Surgery Flashcards

Pre-op, peri-op, post-op care Nutrition and Fluids management. Surgical emergencies

1
Q

What are the 6 Ps of Acute Limb Ischaemia?

A
  1. Pain
  2. Pulseless
  3. Pallor
  4. Perishingly cold
  5. Parasthesia (which supersedes pain)
  6. Paralysis

N.o. 5,6 distinguish it from partial limb ischaemia (which can be treated medically first. Complete ischaemia needs revascularisation within 6 hours.

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2
Q

What is reperfusion injury?

A

Tissue damage as a result of reperfusion (vs ischaemia), due to production of free radicals

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3
Q

Complications of reperfusion (other than reperfusion injury)?

A

Reperfusion washes out byproducts of anaerobic metabolism - e.g. lactic acid - and cell lysis - e.g. K+, myoglobin. This can cause arrythmias, ARDS and acute renal failure (see below)

Cardiac arrythmias - because metabolic acidosis and hyperkalaemia

Acute renal failure: Rhabdomyolysis -> myoglobin -> acute tubular necrosis

Acute respiratory distress syndrome

Compartment syndrome - increased cap permeability + oedema = muscle swelling. Fascia restrict -> muscle necrosis. Treat with fasciotomy.

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4
Q

Open # Rx

A

6 A’s

1) Analgesia - M+M
2) Assess NV status, soft tissue, photo. Likely high energy injury, consider [head] CT.
3) Antisepsis - Wound swab, saline sterile gauze to cover
4) Alignment - reduce/splint. Reassess N+V, imaging - orthogonal, + joint above/below.
5) Antitetanus status
6) Abx - Co-amoxiclav

In theatre, debride, clean with wash-out and fixate externally.

When swelling reduced, fixate internally

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5
Q

Complication of an open #

A

Clostridium perfringens - gas gangrene + Sepsis

Debridement
Clindamycin

Open fractures are associated with high rates of morbidity and mortality
The most common fractures that are open are tibial, phalangeal, forearm, ankle, and metacarpal
Check the overlying area for skin breakdown or tissue loss
All patients with open fractures need antibiotic cover and up-to-date tetanus vaccination
Timely surgical management, with input from plastic and vascular surgery as required, will ensure optimal outcomes

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6
Q

Classification of an open #

A

Gustillo-Anderson

  1. <1 cm and clean
  2. 1-10cm and clean
    3a. >10cm but high energy, but soft tissue coverage (Ortho can manage)
    3b. ^ but not adequate soft tissue coverage (Need plastics)
    3c. All injuries with vascular injury (Need Vascular team as well).
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7
Q

What is compartment syndrome?

A

inc. pressure in a fascial compartment

due to oedema/bleeding

can lead to muscle necrosis

Rhabdomyolysis -> Acute Tubular Necrosis

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8
Q

Symptoms of Compartment syndrome

A

Pain - even passive stretch

Parasthesia

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9
Q

Why is it important to remove pus from septic joint?

A

Pus - neutrophils
Destroy cartilage
Arthritis don’t want

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10
Q

Why intracapsular # matter?

A

Callus formation in joint not ideal

post-trauma arthritis

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11
Q

Types

A

Transverse

Oblique

Spiral

Comminuted

Avulsion

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12
Q

Don’t aspirate prosthetic knee joint that looks septic

A

no idea why

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13
Q

How to distinguish between transient synovitis vs septic arthritis

A

Kocher’s criteria

Non WB
Febrile
ESR raised >40
WBC raised >12000

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14
Q

Chorda equina red flags

A

Back pain
Saddle anaesthesia
Faecal incontinence/loss of anal tone
Urinary retention and subsequent stress incontinence

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15
Q

Shoulder trauma types

A
# Clavicle, prox humeral/humeral shaft # 
Dislocations - humeral and AC joint
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16
Q

Clavicle important points

A

FOOSH. Middle 3rd common site. Rx: Broad arm sling 3 weeks

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17
Q

Humeral shaft

A

Usually FOOSH, oblique and displaced….

Worry about radial nerve damage.

Rx: Collar/cuff

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18
Q

Definition of open #

A

A fracture is ‘open’ when there is a direct communication between the fracture site and the external environment. This is most often through the skin – however, pelvic fractures may be internally open, having penetrated in to the vagina or rectum.

