Spinal Cord Injury Flashcards
Traumatic spinal cord injury: what population is it most common in
- most common in young men (80%)
- most common cause: car accidents, violence, falls, sports injuries
- most injuries cause crushing, edema, hemorrhage or infarct
Look over types of
forces applied to the cervical spine and resultant injury
DONT MEMORIZE
- rotational hyperflexion mechanism: facet dislocation, wedged compression FX
- hyperextension mechansim: posterior arch fx, handman fx
- Hyperflexion or hyperextension mechanism: teardrop fx, spinous process fx, odontoid process fx
- Lateral flexion mechanism: transverse process fx, uncinate process fracture
- axial compression mechanism: burst fx of atlas, burst or vertical fx of C2-C7
Cervical wedge fx from hyperflexion injury
- multiple vertebral bodies fx
- disrupts structures its protecting
- hyperflexion injury
burst fracture
- from vertical compression
- bulk of inside of body gets compressed fx
acute treatment of SCI
- people go into spinal shock
- stabilization of vital signs due to possible autonomic dysfunction is first
- administration of anti-inflammatory drugs to limit swelling (recently shown to no have a significant long term improvements)
- stabilization/traction of spine
Acute treatment: repair and stabilization of fracture
- repair and stabilize fx
- fusion, rods, plates, external stabilization via Halo, SOMI, TLSO, jewitt etc
Halo immobilizer
- screws into skull and a vest around the shoulders that provides traction.
- can have a supine traction for just stabilization or a brace they wear if they still need traction
- halo brace is stable and hard to dislodge but do not pull directly on bars
- isometrics can be helpful to maintain strength since they cannot move their neck
Minerva brace (SOMI)
- can go right into this or transition to this from a Halo
- Sterno-occipital-mandibular immobilizer
TLSO
- can be used for children w/ scoliosis
- not as many precautions w/ it off
- taken off in bed but you must log roll in and out of it
Examination with SCI
- medical history
- medications
- cognition: may have hit their head
- social history
Respiratory management with SCI
- diaphragmatic breathing: may not be able to use all the respiratory muscles
- may need a ventilator
- Glossopharyngeal breathing: recue breathing for short periods of time
- maximizing function
Skin management with SCI
- bed and wheelchair positioning
- patient education
- high risk areas = bony prominence
- treatment of pressure ulcers
Autonomic dysfunction
- most frequent in complete SCI above T6
- orthostatic hypotension
- thermoregulation
- autonomic dysreflexia
Autonomic dysreflexia signs and symptoms
- hypertension: can be life-threatening
- sweating above level of lesion (where they can sweat)
- flused skin above level of lesion
- nasal congestion: due to HTN
- Headache
- blurry vision or seeing spots
- goose bumps
Autonomic dysreflexia: causes
- Noxious or potentially noxious stimuli
- bladder distension
- UTI
- bowel impaction
- wheelchair or bed positioning causing pressure (cant feel it but body reacts to it)
- invasive testing
- DVT
- pulmonary embolus
- blister
- hetertopic ossification
- fx
- surgery
- sexual intercourse
- ingrown toenail
- insect bit
- burn
- temperature flucuations pain
- pregnancy
- ETC
Autonomic dysreflexia treatment
- get patient upright (cause orthrostatic hypotension)
- look for cause and correct it
- emergency procedures if you cannot find the problem
- contact nursing - catheterization PRN
- DO NOT CONTINUE WITH THERAPY
Autonomic dysreflexia treatment afterwards
- pt may need medication to reduce BP: nitroglycerin, nifedipine
- monitor BP and symptoms for abour 2 hours after episode
Heterotopic ossification
- extra bone gets laid down in response to trauma
- complete injuries it is common
- hips and knees
- red, swollen joint/limb
- prevention: ROM (gentle to maintain), NSAIDS
- treatment: bisphosphonates
Spinal shock
- occurs shortly after injury
- hypertension => hypotension
- flaccid paralysis below level of lesion (complete or incomplete injury)
- last hour to weeks
- just tells you there is injury to SC but not how severe
end of spinal shock
- return of spinal reflexes
- stretch reflexes
- bulbocavernosus reflex: center reflex - pulling on the their catheter to see if there is movement in gential region
- hypertonia and spasticity below level of lesion may begin to develop
Spasticity
- impairments
- treatment: rhythmic rotation
- medications: baclofen, tizanidine, benzodiazepines (addictive and cause sleepy feeling), BoTox
- measurement
post stroke hypertonia
vs hyperreflexia in complete SCI
- post stroke: paresis - myoplastic changes: contractures, weak actin-myosin bonds, atrophy
- Complete SCI: stretch reflex hyperreflexia, myoplastic changes: contracture, weak actin-myosin bonds, atrophy
Classification of SCI
- quadriplegia: impairments or arm, trunk and leg function: all four some could be just weakness
- paraplegia: impairment of trunk and leg function
- complete: lack of sensation/mmotor function at lowest scaral segment (S4-S5)
- incomplete: preservation of some sensation and or motor function including S4-S5
Neurological level of injury
- lowest level where both sensory and motor function are normal
- zone of partial preservation: might have some sensory/motor function usually occurs with complete injury
- determined by testing (ASIA): left and right, motor and sensory function