Stress Flashcards
What are the two main ways the body responds to stress?
The sympathomedullary pathway
The hypothalamic pituitary-adrenal system
Outline the sympathomedullary pathway/ diagram
Hypothalamus activates sympathetic branch of ANS(preparing body for f/f)
Then it stimulates the medulla of the adrenal gland to release stress hormones -adren+noradren- into the blood stream.
:heart rate and blood pressure increase, blood is directed to muscles, blood sugar level increases, bone marrow produces more white blood corpuscles, increased perspiration, slowing down digestion, muscles tense, saliva and mucus dry up increasing size of air passages to the lungs, the body’s natural pain killers endorphins are secreted and surface blood vessels construct to reduce blood loss in case of injury.
If stressor is short term and goes away, the parasympathetic branch of the ANS is activated. - reducing arousal and reversing the changes brought about by the sympathetic system and stress hormones and the body returns to normal. (Necessary to conserve energy)
But if stressor persists and arousal remains high for while, we eventually become ill. Bc of prolonged high levels of cortisol may weaken the immune system and prolonged high levels of adrenaline increase levels of blood cholesterol - linked to heart disease.
How would the body respond to an acute stressor
by activating the sympathomedullary pathway
How would the body respond to a chronic stressor
By activating the hypothalamic pituitary-adrenal system
What is stress?
A pattern of physiological and psychological responses to a stressor
When a person has to cope with something in the environment and feels unable to do so
Outline the hypothalamic pituitary-adrenal system
Hypothalamus releases corticotrophin-releasing factor, which stimulates the anterior lobe of the pituitary gland to release the hormone Adreno-cortico-trophic hormone into the bloodstream.
Reaches cortex of adrenal gland causing it to release cortisol into the bloodstream, which stimulates the liver to release glucose into the bloodstream, to provide energy.
Outline the role of cortisol
CORTISOL IS A GLUCOCORTICOID.
It affects glucose metabolism by mobilising and restoring energy supplies to power the stress response-meaning that individuals have a steady supply of bloody sugar, thus a constant supply of energy, allowing the body to deal with stressors.
Another positive effect is that it lowers our sensitivity to pain. - negative effect of suppressing the immune system and impairing cognitive function.
Cortisol is in a negative feedback loop. Levels of cortisol are measured by the pituitary and hypothalamus. High levels of cortisol trigger a reduction in both corticotrophin-releasing factor and adreno-cortico-trophic hormone-resulting in a corresponding reduction in cortisol.
Outline the general adaption syndrome
PROPOSED BY SELYE
1. Alarm reaction-stressor is perceived and the physiological responses of the body are activated in preparation for fight or flight
- Resistance-body tried to adapt itself to the stressful situation by resisting the stressor. The individual appears to be coping, but the bodies resources are being consumed at a potentially harmful rate. E.g high levels of stress hormones that could potentially cause damage to heart and blood vessels. The pns is activated to conserve energy for long term bc the stressor is becoming chronic
- Exhaustion- Adaptation to a chronic stressor is now failing. Resources are drained, the immune system may be compromised. Stress-related illnesses such as high blood pressure, coronary heart disease and depression are now likely.
Evaluate GENERAL ADAPTION SYNDROME
+ supporting research evidence - SELYE experimented on rats. He subjected them to stressors such as extreme cold+ surgical injuries and monitored their response. Identified a general response to stress which could not be attributed to other factors.
- not really that general- MASON replicated the study using 7 stressors(monkeys not rats) - levels of urinary cortisol differed depending on the stressor. Extreme cold increased cortisol, extreme heat reduced it.
- male bias in research- females don’t fight or flight, they tend and befriend. Studies using rats suggest that the release of oxytocin in females during the stress response inhibits fight/ flight whilst increasing relaxation and fearlessness.
- ignores psychological factors - much of research carried out on animals. LAZARUS argues we make cognitive appraisals of a stressor by actively working out if it is a threat/ if we can cope. SPEISMAN ET AL asked students to watch a primitive and gruesome medical procedure on film whilst heart rate was measured. Changed to heart rate depended on how individuals interpreted the film.
- lack of resource depletion in the exhaustion stage- recent research shows that sugars and hormones do not deplete under extreme stress, but increase during the exhaustion phase. The current thinking is that it is the increase in stress hormones such as cortisol during the exhaustion phase that leads to stress-related illnesses.
What helps to prevent illness and how
The immune system is our defensive barrier against invading germs and other foreign bodies (antigens).
One line of defence is innate, such as white blood cells (leucocytes) and natural killer cells.
Second line of defence is acquired immunity which is specific to the invading antigen. This includes lymphocytes such as B cells that produce antibodies to destroy the invading antigens; and T cells which destroys cells infected with antigens.
Stress can suppress the immune system directly (immunosuppression) e.g cortisol inhibits production of lymphocytes
Stress can work indirectly by influencing lifestyle patterns that in turn have a detrimental effect on the immune system e.g smoking, drinking
What are the titles of both studies by Kiecolt-Glaser et al into immunosuppression?