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19
Q

Painful arch

A

Subacromial impingement syndrome refers to the inflammation and irritation of the rotator cuff tendons as they pass through the subacromial space
Presents with progressive pain in the anterior superior shoulder, typically worsening by abduction and relieved by rest
Diagnosis is a clinical one however MRI imaging can be useful to confirm the diagnosis and assess for further complications of the condition
Mainstay of management is conservative, with limited evidence advocating surgical intervention

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20
Q

Olecranon

A

Fractures of the olecranon process are relatively common fractures of the upper limb
Will often follow falling on an outstretched hand, with tenderness over the posterior aspect of the elbow and a potential palpable defect
Lateral and AP radiographs remain the mainstay of initial investigations
Management can be either operative or non-operative, heavily influenced by degree of displacement

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21
Q

What leads to mal/non-union

A
Fragments may have moved
Soft tissue got in the way
Too much movement
Poor blood supply
Infection
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22
Q

Rx Compartment Syndrome

A

Remove all bandages/splints

Lie limb flat (don’t elevate as decreases blood flow)

Monitor pressure in compartment using cathether - if difference between diastolic pressure and osteofacial compartment is less than 30mmHg, immediate decompression.

If you don’t have ^ equipment, then monitor symptoms after removal of bandaging, and if not improved 2 hours, immediate decompression

Fasciotomy. Open and leave for 2 days.

If muscle necrosis, debride
If none, then suture/skin graft

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23
Q

How long does it take for ischaemia to kill off muscle tissue?

A

4-6 hours

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24
Q

Why can compartment syndrome lead to renal problems?

A

Muscle death
Rhabdomyolysis
Acute Tubular Necrosis

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25
Q

Proximal humeral #

What nerve are you worried about?

A

TWO - Radial and (more commonly) Axillary

The axillary nerve is most commonly damaged by trauma to the shoulder or proximal humerus – such as a fracture of the humerus surgical neck.

Motor functions: Paralysis of the deltoid and teres minor muscles. This renders the patient unable to abduct the affected limb.
Sensory functions: The upper lateral cutaneous nerve of arm will be affected, resulting in loss of sensation over the regimental badge area.
Characteristic clinical signs: In long standing cases, the paralysed deltoid muscle rapidly atrophies, and the greater tuberosity can be palpated in that area.

_____________________________________________________

The radial nerve can be damaged in the axilla region by a dislocation at the shoulder joint, or a fracture of the proximal humerus. Occasionally, it is injured via excessive pressure on the nerve within the axilla (e.g. a badly fitting crutch).

Motor functions – the triceps brachii and muscles in posterior compartment are affected. The patient is unable to extend at the forearm, wrist and fingers. Unopposed flexion of wrist occurs, known as wrist-drop.
Sensory functions – all four cutaneous branches of the radial nerve are affected. There will be a loss of sensation over the lateral and posterior arm, posterior forearm, and dorsal surface of the lateral three and a half digits.

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26
Q

Complications of #s

A

CNS ‘n’ VIV

Compartment Syndrome
Nerve damage
Sores - pressure and plaster
Non-union
Vascular injury
Infection
Visceral injury
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27
Q

Rx of anteromedial shoulder dislocation

A

! Axillary N injury

Analgesia
Assess
Radiograph
Reduce
Reassess
Radiograph

Reduction is either traction/countertraction or modified kocker’s (ext. rota, adducted)

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28
Q

What are the types of non-union, and what can be done to treat?

A

Hypertrophic - Bone ends enlarged, so osteogenesis IS taking place, but not enough.

Functional bracing, Pulsed EM fields, Pulsed USS to stimulate along with rigid fixation

OR

Atrophic - Bone ends rounded and sclerosis present. No more osteogenesis will take place without intervention. Need to excise fibrous tissue and sclerotic edge of the bone + bone graft.

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29
Q

Clostridium perfringens infection symptoms

A

Pain
Brown discharge
Tachycardia
SMELL

Rx: Fluid resuscitation, decompress wound/debride

+/- hyperbaric O2 chambers

30
Q

At what point after a fracture and delayed reunion would you intervene ?

