Spinal Cord Injury Flashcards

Weeks 3 & 4

1
Q

How many spinal nerve pairs exist in the human body?

A

31 pairs: 8 cervical, 12 thoracic, 5 lumbar, 5 sacral, 1 coccygeal.

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2
Q

Which spinal levels are most susceptible to injury?

A

C5-C7 and T12-L2 due to increased mobility and mechanical stress.

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3
Q

What is the difference in nerve root exit between cervical and other spinal levels?

A

C1-C7 nerves exit above their corresponding vertebrae, while C8 and below exit below their corresponding vertebrae.

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4
Q

What is the functional loss pattern for a lateral corticospinal tract lesion?

A

Ipsilateral motor loss due to 80% of fibers decussating in the medulla.

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5
Q

What happens when the DCML is damaged?

A

Ipsilateral loss of discriminative touch and proprioception.

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6
Q

What symptoms occur with a spinothalamic tract lesion?

A

Contralateral loss of pain and temperature sensation.

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7
Q

What is the ASIA Impairment Scale (AIS) used for?

A

To classify the severity of spinal cord injuries based on motor and sensory function loss.

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8
Q

What defines a complete SCI (ASIA A)?

A

No sensory or motor function preserved in the sacral segments S4-S5.

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9
Q

What is sacral sparing and why is it important?

A

Sacral sparing (DAP - deep anal pressure & VAC - voluntary anal contraction) indicates an incomplete SCI and potential for better recovery.

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10
Q

What is the Zone of Partial Preservation (ZPP)?

A

Dermatomes and myotomes below the neurological level that retain some function in a complete SCI.

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11
Q

How is sensory function tested in SCI patients?

A

Light touch and pinprick testing across 28 dermatomes bilaterally.

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12
Q

How is motor function graded in SCI patients?

A

Using the ASIA scale, which grades key muscles from 0 (paralysis) to 5 (normal strength).

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13
Q

Which key muscles correspond to C5, C6, and C7?

A

C5: Elbow flexors (biceps), C6: Wrist extensors (ECRL, ECRB), C7: Elbow extensors (triceps).

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14
Q

Which key muscles correspond to L2, L3, and L4?

A

L2: Hip flexors (iliopsoas), L3: Knee extensors (quadriceps), L4: Ankle dorsiflexors (tibialis anterior).

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15
Q

What is Central Cord Syndrome?

A

The most common incomplete SCI, affecting upper limbs more than lower limbs due to corticospinal tract somatotopy.

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16
Q

What is Brown-Séquard Syndrome?

A

Hemisection of the spinal cord causing ipsilateral loss of motor/proprioception and contralateral loss of pain/temp.

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17
Q

What is Anterior Cord Syndrome?

A

Bilateral loss of motor function, pain, and temperature sensation, but preserved light touch and proprioception.

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18
Q

What differentiates Conus Medullaris from Cauda Equina Syndrome?

A

Conus Medullaris affects both UMN and LMN; Cauda Equina is purely LMN with areflexia, saddle anesthesia, and bowel/bladder dysfunction.

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19
Q

What is Autonomic Dysreflexia?

A

A life-threatening hypertensive response to noxious stimuli in SCI patients above T6.

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20
Q

What are common causes of Autonomic Dysreflexia?

A

Bladder distension, bowel impaction, pressure ulcers, ingrown toenails, tight clothing.

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21
Q

What is the first step in managing Autonomic Dysreflexia?

A

Sit the patient upright, identify and remove the noxious stimulus, check blood pressure.

22
Q

What is Orthostatic Hypotension and why is it common in SCI?

A

A drop in BP >20 mmHg systolic or >10 mmHg diastolic due to impaired sympathetic vasoconstriction.

23
Q

Why is pulmonary function often impaired in SCI?

A

Loss of diaphragm, intercostal, and abdominal muscle function depending on injury level.

24
Q

What muscles are primary for inspiration and expiration?

A

Inspiration: Diaphragm, intercostals; Expiration: Abdominals, internal intercostals.

