of long bones with no lysis. Often large breeds with excess protein or calcium
Dx: increased bone opactiy
T: Pain relief

Hypertrophic osteodystrophy
Radius + ulna + tibia
Usually bilateral in the metaphysis –> necrosis + haemorrhage
X-Ray + histopathology (haemosiderin deposits)

Ricketts
Defective bone calcification in growing bones;
-persistant hypertrophic cartilage
-enlargement of epiphysis + costochondral junctions
Cs: bone curvature
T: Ca + P supplement

Osteochondromatsis
Benign bone nodules with cartilage caps
Often on growth plates during ossification of GP
Surgical removal of Exostosis

Avascular necrosis of the femoral head

  • Traumatic: joint/hip trauma
  • Non-traumatic: embolism, hypovolemia
Neoplasia
Characterised 
-Lytic
-Proliferative
-Mixed

Primary
Benign: osteoma, osteochondroma, osteoblastoma

Malignant: Chondrosarcoma, Fibrosarcoma, osteosarcoma

Secondary
Often humeral or femoral

X-Ray of neoplasia

  • Periosteal reactions
  • -Solid
  • -Lamellated
  • -Sunburst
  • -Codman’s triangle
33
Q

Osteochondrosis dissecans. Osteoarthritis, osteoarthrosis, DJD. Arthrotomy.

A

Osteochondrosis
Disruption of endochondrial ossification –> angular deformities, valgus, OCD

OCD
-4-6mnths. Cartilage thickens and blood flow becomes impeded to deeper cartilage –> necrosis and fragmentation

Grading
1 - small defect in subchondral bone
4 - Vertical fracture + separation of the flap
T: remove flap (arthroscopy) + cauterise

Osteoarthritis
Inflammation of bone due to
-Septic: Bacteria –> toxins –> cartilage destruction –> osteomyelitis –> osteophytes + exostosis
-Aseptic: Ca2+
-Autoimmune: Rheumatoid arthritis (like lupus)
Results in osteoarthrosis (DJD)

Osteoarthrosis (DJD)
Degeneration of cartilage 
remodelling of bone 
synovial membrane and peri-articular tissue damage 
Bone hypertrophy

Primary - aging, wear and tear
Secondary - OA, joint instability

Loss of cartilage causes bone on bone action (osteophytes) –> inflammation (destruction > production)

Arthrotomy

  • Surgical exploration of the joint
  • Drainage of inflammatory joint fluid
34
Q

Diseases of the shoulder joint

A

Shoulder joint
Glenoid cavity + humeral head

Luxation (displacement of bone from joint)
Lack of collateral ligaments (glenohumeral ligs) –> instability
Subluxation - partial displacement
Categorised:
-Medial
-Lateral
-Cranial

Congenital - minitures
Acquired - RTA, Fighting

T: Manual reposition, velapeu sling
Surgical reconstruction of glenohumeral ligs (bicep tendons)

Instability
Abnormal movement of humeral head with destruction of periarticular tissues
Graded: Low –> High
Dx: ROM
T: NSAID’s physiotherapy, fusion, prosthesis, lig recon

Bicep tendon disease
Bicep brachii tendon affixes to spina scapulae. Instability of the joint can cause damage to the bicep tendon
T: Tendonotomy/tenodesis

OCD
-cartilage –> necrosis –> fragment

35
Q

Diseases of the elbow joint. ED. Screening ED. Short radius + short ulna

A

Elbow dysplasia
Group of diseases found in growing dogs (esp. giants)

Developmental - nutritional (Ca + P)
Genetic - Lrg + giant breeds

Pathogenesis
Multiple primary lesions –> osteoarthritic process

Fragmented coronoid process
-Mild –> full fragmentation

OCD
-Medial humeral condyle

Ununitied anconial process 
-Ulnar metaphysis 
-Physiological fusion at 4-5 months 
-Short ulna and radius can exaserbate or prevent fusion 
T: Lag screw fixation 

Incongruence of elbow
-Coronoid above radial head (asynchronus growth)

Begins at 4 months Cs at 6

Dx: 
Flex/Extension test
Abducted stance (wide)
X-Ray
ML
-110-120 - Neutral 
-45 - Flexed
Cr-Cau
-15 (OCD)

Grading - IEWG
0 - Normal
1 - Mild dysplasia + osteophytes (<2mm)
2 - Moderate osteoarthritis + osteophytes (2-5mm)
3 - severe OA + osteophyets (>5mm) + UAP

T:
Chondroprotectives (glycoasaminoglycan + chondroitin)
Lag screw - UAP
arthrostomy

Short ulna/radius syndrome 
Growth plates fuse prematurely Due to 
-Trauma 
-HyperVit A + D
Other bone continues to grow 
Shortened bone pulls on the normal one --> bowing and luxation from the joint --> OA

Luxation/subluxation
Irritation + inflammation –> cartilage destruction –> bone irritation –> inflammation (OArthritis) –> bone remodelling (OArthrosis)

T: Arthrodesis + olecranon osteotomy

36
Q

Diseases of the hip joint. Arthritis, Arthrosis (DJD).

