spinal surgery Flashcards

1
Q

spinal neuroanatomy

A
Cervical  and thoracic central canal
Spinal cord
Spinal cord terminates at L1.
Cervical enlargement c5-T1
Lumbar central canal
Lumbosacral nerve roots 
Lumbar enlargement L2-S3
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2
Q

UMN lesion

LMN lesion

A

Anupper motor neuronlesionisa lesionoftheneuralpathway above the anterior hornofthe spinal cord ormotornucleiofthe cranial nerves. ALower motor neuronlesionisa lesion which affects nerve fibers traveling from the anterior hornofthe spinal cord to the associated muscle(s).

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3
Q

cervical cord compression
thoracic cord compression
compression of the lumbosacral nerve roots

A
Cervical cord compression
Lesion of the CNS
Upper limb involvement (UMN)
Lower limb involvement (UMN)
Sphincter dysfunction (UMN)
Thoracic cord compression
Lesion of the CNS 
Upper limbs are spared
Sphincter dysfunction (UMN)
Lower limb involvement  (UMN)
 Compression of the lumbosacral nerve roots
Lesion of the PNS 
Lower limb involvement
Sphincter Involvement
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4
Q

spinal cord compression
vs
nerve root compression

A
spinal cord compression
Weakness (non myotomal)
Sensory loss (non dermatomal)
Increased tone
Hyperreflexia
Clonus
Extensor Plantar response(+ve babinksi)
 \+ve Hoffmans

nerve root compressionWeakness localized to the specific Nerve root.
Sensory loss localized to the specific nerve root
Muscle wasting (localized)
Fasciculations
Hypotonia
Hyporeflexia/ areflexia
Normal plantar response

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5
Q

SCI

A
Traumatic 
Atraumatic
Spinal Tumours
Epidural haematoma
Spinal infection
Compression related to degenerative narrowing of the central spinal canal
Degenerative cervical myelopathy
Large posterior disc bulge (Cervical or thoracic spine)
Syrinx
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6
Q

nerve root injury

A

Cauda Equina Syndrome (lumbar spine only)
Radicular pain
Sensory radiculopathy
Motor radiculopathy

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7
Q

primary and secondary role of surgeon

A

Primary role
Intervene to preserve neurological function
Surgical removal of a spinal tumour
Surgical fixation of a spinal fracture
Secondary role
Intervene to treat disabling nerve root or axial spinal pain related to specific structural pathology
Surgical fixation of a persistently painful osteoporotic fracture
Discectomy for persistently disabling radicular leg pain

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8
Q

time for spinal problems

A

Persistent spinal cord or nerve root compression can cause irreversible neurological deficits
Small surgical window of opportunity to intervene to preserve neurological function.
Foot drop
Cauda equina syndrome
Spinal cord injuries
It is important to recognize when your patient needs time sensitive care.
Emergency spinal imaging +/- surgical opinion is usually accessed through the Emergency department (ED).

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9
Q

%

A
90% of patients non specific spinal pain
No clear structural cause
No specific surgical target
5-10% specific spinal pathology 
Nerve root symptoms secondary to disc herniation/ spinal stenosis
Deformity
Scoliosis
Degenerative cervical myelopathy
Thoracic myelopathy (Calcified thoracic disc protrusion)
<1% serious spinal pathology
Cauda Equina syndrome
Spinal tumour
Spinal infection
Spinal fracture
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10
Q

role of physio

A

Identify features suggestive of serious pathology
Recognise when an indication for emergency referral exists
Surgical opinion via the Emergency Department
Identify features suggestive of specific spinal pathology
Recognise when an indication for a spinal surgical referral exists.

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11
Q

spinal pathologies requiring surgical management

A
Nerve root compression
Spinal Cord Compression
From the outside (i.e. degenerative stenosis/ vertebral fracture/ vertebral metastasis)
From the inside (i.e. syringomyelia)
Cauda Equina Compression
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12
Q

cauda equina syndrome

A

Prevalence 4 in 10,000 ( in CLBP population)
Usually caused by a central disc herniation or a spinal tumour
It is a surgical emergency
Produced by compression of the sacral nerve roots of the cauda equina ( S1-S4)
All patients with acute or deteriorating back (+/- radicular) pain should be assessed for CES in primary/ secondary care.
NB: Explain the context of your questions to the patients to ensure they understand their relevance.

identify
Perianal, perineal or genital sensory loss.
Difficulty initiating micturition (passing urine) or impaired sensation of urinary flow (if untreated this may lead to irreversible urinary retention with overflow urinary incontinence).
Bilateral radicular leg pain.
Severe or progressive neurological deficit of both legs, such as major motor weakness of knee extension, ankle eversion, or foot dorsiflexion.
Loss of sensation of rectal fullness (if left untreated this may lead to irreversible faecal incontinence).
Laxity of the anal sphincter (Does not need to be assessed in primary care)
Erectile dysfunction (Achievement of erection or ability to ejaculate).

