Stroke/CVA Flashcards

1
Q

Cerebral thrombosis-

A

blood clot within cerebral arteries or branches

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2
Q

Cerebral infarction

A

Tissue death resulting from ischemia or occlusion of blood supply

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3
Q

Cerebral Emboli

A

composed of bits of matter formed elsewhere that travel through the blood stream where it lodges in a vessel causing occlusion and infarction

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4
Q

Intercerebellar hemorrhage

A

caused by a rupture of a cerebral vessel with subsequent bleeding into the brain

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5
Q

Primary cerebellar hemorrhage

A

non-traumatic spontaneous hemorrhage, often occurs in small blood vessels weakened by atherosclerosis which causes an aneurysm

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6
Q

Subarachnoid hemorrhage

A

occurs from bleeding into the subarachnoid space typically from a saccular or berry aneurysm affecting primarily large blood vessels

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7
Q

Arterial venous malformation

A

a congenital defect that can result in a stroke, characterized by a tangle or arteries and veins with agenesis of interposing capillaries

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8
Q

Anterior Cerebral Artery Syndrome

A
  • Contralateral HEMIPARESIS and HEMI SENS LOSS involving LE more than UE-because somatotopic organization of medial aspect of cortex includes functional area for LE
  • Urinary Incontinence
  • Difficulty with imitation and bimanual tasks, apraxia
  • Abulla (akinetic mutism), slowness, lack of spontaneity, motor inaction, limited initiation (delayed-need to direct them)
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9
Q

Middle Cerebral Artery Syndrome

A
  • Contrateral hemiparesis involving UE, LE less effected and more spared
  • Contralateral hemisensory loss involving face and UE, LE less effected and more spared
  • Motor speech impairement: Broca’s or non-fluent aphasia with limited vocabulary, slow hesitant speech
  • Receptive speech impairement: Wernicke’s or fluent aphasia with impaired auditory comprehension
  • Perceptual deficits: unilat neglect, depth perception, spatial relations, agnosia
  • Limb-kinetic apraxia
  • Contralateral homonymous hemianopsia (visual field defect-lose both sides of one eye)
  • Loss of CONJUGATE gaze to opposite side
  • Most common site of stroke.
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10
Q

Posterior Cerebral Artery Syndrome (peripheral territory)

A
  • Contralateral or bilat homonymous hemianopsia (macular sparing if bilat-lose outside vision/central still functional).
  • Visual agnosia (can’t recognize visual objects)
  • Prosopagnosia (can’t recognize faces)
  • Dyslexia (diffic reading) without agraphia (can’t write), color naming (anomia), and discrimination problems
  • Memory defect
  • Topographic disorientation
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11
Q

Posterior Cerebral Artery Syndrome (Central Territory)

A
  • Thalamic pain (cent-post stroke), spontaneous pain and dysesthesias (tingle/burning), sens impairements
  • Involuntary movements, choreoathetosis (rapid, jerky, very irreg)
  • CONTRALAT hemiplegia
  • Weber’s syndrome (oculomotor nerve paralysis. Ipsilateral but parotic contralateral)
  • Paresis of vertical eye movements, slight miosis (excessive constriction of pupil of eye) and ptosis (droopy eye lid), sluggish pupillary light response
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12
Q

Vertebral Artery Strokes

A
  • Supply the cerebellum, pons, and medulla
  • Sy/Sx- ipsilateral and contralateral presentation, vertigo, nausea, vomiting, dizziness, ataxia (loss of control of body movements, nystagmus (involuntary eye movement), visual changes, upper and lower limb paralysis
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13
Q

Basilar Artery Stroke

A
  • “Locked-in Syndrome”
  • Bilateral CVA of the Pons
  • Commonly from High BP or aneurysms
  • Sy/Sx- Paresis rapidly progressing to tetraplegia (nothing moves), lower bulbar paralysis (CN V-XII), Anarthria (can’t speak or control facial muscles), preserved consciousness and sensation, blinking preserved, 59% of those with locked in syndrome die
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14
Q

Synergies

A

mass movement patterns associated with neurological deficit; primitive automatic, reflexive, stereotyped. When stimulated at one joint the limb responds at multiple joints or all joints. Outside of conscious control. Difficult to manage.

