Study Q's - SSL Flashcards

1
Q

What percentage of Americans have a sliding hiatal hernia?

A

10-80% (uptodate)
40% hiatal hernia (>60% if you add HHS)

95% sliding / 5% paraesophageal

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2
Q

What differentiates hiatal hernia (HH) from hiatal hernia syndrome (HHS)?

A
upward pressure without protrusion
a picture (endoscopy, barium, CT)

with no visualization, you have a collection of sx -> syndrome

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3
Q

What are the common sx hiatal hernia / HHS?

A
fatigue
anxiety
shallow breathing
chest oppression
stitching pain
tickling cough
easy satiety
reflux / regurg
dysphagia
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4
Q

What are the common dx hiatal hernia / HHS?

A
Onset of sx following:
constipation
pregnancy
illness w/vomiting or violent coughing
new exercise program
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5
Q

What are the common tx hiatal hernia / HHS?

A
  • visceral manipulation (caudal pull)
  • functional breathing
  • NMT or myofascial release (focus at occiput and T10-11)
  • energetic clearing
  • post manip exercises
    • heel drop (drink h20, drop 11x)
    • core exercise
      • leg raise, knee raise
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6
Q

What is the relationship between spinal levels and hiatal hernia syndrome?

A
T 10-11 left paravertebral area
C 0 (occiput)
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7
Q

What are the various options for determining if a pt has hypochlorhydria?

A
  • Heidelberg test
  • gastric string test (screening)
  • riddler’s gastric acid point (fxnl)
  • BL weakness of pec major clavicular (fxnl)
  • clinically
  • HCl challenge
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8
Q

What is the relationship between hypochlorhydria and gastroparesis? How could gastroparesis be life-threatening?

A

poorly digested food can sit in the stomach

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9
Q

How does the temperature of food and drink affect orocecal transit time?

A

cold - slows

hot - speeds/increases motility

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10
Q

What is the explanation for reflux causing sx (including heartburn) if a pt has hypochlorhydria?

A

x

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11
Q

What are the available txs for hypochlorhydria?

A

x

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12
Q

What are the sx/signs that change in the intermediate phase of perforated peptic ulcer that might incorrectly cause you to think that the patient’s condition is improving and that a surgical consult is not needed?

A

vomiting ceases
abd pain lessens
appearance better - regain color
temp and pulse normal

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13
Q

What is the finding found on percussion of the abdomen that is highly correlated with perforated peptic ulcer?

A

resonance over the liver in the midaxillary line 2” above the costal margin

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14
Q

To what location is pain referred from a perforated peptic ulcer?

A

top of shoulder, supraspinous fossa, acromion, clavicle

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15
Q

What is the finding on a plain x-ray film that is a highly likely sign of a perforated peptic ulcer (80-85% of cases are positive for this?)

A

free air btw liver and diaphragm

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16
Q

Summarize the prevention of dysplasia and adenocarcinoma of the esophagus in patients with long segment Barrett esophagus.

A

x

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17
Q

What is NERD? What are dilated intercellular spaces? What is DGER?

A

Non-erosive reflux disease - NERD
DIS - “leaky esophagus”
Duodenal-gastro-esophageal reflux - DGER

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18
Q

Understand the spectrum of GERD including NERD, erosive and non-erosive esophagitis, Barrett esophagus, dysplasia and adenocarcinoma.

A

x

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19
Q

What are the components of the mnemonic – “cut out the CRAP?”

A

C - coffee, cigs, chocolate
R - rx, refined carbs (carbs in general)
A - alcohol, allergic foods, acid foods
P - pop, peppermint, progesterone, packin food in hs

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20
Q

Understand the flowchart of Naturopathic GERD evaluation.

A

x

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21
Q

What is the most distal site that may be assessed by upper endoscopy (EGD)?
What can be assessed, beyond PUD status?

A

first two portions of duodenum

  • H. pylori status
  • celiac disease biopsies (D3, D4)
  • aspirate for SIBO
  • flap valve assessment
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22
Q

Discuss why H. pylori screening of patients who do not have sx of PUD may lead to unnecessary treatment.

A

Because it’s normal flora, and protective against conditions such as GERD, Barrett’s, adenocarcinoma, Crohn’s, etc.
Labs can’t test commensal vs virulent, only research is doing that at this time.

23
Q

What cause of GERD might be managed with phosphatidylcholine and Huperzine A?

A

sphincter tone
mucosal health
GI motility

24
Q

Why might both hyperchlorhydria and hypochlorhydria cause pyrosis?

