Stroke Flashcards

1
Q

Stroke occurs when there is _________ to a part of the brain that reults in the death of brain cells

A

Ischemia

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2
Q

Stroke is the ____ leading cause of death in canada

A

3rd

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3
Q

Cerebral blodd lfow myust be mainatianed at ______ % of cardiac output

A

20%

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4
Q

Cellular death in the brain can occur in as few as ______ in the event of no blood flow

A

5 minutes

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5
Q

Cerebral autoregulation

A

Changes in diameter of cerebral blood vessels in response to changes in pressure so the pressure in the brain stays constant
May be impaired following cerebral ischemia

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6
Q

Circle of Willis

A

A redundancy of the brain that allows blood to continue to flow through the brain, even if a major vessel is blocked

Not necessarily entirely effective, especially over long period of time

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7
Q

Common areas where athersclerosis will build up

A

Plaque builds up along forks in the blood vessels

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8
Q

How does HTN affect blood flow to the brain?

A

Reduceds blood flow by vasoconstirction and vessel stiffening (stenosis)

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9
Q

Factors affecting blood flow to brain

A

Cardiac output (Must decrease by 1/3 before brain is affected
Blood viscosity (Increased = less flow to the brain)

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10
Q

What causes cerebral ischemia

A

Thrombosis, mostly caused by athersclerosis

Plaque builds up in stiffened vessels, platelets form thrombus on plque until vessel is obstructed

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11
Q

Embolic stroke

A

Embolus that forms outside of the brain (usually plaque), then travels up and occludes vessel in brain

associated with chronic Afib
- Blood pools in atrium forming small clots that travel through circulation and enter brain

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12
Q

Sources of embolism

A

Afib (Allowing for pooling of blood in atrium forming clots)

Mechanical valve (rougher surface increasing risk for clots)

Bacterial/non endocarditis

Tumors that break

Air

Fat

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13
Q

Hemmorhagic Stroke

A

Rutprue of vessels caused by increased pressure

Most common cause is HTN

Slowest recovery

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14
Q

Ischemic strokes include

A

TIA (precursor)
Thrombotic (develops in the brain)w
Embolic (embolus

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15
Q

Hemmorrhagic Stroke, types and commonality

A

Intracerebral
Subarachnoid (bleeding in this space)

15% of all strokes

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16
Q

Anuerysm

A

Pouching of artery due to HTN

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17
Q

Which type of stroke results in decreased crervral perfusion and incread ICP?

A

All strokes can

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18
Q

TIA

A

Precursor

Temporary loss of neuro function

Usually lasting less than 15 minutes

Don’t know if it’s going to resolve or if it will progress into full Ischemic stroke

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19
Q

Ischemic stroke

A

85% of strokes

61% are thrombotic

24% are embolic

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20
Q

Symptoms of embolic strokes

A

Symptoms sudden, no chance to develop collateral circulation

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21
Q

Thrombotic stroke symptoms

A

Slow and progressive

may not have changes in LOC in 1st 24 hrs.

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22
Q

Extent of stroke dependent on

A

Size of lesion
presence of collateral circulation

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23
Q

Prognosis of embolic stroke?

A

Dependtant on amount and location of brain tissue affected

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24
Q

Intracerebral stroke

A

10% of H strokes

HTN is sig risk factior

Occurs DURING activity

Sudden onset with progression of symptoms of min-hours b’c of bleeding
- headache, n/v, sudden alteration in LOC, hypertension
Poor prognosis

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25
Q

Subarachnoid stroke

A

Intracranial bleeiding into CSF space

Often rupture of cerebral aneurysm (silent killer), can also be trauma or cocaine
headache, n/v, sudden alteration in LOC, hypertension
Poor prognosis

Hemiplagia, neck stiffness, Loss of consciousness

40% will die within first episode

26
Q

Brocas aphasia

A

People with Broca’s aphasia have a clear understanding of language, but have trouble finding and saying the right words.