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15
Q

Flexor Synergies: UE FLX SYN

A
  • Scapular retraction/elevation
  • Shoulder ABD, ER
  • Elbow flexion-strongest component
  • Wrist flexion
  • Finger flexion
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16
Q

Flexor Synergies:

LE FLX SYN

A
  • Hip flexion-strong component, ABD, ER
  • Knee flexion
  • Ankle dorsiflexion, inversion
  • Toe extension
17
Q

Extensor Synergies-

UE EXT SYN-

A
  • Scapular protraction
  • Shoulder ADD, INT ROT-strong component
  • Elbow EXT
  • Forearm pronation-strong component
  • Wrist Flexion
  • Finger Flexion
18
Q

Extensor Synergies-

LE EXT SYN-

A
  • Hip ext, ADD-strong component, INT ROT
  • Knee EXT
  • Ankle dorsiflexion-strong component, Inversion
  • Toe Flexion
19
Q

Spasticity

A

a. Hypertonicity
b. Present ~ 90% of stroke patients
c. Occur in antigravity muscles (stand or sit upright)
d. Velocity dependent
e. Spasticity does not equal tone

20
Q

Apraxia

A

the inability to plan and execute coordinated movement. Speaking in L hemisphere. (more common in L hemisphere strokes than R)

21
Q

Ideational apraxia

A

inability to produce movement either on command or automatically, a total breakdown of the ability to conceptualize a task. (hand tooth brush and paste-wont now what to do w/o command)

22
Q

Ideal motor apraxia

A

inability to produce a movement on command but can produce the movement automatically. (tell to brush teeth-don’t know, but give paste and brush teeth)

23
Q

FUGYL-MEYER ASSESSMENT OF PHYSICAL PERFORMANCE

A

a. Impairment based test organized by recovery
b. Scoring 0-2 (0 can’t do the task, 2 independent performance)
c. Test includes separate sections: LE, UE, balance, sensation, ROM, pain

24
Q

FUGYL-MEYER MOTOR SCALE

A

a. Shortened version
b. Only the UE and LE sections of the form
c. Still takes 20 min to give
d. Still consistent with other tests

25
Q

STROKE REHABILITATION ASSESSMENT OF MOVEMENT

A

a. STREAM
b. Clinical measure of voluntary movements
c. 30 item test (UE, LE, Mobility)
d. Can predict discharge destination
e. Can document change over time

26
Q

CHEDOKE_MCMASTER STROKE ASSESSMENT

A

a. Impairment and disability measure
b. Two parts- impairment inventory and activity inventory
c. 14 score items/All has sub scales
d. Not free and pay per use
e. 45-60 mins

27
Q

STROKE IMPACT SCALE

A

a. SIS
b. Self-report measure
c. Meant to assess quality of life post stroke
d. 8 subgroups (last question-how much do you feel you have recovered from your stroke)
e. 30 mins to complete
f. They perform test themselves
g. Questions with yes/no or draw on scale (has many drawbacks for post stroke patients)

28
Q

FUNCTIONAL INDEPENDENCE MEASURE

A

a. FIM
b. Measure of burden of care
c. 15 items
d. Score 7-1
e. Can score a 0 if the task was not completed
f. Medicare requirement
g. Difficult to progress

29
Q

History and Examination

A

aka patient eval

Hx- PMH, PLOF, DME, assistance at home?

Sensation- sharp & 2 pt discrimination

Tone/reflexes- variable based on presentation

MMT- UE/LE

Supine to sit

Sitting balance

Stand pivot or side board to chair

W/C?- cranial nerve integrity, vision, w/c mobility

Stand in // bars- stand balance, steping, wt shift

Litegait- symmetry, step length, stance time, distance, assistance of LE mngmt

Stairs- if cant stand no stairs

Tests and Outcome measures- stream (short), Fugyl-meyer (long), FIM (required)

30
Q

Evaluation

A

evaluate results, identify impairments, identify functional limitations, establish PT Dx, establish prognosis

31
Q

Solid ankle AFO

A

keeps neutral covering malleolus. Doesn’t allow tibia to advance on foot

32
Q

Dorsal spring leaf-

A

allows tibial advance. Spring pops you into DF/some PF

33
Q

DF assist AFO-

A

In DF when in neutral. Not much PF.

34
Q

DF stop AFO-

A

Prevents knee hyper extension

35
Q

Pusher syndrome causes

A

ipsilateral pushing or contraversive pushing

CVA causing injury to the thalamus. Postero-lateral thalamus most likely damaged. ushers always have these effected but could be and not have pushers.

36
Q

Pusher syndrome systems

A

Neglect, apraxia, and aphasia occurs inconsistently with presentation of pusher

visual system and input to vestib system intact

Graviceptive (proprioception to vertical) system appear most affected- postural vertical is disturbed.

37
Q

Pusher syndrome observation

A

individual using uninvolved side actively pushing toward the involved

Actively seek any stabe surface with their uninvolved side to push to involved side

avg of 20 deg tilt present

38
Q

Pusher syndrome tx

A

find a vertical

Perching-raise up to LE in EXT. Can’t push with bad then

Reaching tasks