A

If there is point tenderness and pain after 6months, internal fixation and bone grafts

31
Q

Methods to immobilise #s

A

External and Internal. Non-op and Op.

External - Splints (relieve pain + prevent further damage); Slings/Collar and cuff; Casts, Traction (gravity, skin, skeletal); cast bracing (plaster cast on thigh and calf, joined by brace). Operative external: Ring fixators for tibia.

Internal - ORIF: Plates and screws (articular/comminuted); IM nails (#s of long bones - femur, tibia, humerus); K-wires (foot, wrist, hand #s)

32
Q

How do bones heal?

A

1) Inflammatory phase
2) Reparative phase
3) Remodelling

Torn tissue + blood vessels = haematoma. Lack of blood supply so bone regresses a bit.

Inflammation + soft callus (temporary join + stability, we don’t want this in joints (!) and fixation reduces the need for a callous).

Consolidation: Soft callus becomes woven bone and is mineralised. This is then replaced with lamellar bone.

Over time this is remodelled.

33
Q

Osteoarthritis ttmt

A

Advice, Exercise, Weight loss, pain relief

1st line: Paracetamol/topical NSAID

2nd Line: Weak opioid

3rd line: Oral NSAIDs + PPi

4th line: Corticosteriod IA

Surgery

Locking/poor QoL - TKR, THR

34
Q

Septic Arthritis organisms

A

Staph aureus/Strep epid (prosthetics)

Prosthetics
Inarticular injection - e.g. for OA
IVDUs
UTIs

35
Q

Septic Arthritis diagnosis

A
Kocker's Criteria
Fever
Non-weightbearing
ESR
WBC
36
Q

Septic Arthritis rx

A

Aspirate under USS - mcs, polarised light microscopy, WCC, gram stain

blood culture
blood test - esr, wcc, crp, lfts, electrolytes
x-ray

37
Q

Pseudogout

A

Positively bifringent
Crystals
Calcium pyrophosphate

38
Q

Gout

A

Negatively bifringent needles

Sodium monourate

39
Q

Septic Arthritis Abx

A

6 week initially IV Flucloxacillin, then oral

40
Q

Spondyloarthropathies

A

PAIR

Psoriatic
Enteropathic
Ankylosing spondylitis
Reactive arthritis

41
Q

Ank Spond

A

NSAIDs

42
Q

Reactive Arthritis

A

Strep A
Chlamydia
Salmonella, shigella, campylobacter

Reiter’s syndrome - conjunctivitis, urethritis, and arthritis

Can’t see, can’t pee, can’t bend your knee

43
Q

Reactive Arthritis rx

A

NSAIDs

44
Q

Gout causes

A

Monoarthritis

Diuretics
Alcohol
Starvation
Surgery
Infection
Trauma
45
Q

Gout ttmt

A

NSAIDs
if contraindicated: colchicine

Steroids

Wait two weeks, then give Allopurinol - inhibits xanthine oxidase.

!Caution! Azathioprine interacts because xanthine oxidase is involved in breaking down azathi. Reduce dose.

46
Q

Gout ttmt

A

NSAIDs
if contraindicated: colchicine
if not getting better
Steroids

Wait two weeks, then give Allopurinol - inhibits xanthine oxidase. Titrate to urate levels.

!Caution! Azathioprine interacts because xanthine oxidase is involved in breaking down azathi. Reduce dose.

47
Q

Gout extraarticular manifestations

A

Tophi

48
Q

Pseudogout - calcinosis in tendons in x-ray

A

Rest, Icepacks, NSAIDs, Colchizine

49
Q

Kocher’s criteria for septic arthritis

A

Non wb
temp 38.5
ESR>40
WBC>12000

50
Q

Risks of surgery

A

Bleeding, infection, DVT/PE, anaesthetic risks, damage to nerves/muscles

51
Q

Where does crohn’s start?

A

Terminal ileum

52
Q

Where does ulcerative colitis start?

A

Rectum

53
Q

Which has skip lesions Crohns or UC?

A

Crohn’s

54
Q

Crohn’s - how deep does it go ?

