Spine Flashcards

1
Q

Identify the anatomic structures of the entire vertebral column. (Concave vs convex; number of vertebrae; label vertebrae in coronal and lateral views)

A

xx

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2
Q

Spondylolithesis

A

slipping of one vertebra on another

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3
Q

Spondylolysis

A

pars interarticularis defect

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4
Q

Spondylosis

A

degenerative change, osteophytes, disc space changes

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5
Q

Identify which carcinomas are known to metastasize to the spine

A

Multiple myeloma: Lung 31%, Breast 24%, GI 9%, Prostate 8%

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6
Q

Cervical strains

A

aka Whiplash, Result of sudden hyperextension followed by hyperflexion of the neck.

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7
Q

Clinical Presentation of Cervical Strains

A

Most common cause is from rear-end collisions, sports trauma, repetitive C-spine stretching beyond their physiologic capacity, generating inflammation within the local soft tissues.

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8
Q

Symptoms and Exam of a patient with a Cervical Strain

A

Neck pain, trapezial pain, back pain, muscle spasm, heacache, and limited ROM. (*bogginess of post neck musculature, c-tissue edema (not pitting!), and limited ROM secondary to m. spasm). Often begin ACUTELY, hours after the injury (usually the sx occur the next day). Very minimal long-term sequelae.

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9
Q

Diagnosis of Cervical Strains

A

CLINICAL! Based on H and P and simple tests. Use Xray to help exclude more severe injuries.

NLC- Low Risk Criteria and CCR- Canadian C-Spine Rule. Both the CCR and NLC are proven effective with excellent negative predictive values. Helical CT is faster with better sensitivity (but $$). MRI-used for soft tissues, spinal cord, and ligament.

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10
Q

Physical Exam of Cervical Strains

A

PE- ALWAYS assume DANGEROUS underlying patho and consider c-spine strain ONLY as dx of exclusion. Many times, pt will present w/ C-collar.

First- assess pt for: sobriety, cooperatively, and presence or absence of distracting injury. Check neck for skin integrity, edema, eccyhymosis, and asymmetry. Next, perform detailed neuro exam in UE and LE and then test Cervical ROM.

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11
Q

Treatment for Cervical Strains

A

Clinical course is benign. Most pts will be able to return to normal function in 1-2 weeks. Heat and Ice packs are beneficial (altering 2 for 15-20 minutes per session). C-collar and time off from work are not recommended for C-strain. NSAIDS and analgesia (pain control), Encourage normal use as tolerated, PT.

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12
Q

When to refer

A

Persistent pain over several weeks, abnormal neuro exam or abnormal radiologic findings.

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13
Q

Cervical Disc Degeneration

A

Common cause of neck pain in the elderly, showing decreased ROM, and stiffness. It’s MUCH LESS COMMON than a disc degeneration of lumbar spine (because it’s subjected to much less force). It’s caused from Wear and Tear over time!

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14
Q

Clinical Presentation of Cervical Disc Degeneration

A

Low-grade neck pain, stiffness, and instability. Can present with numbness, tingling, weakness in the neck, arm, or shoulders→ from nerves being irritated/pinched.

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15
Q

Imaging for Cervical Disc Degeneration

A

X-ray to help define problem more clearly. They show a decrease in intervetebral disc space, osteophyte formation, and loss of normal cervical lordosis. Get flexion, extension, AP (for tumor, fractures), and lateral view (anterior osteophytes and disc space narrowing). Use MRI for pts with neuro symptoms.

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16
Q

Physical Exam of Cervical Disc Degeneration

A

Ask the pt to perform flexion, extension, and rotational movements (report if pain decreases/increases). Can become so severe that surrounding osteophytes may encroach spinal canal, leading to spinal stenosis, and myelopathy. Symptoms include stumbled gait, difficult w/ fine motor skills in hands/arms, and shock-type feelings down legs to toes→ NEEDS REFERRAL!

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17
Q

Treatment of Cervical Disc Degeneration

A

Nonsurgical: NSAIDS, PT (freestyle swimming w/ snorkeling gear), Epidural steroids (if needed),
Restoring flexibility helps further repetitive microtrauma, Intermittent C-traction.