A

Coxofemoral joint (acetabulum + femoral head + ligs + glutes)

Arthritis
Joint inflammation
-Septic: myocplasma, pasturella, staph
-Aseptic: AI (rheumatoid)

Arthrosis
Degeneration of cartilage + formation of osteophytes
2nd to luxation, osteochondrosis, osteoarthritis and hip dysplasia

Luxation of the femoral head
Part of HD
Avascular femoral head necrosis

Salter-harris fractures
OCD

Legg-calve perthes disease
-Thrombosis –> ischemia of subchondral bone –> necrosis + osteolysis
X-Ray: flattened femoral head

T:
Physical therapy 
Arthrodesis 
Chondoprotectants (GAG, Ch)
(open) Arthrotomy if osteophytes 
(closed) Myorelaxants + repositioning 
Prosthesis
37
Q

Hip dysplasia. Screening program. Grading.

A
Laxity + arthritis of hip 
Multifactorial;
-Genetic (German sheps)
-Lifestyle (work load)
-Nutritional (Ca + P)
-Hormonal (oestrogen)

Cs: Bunny hopping, muscle atrophy, asymmetrical effusion

Laxity

  • Subluxation (decreaed norberg angle)
  • Femoral head should be 50% covered by craniolateral acetabulum

OA - “Morgan’s line”

Dx: ortolani signs - abduct + adduct femer = “click”

X-Ray

  • VD - Rotate inwards
  • Stress VD
  • Frog legs (hip + stifle flex)
  • Norberg = 105degrees (physiological)

T: Tripelvic osteotomy - bone plate that holds 3 parts of bone (ischi, ilii, pubis)

Pectinal myectomy - Remove m.pectinus = reduced pressure

Juvanile pubic symphsiodesis (>2 years)

Screen and grade
Minimum 2 views (VD + L)

FCI (>1 year age)
A - normal 105 NA
B - Asymptommatic but pathological 
C - Mild HD <100 NA
D - Moderate HD <95 NA + Morgan's line + subluxation
E - Severe HD <58 NA + ML + Luxation 

OFA similar to FCI
-Excellent, good, fair, transitional, mild, moderate. severe

BVA 
9 radiographic criteria 
1-NA
2-Subluxation
3-Position in acetabulum (cranial)
4-Position in acetabulum (dorsal)
5-Position of acetabulum edge (cranial)
6-Acetabular fossa
7-Femoral head exostosis 

58 per hip
116 total - high score = higher degree of HD

38
Q

Diseases of the stifle joint. Patella luxation and fractures

Screening and radiological exam of DJD in the stifle

A

Stifle (femoropatellar + femorotibial Med + Lat)

Luxation of the patella 
Common cause of lameness 
Patella slipping from trochlear groove 
Medial, lateral, bidirectional
Results in varus/valgus

Medial luxation
Quads hold the patella in place by inserting onto the tibial tuber
causes varus

Lateral luxation
often causing valgus

Grading
1 - No lesions
2 - Push patella easily returned
3 - Out of groove, medial movement, pushing limb laterally. Can be replaced
4 - Patella is STUCK medially, cannot be repositioned

X-Ray: patella is superimposed on condyles

T:
2-3 Soft tissue surgery
4 - Osteotomy of femer and tibia

Patella fracture
Trauma + extreme quadricep contraction
T: Kirschner wires

Radiographic exam
ML - Flexed 90
Cau-Cra
Lateral-oblique (dorsal recumbency)

39
Q

Diseases of the stifle joint. Diseases of the ligaments of the stifle (LCC, LCCa, collateral ligaments)

A

Stifle stabilised by
Medial and lateral collateral ligs around the joint (prevents twisting of the joint)

Cruciate ligs inside the joint (prevents cranial and caudal movement.

Meniscus - c-shaped cartilages in the stifle

Cranial cruciate ligament rupture
-Partial or full
-Causes pinching on the medial meniscus
Dx
-Orthopedic exam (gait, palpation)
-Tibial compression test (Tibia will pop forward when pressed on)
-Cranial draw exam (instability side to side)

Caudal cruciate ligament rupture
-Less common
Dx: Caudal draw exam (place dorsally –> tibia parallel to ground –> if ruptured –> caudal aspect of tibia will depress)

Rupture of collateral ligaments
Prevent abduction/adduction
Varus - inward turning (medial)
Valgus - outward turning (lateral)

T:
Hyaluron + NSAID's
Muscular flap (fascia latae)
Meniscotomy 
Stabilising sutures 
Prosthetic ligaments
40
Q

Fractures. Biological, physical, clinical assessment. Cerclage. Intramedullar pinning. Bone plate. External fixation.

