Sudden Death in Lambs Flashcards

1
Q

List 11 causes of sudden death in lambs other than clostridia

A
Acidosis
Septicaemic pasteurellosis
Systemic pasteurellosis
Polioencephalomalacia (CCN)
Obstructive urolithiasis
Nematodirus and cocci
Red gut
Poisoning
Nephrosis
Trauma
Anthrax
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2
Q

Describe the pathology of septicaemic pasteurellosis

A

Mannheimia haemolytica

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3
Q

Describe the pathology of systemic pasteurellosis

A

Pasteurella trehalosi
Recently weaned lambs
Brought on by stress

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4
Q

What are the clinical signs of septicaemic pasteurellosis?

A

Depression
Pyrexia > 41 degrees
Toxic mm
Dyspnoea

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5
Q

What are the clinical signs of systemic pasteurellosis?

A

Begins with pneumonia which quickly progresses to septicaemia

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6
Q

How is pasteurellosis diagnosed?

A

Hx
CS
PM - oedematous and congested lungs, haemorrhagic LN, petechiae on serosa, hepatitis, pleurisy, pericarditis

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7
Q

How is pasteurellosis treated?

A

1 large dose of Oxytetracycline

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8
Q

How can pasteurellosis be prevented?

A

Vaccination - takes 6-8wks for it to become effective, doesn’t cause ultimate prevention but will reduce incidence

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9
Q

Describe the pathology of acidosis

A

Sudden and new intake of high quantities of fermentable CHO
Sheep and lambs on stubble pasture
Causes ruminal and metabolic acidosis

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10
Q

What are the clinical signs of acidosis?

A

Death

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11
Q

How is acidosis daignosed?

A

Hx
CS
PM - rumen contents = porridge, rancid odour, sloughed epithelium, pH

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12
Q

How is acidosis treated?

A

Is tx cost effective?
Correct acidosis and dehydration with fluids
Give a rumen function stimulant

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13
Q

How can acidosis be prevented?

A

Introduce grain gradually over a period of about 3 weeks, ensure access to good quality roughage.

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14
Q

Describe the pathogenesis of polioencephalomalacia

A

Occurs in adults and weaned lambs
Preceeded by diet change (?), disruption in normal feeding within the past 2 weeks
Induces a thiamine deficiency

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15
Q

What are the clinical signs of polioencephalomalacia?

A
Start by going blind
Wandering
Stargazing
Head pressing
High, stepping gait
Dorso-medial strabismus
Recumbency, seizures
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16
Q

How is CNN diagnosed?

A

Faecal/ruminal fluid/blood sample - decreased erythrocyte transketolase and increased thiaminase
Based on CS and response to tx
PM - cerebral cortex flurousces, bilateral laminar necrosis

17
Q

What are the ddx for CCN?

A

Acidosis, PT, Listeriosis, focal symmetrical encephalomalacia

18
Q

How is CCN treated?

A

IV VitB1 and dexamethasone
Usually improve within 24hrs
May remain blind for 2-3 weeks