25
What is Glossopharyngeal Breathing (GPB)?
A technique used by high cervical SCI patients to swallow air for breathing assistance.
26
What are the primary PT interventions for pulmonary function in SCI?
Diaphragmatic breathing, incentive spirometry, assisted coughing, respiratory muscle training.
27
What are the primary PT goals in acute SCI management?
Maintain neutral spine, prevent secondary complications, early mobilization, respiratory support.
28
What is the role of a cervical orthosis (CO) in SCI?
To provide spinal stabilization post-injury or surgery (e.g., Miami J, Philadelphia collar, Halo).
29
What is the role of a TLSO in SCI management?
Thoracolumbosacral orthoses stabilize lower spinal injuries and prevent excessive motion.
30
What is the expected functional outcome for a C5 complete SCI?
Requires dependent ADLs, power wheelchair with hand controls, limited functional hand use.
31
What is the expected functional outcome for a T10 complete SCI?
Independent transfers, wheelchair mobility, potential for therapeutic ambulation with bracing.
32
What are the most common secondary complications of SCI?
Pressure ulcers, DVT, contractures, osteoporosis, heterotopic ossification, chronic pain.
33
How often should weight shifts be performed in SCI patients?
Every 15 minutes in a wheelchair, every 2 hours in bed to prevent pressure sores.
34
What is the primary cause of heterotopic ossification in SCI?
Abnormal bone growth in soft tissues due to prolonged immobility and inflammation.
35
What pharmacologic interventions help manage spasticity in SCI?
Baclofen, tizanidine, botulinum toxin, intrathecal baclofen pump.
36
What is Tenodesis Grasp and why is it important?
A passive grasp mechanism used by C6-level SCI patients for functional hand use.
37
What are positive prognostic factors for walking recovery post-SCI?
Preserved pinprick sensation in LEs, younger age, ASIA C/D classification.
38
What percentage of ASIA C patients regain some walking function?
Approximately 65% regain some ability to walk.
39
What is the plateau period for functional recovery post-SCI?
12-18 months post-injury.
40
How does motivation and psychosocial support impact SCI recovery?
Higher motivation, strong social support, and access to rehab services improve outcomes.
41
What are the major spinal cord syndromes?
Includes Central Cord Syndrome (UE > LE weakness) Brown-Séquard Syndrome (ipsilateral motor & proprioception loss, contralateral pain/temp loss) Anterior Cord Syndrome (loss of motor, pain/temp, preserved proprioception) Cauda Equina Syndrome (LMN signs, bowel/bladder dysfunction)
42
What is the neurological level of injury (NLI) in SCI?
The most caudal segment of the spinal cord with normal motor and sensory function bilaterally.
43
What are the functional outcomes based on lesion levels?
Higher cervical injuries (C1-C4) require ventilators; C5 has partial UE movement; C6 allows tenodesis grasp; T1 and below allow full UE function; L2 and below have potential for ambulation with bracing.
44
What is spinal shock and its phases?
A temporary loss of reflexes, motor, and autonomic function below the injury level. Phases: 1 (areflexia) 2 (initial reflex return) 3 (hyperreflexia) 4 (spasticity/variable function).
45
How do you differentiate between complete and incomplete SCI?
Complete SCI has no motor/sensory function at S4-S5 (no sacral sparing); incomplete SCI retains some function below the lesion, including sacral sparing.
46
What are common bladder and bowel dysfunctions in SCI?
Above T12 = UMN bladder (spastic, reflexive emptying). Below T12 = LMN bladder (flaccid, requires catheterization). Bowel dysfunction requires bowel programs.
47
What respiratory complications arise from SCI?
C1-C4 injuries may need ventilators; C5-T12 injuries impact cough function and breathing control. Patients benefit from assisted cough techniques and incentive spirometry.
48
What are key wheelchair skills for SCI patients?
Includes propulsion, pressure relief techniques, curb negotiation, floor recovery, and maneuvering uneven surfaces.
49
What are transfer techniques for SCI patients?
Includes slide board transfers, dependent lifts, anterior/posterior transfers, head-hips relationship for movement control.
50
What are key bed mobility techniques for SCI patients?
Rolling, supine to sit, long sitting balance, and leg management techniques based on lesion level.
51
What factors influence gait training potential in SCI patients?
Level of injury, presence of spasticity, bracing needs, assistive devices, and neuroplasticity training principles. ASIA C/D have better chances of walking.
52
What are PT implications for SCI rehabilitation?
Includes preventing pressure ulcers, managing autonomic dysreflexia, respiratory training, mobility training, strength & endurance exercises, and patient education on long-term care needs.