Stress Flashcards
What is the G.A.S ?
General adaptation syndrome:
-3 stages which explain how the body reacts to stress.
What is the alarm reaction?
the psychological response activated once a stressor is perceived in preparation for fight or flight.
What is resistance?
when the body attempts to react to the stressor.
-physiological activity is higher than normal, so it uses lots of energy.
-the body’s resources are consumed at a potentially harmful rate.
-the parasympathetic nervous system is activated to conserve energy for the developing chronic stress.
What is exhaustion?
when resources are drained as adaptations fail.
-symptoms of sympathetic arousal (like in alarm stage) appear.
-adrenal glands can become damaged and stress related illness is likely.
Describe the sympathomedullary pathway for acute stress.
- the sympathetic branch of the ANS is activated by the hypothalamus when an individual perceives a stressor as threatening.
- sympathetic arousal stimulates the adrenal medulla to release adrenaline and noradrenaline.
What does adrenaline and noradrenaline do the the body?
causes:
- increased heart rate
- tense muscles
- liver convert stored glycogen into glucose (provides fuel for fight or flight response)
When does the sympathomedullary pathway stop?
ends when stress becomes chronic or when the parasympathetic nerve takes over.
Describe the hypothalamic-pituitary-adrenal system for chronic stress?
- HPA takes longer than SAM to activate, but lasts for longer.
- the hypothalamus is activated when the stressor sends a signal to activate the sympathetic nervous system, but also produces corticotropin releasing factor (CRF).
- CRF is detected by the anterior lobe of the pituitary gland, causing the release of adrenocorticotropic hormone (ACTH).
- ACTH levels are detected by the adrenal cortex, which secretes cortisol in response.
What is the role of cortisol?
- affects glucose metabolism by restoring and mobilising energy supplies to power the stress response.
- but, does have damaging effects (e.g: suppress the immune system).
What is the negative feedback loop?
- the HPA is self-regulating via a negative feedback loop.
- levels of cortisol are monitored as the pituitary gland and hypothalamus.
- high levels of cortisol trigger a reduction in CRF and ACTH, resulting in a corresponding reduction in cortisol.
What is the support for G.A.S ?
Selye (1936):
- research support on rats, subjecting them to physical stressors (i.e: extreme cold, surgical injury, etc.).
- same collection of responses (after 6-48hrs) regardless of the stressor.
- tracked rats continuing responses through resistance and exhaustion stages.
What is the challenge for G.A.S ?
Mason (1971):
- replicated Selye’s procedure on monkeys, measuring response to stressors by measuring urinary cortisol (produced by HPA).
- outcomes depended on the stressor, which reduces validity of GAS by showing specific stressors produce specific responses.
What is the strength for the physiological stress response?
Knowledge has practical value:
- Addison’s disease is a rare disorder of the adrenal glands where they can’t produce cortisol, so the body can’t mobilise energy to deal with a stressor.
- lack of cortisol can trigger a life-threatening Addisonian crisis (mental confusion, abnormal heart rhythm, drop in blood pressure) when a stressor occurs.
- knowledge has revolutionised treatment, where individuals self-administer daily cortisol replacement therapy.
- they should be aware of stressful situations when they might need an ‘extra’ injection.
What is a weakness for the physiological stress response?
Ignores psychological factors in stress like cognitive appraisals:
Speisman et al. (1964)
- asked students to watch a gruesome medical procedure on film, while heart rates were measured.
- changes to heart rate depended on how the students interpreted what was happening.
- but if told the procedure was part of a voluntary and joyful rite of passage, heart rate decreased, which can’t be explained by physiological theory.
How does immunosuppression happen?
- chronic stress produces cortisol continuously (during activation of the HPA), which interferes with the production of antibodies and so, reduces the immune system’s ability to fight off antigens