clinical reasoning for spinal patient Flashcards

1
Q

define clinical reasoning

A

thought processes used in patient diagnosis and management

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2
Q

model of chronic pain

A

psychological factors in response to acute pain are predictive of chronic incapacity

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3
Q

MSK clinical translation framework

A
individuals perspective 
individual problems 
functional capacity 
goals/ expectations 
Diagnosis: specific diagnosis 
non specific 
red flags 
stage of disorder 
acute 
sub 
chronic 
recurrent 

pain features
types - nociceptive neuropathic nociplastic missed
characteristics - mechanical /non
sensitisation - low to high

psychological characteristics 
cog 
affective 
social 
low to high 

work considerations
workplace factors
low to high
blue flags

lifestyle considerations
lifestyle factors
low to high

whole person consideration
general health and co-morbidities

functional behaviours 
helpful - protective 
unhelpful - provocative 
impairment of movement 
impairment of control 
pain behaviours 
reconditioning 

clinical decision making
diagnosis
stages
important contributing factors

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4
Q

MSK clinical translation framework

individuals perspective

A

individual
key questions
main problems?
pain function QOL?

functionals capacity
how do problems affects ADLs / QOL
what can you do?

goals/ expectations
addressed goals is important to align with PCC

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5
Q

early clinical reasoning categories

  1. diagnosis
  2. pain mechanisms
A
  1. red flags vs specific diagnosis vs non-specific disorder
    consider source of symptoms
  2. pain mechanisms - mixed mechanisms can co-exist concurrently
    nociceptive - pain from acute or threatened damage to non-neural tissue and is due to activation of nociceptors
    Neuropathic:
    Pain caused by a lesion or disease of the somatosensory nervous system
    Superficial, burning, shooting, within all or part of nerve innervation field.
    +/- Paraesthesia or corresponding muscle weakness.
    Nociplastic:
    Pain arising from altered nociception despite no clear evidence of actual or threatened tissue damage causing the activation of peripheral nociceptors, or evidence for disease or lesion of the somatosensory system causing the pain.
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6
Q

subjective Ax

A
body chart 
behaviour of symptoms 
special questions 
HPC 
PMHx 
Social Hx - physical activity profile 
family Hx 
patients goals and expectations 
sleep patterns
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7
Q

body chart

A
Type & Area of current symptoms
Pain, stiffness, instability etc
Knowledge of pain patterns NB
Quality of pain
Severity & Frequency
Irritability
Abnormal sensation
P&Ns 
Numbness
Relationship of symptoms
Hierarchy (usually label worst as P1)
Temporal 
Continuity re. aggr. factors
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8
Q

somatic referral pain patterns

A
bone - deep nagging dull 
muscle dull ache 
nerve root sharp shooting 
nerve sharp lightning like 
sympathetic nerve burning pressure like stinging aching 
vascular throbbing diffuse
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9
Q

Pain - aggravating factors

mechanical

A

What activities / positions e.g. LBP - sitting, standing, walking, bending
How do individual structures handle ‘load’
severe enough to stop activity?

24 hour pattern
am
night - sleep position
EOD weekday vs weekend

easing factors
mechanical - activities / positions
drugs heat cold

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10
Q

mechanical vs inflammatory pain

A
SCREEN EM
skin - psoriasis rash
C- colitis, Crohn's 
R - relative - any family hx
E - eyes uveitis, dry eyes., photosensitivity 
E - early morning stiffness 
N - nocturnal pattern, nails and no. of joints 
E - exercise response/ effect 
M- medication effect
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11
Q

behaviour of symptoms - OTHER

A
stiffness 
giving way / instability of spine 
locking 
clicks - painful / painfree
weakness 
paraesthesia - sensory gain 
numbness - sensory loss
deadness / heaviness
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12
Q

special questions

A
general health 
unexplained weight loss 
severe unremitting pain 
night pain 
cancer pain 

spine - cord compression symptoms
cervical spondylitis myelopathy

LxSp: Cauda equina syndrome -gait saddle anaesthesia B+B symptoms

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13
Q

patient goals / expectations

A
symptoms tpainfree or less pain 
function = return to all activities or delay return until all symptoms are gone 
social - return to work/ sport / take time off
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14
Q

subjective Ax

A

Attune your language & approach to pt
Make sure pt understands the question
Listen & look – give pt your full attention
Make them feel you are not in a hurry, even when you are
Explore their fear; anxiety = pain magnifier

NB: Watch for signs of incongruity between what
they say & what you see – body posture, non-
verbal communicationExamine methodically – go back over doubtful points i.e. question again
Explain things clearly
Check patient’s understanding
Handle the pt considerately and with confidence
The 1st thing you say is what they will probably remember
Always be aware that the pt may hang on every word, facial expression & change of manner

Be careful of attempting to gain sensitive info in an unsuitable environment

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15
Q

reflection after subjective examination

A

Diagnostic Triage:
1% pts with spinal pain will have serious pathology e.g. tumour or infection
Identify other conditions requiring urgent specialist investigation & treatment (e.g. fracture, cardiac pathology)
Generate hypothesis for musculoskeletal diagnosis
Structures involved – articular / muscle / neural systems
If 2 pains – are they related or separate sources?
Pain mechanisms
Contextual factors – pain behaviour DRIVERS
Plan Physical examination –
Tests to be included
Priority areas for day 1
Precautions? e.g. SIN
Clues for Rx

