spinal cord compression Flashcards

1
Q

definition of cord compression

A

injury to the spinal cord with neurological sx that depend on the site and extent of injury

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2
Q

aetiology of cord compression

A

trauma and tumours are most common causes

  • trauma can cause direct cord contusion or compression by bone fragments, haematoma or acute disk prolapse
  • tumours can be primary, more commonly are mets (breast, lung, prostate, kidney, thyroid, melanoma) = collapse or compression of a vertebral body

vascular malformationeg cavernous haemangiomas

infection - spinal abscess

TB (Pott’s disease)

cervical disc prolapse

haematoma - warfarin

atlanto-axial subluxation

myeloma

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3
Q

associations and RF of cord compression

A

sig spinal cord injury is associated with severe trauma, often with head injuries

in the presence of a tumour or vascular malformation, relatively minor trauma can cause severe sx

predisposing factors

  • osteoporosis
  • metabolic bone disease
  • vertebral disk disease
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4
Q

epidemiology of cord compression

A

common

trauma at any age

malignancy and disc disease - more common in older pop

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5
Q

sx of cord compression

A

Hx of injury or trauma

back pain - nocturnal and with straining

bilateral leg weakness (arm weakness less severe and suggests cervical lesion)

limb weakness

sensory loss

difficulty walking

clumsy

disturbance of bowel/bladder func - bladder and anal sphincter involvement is late - hesitency and frequency and then painless retention

large central lumbar disk prolapse may cause bilateral sciatica, saddle anaesthesia and urinary retention

may be a hx of malignancy

ask about nocturnal pain and pain with straining

worry if cervical/thoracic pain

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6
Q

signs of cord compression

A

diaphragmatic breathing

reduced anal tone

hyporeflexia

priapism

spinal shock - low BP w/o tachycardia

look for sensory, reflex and motor level

  • normal above
  • sensory signs at level of lesion
  • UMN below (except in acute) - spastic weakness, upgoing plantars

motor signs

Brown-Sequard syndrome

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7
Q

motor signs of cord compression

A

weakness/paralysis (LMN signs), downward plantar in acute phase and at level of spinal cord injury

UMN signs and upward plantar reflexes seen in the later phase below the level of the spinal cord injury

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8
Q

brown-sequard syndrome

A

seen in hemisection of the spinal cord

below level of lesion - ipsilateral spastic paralysis and loss of postural sense and contralateral loss of pain and thermal sense#

(spinothalamic dessucates at level of exit)

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9
Q

pathology of cord compression

A

spinal cord exits the skull at the foramen magnum and ends at L1

8 cervival, 12 thoracic, 5 lumbar, 5 sacral and 1 coccygeal pairs of spinal nerves

the lumbar, sacral and coccygeal roots form the cauda equina

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10
Q

role of C5

A

shoulder abduction - deltoid

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11
Q

role of C6

A

forearm flexion - biceps

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12
Q

role of C7

A

forearm extension - triceps

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13
Q

role of C8

A

wrist/finger flexion

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14
Q

role of T1

A

finger abduction

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15
Q

role of L2

A

hip flexion - iliopsoas

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16
Q

role of L3

A

knee extension - quadriceps

17
Q

role of L4

A

ankle dorsiflexion (tibialis anterior)

18
Q

role of S1

A

ankle plantar flexion - gastrocnemius

19
Q

Ix for cord compression

A

radiology:

  • AP and lateral XR of cervical (also peg view), thoracic, or lumbar spine - look for loss of alignment, loss of height or wedging due to compression fracture, detatched fragments
  • emergency MRI or CT necessary

blood - FBC, UE, Ca, ESR, immunoglobin electrophoresis, B12, syphilis serology, PSA

urine - Bence Jones protein (indicative of multiple myeloma)

biopsy or surgical exploration may be needed to idnetify nature of a mass

CXR - primary lung malignancy, lung secondaries, TB

20
Q

ddx for cord compression

A

transverse myelitis

MS

carcinomatous meningitis

cord vasculitus (PAN, syphilis)

spinal artery thrombosis or aneurysm

trauma

Guillain-Barre syndrome

21
Q

sx with cauda equina and conus medullaris lesions

A

leg weakness is flaccid and areflexic (not spastic and hyperreflexic as is the case with lesions higher up)

alternating or bilateral root pain in legs

saddle anaesthesia (perianal) - lack of sensitivity in S3-S5 around anus, genitalia, and inner thighs

loss of anal tone on PR

bladder +- bowel incontinence

22
Q

causes of cauda equina and conus medullaris lesions

A

same as lesions higher

congenital lumbar disk disease adn lumbosacral nerve lesions

23
Q

signs of conus medullaris lesions

A

mixed UMN and LMN signs

leg weakness

early urinary retention and constipation

back pain

sacral sensory disturbance

erectile dysfunction

24
Q

cauda equina lesions signs

A

back pain

radicular pain down the legs

asymmetrical

atrophic

areflexic paralysis of legs

sensory loss in a root distribution

reduced sphincter tone - do PR

saddle anaesthesia

25
Q

mx of neoplastic cord compression

A
  1. steroids
  2. analgesia
  3. urinary catheterisation and consideration of bowel incontinence
  4. VTE prophylaxis
  5. surgery within 24 hrs
  6. radiotherapy as rx or after surgery
  7. physio
26
Q

when is surgery done for neoplastic spinal cord compression

A
  • ASA good
  • survival >3 mo
  • if neuro impairment >24hrs - surgery just for pain relief
27
Q

when should you not give radiotherapy for neoplastic cord compression

A

severe neuro impairment >24hrs and pain undder control
or px too poor for any rx - ie palliative

28
Q

complications of neoplastic cord compression

A
  • immobility
  • incontinence
  • pressure ulcer
  • dvt
  • infection
  • surgery complications
  • depression
  • shock and anxiety
29
Q

scoring system to assess px in neoplastic cord compression

A

Tokuhashi scoring system