Palsy Flashcards

1
Q

what is a palsy

A

paralysis usually accompanied weakness, loss of feeling and with involuntary tremors or movements

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2
Q

what is cerebral pasly

A

non-progressive disorder of tone and posture from an acquired pre- or post-natal insult (up to 30 days of life)
not usually apparent at birth, may be noted as early as 4 months, definitely by 2 years

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3
Q

what are the risk factors of CP

A

include permaturity, perinatal deprivation of blood or oxygen to the brain via:
PVL(periventricular leukomalacia), IVH (Intraventricular hemorrhage) or Hypoxic-ischemic encephalopathy (HIE), maternal infection, trauma, and other processes involving ischemia, inflammation, infection, genetic predisposition

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4
Q

what maternal infections increase the risk of CP

A

Zika
TORCH -
Toxoplasmosis
Other
Rubella
CMV
HSV

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5
Q

what are the classifications of CP

A

Hypotonic
Hypertonic or Spastic
Dystonic
Cerbellar ataxia

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6
Q

what are the types of Hypotonic CP

A

quadriparesis or tetrapaesis
diaparesis or diplegia
hypotonia with choreoathetosis

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7
Q

what are types of Hypertonic or spastic CP

A

quadriparesis
hemiparesis
diparesis or diplegia

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8
Q

what is dystonic CP

A

facial rictus, stimultaneous contaction of agnoist and antagonist muscles, opsithotonus

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9
Q

what is cerebellar ataxia CP

A

unsteady wide - based gate, gross motor delay, coordination and balance problems (~3%)

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10
Q

how is CP diagnosed

A

labs - look for congenital infection in mothers and kids
imaging - mri for brain changes or normal

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11
Q

what are differential diagnoses for CP

A

some neurodegenerative and neurometabolic disorders can mimic, genetic testing is helpful in differentiating

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12
Q

what is the prognosis of CP

A

spasticity tends to worsen with time vs hypotonia can improve with age

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13
Q

what is the intelligence level in CP patients

A

cognitive impairment does not always match physical deficits. can have normal intelligence with more severe hyper- or hypotonicity

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14
Q

what is the treatment of CP

A

multidisciplinary approach:
PT/OT
Bracing
Pharmacologic treatment of spasticity

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15
Q

what are generalized medications treatments for CP

A

oral antispasmodics (baclofen, tizanidine, benzodiazepeine)

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16
Q

what are localized treatments for CP

A

botulinum toxin (botox), bracing, orthopedic surgery, neurosurgery

17
Q

what is a selective dorsal rhizotomy

A

effective in young children with spastic dispraises who can walk - involved cutting abnormal nerve in lower spinal cord to relax muscles

18
Q

what is radial nerve palsy

A

paralysis of radial nerve due to compression of nerve against humerus

19
Q

what is the common cause of radial nerve palsies

A

fracture of the humerus - during fracture or repair
sedated with ETOH, falling asleep with chair over arm
during sleep with a parner resting on your arm

20
Q

when are radial nerves more likely to be severed and need repair

A

open fractures, high energy traumas, open wounds

21
Q

what are the symptoms of radial nerve palsy

A

pain, weakness, loss of function in wrist, hand and fingers
numbness over the lateral dorsum of hand
wrist drop or finger drop

22
Q

what is the cause of axillary radial nerve compression

A

crutches and the triceps muscle is affected and interferes with elbow extension

23
Q

how is radial nerve palsy diagnosed

A

ultrasound can be used to distinguish between nerve ruptures, swelling around nerve, neuroma formation

24
Q

what is the initial treatment for radial nerve palsy

A

brace or spline and PT plus NSAIDs - 90% will improve with observation alone and can take 3-6 months

25
Q

what is bells palsy

A

weakness or paralysis of facial muscles caused by damage to CN7, facial nerve (mononeuropathy)

Idiopathic

26
Q

what are known conditions associated with Bells palsy

A

associated with viral infections (HSV, EBC, VZV)
associated with lyme disease
if caused by stroke, trauma it is NOT bells palsy

27
Q

what neurons are responsible for saliva production as well as lacrimal glands and nasal oral mucous membranes

A

neurons from the motor complex

28
Q

what carries info from anterior 2/3 of the tongue

A

neurons from the motor complex

29
Q

are patients with Bells Palsy able to lift their eyebrow

A

NO

30
Q

what must be excluded for bells palsy diagnosis

A

stroke, tumor or trauma

31
Q

what are risk factors for Bells Palsy

A

PREGNANCY, DIABETES, OLD AGE , immune compromise - can also be caused by HIV, Sarcoidosis, lyme, meningitis

32
Q

what is the presentation of Bells Plasy

A

sudden onset, muscle weakness maximal at 48 hours after onset
onset can be preceded or accompanied by retro-aural pain which lasts a few days

33
Q

what labs and studies are done for the diagnosis of Bells Palsy

A

CBC, BMP, magnesium, and calcium, HgbA1c, ESR, Lyme titer, ANA,RF, HIV, +/- MRI if clinical exam suggests lesion

34
Q

what is the treatment of bells palsy

A

usually resolves within 6 months - but can be permanent
treatment is not usually required but if severe - can use corticosteroid to reduce inflammation of the nerve (high dose pred)

35
Q

where does the facial nerve originate

A

in the brainstem

36
Q

what neurons travel along with the facial nerve

A

motor and sensory neurons