A

Transmural - therefore fistulas

55
Q

Types of fistulas in Crohns

A

Colovesical

Enterocutaneous

Colovaginal

56
Q

Defnition of fistulas, strictures, adhesions

A

fistula = abnormal connection between two epithelial surfaces

stricture = abnormal luminal narrowing

adhesions =

57
Q

Crypt abscesses are a feature of…

A

Ulcerative colitis

58
Q

UC hx

A

most common manifestation of ulcerative colitis is proctitis, whereby the inflammation is confined to the rectum. Patients will complain of PR bleeding and mucus discharge, increased frequency, urgency of defecation, and tenesmus.

59
Q

Test to differentiate IBS from IBD

A

Faecal calprotein

60
Q

Tests if someone presents with IBD symptoms

A

Routine bloods* (FBC, U&Es, CRP, LFTs, and clotting) are required to examine for anaemia, low albumin (secondary to malabsorption), and raised CRP and WCC.

In the UK, NICE guidelines recommend that faecal calprotectin testing is carried out in patents with recent onset lower gastrointestinal symptoms; it is raised in inflammatory bowel disease, but unchanged in irritable bowel syndrome. A stool sample should be sent for microscopy and culture.

*Liver function tests may become deranged in patients on medical treatment and clotting can become deranged in severe attacks due to the large inflammatory response affecting the coagulation cascade.

61
Q

Extra-articular manifestations of Crohns

A

Musculoskeletal
Enteropathic arthritis (typically affecting sacroiliac and other large joints) or nail clubbing
Metabolic bone disease (secondary to malabsorption)

Skin
Erythema nodosum – tender red/purple subcutaneous nodules, typically found on the patient’s shins

Pyoderma gangrenosum – erythematous papules/pustules that develop into deep ulcers and can occur anywhere (yet typically affect the shins)

Eyes – Episcleritis, anterior uvetitis, or iritis
Hepatobiliary – Primary sclerosing cholangitis (more associated with UC),
cholangiocarcinoma (due to association with primary sclerosing cholangitis), and gallstones

Renal: Renal stones (reduced absorption of bile salts which leads to increased free oxalate)

62
Q

Mucus in poop

A

suspect UC

63
Q

Diarrhoea definition

A

> 3 loose stool/wk

64
Q
Rota virus
Norovirus
Capylobacter
Shigella
Salmonella
E.coli
C dif
A

65
Q

Diverticulosis

Diverticular disease

Diverticulitis

A

A diverticulum is an outpouching of the bowel wall that is composed of mucosa. The are most commonly found in the sigmoid colon yet can be present throughout the large bowel and (less commonly) small bowel.

There are three different manifestations of diverticiulum:

Diverticulosis – the presence of diverticulum
Diverticular disease – symptomatic diverticulum
Diverticulitis – inflammation of the diverticulum
Diverticulosis is present in around 50% of >50yrs and 70% of >80yrs, yet only 25% of these cases become symptomatic. The disease affects more men than women (1.6:1) and is most prevalent in developed countries.

66
Q

GI Symptom red flags

A

ALARM

Anaemia
Loss of weight
Anorexia
Recent onset
Malaena
67
Q

Achalasia

A

Achalasia is a serious condition that affects your esophagus. The lower esophageal sphincter (LES) is a muscular ring that closes off the esophagus from the stomach. If you have achalasia, your LES fails to open up during swallowing, which it’s supposed to do. This leads to a backup of food within your esophagus.

68
Q

What neurological syndrome is associated with lower oesophagel..f.dfmlksnkfsjbgn

A

CREST ??

Calcinosis
Raynauds
Esophageal dismotiliy
Schlerodactyly 
Telangictasia??????????????
69
Q

Oropharyngeal dysphagia

A

Barium swallow

Endoscopy - Abx co-amox prophylaxis

70
Q

Referral for OGD

A

New-onset dysphagia
Aged >55 years with weight loss and either upper abdominal pain, reflux, or dyspepsia
New onset dyspepsia not responding to PPI treatment

71
Q

What is whipple’s procedure

A

Remove bile duct gallbladder
head of pancreas
duodenum

anatamose everything

rx for pancreatic ca

72
Q

Hartmann’s procedure

A

Hartmann’s Procedure

This procedure is used in emergency bowel surgery, such as bowel obstruction or perforation. This involves a complete resection of the recto-sigmoid colon with the formation of an end-colostomy and the closure of the rectal stump.