A

reflux without acid - “neutral” reflux, still get sx d/t DIS (“leaky esophagus”)

25
List the effect of the following on gastric acid levels: a) H. pylori pangastritis b) H. pylori antral gastritis c) early (first 3 months) of any H. pylori gastritis
a) hypo- or achlorhydria b) hyperchlorhydria c)
26
Know the mnemonic PATELLA and the typical order of symptoms in acute appendicitis.
``` P - pain A - anorexia (or N/V) T - tenderness (somewhere) E - elevated temp L - leukocytosis L - lying still A - asleep ```
27
Be familiar with the ascending, pelvic and retrocecal appendicitis presentations. How are they different from the typical iliac appendicitis?
Ascending - QL most likely irritated Retrocecal - little pain or rigidity, early distention Pelvic - epigastric / periumbilical pain, rectal exam, no rigidity Iliac - psoas irritability temp is less than 102.5 before perforation.
28
How is functional testing used to check for open and closed ileocecal valve syndrome? Possible sx? Possible causes?
Sx: RLQ pain, dizziness, chronic right shoulder problems and tinnitus Cx: adrenal imbalances and lack of adequate calcium
29
What is the function of the appendix?
storehouse for commensal flora
30
What are 3 homeopathic remedies for acute appendicitis?
Bryonia Belladonna Iris tenax
31
Why does acute appendicitis usually causes constipation?
irritation of the cecum -> increased IC valve tone
32
What are important differentials in women with ssx of appendicitis? What dx may mimic acute appendicitis in children?
Ddx: Acute salpingitis and ectopic pregnancy! Dx: mesenteric lymphadenitis
33
How are Crohn and ulcerative colitis differentiated (see chart)?
x
34
What is indeterminate colitis?
x
35
What are the 2 forms of microscopic colitis and why are they called microscopic?
Lymphocytic (MC) and Collagenous (rare) | Look normal on colonoscopy, but seen on Bx
36
Understand the difference between ulcerative proctitis and more widespread UC including pancolitis. What exam can you often find it on? What are general sequelae of UC?
``` Proctitis - rectum Sigmoproctitis - rectum, sigmoid Left sided colitis Pancolitis (inc risk of colon cancer, even in remission) anuscopic exam toxic megacolon, systemic effects ```
37
Understand the locations of Crohn disease (ileitis, Crohn colitis) and its sequelae in general.
Crohn's - skip lesions, mouth to anus; Abs to yeast (ASCA) UC - rectum up, contiguous; neutrophil cytokine (P-ANCA) transmural thickening leading to obstruction of the terminal ileum, systemic effects
38
Know the anatomical limits of EGD and colonoscopy. Which type of endoscopy is used to biopsy for Celiac disease? Which is used to definitively diagnose IBD?
EGD - first 2 portions of small intestine | Colonoscopy - to cecum and terminal ileum (have to see terminal ileum to definitively dx Crohn's)
39
Know the common sx/signs of UC and Crohn.
x
40
Know the lab tests that may help in the diagnosis of IBD and its differentiation from IBS. Know that IBD is considered to be autoimmune.
ASCA (yeast abs) - Crohn's P-ANCA (neutrophil cytokine) - UC Neutrophilic hypersegmentation index - folate def.
41
Be familiar with dietary, nutritional supplements, botanicals, and immune suppressives in the treatment of IBD.
specific carbohydrate diet
42
If a pt comes in with ileocecal valve resection, guess what disease they have:
Crohn's - most likely place to obstruct
43
What are the three key symptoms/signs of IBS?
pain (distension)
44
How does the menstrual cycle and post-menopausal status affect IBS symptoms?
x
45
What stool tests may reveal infectious etiologies for IBS?
x
46
What are the alarm signs (red flags) in IBS?
x
47
What non-invasive tests are used to determine need for colonoscopy to r/o more serious dx in pts with IBS-like sx?
x
48
What are 7 clinical indicators that increase the chances of SIBO being the etiology for IBS?
x
49
List the physiological mechanisms by which bacterial overgrowth is normally prevented?
x
50
Briefly describe the typical effects of H2 and CH4 on gastrointestinal motility.
x
51
Which gas produced by SIBO is associated with increasing symptoms of fibromyalgia?
x
52
What is the likely mechanism by which SIBO leads to fat soluble vitamin deficiencies?
x
53
What are the 4 main categories of treatment for SIBO?
x
54
Discuss the MOA and use of enteric coated menthol for IBS
x