Stroke Flashcards

1
Q

Motor Learning

A

Acquisition/modification of learned movement patterns over time. Cognitive/perceptual processes code motor programs.

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2
Q

Motor Control

A

Outcome of motor learning; ability to produce purposeful movements of extremities and postural adjustments in response to activity/environmental demands.

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3
Q

Cortical Map Reorganization

A

Form of brain plasticity that recent studies show can play a major role in regaining functional use of a hemiparetic upper extremity after a CVA.

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4
Q

Dynamic Systems Theory

A

Motor control approaches are based on this; views motor behavior as dynamic interaction betw client factors (ie: sensorimotor, cognitive, perceptual, psychosocial), and occupations that must be performed to enact the client’s roles. Based on HETERARCHICAL MODEL in which component (client, environment, occupational performance) is viewed as being critical in a dynamic interaction to support client’s ability to engage in occupation.
Ex: A CVA can lead to changes in client’s sensorimotor, cognitive, and perceptual skills, which affects motor control, engagement in occupation, and ability to master the environment.

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5
Q

Hierarchical Model

A

Contrast to heterarchical model in dynamic systems theory. Views higher centers in CNS as having control over the subordinate lower centers. (ie: neurodevelopmental treatment or proprioceptive neuromuscular facilitation).

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6
Q

Task-Oriented Approach

A

Approach to motor recovery based on dynamic systems principles in which occupational performance and motor recovery are achieved by dynamic interaction of person, environment, and occupations being performed. Uses real objects, environments and meaningful occupations.

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7
Q

GRASP

A

Graded Repetitive Arm Supplementary Program. A self-administered delivery model that improves arm function and upper limb recovery.

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8
Q

Constraint-Induced Movement Therapy (CIMT) - Definition

A

Forced use; therapeutic strategy designed to promote functional use of a hemiparetic UE and credited with speeding up cortical map reorganization. Based on principles of dynamic systems theory and task-oriented approach to acquisition of motor control. Avoids LEARNED NON-USE (neglect) of affected UE.

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9
Q

Shaping

A

Therapeutic strategy using behavioral techniques that approach a desired motor outcome in small, successive increments providing explicit feedback. Allows subjects to experience successful gains in performance with small motor improvement.

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10
Q

Rehabilitation methods for CVA or other neurological insult to the brain

A
  • Biofeedback
  • Neuromuscular facilitation
  • Operant conditioning
  • CIMT
  • Compensatory rehab strategies (often used in combination with one of the above; may cause learned non-use)
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11
Q

How CIMT is practiced

A

In rehab of hemiparetic UE, CIMT forces client to use the more involved UE by immobilizing the less affected UE in a sling, mitt, or combination. Clients then practice using affected UE on an intensive basis for several consecutive weeks by using SHAPING movements with the affected UE. Shown to be most effective in the CHRONIC stage of stroke (at least one year post stroke, not during acute phase), and with
those who have some active wrist/hand movements.

Requires repetitive, supervised, constant practice, and calls for 6 hours of continuous task practice per day.

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12
Q

WMFT and MAL

A

Two measures for effectiveness of CIMT.

Wolf Motor Function Test (WMFT): 15 motor items examining contributions from distal and proximal muscles of arm. Rated from 0 to 5, and timed.

Motor Activity Log (MAL): Assesses activities attempted outside clinical setting. Self-reported, 30-item test administered in an interview format. Rates functional use 0 to 5, and quality of movement 0 to 5.

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13
Q

Robotics

A

Robot-assisted therapies; robot used to assist in initiation of movement, guidance, and resistance to movement and to provide feedback. Studies show sensorimotor training with robotic devices improves UE functional outcomes and motor outcomes of shoulder and elbow.

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14
Q

Mirror-Image Motion Enabler (MIME)

A

Robotic devices developed to enable unrestricted unilateral or bilateral shoulder or elbow movement. Applies force to the affected forearm during goal-directed movements.

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15
Q

Bilateral Training Techniques

A

Technique in which clients practice the same activities with both upper limbs simultaneously. Use of intact limb assists in promoting functional recovery of impaired limb through facilitative coupling effects between UEs. Activation of intact hemisphere facilitates activation of damaged hemisphere through neural networks linked via the corpus callosum. Best used in subacute and chronic phases of motor recovery.

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16
Q

LEFT CVA

A

Causes RIGHT sided deficits!

