Stroke Flashcards

1
Q

What are the benefits of admission to a specialist acute stroke unit for all suspected stroke?

A

Early recognition and treatment of complications.
MDT working
Co-ordinated and organised in-patient care with weekly MDT meetings
Involvement of carers in rehabilitation
Staff interest and expertise

Patients are more likely to receive measures to reduce aspiration, early nutrition, shorter length of stay, less likely to die and more likely to discharge independent

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2
Q

What is aphasia

A

inability to comprehend or formulate language. Comprehension and formulation of language is affected.

Generally with left hemisphere stroke. Traditionally Broca’s or Wernicke’s are the site of problem, bzut can also be right hemisphere.

Reading and writing is affected. Gestures and drawings as well

difficulties with communication affect how a person maintain and sustains their relationships, how they control their live through making choices and how they see themselves

The emphasis should be on us to provide help rather than relying on the person with aphasia to produce alternative ways of communicating. without this support someone with aphasia may not be able to participate in discussion or consent to treatment, but with it they may be enabled to reveal more ability than appears on the surface

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3
Q

What is dysphasia?

A

Expressive: understands language, but cannot find the right words. Recognises incorrect language. Reading and writing may be affected

Receptive (Wernicke’s):
inability to understand language. Does not recongize error in speech. Reading and writing unaffected

there is often a combination of expressive and receptive dysphasia, as the two areas are closely related anatomically

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4
Q

What is dysarthria?

A

know what they want to say, but cannot get the words out corretly (weakness of tongue or facial muscles).
Flat sounding voice. May sound slurred

Understanding, reading and writing not affected

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5
Q

What is apraxia

A

Difficulty in performing tasks, despite intact motor function

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6
Q

What is asterognosis

A

Inability to identify objects in both hands by touch alone, despite intact sensation

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7
Q

What is agnosia

A

Inability to recognize objects

Persons, sounds, shapes or smells

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8
Q

What is inattention

A

Inability to attend to stimuli bilaterally, despite intact sensation

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9
Q

What is dyspraxia

A

Dyspraxia affects the person’s ability to respond voluntarily in conversation, but they may be able to do things automatically

Can greet normally, but then unable to answer any questions

Generally, unable to repeat things

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10
Q

How is dysphagia recognized and managed?

A

Doctors and nurses are often the first member of a team to notice. Nurses can be trained to carry out intitial screening assessment.

Speech and Language therapist (SALT) does further assessment to check risks for oral intake.

Physiotherapist will assess appropriate positioning for swallowing (sitting balance, head control)

Occupational therapist (OT) will assess seating for eating and drinking to ensure optimum positioning. Look at hand-mouth coordination and adaptations to help

Dietician will assess and monitor intake to ensure nutritional requirements are met

The healthcare Assistants and nurses may feed/supervise the patient when eating/drinking.

The support team assistant needs to be aware whether the patient is on a modified diet or thickened fluids and give appropriate meals/drinks

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11
Q

What are the signs of dysphagia

A

coughing
choking
becoming very short of breath (aspiration)

however, some people can aspirate silently and there are no obvious clinical signs to indicate this

Silent aspiration can only be detected using videofluoroscopy (similar to a barium meal)

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12
Q

What measures can be taken to reduce the chance of aspiration in dysphagia

A

Modified diet and/or thickened fluids to minimise the risk

Risk may be considered too high: NBM
Non-oral feeding needs to be considered. eg. NG tube or PEG

Considering the risk of malnutrition is as important as considering the risk of aspiration. Taking oral drugs are also affected. Need to find a different way

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13
Q

Management of daysphagia..

A

Consistency of food
Quantity of food (small portions)

Strategies such as chin tuck, extra swallows

Positioning, alertness

Therapy

Advise re prognosis. Liaison re non-oral feeding

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14
Q

Other than dysphagia, what other dysfunction may arise from a parietal lesion?

A

Sensory neglect (when bilateral confrontation)

Agnosia

Astereognosis

Dyspraxia (problem with performing tasks)

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15
Q

What are the risk factors for stroke

A
HTN
Hypercholesterolaemia
DM
Smoking
Alcohol
Dietary
Low exercise
Increased weight
AF
Drugs - illicit, Warfarin
Increasing age - but young people can also have stroke
Male
Personal Hx of stroke, TIA or migraine
FHx of stroke or clotting disorders
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16
Q

Describe the oxford classification of stroke

A

Neurological deficit of cerebrovascular cause that persists beyond 24hrs or is interrupted by death within 24hrs

TIA - persists less than 24hrs

Total anterior circulation stroke (TACS) - 20% of stroke

Partial anterior circulation stroke (PACS) - 35% of stroke

Lacunar stroke (LACS) - 20%

Posterior circulation stroke (POCS) - 25%

17
Q

Describe total anterior circulation stroke

A

ALL OF THESE

•Form of higher dysfunction:
Dysphasia
Reduced consciousness
Visuospatial neglect
Asterognosis
  • Homonimous hemianopia
  • Motor/sensory deficit (>2/3 of face/arm/leg)
18
Q

