Stroke Flashcards

1
Q

List of non-modifiable risk factors for stroke? What’s the greatest?

A
  1. Age
  2. Sex (M more than F)
  3. Race (AA’s more than Caucasians than Asians)
  4. FH
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2
Q

Greatest modifiable risk factor for stroke?

A

HTN

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3
Q

CSF produced in brain largely by

A

modified ependymal cells in choroid plexus in lateral, third and fourth ventricles

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4
Q

Pathway of CSF

A

Lateral ventricles to foramina of Munro, third ventricle, aqueduct of Sylvius, fourth ventricle, then foramen of Magendie and Luschka

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5
Q

Which stroke type is more common, ischemic or hemorrhagic? Of the ischemic sub-type, which is most common?

A

Ischemic;

thrombotic

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6
Q

____ % of cardiac emboli go to the brain

A

75%

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7
Q

Superior division of MCA affects what area? Inferior division affects what area? What is most common cause of occlusion of superior division of MCA?

A

Broca’s;
Wernicke’s;
embolus

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8
Q

ACA occlusion results in what deficits?

A

Contralateral weakness and sensory loss affecting distal contra leg;
Could see urinary incontinence with contralateral grasp reflex and paratonic rigidity;
Gait apraxia

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9
Q

What CN’s are supplied by the interpeduncular branches of the PCA?

A

CN III and IV

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10
Q

Where are nuclei for CN I and II located?

A

Forebrain

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11
Q

Triad of medial medullary syndrome?

A
  1. Ipsilateral CN 12 palsy
  2. Contralateral hemiplegia
  3. Contralateral lemniscal sensory loss
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12
Q

Region of brain affected when one has dysarthria/clumsy hand?

A

Internal capsule (anterior limb), and basis pontis

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13
Q

Most common location for hypertensive ICH?

A

Putamen; also thalamus, pons, cerebellum, cerebrum

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14
Q

SAH: where are aneurysms most likely to occur? When most likely to rupture? Peak age for rupture?

A

Anterior part of circle of Willis;
10 mm or larger;
5th or 6th decade

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15
Q

Risk of rebleeding within 1 month of SAH?

A

30%

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16
Q

What is most common presentation of AVM rupture?

A

Hemorrhage, usually parenchymal;

could be seizures, HA’s

17
Q

Ischemic stroke BP management, what are IV agents used? What about hemorrhagic?

A

Labetalol, nicardipine, clevidipine;

labetalol

18
Q

Exclusion criteria for tPA?

A
  1. Head CT positive for blood
  2. BP over 185/100 despite medical treatment
  3. Anticoagulation (INR over 1.7, on warfarin, etc)
  4. Plt less than 100k
  5. Glucose less than 50 or over 400
  6. H/o stroke or severe head injury last 3 months
  7. H/o ICH, AVM, aneurysm
  8. H/o GI or GU bleed last 21 days
  9. Major surg within past 14 days
  10. Seizure at onset of stroke
  11. Acute MI
19
Q

Best reason to anticoagulate after stroke?

A

Cardiac emboli

20
Q

CEA for symptomatic lesions and greater than ______ % stenosis

A

70

21
Q

What can help decrease cerebral vasospasm after SAH?

A

Nimodipine

22
Q

What is concept behind proprioceptive neuromuscular facilitation?

A

Uses spiral and diagonal components of movement rather than traditional movements in cardinal planes of motion with goal of promoting functional relevance

23
Q

Goal behind Bobath technique?

A

Normalize tone;
inhibit primitive patterns of movement;
facilitate automatic and voluntary reactions prior to normal movement patterns

24
Q

Goal behind Brunnstrom approach?

A

Uses primitive synergistic patterns in training in attempt to improve motor control through central facilitation

25
Q

Goal behind sensorimotor/Rood approach?

A

Modification of muscle tone and voluntary motor activity using cutaneous sensorimotor stimulation

26
Q

CIMT requisites?

A

At least 10 degrees active wrist extension, 10 degrees thumb abduction/extension, at least 10 degrees extension in at least 2 additional digits

27
Q

What phase of recovery after stroke is shoulder pain most likely to develop?

A

Spastic

28
Q

When is stellate ganglion block considered successful?

A

Ipsilateral Horner’s

29
Q

Sling for shoulder subluxation indicated?

A

Routine use is not indicated

30
Q

Gold standard for evaluating dysphagia? What other studies can be used?

A

VFSS (modified barium swallow);

FEES (visualize vocal cords)

31
Q

Predictors of aspiration on bedside swallow

A

Abnormal cough, cough after swallow, dysphonia, dysarthria, abnormal gag, voice change after swallow

32
Q

Four phases of swallowing?

A
  1. Oral prep phase
  2. Oral phase
  3. Pharyangeal phase
  4. Esophageal phase

OOPE

33
Q

What’s required in oral phase of swallow?

A
  1. Tension in labial and buccal musculature to close anterior and lateral sulci
  2. Anterior-posterior tongue movement to transport bolus to pharynx
  3. Soft palate elevation and velopharyngeal port closure to close off nasal cavity
34
Q

When is aspiration most likely to occur? What does this phase require?

A

Pharyngeal phase;

  1. laryngeal elevation with forward movement of hyoid bone and folding of epiglottis
  2. Adduction of ventricular and true vocal cords to protect the airway
  3. Coordinated pharyngeal constriction and cricopharyngeal relaxation to transport bolus into esophagus
35
Q

Strategies to prevent aspiration

A

Chin tuck;

head rotation to PARETIC side; tilt to strong side

36
Q

What intervention can be used for aphasia recovery?

A

Melodic intonation therapy: recruit right hemisphere for communication by incorporating melodies or rhythms with simple statements; particularly good for non-fluent Broca’s

37
Q

When does greatest amount of recovery occur with aphasia?

A

First 2-3 months

38
Q

Negative factors for return to work post-stroke

A
  1. Low score on Barthel index at time of rehab discharge
  2. Prolonged rehab length of stay
  3. Aphasia
  4. Prior EtOH abuse