27
Q

Global Aphasia

A

Loss of expressive and receptive

28
Q

Clinical manifestations of stroke

A

Motor function
Communication
Affect
Intellectual function
Spatial-perceptual alterations
Elimination

29
Q

Risk factors for stroke (Non-mod)

A

Age
Gender
Race
Family Hx (First degree releative)

30
Q

Modifiable Factors for stroke

A

Smoking
Alcohol
Obesity
Inactivity
High Cholesterol
Illicit drug use
Oral contraceptives & HRT

31
Q

Secondary dx that contirbute to increased risk factor for stroke

A

HTN - single most contributing factor to stroke
DM
Heart
Disease/CAD

32
Q

All symptoms of stroke are contralateral EXCEPT

A

pupil dialation

33
Q

Right-brain damage

A

Left hemiplegia
Left side neglect
Spatial perceptual deficits
Tends to deny/minimize problems
Rapid performance, short atention span

34
Q

Diagnostics for stroke (IMPORTANT) **

A

When s/s of stroke appears
- Diag. studies confirm it’s a stroke
- Identify likely cause of stroke
CT is primary diagnostic

35
Q

Spinal tap for a stroke?

A

RBC in CSF

36
Q

LAb diagnostics for stroke

A

APT, PT, INR (Low more at risk for stroke, high means more at risk for bleeding) Platlets

37
Q

Collaborative Care prevention

A

Management of those with high risk

Drug therapy
Antiplatelet drugs
Aspirin
Statins

Surgical therapy
carotid endarterectomy.
transluminal angioplasty.
stenting.
extracranial–intracranial bypass.

38
Q

Carotid endarterectom

39
Q

Brain Stent

40
Q

Hallmark s/s of stroke

A

Weakness/numbness
Trouble speaking
Vision problems
Headache
Dizziness

41
Q

Goals in stroke response

A

Preserving life
Prevent further brain damage
Reduce disability

42
Q

Single most improtant point in a stroke pt hx is___hours after onset

43
Q

What is important to assess when you suspect a pt has a hd a stroke

A

ABCs
VS
GCS (Neuro exam), PERRLA, LOC

44
Q

Assessment findings in stroke

A

Altered LOC
Weakness, numbness, or paralysis
Speech/visual disturbances
Severe headache
HR change
Pupil differsences
Resp Distress
HTN
Facial droop
Difficulty swallowing
Seizures
incontinence
N/v
Vertigo
Fever

45
Q

Projectile vomit is a sign of

A

Increased ICP

46
Q

Our stroke intervention goal is

47
Q

Additional interventions for stroke

A

Open Airway (remove dentures)
Start IV
CT scan

48
Q

Why is baseline importnat to establish at onset of stroke

A

To see if they are improving or getting worse

49
Q

Interventions to HTN for ACUTE ischemic stroke

A

Response to maintain cerebral perfusion
- Use of BP medications are not recommended unless BP is markedly increased (systolic >220mmhg or MAP > 130mmhg why?

`

50
Q

Interventions for fluid electrolyte blaance in stroke

A

Adequate hydration to help perfusion

Hypotonic fluid can WORSEN cerebral edema

Fluid restriction (1.5-2L

51
Q

Nursing response to cerebral edema

A

lift HOB
Decrease pain
Avoid seizures
don’t increase metabolsim of brain

51
Q

Preventing aspiration in a stroke pt

A

Raise HOB
Pureed Diet (NPO until swallowing assessment)
Thickened fluids

52
Q

Temperature and the brain

A

Every temp increase is 10% more increase metabolism in the brain

53
Q

Medication intervention for ischemic stroke

A

tPA (alteplase)
Must be given 3-4.5 hours of onset of symptoms
Aspirin
Anticoagulant and antiplatlet

Assess neuro
Assess BP

54
Q

Surg interventions for ischemic

A

Endovasculat TS (allows clot to be removed

55
Q

Interventions for hemorrhagic stroke

A

Keep blood pressure in a normal to high normal range <160
Close monitoring for patients on blood thinner
May have reverse medication
Medication Treatment
Nimodipine
Surgical intervention can be considered
Clipping & coiling

56
Q

Manifestations of acute changes in cerebral perfusion

57
Q

Cushings Triad

A

Systolic BP increase
Pulse decrease
Resp decrease

58
Q

Nursing invterventions for ICP

A

Medically induced coma
Decreased stimulation
Manitool
Hypertonic NS
Steroids (Reduce inflam)
BP meds

59
Q

Why does ischemic stroke still increase ICP

A

primarily due to cerebral edema, which is brain swelling caused by the lack of blood flow to the affected area, leading to an increase in brain tissue volume within the rigid skull, thereby elevating pressure