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16
Q

summary of physical exam

A
Observation
Movement
Functional Testing
ROM – 
active physiological
Movement differentiation tests
passive physiological
Muscle tests – 
Isometric / Resisted
Strength 
Endurance
Flexibility
Motor Control
Neurological tests
function / conduction
Neurodynamic tests
nerve compliance to movement  stretch
Nerve trunk palpation
Palpation
Accessory movements –
passive
Combined movement tests – spinal
Ligamentous tests
stability / function
pain
Special tests 
Screen adjacent regions – how?
17
Q

spinal screen

A

From now on…….
All peripheral pain problems will require ‘clearing’ of the spine for
Direct involvement (referral / source)
Indirect involvement is also possible in longstanding cases due to compensation
Examples:
Hip pain – clear LxSp and SIJ
Knee pain – clear LxSp and SIJ and Hip

18
Q

observation

A

Willingness to move / functional level
Gait – antalgic?

Posture – ant, post, lateral
Bony structure & alignment

Muscle bulk, activity / tone
Skin – incisions, texture, moisture, colour (inflammatory / vascular)

19
Q

ROM

A

active physiological
normal
hyper/hypo
(+/- pain)

passive physiological 
peripheral 
spine PPVIMs
passive physiological 
intervertebral movements
20
Q

muscle testing

A

isometric/ resisted
resistance applied in joint rest position
pain provocative - implies contractile unit lesion as nociceptive source

weak and painful 
strong and painful 
weak and painless 
strong and painless 
Strength & Endurance–
Graded isotonic contraction – (MMT)
S&C tests - functional
muscles react differently to pain / stress
spasm / over activation
inhibition / underactivity

Length / Flexibility
Motor Control

21
Q

muscle reaction to pain

overactive / tight

inhibited / weak

A

overactive / tight Masseter, temporalis, digastric, suboccipital muscles, levator scapulae, pec major & minor, rhomboids, upper traps, SCM, UL flexors, erector spinae, quadratus lumborum, piriformis, TFL, rectus femoris, hamstrings, short adductors, tib post, gastrocs

Serratus anterior, mid & lwr traps, deep neck flexors, mylohyoid, subscapularis, UL extensors, Gluteus maximus, medius, minimus, deep lumbar multifidus, iliopsoas, vastus medialis & lateralis, tib ant & peronei

22
Q

neuro tests

neurological function

A

neurological function
DTRs
MYOTOMES
DERMATOMES

Balance
Coordination
Tone
Babinski / Clonus

neurodynamic tests 
compliance / flexibility of nerve
Slump, SLR, PKB etc.
Consider  mechanical interfaces
measure
pain response &
resistance – joint angle at onset e.g. Hip flex in SLR
23
Q

nerve protective features

A
Course
Slack 
undulations in nerve trunk, fasciuli & fibres
Mechanical Properties 
Tensile strength
 fasciuli = stronger Nerve

Cushioning
Epineurium as needed
Muscle Tone
Muscle protects nerve

24
Q

neurodynamics

A

Undulations/slack taken up first
Sliding movement of nerve against / between other tissues
Median N in forearm
elongation of nerve

ICP 
changes 
compromises blood flow 
stasis 
increase permeability 
endometrial oedema 
changes in CSA
25
Q

compromises at mechanical interface impedes nerve ability to glide and slide

A

Treatment Strategies

Target mechanical interface
E.g. Contral Sideflexion PPIVM
Target nerve mobilisation
E.g. Sliders vs. Tensioner exercises

26
Q

palpation

A
Useful to record also on body chart or palpation chart or o/palp=
NOTE:
Temp
Skin moisture
Oedema / effusion
Superficial tissues: mobility, ganglions / cysts
Muscle spasm
Trigger points: 
Bony prominence / tenderness
Pain response for individual structures
27
Q

trigger point

A

Trigger point = a focus of hyperirritability in a tissue that, when compressed, is locally tender and, if sufficiently hypersensitive, gives rise to referred pain and tenderness and sometimes to referred autonomic phenomena and distortion of proprioception (Travell & Simons 1983) – mostly myofascial

establish baseline
opposite side first
superficial to deep
go lightly

28
Q

accessory movements

A

PAIVM:
Passive Accessory Intervertebral Movements = central PA & unilateral PA +/- Transverse movements
Historically: graded mobility – poor intertester reliability for motion testing
Define nature of motion abnormality
Normal
Hypermobile
Hypomobile
Excellent reliability for pain provocation  identifying symptomatic spinal segment(s) Jull 1994

29
Q

accessory movements

assess

A
Assess – 
quality of motion
motion abnormality
pain behaviour through PAIVM: local P1 & referred  
   P2
resistance through range
reactivity of muscle 	
Example:   ↓ L5 - P1 & P2
30
Q

special tests

A
SPECIAL TESTS 
REGION SPECIFIC
Vascular: check peripheral pulses
Knee: McMurray’s meniscal tests
PF joint: Apprehension test
Hip: Flexion/Adduction Scour test
CxSp: Cervical Artery testing
ETC
31
Q

diagnostic triage

A

CSAG (1994) – Clinical Standards Advisory Group: UK Dept Health
Simple / Mechanical LBP: 95% cases
Nerve root pain / Radiculopathy: 5% cases
Serious spinal pathology: <1%

Recognised by all disciplines – Doctors, Physios etc.
Prioritises RX