Surgical: Only when conservative treatment has failed- Surgery= disc removal, placement of intervetebral bone graft, and fusion. Plate fixation is usually for multiple levels of involvement.

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18
Q

Cervical Disc Herniation

A

Very common cause of neck and arm pain in young adults. Usually caused by traumatic events but can also occur spontaneously

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19
Q

Clinical Presentation ofCervical Disc Herniation

A

Frequent headaches, Pain originating around paraspinal muscle that radiates down one extremity. Finger numbness and/or tingling. Can cause spinal cord compression - disc material pushes directly on spinal cord (more serious- with awkward stumbling gait, tingling, and shock-type feelings down the torso or legs).

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20
Q

Special Tests for the Diagnosis of Cervical Disc Herniation

A

Spurling maneuver→ the head is placed into an extended position, and the patient’s chin is rotated toward the affected side. A compressive force is then placed onto the patient’s head, and symptoms of nerve impingement are reproduced. On the flip side, patients typically get relief of symptoms when asked to place their hands on top of their heads (abduction relief sign)

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21
Q

Physical Exam for Cervical Disc Herniation

A

Perform careful exam of shoulder and wrist to r/o shoulder/wrist pathology. Numbness, tingling, begins in shoulder and extends down into the fingers. ROM is usually limited secondary to pain. Presents with biceps weakness and pain/numbness in index and thumb. Pain can often be reproduced by spurling maneuver.

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22
Q

Imaging for Cervical Disc Herniation

A

Dx studies → X-rays and MRI. MRI is imaging of choice.

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23
Q

Treatment of Cervical Disc Herniation

A

Nonsurgical: NSAIDS, Cervical epidural steroids, Surgical discetomy and fusion.

Surgical: Only when nonsurgical tx has not worked. Removal of disc through and anterior approach or autograft (bone graft taken from the pt’s iliac crest) can be used.

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24
Q

Cervical Spinal Stenosis

A

The NARROWING of spinal cord-may lead to neural compression and radiculopathy or myelopathy. Some of later sx are bowel/bladder incontinence in a rapid progression.

***Note: it’s very important to recognize possible cervical cord compression early to prevent IRREVERSIBLE damage to cord.

25
Q

Etiology of Cervical Spinal Stenosis

A

Stenosis=narrowing of the spinal canal and may lead to neural compression, subsequent radiculopathy or myelopathy

Radiculopathy- root compression and symptoms follow dermatomal pattern

Myelopathy – cord compression and symptoms are more diffuse

26
Q

Special Tests for Cervical Spinal Stenosis

A

Positive Lhermitte sign→ Electric Shock like sensation radiates down spine or extremities w/ certain movements of neck esp flexion/extension.

27
Q

Physical Exam for a patient with Cervical Spinal Stenosis

A

Neck and arm pain, Numbness and tingling, Clumsiness, loss of fine motor skills, and sensory complaints in UEs. Difficulty walking and imbalance in Les Weakness, bowel and bladder incontinence, and proprioceptive dysfunctions=advanced disease.

+ Lhermitte sign, Cord compression-upper motor lesion-hyperreflexia and upper and lower extremities and ankle clonus (Babinski), Hoffman Spurling.

Jaw jerk test-differentiates cervical myelopathy from lesions in the brain

28
Q

Diagnosis of Cervical Spinal Stenosis

A

MRI-diagnositc TEST OF CHOICE/CT scan with myelocram is next best if MRI not available

29
Q

Treatment for Cervical Spinal Stenosis

A

NSAIDs and PT, Epidural steroid injection, Surgical evaluation with abnormal neurologic exam

30
Q

Cause of Cervical Spinal Stenosis

A

Depends on compression of ant or post structures:
Anteriorly herniated disks; ossification of posterior longitudinal ligament (OPLL); and osteophytic spurs from back of vertebral bodies, endplates, or uncovertebral joints are the common culprits of cord and root compression. Osteophytic spurs develop as reactive response to hypermobility of adjacent degenerative disk (extra note: increased stress on articular cartilage and endplates of vertebrae stimulates osteophytic spur formation as body naturally tries to stabilize spine).