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13
Q

CES

A

explore
Onset and progression of symptoms
Other causes for leg pain
History of bladder/bowel disturbance/ sexual dysfunction

consider
Side effects of pharmacology
Age: Older patients may have spinal stenosis and are less likely to have acute CES.
Functional symptoms: psychosocial presentation and prior healthcare utilization.

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14
Q

presentations - increase prob of acute / threatened CES

A

Presence of new SA/ Bladder or bowel disturbance
Age < 50
Radicular pain
Unilateral radicular pain progressing to bilateral
Sudden onset bilateral radicular leg pain
Alternating radicular leg pain
Presence of new motor weakness
Obesity
Previous CES
Congenitally narrow spinal canal

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15
Q

presentations not managed with emergency spinal surgery

A

Urinary or bowel disturbance that has been present for more than 4 weeks and is not deteriorating.
Let the ED/surgeon make that decision
Should still be referred as an emergency
Older people with symptomatic spinal stenosis in the absence of acute bladder disturbance.

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16
Q

prognosis of CES

A

If symptoms are suggestive of CES: immediately referral to the ED should be made.
If you suspect a patient may be at risk of developing CES
Communicate clearly what the person should do if symptoms develop or progress.
NB: This must be documented.

17
Q

nerve root compression

A
Cervical/ thoracic spine
Single site of compression at each level
Exit foramen
Lumbar spine
2 potential sites of compression at each level
Lateral recess
Exit foramen
18
Q

nerve root symptoms

A

Axial spinal pain +/- limb pain in a dermatomal distribution
+/- sensory disturbance in a dermatomal distribution
+/- muscle weakness in the muscle groups innervated by the specific nerve root (myotomal).
LBP+/- leg pain (not necessarily in a specific dermatomal distribution)
Aggravated with walking/ standing
Eased with sitting or forward bending.
+/- Symptoms of nerve compression
Sensory disturbance
Motor weakness
Sphincter disturbance

19
Q

natural history of nerve root symptoms

A

Rate of resolution (Weber et al 1993, Vroomen et al 2000)
50% recovered at 2/52
60% recovered at 3/12
70% recovered at 12/12
Advanced imaging is only indicated when a patient fails to respond to a reasonable algorithm of conservative care (minimum 6 weeks)
Pain medication
Limiting activities or positions which aggravate nerve root symptoms
Rehabilitation
Timing of rehabilitation is important

20
Q

features concerning for serious pathology

A
Presence of features concerning for serious pathology
Infection
Fracture
Cancer
Bleed
Myelopathy
CES
21
Q

factors to consider

A
Age 
Disc versus stenosis
Dominant symptom
Axial spine pain
Limb  pain
Distribution of  peripheral symptoms
Dermatomal
Non- dermatomal
Unilateral or bilateral 
SIN
Severity
Irritability
Nature
Extent of neurology
Grade of weakness
Correlates with specific nerve root
Functional impact
Baseline level of physical activity
Duration of symptoms
Stability of symptoms
Response to treatment to date
Pharmacy
Conservative Treatment
Imaging findings*
MRI findings which correlate with the distribution of symptoms and signs.
Timing of imaging
Patient preference
22
Q

onward referral

A

Radicular leg pain with significant neurological deficit (MRC >4/5) but no concerning features for serious underlying pathology
Urgent MRI
Correlating pathology – surgical referral
Radicular leg pain with major motor radiculopathy(<3/5)/ Suspected serious pathology/ Severe unrelenting radicular leg pain unresponsive to pain medication.
Emergency Department

Radicular pain/ normal neurology/ no features concerning for serious pathology
Conservative management
Radicular pain <6/52 + mild stable neurological deficit/ no features concerning for serious pathology
Isolated diminished or absent reflex/ dermatomal sensory deficit or motor deficit which does not impact on function (EHL weakness/ some difficulty with SLHR but normal gait, mild triceps weakness)
Conservative management
Radicular pain > 6/52 with severe pain and functional deficit / not resolving
Link in with GP regarding referral for advanced imaging
Correlating pathology on imaging – surgical referral

23
Q

patient with axial LBP who may benefit from spina surgical opinion

A

Young athlete/ dancer (sport involves hyperextension) who develops LBP
Disabling LBP+/- nerve root symptoms and imaging evidence of pars defect +/- spondylolisthesis.