A

Trauma
metabolic - Osteoporosis/chondrosis/malacia
Neoplasia

Classification
Types: Simple, complex, communicated, compound(open)
Salter-Harris
Localisation - Spiral, oblique, transverse, communicated, avulsion, greenstick, fissure

Fracture assessment score 
Evaluates 
Mechanical 
-Configuration (simple vs communicated) 
-Patient size (Large dogs are heavier)

Clinical

  • Post-op co-op - active dogs (poor candidates)
  • Comfort
  • owner compliance

Biological
-Age, soft tissue injury, region on bone (distal heals slower)

1-10 (higher scores) = faster healing, lighter load on prosthesis and lowered rejection risk

Treatment
Incomplete fractures in young dogs can be splinted/cast

Anyting more will require fixation, a bone graft can be used to accelerate healing

Cerclage wire (kirschner wire)

  • Full circlage: around the bone
  • Hemi-circlage: through the bone and into medullary cavity

Intramedullary pinning

  • Steinmann pin/kirschener wire
  • Stops bending pressure but does not prevent rotation so often done with another form of fixation (6-8wks)

Bone plate
-Versatile and often left in place

External fixators

  • Pinned both above and below the break, often with an intramedullary pin
  • Often with ilizarov fixators
41
Q

Primary and secondary bone healing. Complications of fracture therapy and arthrodesis

A

Primary (cortical) healing
Requires rigid fixation that inhibits callous formation
-Gap primary healing
-Contact primary healing

Gap

  • Stage 1: bone fills the gap (not connective tissue)
  • Stage 2: Haversian remodelling replaces necrotic bone

Contact

  • Direct apposition allows cavity to form across the break by osteoclasts.
  • These canals/cavities then become filled with new bone by osteoblasts

Secondary bone healing
Absence of fixation
3 stages
-Inflammation: immediately from break. oedema and pain stabilises movement of the break

  • Reparative: first few days. Callus forms + chondrification begins –> invasion of osteoblasts and blood vessels –> CT replaced with woven (fibrous) bone
  • Remodelling: Woven bone is replaced with lamellar bone

Assessment of fracture healing
2 weeks - Haematoma at site
3-4 weeks - Bridging callus with mineralised edge
8-12 weeks - remodelling + cortical repair

Complications
Delayed union - poor fixation, little blood supply, nutritional
Malunion - inadepqute fixation
Excessive callus - movement, periosteal stripping
Osteomyelitis - infection during malunion

Arthrodesis
Removal of cartilage, fusion of joint –> increases stability and strength + decreases pain

Use of bone grafts, pins/plates or synthetic bone can assist healing

42
Q

Fractures of the pelvis

A

Often occur in multiple places due to shape

Types:
Stable - main joints in tact (symphesis pelvini, lumbosacral, hip, sacroilieac)

Unstable - break of any main joint of the pelvis

Cs: HL Paralysis (ensure there is no siatic n. damage)

T:
Conservative - rest and NSAID
-Stable

Surgical - Plates, screws, wires

  • Non-stable:
  • -Ileal - long oblique of the midline (plates required)
  • -Acetabulum - usually with femoral head displacement
43
Q

Surgical oncology. Amuptation of digits and limbs.

A

Type and grading
Benign - well divided cells
Malignant - mesenchymal (sarcoma), epithelial (carcinoma)

Aspects of tumours

  • Structure (differentiation)
  • Growth type (invasion (outside capsule with branches) or expansion (inside capsule))
  • Growth speed (slow or fast)
  • Growth extent (spontaneous or continual)
  • Degree of Mx

Surgical types
Incision biopsy - histopath
excision biopsy - CI’ed as can cause PNPS + metastasis
Intracapsular resection - cannot be fully exsised due to location near a vital organ
Marginal resection - Removal of tumour with no margins (+ chemo and radio)
wide resection - Tumour and a wide section of healthy tissue around the tumour

Amputation 
FL 
-Scapula + humeral resection 
-Scapulectomy 
Humeral amputation 

HL

  • Coxo-femoral disarticulation
  • Mid-femoral amputation

Limb-sparing technique
-<50% of bone, no joint or Mx

Digital amputation

  • For neoplasia, fractures, toe deformities
  • Disarticulation to ensure no adverse reaction to necrotising cartilage
44
Q

Diagnostic and therapeutic arthroscopy. Rehabilitation & physical therapy.

