Spinal Cord Injuries - Collins (incomplete) Flashcards

1
Q

what is the most common population that presents with SCI

A

50% of acute injuries involve young patients (16-30)
second peak at age 60+
MEN>females

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2
Q

what are patients with SCI at higher risk of

A

more likely to die prematurely by 2-5x

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3
Q

what is the most common location of SCI injuries

A

cervical C5 most common

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4
Q

what is the most common manifestation of SCI

A

incomplete paraplegia

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5
Q

what are the most common mechanisms of SCI

A

MVC
falls
sports-related injuries
violence
secondary to compression (contusion)
complete transection rare

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6
Q

what is a complication of SCI

A

neurologic regulations are worse the higher the level of the injury within the spine

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7
Q

what is a primary injury

A

the initial mechanical insult

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8
Q

what is a secondary injury

A

persistent physiologic insult

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9
Q

what is the acute timeline

A

first 48 hours
cytotoxic, inflammatory, vascular, necrosis, nerve depolarization

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10
Q

what is the subacute timeline

A

48 hours to 14 days
macrophage infiltration and scar initiation

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11
Q

what is the intermediate timeline

A

14 days to 6 months
continued scar formation

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12
Q

what is the chronic timeline

A

6+ months
degeneration of spared components

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13
Q

when is the most important time to worry about hypotension s/p SCI

A

acute phase - compensation for vascular changes

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14
Q

what are the role of steroids in SCI

A

steroids are anti-inflammatory and targeting acute phase but not clinically understood

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15
Q

what is the initial treatment for a spine trauma patient

A

immobilization until cleared

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16
Q

what trumps immoblization

A

treat life-threatening injuries first

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17
Q

what is assessed in the initial survey for spinal trauma patients

A

gross motor/sensory deficits
tenderness - especially midline
step-offs
palpable fluid collections/hematoma
bruising or abrasions/wounds

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18
Q

what is assessed later in the ED for spine trauma patients

A

complete neurological exam including perineal sesation and anal sphincter tone

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19
Q

how are spine trauma patients worked up

A

if they are “walking/talking” and no pain/distracting injuries - dont need imaging

20
Q

what is first line imaging for spinal injuries

A

book answer: AP and Lateral x-rays + odontoid for c-spine
Practice: CT usually entire spine

21
Q

how are stable fractures typical treated

A

conservative management usually with brace for immobilization

22
Q

how are unstable fractures typically treated

A

ORIF, usually fusion procedure

23
Q

does the degree of the SCI correlate with the stability of the fracture

A

NO, not necessarily correlated

24
Q

what level injury is Neurogenic shock seen most commonly

A

most often with thoracic level injury

25
Q

what part of the nervous system is disrupted with Neurogenic shock

A

Sympathetic NS - inability to maintain vascular tone

26
Q

what is the treatment of neurogenic shock

A

fluid resuscitation and vasopressors

27
Q

what is the resolution timeline of neurogenic shock

A

24-48 hours

28
Q

what occurs during neurogenic shock

A

inability to maintain vascular tone
hypotension with BRADYCARDIA and without vasoconstriction

29
Q

what are the SCI patterns

A

complete, incomplete or transient

30
Q

what is transient SCI

A

temporary: spinal shock
NOT necessarily neurogenic shock though often co-occurring
spinal cord “stinger”

31
Q

what is the ASIA scale

A

american spinal injury association system to classify SCI

32
Q

what is the ASIA grading scale

A

Grade A (complete injury) through E (normal)

33
Q

when is the Bulbocavernossus reflex absent

A

during Transient SCI

34
Q

when do Transient SCI resolve

A

24-72 hours - must be resolved to definitively classify SCI

35
Q

What is an incomplete SCI

A

some degree of neurologic function present distal to injury

36
Q

What is ‘sacral sparing’

A

voluntary anal sphincter tone, perineal sensation, great toe flexion

BARE MINIMUM

37
Q

what is seen with greater initial function

A

better prognosis

38
Q

what is the timeline of neurologic improvement following a SCI

A

up to 12-18 months

39
Q

what are the incomplete SCI patterns

A

Brown-sequard syndrome
Central Cord syndrome
Anterior cord Syndrome
Posterior Cord Syndrome

40
Q

Ascending

A

carry information to the brain - sensory

41
Q

descending

A

carry information from the brain to target tissue/organs- motor

42
Q

what should the SCI patients have for treatment

A

transfer to specialized SCI injury rehabilitation facility - the earlier the better
shorter overall LOS
lower morbidity and mortality
Extensive PT, assistive technologies/devices
often far away, limited beds and expensive

43
Q

what are common complications for SCI

A

Gastritis/illeus - NG tube and H2 blockers
Urinary dysfunction - foley or intermittent caths - risks for UTI
Breathing difficulty - “C3,4,5 Keep the diaphragm alive”
Skin Breakdown - reposition every 2 hours, regular inspections, pad pressure points - decreased sensation and loss of motor function
Vascular complications

44
Q

what are the concerns with vascular complications s/p SCI

A

most common cause of morbidity and mortality
worse the higher the injury
arterial hypotension, orthostatic hypotension
autonomic dysreflexia

45
Q

what is autonomic dysreflexia

A

rapid increased BP (imbalanced sympathetic SN stimulation)
precipitated by some stimuli below level injury
often bowel and bladder dysfunction