Ligamentum flavum is the main culprit causing posterior compression, losing its tension and buckling into the canal as the disc degenerates anteriorly. It may also hypertrophy or ossify to cause more compression to the spinal cord.

Spinal cord may stretch over anterior osteophyte during flexion or be compressed by ligamentum flavum during extension

31
Q

Radiculopathy vs Myelopathy

A

Radiculopathy results from root compression and symptoms usually follow a dermatomal pattern BUT myelopathy is from cord compression and symptoms are more diffuse and usually happen progressively in a step-wise fashion.

32
Q

Side Note On Cervical Spinal Stenosis

A

NOTE!! Not all patients have the same degree of symptoms even with similar levels of cervical stenosis. With age, however, degenerative changes cause the canal to become stenotic, with inability to compensate for the narrowing space. Some congenital cervical stenoses are severe, causing symptoms in patients as young as 30 and 40 years old. Other uncommon but important diagnoses to consider include epidural abscess, amyotrophic lateral sclerosis, multiple sclerosis, syringomyelia, primary or metastatic spinal cord tumors, and stroke.

33
Q

Atlas and axis fractures (Hangman’s Fracture)

A

Atlas and axis fractures (Hangman’s Fracture)

34
Q

Lumbar Strains and Sprains (Whiplash)

A
  • If fever, chills, pain in young individual or pain in someone elderly, weight loss, malaise- MUST be concerned may be infection or malignancy
35
Q

Mechanism of Injury/Etiology for Lumbar Strains and Sprains (Whiplash)

A
  • Heavy Lifting
  • Vibrational stresses
  • Prolonged sitting
  • Job dissatisfaction
  • Obesity
  • Smoking
36
Q

Clinical Presentation of Lumbar Strains and Sprains (Whiplash)

A

Low Back pain, Tension like pain fairly localized, Radiation usually localized if present, Difficulty with standing erect/upright

37
Q

Diagnosis of Lumbar Strains and Sprains (Whiplash)

A

PE and Xrays only if worried about serious underlying pathology.

38
Q

Physical Exam for Lumbar Strains and Sprains (Whiplash)

A

Spasm of paraspinous muscles, Tenderness, Limited ROM secondary to pain. Neuro, strength, sensation, and reflexes normal.

39
Q

Treatment for Lumbar Strains and Sprains (Whiplash)

A

Rest, Ice first 48 hrs and heat thereafter, NSAIDS and Possible muscle relaxants or opioids.

40
Q

Low Back Pain

A

60-90% lifetime prevalence; 10% annual incidence. Most improve with little intervention. 10% develop chronic LBP/disability → utilize up to 90% of resources spent on back pain.

41
Q

Function of the Lower back:

A

Connects lower and upper limbs, Bears weight of the upper body and transmits forces, Stabilization, Shock absorption, Flexibility, Protects the spinal cord/neural structures

42
Q

Diagnosis and Treatment of Low Back Pain

A

Acute if 12w. Most resolves in 4-6 weeks
XRay not necessary unless S/S don’t resolve or red flags are present (then consider MRI)

Pharmacological management = mainstay of tx. Acetaminophen is first line analgesic therapy for acute or chronic LBP. Anticonvulsants-gabapentin for pts suffering from radiculopathy.

43
Q

Patient Presentation and Exam for Low Back Pain

A

Pts usually present with localized pain in lumbosacral area, in some cases with pain radiating into the buttocks.. Palpation of the paraspinal muscles reveals spasms, and motion is limited. LBP that radiates into one or both legs may be due to degeneration fo the annulus fibrosus

Results of the neurologic examination are normal, and the straight leg–raising test is negative. Sensation and reflexes are symmetric.