A

Arthroscopy
Visualise joint with minimal risk compared to open arthrotomy

Indications

  • Shoulder
  • Elbow
  • Stifle
  • Tarsus

Procedure
-GA –> fluid egress + camera ingress. File down pathology or remove pathology

Osteoarthritis
-Aseptic: <4g of TP in synovial fluid

Synovia fluid types

  • Non-inf - yellow, low TP
  • Inf - Yellow, opaque, high TP
  • Haemorrhagic - red
  • Infectious - green/purulent

Rehabilitation and physiotherapy
Reduction of pain, increase in mobility and strength. Prevent necrosis or atrophy

Methods
Cryotherapy - lowered inflammation

Heat therapy - chronic joint stiffness (CI’ed in acute)

USG - increases blood and lymph flow

Massage - increases blood and lymph flow

ROM - prevents atrophy

Electrical stimulation - muscle contraction

Exercise - weight baring, wheelbarrow

Hydrotherapy - allows strengthrning without pain due to upper thrust

45
Q

Principles of general anaesthesia. Perioperative pain management and perioperative monitoring. ASA system

A

Induction
Pre-med
-medetomidine + buprenorphine
-medetomidine + midozolam + acepromazine

Induce

  • Propofol: 5mg/kg (0.5ml/kg)
  • alfaxalone: 0.3ml - 0.6ml/kg

Peri-op care
Fluids
Fluids (10mk/kg/hr) and O2
-IV, IM, SubQ, IO, IP, PerOs

Crystalloids
-Water based
-Lactated ringers (0.9% glucose)
KCl - for acidosis from hypokalaemia (diabetes)
Hypertonic - oedema or shock (pulls fluid into vessels)

Colloids

  • Large particals that stay in the blood (regulates oncotic pressure)
  • Hexastarch, albumin, whole blood or plasma

Dosage

  • Maintainence: 50-60ml/kg/day
  • Shock crystalloids
  • -80-90ml/kg (dog)
  • -50-60 ml/kg (cat)
  • 1/2 or 1/3 given over 10-20mins usually
  • Shock colloids
  • -3-5ml/kg/hr
Monitoring 
HR - 60-140 (110)
Temp - 36-38 degrees 
Resp - 6-20 bpm
O2 Saturation - >95%
CRT - <2 Seconds 
BP - 60/90 
General anaesthesia 
Requires:
Analgesia, 
Anemnesis, 
Immobility, 
Unconciousness, 
Muscle relaxation 
Barbituates - old but gold 
Non-barbituates (propofol) - lowered liver damage and clears faster 
Dissociatives - ketamine, zolazepam 
Inhalation - sevo and iso 
--Much safer and faster clearing 
ASA system 
1 - Minimal risk 
2 - slight risk: young, old, obese
3 - moderate risk: fever, anaemia, murmur 
4 - high risk: systemic disease 
5 - extreme risk: Moribund
E - Emergency 

ASA score of >3 are 10x more likely to crash under GA

Pain management
NSAID’s - meloxicam, ketoprofen, carprofen, paracetamol (no cats)
Opioids - buprenorphine, butorphanol, tramdol, morphine

Behaviour is the idicator for pain, some vet associations have made questionaire sheets to assess pain using body positioning and behaviour (willingness to eat or drink)

46
Q

Lameness in dogs and cats.

A

Lameness
Disharmony of swing and stand phase
Causes;
-Pain: usually soft tissue
-Neurological: ataxa / MG
-Mechanical: scarring or shortening of ligaments
-Orthopedic: Fracture, joint incongruency or osteomyelitis
-Metabolic: Ca/PTH/P/Vit D
-Circulatory: Ischemia/infarcts
-Infectious: Inflammation is painful (osteomyelitis)

Grading
0 - Normal weight baring
1 - weight bare at rest (stand), lame on trot
2 - partial weight baring at rest + walk
3 - no weight baring at walk
4 - can weight bare at rest or walk
5 - Reluctant to rise. walks <5 steps

Dx lameness
History 
-When (walk, rest, run)
-Onset
-Which limb
-Timing 
-BCS, Age and breed

Eliminating neurological, arthritis, muscular issues
Proprioception, spinal reflexes, pain responce (or lackthereof)

Observation
Short stride (reduced ROM)
Goose-stepping 
FL - Head bob 
HL - hip raising 

Palpation
Standing - superficial
Laying - deep
Check for crepitus, pain, atrophy, malformed joints

Drawer test

  • Cranial: thumb on patella and finger on tibial crest
  • Looseness compared to femer = torn cranial cruciate
  • Caudal: tibia parallel to ground, if caudal aspect drops = torn caudal cruciate

Tibial compression test
-Pressure on tibia –> popping forward = cruciate torn

Imaging 
X-Ray - breaks
USG - arthritis 
Arthroscopy 
Muscle enzymes 
neurological exam and deficits