  • Hemiparesis
  • Aphasia
  • Dysphagia
  • Depression

Acute Phase:
• Right hemiparesis upper and lower body
• Flaccid stage may last much longer which leads to edema
• More significant sensory impairment
• Joints must be supported
• ROM and soft tissue length MUST be maintained
• Aphasia-inability to speak
• Global Aphasia-inability to speak or comprehend
language (spoken or written)
- Words may come out automatically
- When patient concentrates-words jumble
- Patient IS aware that they are unable to speak the
way they are intending

Dysphagia-difficulty swallowing:
• Patient may cough with thin liquids or tough consistencies (meat)
• Silent aspiration-food/liquids go into lungs instead of stomach due to malfunctions in the throat
• VERY dangerous if left untreated
• Swallow study can diagnose

• Due to language deficits, motor impairments and swallowing impairments:

  • Patients often become depressed
  • Emotionally Labile
  • Family members need support and education
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17
Q

Early medical tx for stroke

A
  • Maintenance of open airway
  • Hydration with intravenous fluids
  • tx of hypertension
  • Evaluation/tx of coexisting cardiac/systemic diseases
  • Prevention of DVT (pulmonary embolism most common cause of death in first 30 days post-CVA)
  • Monitor for respiratory problems/pneumonia (increase inspiratory/expiratory muscles and cough effectiveness)
  • Bowel and bladder dysfunction common
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18
Q

Impairment of Trunk Control May Cause:

A
  • Dysfunction of limb control
  • Increased risk of falls
  • Impaired ability to interact w/environment
  • Visual dysfunction secondary to resultant head/neck malalignment
  • Symptoms of dysphagia secondary to proximal malalignment
  • Decreased independence in ADLs
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19
Q

Effects of Stroke on the Trunk

A
  • Inability to perceive midline (spatial relationship dysfunction), leading to misaligned sitting postures
  • Assumption of static postures that do not support engagement in functional activities
  • Multidirectional trunk weakness
  • Spinal contracture secondary to soft tissue shortening
  • Inability to move trunk segmentally (cannot twist)
  • Inability to shift weight through pelvis anteriorly, posteriorly and laterally.
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20
Q

Global Aphasia

A

Aphasia typical with left CVA. Loss of ALL language skills. Oral expression is lost, except for some persistent or recurrent utterance.

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21
Q

Broca’s Aphasia

A

Poor speech production and agrammatism. Manifested as slow, labored speech with frequent misarticulations. Good auditory comprehension except with rapid/lengthy speech. Reading, writing and calculating difficulties as well.

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22
Q

Wernicke’s Aphasia

A

Impaired auditory comprehension and feedback, along with fluent, well-articulated paraphasic speech (word substitution errors). Speech may occur at excessive rate and hyperfluent. Uses many function words. Running speech in meaningless sequence. May mix English with nonsense words. Also has reading, writing, math impairments.

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23
Q

Anomic Aphasia

A

Difficulty with word retrieval. Occurs in all types of aphasia, but can be the only symptom (hence Anomic Aphasia). Fluent, grammatically correct, but accompanied by difficulty finding words. Results in hesitant/slow speech, substitutions. Mild to severe reading, writing, math deficits.

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24
Q

Dysarthria

A

Articulation disorder, in the absence of aphasia, because of dysfunction of CNS speech mechanisms. Paralysis and incoordination of organs of speech, which causes thick, slurred, sluggish sounding speech.

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25
Q

Neurobehavioral Deficit

A

Functional impairment of an individual manifested as defective skill performance resulting from a neurological processing dysfunction that affects performance components such as affect, body scheme, cognition, emotion, gnosis (perception), language, memory, motor movement, perception, personality, sensory awareness, spatial relations, and visuospatial skills.

Determine which of these deficits is blocking a CVA patient’s function of occupations? OT interventions may be based on adaptive/compensatory approach or restorative/remedial approach.

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26
Q

Examples of Neurobehavioral Impairments:

A
  • Spatial dysfunction
  • Spatial neglect
  • Body neglect
  • Motor apraxia
  • Ideational apraxia
  • Organization and sequencing
  • Attention
  • Figure-Ground
  • Initiation and Perserverence
  • Visual agnosia(inability to interpret sensations)
  • Problem solving
27
Q

Methods to Transfer Learning to Other Contexts

A
  1. Vary treatment environments
  2. Vary nature of task
  3. Help clients become aware of how they process info
  4. Teach processing strategies
  5. Relate new learning to previously learned skills
28
Q

Levels of Learning Transfers

A

Near Transfer = between 2 tasks that have 1-2 differing characteristics (possible for all pts with brain injury)

Intermediate Transfer = transfer learning to task that varies by 3-6 characteristics (only in pts with localized lesions and good cog skills and training)

Far Transfer = to task conceptually similar but has one or no characteristics in common (only in pts with localized lesions and good cog skills and training; not for diffuse injury/severe cog deficits)

Very Far Transfer = spontaneous application of what was learned in tx to everyday living (only in pts with localized lesions and good cog skills and training; not for diffuse injury/severe cog deficits)

29
Q

Reasons CVA Clients Do Not Integrate Affected UE:

A

1) Pain
2) Contracture/deformity
3) Loss of selective motor control
4) Weakness
5) Superimposed orthopedic limitations
6) Loss of postural control to support UE control
7) Learned nonuse
8) Loss of biomechanical alignment
9) Inefficient/ineffective movement patterns

30
Q

Self-Reported Measures of UE Function (examples):

A

1) Motor Activity Log: related to actual use of affected UE outside structured therapy time. Interview format. Things like holding a book, using a towel, steadying oneself.
2) 36-Item Manual Ability Measure (MAM-36): contains 36 gender-neutral common hand tasks; pt reports ease/difficulty of performing.