44
Q

Lumbar Disc Degeneration

A

Intervertebral disks bear at least one-third of the weight of the spinal column. The central portion, nucleus pulposus, is composed of gelatinous material early in life. This material degenerates and becomes fibrotic w/ age and trauma. Degenerative changes lead to disc herniation
Most commonly lumbar spine bc subject to greatest motion and post longitudinal ligament is thinnest at L2-L5

45
Q

Imaging for Lumbar Disc Degeneration

A

Xrays: evidence of degenerative disk disease

46
Q

Risk Factors for Lumbar Disc Degeneration

A

Risk factors: -smoking, sedentary work and vehicle

47
Q

Patient presentation and physical exam for Lumbar disc Degeneration

A

Deep steady pain in mid or low back that may be episodic. Unilateral and may radiate into the buttocks and posterior thigh. Relieved by bedrest and aggravated by acticity. Escalating use of multiple pain meds.

48
Q

Lumbar Disc Degeneration Treatment

A

NSAIDS, bed rest, muscle relaxers, diet, hospitalization is rarely indicated. Alternative tx: massage therapy, PT, chiropractic therapy, yoga, exercise, etc

49
Q

Herniated Disc

A

Much more common in cervical and lumbar spine, than thoracic. L4-5 followed by L5-S1 disks most common sites for disc herniation.

Prognosis: Even with weakness, majority improve within 4 mo - 1 year

50
Q

Presentation of a Herniated Disc

A

“foot slap”, radicular pain and sciatica (pain radiating down leg in dermatomal distribution is most common symptom and is usually at the level of involvement. Leg and buttock pain.
Complain of back or lower extremity pain, extremity weakness, numbness corresponding to the level of the disk herniation or below, and bowel or bladder dysfunction. They may demonstrate a spastic gait, with long-tract signs, if the disk is more central.

51
Q

Diagnosis and Treatment of a Herniated Disc

A

Tests: Straight leg raise test. Dx made by Myelography, sometimes in conjunction with CT or MRI.

Initial flare: 7-10 day prednisone taper
Injections: epidural steroid injection
PT: decrease forces across the nerve, improve biomechanics. Surgery if refractory pain, progressive weakness, significant initial weakness signs of myelopathy.

52
Q

Spinal stenosis

A

Grocery cart sign! (standing upright hurts). Better walking uphill than downhill. Long hx of lower back pain, bilateral leg pain. Can occur by bulging disc

Can be congenital or acquired (more common), more common in men and elderly (>70)

53
Q

Presentation of Spinal stenosis

A

Neurogenic claudication”: narrowing of the central canal → worsened in lumbar extension, more opening in flexion.

Different from vascular claudication (worse going uphill, relieved without having to sit down. Intermittent flares that are stepwise in presentation. May affect 1 or both limbs.

54
Q

Diagnosis and Treatment of Spinal stenosis

A

XRay: diffuse arthritis (spondylosis), OA of facets, spondylolisthesis shown. MRI:imagaing modality of choice disk bulges, and disk- osteophyte complexes (≠ disk herniation). Axial series with narrowing of the central canal.
CT to evaluate spinal elements and allow for accurate measurement of canal dimensions. Dural sac with AP diameter of <10mm = clinical finding for stenosis. All patients should have distal pulses examined as part of overall neuro evaluation.

Can treat with Decompression: laminectomy, PT, neuropathic pain meds, Epidural steroid if not controlled by above. Surgical decompression for refractory sxs or progressive neurologic deficit, or instability

55
Q

Cauda Equina

A

May be traumatic or relatively nontraumatic. Caused by: compression of nerves in spinal canal. Usually massive disc herniation. L4/L5 or L5/S1

56
Q

Presentation of Cauda Equina

A

COMPLAINS OF saddle anesthesia and loss loss of bowel or bladder. Perform rectal exam

57
Q

Diagnosis and Treatment of Cauda Equina

A

MRI finding demonstrates obliteration of the spinal canal, compressing the cauda equina (DON’T GET THE MRI FIRST!) Surgical emergency → decompression.

58
Q

3 types of back pain:

A

Axial, Axial WITH red flags and Radicular.

Pain may originate from the disk, vertebral body, or posterior elements or may be unrelated to spine. intervertebral disk may be pain generator. Posterior portion of annulus fibrosus is innervated by fibers and irritation of the nerve → axial back pain.

59
Q

3 structures without innervation (no pain):

A

Nucleus pulposis, inner 2/3 annulus fibrosis, ligamentum flavum