Effects of preparing for exams
Effects of looking after relatives that are ill
Outline Kiecolt-Glaser et al’s first study into immunosuppression
Natural experiment using 75 medical students whose blood was sampled one month before their final exams and again on the first day of their final exams.
No. Of T cells was measured as indicators of immune functioning.+ students filled in questionnaires on both occasions which measured psychiatric symptoms, loneliness and life events.
T CELL ACTIVITY DECLINED BETWEEN THE LOWER STRESS AND HIGH STRESS CONDITIONS.
Findings confirm the assumption that stress is associated with reduced immune functioning.
Questionnaire: immune responses were particularly low in ps who reported psychiatric symptoms.
Outline Kiecolt-Glaser et al’s second study into immunosuppression
Longitudinal study comparing health and immune functioning of two groups of people.
Carers looking after a relative with Alzheimer’s disease.
Matched group of non carers
Over 13 months, carers showed an increase in antibodies to EBV. Not the same with non-carers. Carers also had infectious illnesses on significantly more days and higher levels of depression.
Outline Riley
Mice on a turntable for 5hours. White blood cell count decreased.
Outline Wilbert-Lampen et al
ACUTE STRESS
looks into incidences of heart attacks during football matches played in Germany in the 1996 World Cup.
On days when Germany played, cardio emergencies increased by X2.66 compared with a control period. - appears that ACUTE emotional stress of watching your team play football can more than double your risk of suffering a cardiovascular effect.
Evaluation of Wilbert-Lampen et al
+seems to show the effect of acute emotional arousal.
- natural experiment makes it difficult to draw causal relationships.
- gives no indication of the other factors that must be involved. One event is highly unlikely to be the only cause of cardial emergencies.
Outline Yusuf et al
CHRONIC STRESS
Investigated 52 countries, seeking to identify risk factors for CVD’s that exist across different cultures. They compared 15000 people who had suffered a heart attack (myocardial infarction) with a similar number of people who had not.
They found that several chronic stressors had a strong link to MI, including workplace stress and life events. In fact, the contribution of stress was greater than obesity ( but not so much as smoking and high cholesterol). Stress contributes to the development of CVD’s but also makes existing disorders worse.
Evaluation of Yusuf et al
+ international study with large sample size, making generalisation possible
-there is a problem with the retrospective nature of the study. Patients who have experienced a heart attack may be subject to distortion.
Evaluation of research into the role of stress in illness
+ stress can benefit immunity
Rats exposed to mild stressors and found that it stimulated a substantial immune response. Due to lymphocytes flooding into the blood stream and into body tissues in preparation for physical damage. Acute stressors stimulate the immune system and give some protection against short term stress (compared to chronic stressors- the opposite)
~evidence that stress increases risk of heart attack in patients Who already have cardiovascular disorders. Evidence that stress directly causes a patient to develop cardiovascular disorders is more mixed, as many patients to experience a lot of stress never go on to develop cardiovascular disorders.
+ The measure of the immune function is objective and so cannot be biased or subjectively interpreted by the investigator increasing the validity. The experiment to utilise a real life experience of stress which increases its external validity.
-natural experiments therefore, lack of control over the independent variable. Proving that they might in fact be other factors which have affected the results. Study uses specific groups of participants and limits the extent to which the results can be generalised to other groups in society.
-Immunosuppression and cancer
Pereira et al studied women who were HIV+
Women who experienced many stressful life events were more likely to develop cervical cancer than those who experience few stressful life events
Real life application-Kiecolt Glaser and Glasert found that students who took a relaxation training program seriously had better immune functioning during an exam period than those who didn’t bother with it.
Outline rahe et al
Used the social readjustment rating scale to measure correlation between life changes and onset of illness.
Investigated 2500 male US naval personnel over a six month period. Ps were asked to say how many of the life events they had experienced in the past six months. A health record was also kept of each participant during the six months tour of duty, after which they calculated the correlation between total life changes and incidence of illness.
Findings: they found a significant positive correlation of +0.118 between the total LCU Score and illness. The relationship is small but significant.
Evaluation of rahe et al
The correlation they found is weak- significant because large sample size but there may be other factors affecting health as well as life changes.
Cause-and-effect- correlational study, third variable such as anxiety may play a role in stress. For example more anxious people are more likely to report life changes and would be more prone to illness.
Population validity-The sample only included male US Navy personnel; ethnocentric and androcentric. -reduces validity and more difficult to generalise
Individual differences- Study does not take into account individual differences in reaction to stress. Some people may react more to stress than others.
Supporting research-Stone et al
Reliability of recall- rahe used the test-retest message to show a low reliability for the SRRS but other studies have found acceptable levels of reliability
Who devised the hassles scale?
DeLongis et al
Original form had 117 items although shorter versions have been constructed for specific groups.
Some research suggests that the negative effects of daily hassles can be offset by daily uplifts.
Uplifts scale has 135 items that cheer people up.