31
Q

Interventions for No Functional Use of Affected UE

A
  • Teach shoulder protection
  • Self ROM
  • Positioning
32
Q

Interventions for Postural Support/Weight Bearing with Affected UE

A
  • Bed mobility assistance
  • Support upright function (work, ADLs, etc.)
  • Support during reach w/opposite hand
  • Stabilize objects
33
Q

Interventions for Supporting Reach with Affected UE

A
  • Wiping a table
  • Ironing/Polishing/Sanding
  • Smoothing out laundry
  • Applying lotion
  • Washing body parts
  • Vacuuming
  • Locking wheelchair brakes
34
Q

Interventions for Reach with Affected UE

A
  • Multiple possibilities to engage UE in ADLs, leisure, mobility
  • Grade tasks by height/distance reached, weight of object, speed. and accuracy
35
Q

Interventions Shown to Improve Occupational Performance with Motor Control Limitations:

A
  • Repetitive task practice
  • Constraint-induced movement therapy (CIMT)
  • Strengthening and exercise
  • Mental practice (cognitively rehearsing skill)
  • Virtual reality
  • Mirror therapy (mirror placed midsagittally so pt only sees affected UE doing task)
  • Action observation (pt watches another person do task and imitates)
36
Q

Subluxation after Stroke

A

Malalignment caused by instability of GH joint, common after stroke. May be:
• Inferior (head of humerus below glenoid fossa)
• Anterior (head of humerus anterior to fossa)
• Superior (head of humerus lodged under acromion-coracoid)

Weight of arm combined with instability and malalignment contribute to subluxation. To avoid, focus on achieving trunk alignment and scapula stability in position of upward rotation.

37
Q

Abnormal Muscle Activity after Stroke

A

Change of resting state of limb and postural muscles common after stroke. Low tone occurs in acute state of CVA, and limbs/trunk become influenced by pull of gravity. Low/no muscle activity results in deviations from normal alignment. Low tone can cause secondary problems:
• Edema in hand
• Shortening of muscles to support weak limb
• Overstretching of antagonist muscles
• Risk of joint/soft tissue injury during ADLs/mobility (unaware of arm in harm’s way, etc.)

38
Q

Secondary Problems from Spasticity after Stroke

A
  • Deformity of limbs (esp distal end of upper limb)
  • Maceration of palm tissue
  • Possible masking of underlying selective motor control
  • Pain syndromes resulting from loss of normal joint kinematics (ROM feels impinged/painful)
  • Impaired ability to manage BADL tasks (UE dressing and bathing)
  • Loss of reciprocal arm swing during gait
39
Q

How to Prevent Pain Syndromes/Contracture after Stroke

A
  • Protect unstable joints
  • Maintain soft tissue length
  • Positioning programs
  • Soft tissue elongation
  • Orthotics
  • Client management (train client to manage UE through positioning, ROM, etc.)
40
Q

Visual Impairment Remediation Interventions

A
  • Eye calisthenics
  • Fixations
  • Scanning
  • Visual Motor Techniques
  • Bilateral Integration
41
Q

Visual Impairment Adaptive Interventions

A
  • Change in working distance
  • Use of prisms
  • Adaptations for driving and reading
  • Changes in lighting
  • Enlarged print
42
Q

Psychological Manifestations that Occur After Stroke

A
  • Depression (35% of population)
  • Anxiety
  • Agoraphobia
  • Substance abuse
  • Sleep disorders
  • Mania
  • Aprosody (difficulty expressing/recognizing emotion)
  • Behavioral problems
  • Lability
  • Personality Changes (apathy, social withdrawal, irritability, etc.)
43
Q

OT Guidelines to Interventions for Psych Manifestations after Stroke

A
  • Foster internal locus of control related to recovery
  • Use therapeutic activities to improve self-efficacy or confidence in specific activities
  • Promote use of adaptive coping strategies, such as social support, info seeking, positive reframing, and acceptance
  • Promote success in chosen occupations to improve self-esteem
  • Encourage social support networks, such as families/friends, support groups
  • Use occupations to promote social participation
  • Pharmacologic interventions may also help, such as antidepressants, benzodiazepines for anxiety, or neuroleptic meds for post-stroke psychosis.
44
Q

Motor Control Theory

A

Ability to make dynamic changes/responses of body and limb to complete a purposeful activity.