Describe Partial anterior circulation stroke

A

Either:

Higher dysfunction alone (dysphasia, reduced consciousness, visuospatial neglect, asterognosis)

Or

2 out of 3 of TACS (higher dysfunction, homonymous hemianopia, motor/sensory)

19
Q

Describe lacunar stroke

A

Any one of: (with 2 or more of face/arm/leg)
Pure motor
Pure sensory
Sensorymotor

Or
Ataxic hemiparesis

NONE OF THESE:
new dysphasia, visuospatial problem, proprioceptive sensory loss only
No vertebrobasila features

20
Q

Describe POCS

A

Any one of these present:

Cranial nerve palsy AND contralateral motor/sensory deficit

Bilateral motor or sensory deficit
Conjugate eye movement problems
Conjugate eye movement problems
Cerebellar dysfunction
Isolated homonymous hemianopia
21
Q

What are the risk factors for intracerebral haemorrhage

A
On anti-coagulation
Bleeding tendency
Depressed consciousness
Severe headache
HTN +++
Vomiting
BM >11
22
Q

Management of intracerebral haemorrhage

A

Reverse anticoagulants
Stop antiplatelet
Potential rapid deterioration:
roles of neurosurgery/ITU

Lower BP <140/90 ASAP!! (normally they can be very hypertensive)

23
Q

Pre-hospital stroke assessment and later assessment

A

FAST (Face. Arms. Legs. Time)

Confirm history
Exclude mimics:
migraine
SOL
Seizure
Syncope
Metabolic disturbance (hypoglycaemia)

Peripheral neuropathy
Transient global amnesia
Psychiatric conditions

Time of onset is important for thrombolysis

Areas that can be affected usually show negative symptoms: eg. loss of:
Motor
Speech
Vision
Sensation
Coordination
Conscious level
Memory
24
Q

Which scale can be used for quick stroke assessment

A

National Institute of Health Stroke Scale (NIHSS)

Provides insight into location of stroke and severity

Identifies those who would benefit from thrombolysis

25
Q

Investigations in suspected stroke

A
AIM:
define arterial territory
pathology
exclude stroke mimics
guide further investigation
Aids treatment strategies
Aids prognostication

RCP guidelines: all strokes to be scanned within 24hrs (soon to be 12hrs)

CT Brain is easily accessible, fast and has a sensitivity for bleeding, but it does have a high radiation burden. CRITICAL TO EXCLUDE HAEMORRHAGE

Bloods:
FBC, U and E, LFT, TFT
Glucose
Lipids
Coagulation
ESR
Thrombophilia screen
Vasculitic screen

ECG: AF, LVH, ischaemic changes

Echo - valvular disease (SBE), mural thrombus, LVH

26
Q

Indications for urgent CT brain

A
Thrombolysis?
On anti-coagulation
Bleeding tendency
Unexplained progressive fluctuating symptoms
Depressed conscious level
Suspicion of SAH
27
Q

What are the indications for thrombolysis

A

Alteplase (0.9mg/kg)

Up to 4.5hrs
Clear time of onset
Clinical symptoms and signs of acute stroke
Haemorrhage excluded
Age - no upper age limit
28
Q

Contraindications for thrombolysis

A

Rapidly improving or minor stroke symptoms
Stroke or serious head injury 3 months
Major surgery. obstetrical delivery, external heart massage last 14 days
Severe haemorrhage last 21 days
Bleeding tendency
History of central nervous damage (neoplasm, haemorrhage, aneurysm, surgery, haemorrhagic retinopathy)

29
Q

What is a carotid endarterectomy?

A

Incision is made to open the carotid artery. Plaque is removed,. Then, the artery is repaired and closed.

Carotid endarterectomy may prevent a stroke if the carotid is severely narrowed

30
Q

Which antiplatelet drugs are used for prophylaxis

A

Acute: Aspirin 300mg PO/PR for 2 weeks

Stroke - clopidogrel 75mg

TIA - clopidogrel 75mg

31
Q

Which anti-coagulants can be used for secondary prevention of stroke

A

Heparin (UF/LMWH), but increases risk of bleeding

Warfarin for AF

CHAD2VASC score
Anti-platelet for 2 weeks then switch to clopidogrel

32
Q

DVT prophylaxis

A

Because of being bed-bound or less active.

Enoxaparin - started on day 3 in ischaemic stroke

TED stockings. But it increases risk of skin complications

Mechanical compression stockings have evidence for use in stroke

33
Q

What are the potential complications of stroke management

A
DVT
Pulmonary embolism
Aspiration and hypostatic pneumonia
Pressure sores
Depression
Seizures
Incontinence
Post-stroke pain