Adult spinal deformity
Persistent disabling LBP which has not responded to exhaustive conservative treatment.
Claudicant LBP with imaging evidence of high grade stenosis

24
Q

cervical myelopathy

A
Describes any neurologic deficit related to the spinal cord.
Usually caused due to cord compression by osteophyte or extruded disc material.
Examples of other  causes
Spinal tumour
Trauma
Inflammatory or autoimmune disease
Cervical epidural abscess
Vascular pathologies
Arteriovenous malformation
Epidural haematoma
25
Q

degen cervical myelopathy

A

Serious and specific spinal pathology
Most common type of spinal cord injury
Delayed diagnosis and treatment results in an Irreversible spinal cord injury
Surgery is very effective treatment but recovery is typically incomplete
Should always be included in your differential diagnosis of a patient presenting with neck pain/ upper limb sensory disturbance/ hand function disturbance / gait disturbance.
Delayed diagnosis is very common

26
Q

presentation of myelopathy

A

Age
> 40
Often between 50-70 but can begin in elderly
Neck and arm pain (unilateral or bilateral).
Neck stiffness
Gait disturbance (Degeneration of corticospinal tract)
Sensory disturbance in one or both arms or hands
Hand function disturbance
Clumsiness and dexterity loss (interneuronal loss)
Grip Strength weakness (Atrophy of the anterior horns associated with motor neuronal loss)
Urgency of bladder more than bowels, Urinary urge incontinence and urinary frequency

27
Q

typical neurological findings

A
Hyperreflexia
Localised
Global
Long tract signs
Increased tone
Upgoing plantar response
Sustained clonus
\+ve Hoffmans
Non dermatomal sensory loss
Non myotomal weakness
Gait disturbance
Spastic gait
28
Q

diagnosis

A
Diagnosis
1 symptom of DCM
1 neurological sign (UMN)
 MRI evidence of cord compression
Management
Severity of myelopathy (mJOA)
Moderate/ severe : Urgent surgical decompression
Mild
Surveillance
Surgery
Prophylactic surgery not recommended in asymptomatic compression
29
Q

assessment of patients with spinal pain

A
Understanding what pathologies have the potential to cause cord and nerve root compression and how they typically present.
Metastatic spinal cord compression
Age
History of cancer
Unexplained weight loss
Progressively worsening spinal pain 
Thoracic pain
30
Q

symptoms suggestive of neurological dysfunction

A
Listening for symptoms suggestive of neurological dysfunction .
Pins and needles/ Numbness
Weakness/heaviness in limbs
Hand function disturbance (Cervical)
Difficulty walking 
Sphincter disturbance
Bilateral symptoms
Radicular pain
31
Q

taking a comprehensive neuro exam

A

Knowledge of dermatomes/ myotomes.

Differentiate between Cord compression and nerve root compression

32
Q

emergency referral

A
Suspected Cauda Equina Syndrome
Suspected spinal cord compression
Related to serious underlying pathology
Tumour
Infection
Bleed
Fracture
Progressive
Significant neurological dysfunction
Major motor radiculopathy
Suspected  new spinal fracture
Trauma
Atraumatic
Osteoporotic
Pathological
33
Q

urgent referral

A

Suspected spinal cord compression
Moderate/ severe degenerative cervical myelopathy
Severe unrelenting radicular leg pain not responsive to usual pain medication
Suspected new pars defect in adolescent or young adult

34
Q

routine

A
Radicular pain
Sensory radiculopathy
Mild  stable motor radiculopathy
Mild stable degenerative cervical myelopathy
Axial spinal pain 
Imaging evidence of specific structural abnormality
Deformity
Pars defect (spondylolsis)
High grade IVD disease

Persistent disabling axial spinal pain not responding to a reasonable algorithm of conservative care.