45
Q

Control Parameter

A

(As in Motor Control): Anything that shifts a motor behavior from one manner of performance to another type of performance. Can be internal (strength, vision) or external (location of object, lighting).

46
Q

Mechanoreceptors

A

Receptors of somatosensory system (peripheral) that responds to touch, pressure, stretch, and vibration and is stimulated by mechanical deformation.

47
Q

Chemoreceptors

A

Receptors of somatosensory system (peripheral) that responds to cell injury or damage and is stimulated by substances (neuropeptides) that the injured cells release.

48
Q

Thermoreceptors

A

Receptors of somatosensory system (peripheral) that responds to stimulation of heating/cooling.

49
Q

Nociceptors

A

Subset of somatosensory receptors that sense pain when stimulated.

50
Q

Paresthesia

A

Tingling, electrical, or prickling sensation. Disturbance of somatosensation. (Tinel’s Sign—tapping on volar aspect of wrist to create paresthesia in CTS—is an example).

51
Q

Hyperalgesia

A

Increased pain, may occur during nerve regeneration. A disturbance of somatosensation.

52
Q

Hypersensitivity

A

Increased sensory pain. A disturbance of somatosensation.

53
Q

Dysesthesia

A

Unpleasant sensation that may be spontaneous or reactive to stimulation. A disturbance of somatosensation.

54
Q

Allodynia

A

Pain caused by stimulus that would not normally cause pain. (ie: Complex regional pain syndrome, pain with breeze). A disturbance of somatosensation.

55
Q

Neuropathy

A

Impairment of the PNS (vibration, light touch, proprioception, temperature, and/or pain).

56
Q

Cutaneous/Superficial Sensation

A

Pain, temperature, touch. Distal parts more innervated, creating enhanced fingertip sensation.

57
Q

Moberg Pickup Test

A

Stereognosis test in which: Test 1: patient moves small items (nut, screw, nickel, safety pin…) into a box, timed. Or, Test 2: if deficits do not appear severe in test 1, pt, with vision occluded, identifies item placed in palm and times response.

58
Q

Habituation

A

Decrease in response after repeated benign stimuli (desensitization). Used to treat hypersensitivity.

59
Q

Strategies for Compensation for Sensory Loss (Protective Sensory Reeducation)

A
  • Protect from exposure to sharp/hot
  • Try to soften amt of force when gripping
  • Use built-up handles to distribute gripping pressure
  • Do not persist in activity for prolonged periods; change the tool used and rotate work tasks often
  • Visually examine skin for edema, redness, warmth, blisters, cuts or other wounds.
  • If tissue injury, be careful in tx and avoid infection
  • Maintain skin suppleness by applying moisturizing agents
60
Q

Discriminative Sensory Reeducation

A

For clients with intact protective sensation and minor touch pressure ability. Graded by initially using grossly dissimilar objects (spoon and penny) and working toward similar (dime and penny). Uses Localization and Graded Discrimination.

61
Q

Localization

A

In Discriminative Sensory Reeducation, ability to discern where touch is being felt. Moving touch tends to return before constant touch. Use a pencil’s eraser on hand with pt vision occluded, then have them point to where it was felt. Repeat until improved results. As improvement occurs, stimulus grades up to lighter/smaller.

62
Q

Graded Discrimination

A

In Discriminative Sensory Reeducation, use of levels of difficulty in stimulus (gross stimulus to fine). With pt vision occluded, sequence the following: 1) same or different, 2) how they are same/different, 3) identification of material/object. Use various textures like sandpaper, fabric, nuts/bolts.

Another version is tracing geometrical shape/letter/number on fingertip or small area of hand, and client tries to identify the figure.

63
Q

Graded Motor Imagery

A

Treatment regimen that includes interventions such as mirror visual feedback, imagined hand movements, and laterality training to affect cortical reorganization.

64
Q

RIGHT CVA

A

Causes LEFT sided deficits!

  • Hemiparesis
  • Visual impairments
  • Major cognitive deficits

Acute Phase:
• Left hemiparesis upper and lower body
• Typically flaccid or low tone initially
• Hypertonicity can develop quickly
• Joints must be supported and mobilized
• ROM and soft tissue length must be maintained

Visual Impairments:
• Homonymous Hemianopsia-complete disruption 
• Visual field loss-one or more areas
• Cognitive inattention to left side 
• Diploplia-double vision-initially
Cognitive Deficits:
• Poor or NO insight 
• Poor memory
• Poor safety awareness 
• Poor attention to tasks